Alzheimer's & Toxic Mold- Whoohoo!!!- Congress & Funding More Point Blank Facts

[Guest guest]

A reminder of my theory of how people get sick from toxic mold.

Right behind your nose is your pineal gland (responsible for your

melatonin, RNA, DNA, immune system.) The pineal gland is the most

important part of your body and is not protected by a barrier. So

common sense says what happens when you inhale toxic substances

through your nose and they go directly to your unprotected pineal

gland? People get sick..

So here is Alzheimer's disease:

Journal of Pineal Research

Volume 35 Issue 2 Page 125 - September 2003

doi:10.1034/j.1600-079X.2003.00065.x

Early neuropathological Alzheimer's changes in aged individuals are

accompanied by decreased cerebrospinal fluid melatonin levels

Jiang-Ning Zhou1,3, Rong-Yu Liu1,2, Wouter Kamphorst4, Michel A.

Hofman2 and Dick F. Swaab2

Abstract: Neuropathology is the most reliable criterion for

diagnosing Alzheimer's disease (AD). A well-established system for

staging the spread of neuropathological changes in AD is available.

The clinical use of a biomarker that reflects the neuropathological

change occurring in brain tissue has not yet been established.

Melatonin is a product that plays not only a major role in the

regulation of the circadian rhythms but may also exert

neuroprotective effects in AD. Melatonin levels were determined in

ventricular postmortem cerebrospinal fluid (CSF) of 121 subjects.

Braak staging and a modified Braak staging for cortex (MBSC) were

used to evaluate the severity of AD neuropathology. The present

study revealed that not only the Braak stages of AD, but also the

MBSC were negatively correlated with CSF melatonin levels. By using

MBSC, we now demonstrate for the first time that CSF melatonin

levels were significantly decreased in the aged individuals with

early neuropathological changes in the temporal cortex, where the AD

process starts. Those individuals that did not have any

neurofibrillary tangle (NFT) or neuritic plaque (NP) in the temporal

cortex, had much higher melatonin levels (287 ± 68 and 280 ± 64

pg/mL, respectively) than those individuals that had a few NFTs and

NPs (82 ± 4 and 39 ± 8 pg/mL, respectively) in the temporal cortex.

These results suggest that the decrease in CSF melatonin levels may

be an early event in the development of AD possibly occurring even

before the clinical symptoms.

http://www.blackwell-synergy.com/links/doi/10.1034/j.1600-

079X.2003.00065.x/abs/

Do you think Congress needs some more point blank facts? Whoohoo!!!