Guest guest Posted September 12, 2000 Report Share Posted September 12, 2000 , I am not that new to the list but I don't post that often. This was a subject that I thought was very concerning. How do you deal with these pro-actor people who blame us for not doing enough? It makes me feel really bad that we have such a small population, my letters probably mean nothing to the networks. I am not anti-actor LP. I am anti-selfish. I would never do anything to make my children to be or other LP's ashamed of what I do. SauceyKitty@... wrote: > > Another One > > After reading all the posts on the list regarding the MTV Music Awards, and previous topics, I pose this question to the list. > > Is the only thing that connects our whole group, that we actually all have in common, just the simple fact that we all have Dwarfism? Or am I just being cynical in the fact that I don't believe we all have some sort of shared set of morals, self respect and enough will to put forth the effort to conduct ourselves in a civil manner in public in relation to our dwarfism? > > I know I know, I've heard it about a billion times, " It's my life and you can't tell me how to act. What I do doesn't affect you. " The first point is true, " I " can't tell " you " how to act, BUT the second point, is not true, some things you do significantly affects others now and for years to come. > > Can't there be some sort of compromise? Or are some of you so adamant in the belief of the concept " you can't teach an old dog new tricks " and " you can't change society's view as a whole…or could it be that you are so selfish to say, it's my life and you can't tell me what to do? > > And a note only to the demeaning performers, actors and actress' (not ALL performers, actors and actress'), the audience, crowd, society, BP's or whatever you want to call them aren't laughing with you, but are laughing AT you and a lot of other innocent bystander LP's who just want to act " normal " . > > That's all for now..Thanks to those who listen, and for those who disagree and want to attack my letter, don't pick it apart line by line and take it out of context. Bash it as a whole, please. > > > > And again a special thanks to Rose, and for their wonderfully written letters about the MTV topic. -- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 12, 2000 Report Share Posted September 12, 2000 Sorry folks. That was meant to be a private email. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 4, 2002 Report Share Posted November 4, 2002 I actually think that the Lymes has affected my eyesite in terms of detail work. I was fine until June. At 10:30 PM 11/04/2002 -0500, you wrote: >Jeff (and Everyone), > >Can dry eyes be a side effect of any meds? > >I got dry eyes once from reading almost 12 hours a day for my Doctoral >General Exams. The eye doc recommended some drops from the drugstore >called " Refresh. " I think that's what they were called. They were just >saline, very harmless, but VERY helpful. It did the trick and helped me >thru my exams. > >Wellness to all, >Marie > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 4, 2002 Report Share Posted November 4, 2002 Yes, I though about meds affecting eye dryness too. But this was one of the very first symptoms I noticed (right before my bulls eye rash showed up), well before I started taking meds. It may be that the meds made it worse, but it seems like part of the package. I haven't had any other changes physically or in terms of eye strain that should explain it. Incidentally, I was at the eye doc today and he recommended " refresh p.m. " We'll see how that goes tonight. Jeff Jeff Assistant Professor of Management Miami University 307 Laws Hall Oxford, OH 45056 (513) 529-4746 >>> henson2@... 11/04/02 22:31 PM >>> Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 5, 2002 Report Share Posted November 5, 2002 Jeffery~ I'm glad you brought up the dry eye symptom because I too was curious about its occurence in LD patients. I've had dry eyes since the onset of my other symptoms (about 6 months ago) and my doc didn't put me on meds until a month later and Doxy was two months later. Many meds probally cause dry eyes as a side effect but this surely was not my initial case. My symptoms is a little diff. than yours: Eyes become thoroughly dry during the night but then they do not really recover during the day. I'm usually a contact wearer but I've heard LD bacteria can build up frequently and cause more damage in a contact wearer's eye thus I haven't been wearing them. Has anyone w/ LD who wears contacts noticed any problems to support this? I also usually experience tremendous pressure behind my eyes...sometimes so bad that I'm not able to to keep my eyes open and view my surroundings. My only treatment for dry eyes as of now is to just carry a small bottle of eye drops w/ me to try and keep them lubricated. Bridget in MN Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 5, 2002 Report Share Posted November 5, 2002 Bridget: I wear contacts, but aside from the occasional bout of dry-eye, I haven't experienced any other eye, pain, pressure or vision problems. I do not sleep in them though. I have woken up with very dry eyes a few times. Your case sounds pretty bad and I would imagine that contacts might aggravate it. I do know that they decrease the amount of oxygen in the eye, but as to what part of the eye, and whether that would make a difference in lyme I have no idea. E. > I'm usually a contact wearer but I've heard LD >bacteria can build up frequently and cause more damage in a contact >wearer's eye thus I haven't been wearing them. Has anyone w/ LD who >wears contacts noticed any problems to support this? > >Bridget in MN Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 5, 2002 Report Share Posted November 5, 2002 Dear Bridget, I am a contact lens wearer and when I was going through the worst years of my Lyme battle, I had great difficulty wearing my lenses. My vision is pretty bad, so I can only wear the gas permeable lenses. I remember trying to put my lenses in if I was going some place and within a couple of hours, I had to remove them. Thinking back, I'm not sure whether it was from the Lyme or the antibiotics. Also, one time I wore them and just kept lubricating them throughout the evening, and later that night, my vision was very blurry. I was stupid in not taking them out because I had given myself a corneal abrasion. I am telling you this because in retrospect, wearing my glasses with the dry eyes would have been more intelligent than pushing it because of my vanity, and trying to wear my lenses all evening. My ophthalmologist had put antibiotic drops in my eyes and had to patch both of them for 48 hours so they could heal, and that was a pretty draining experience. Sue Rauch Quote Link to comment Share on other sites More sharing options...
Guest guest Posted February 27, 2003 Report Share Posted February 27, 2003 http://www.direct-ms.org/seasonal.html Vitamin D and Seasonal Fluctuations of Gadolinium-Enhancing Magnetic Resonance Imaging Lesions in Multiple Sclerosis Ashton F. Embry, Ph.D., Lloyd R. Snowdon, Ph.D. and Reinhold Vieth, Ph.D. ls of Neurology, 2000, v. 48, p.271-272 Auer et al1 recently showed a striking, near sinusoidal annual variation in the number of active magnetic resonance imaging lesions in 53 multiple sclerosis (MS) patients. Their results provide solid support for past, less-well documented claims of seasonal fluctuation in MS disease activity. Furthermore, the seasonal fluctuation in lesion activity may provide an important clue for identifying environmental factors which are part of MS etiology. Auer et al proposed that seasonal changes in the rate of common infections might partially explain the seasonal fluctuation in lesion activity. They noted that climatic factors such as temperature, amount of sunlight and UV light exposure might also be involved1. We suggest that vitamin D supply, which fluctuates with seasonal UV light exposure, is the main environmental factor involved. Vitamin D and its metabolites have been implicated in MS etiology by epidemiological, experimental and immunological data2. Moreover, circulating 25-hydroxyvitamin D [25(OH)D] also shows a near sinusoidal annual fluctuation at higher latitudes3,4 To investigate a possible correspondence between the fluctuations in vitamin D intake and lesion activity, we compared published monthly 25(OH)D levels in 415 people, aged 50-80, from southern Germany4 with the data of Auer et al1 which were also collected in southern Germany (Figure). Third order polynomial curves fit both the 25(OH)D and lesion data significantly. When the 25(OH)D data are lagged by two months, there is a close correspondence between the two curves with high levels of 25(OH)D correlating with low levels of lesion activity and vice versa. A two-month lag is within reason and is taken to reflect the time for a given 25(OH)D level to affect detectable lesion occurrence. The inverse correlation between lesion activity and 25(OH)D level suggests that vitamin D nutrition may have a notable immuno-modulating effect on CNS inflammation, a conclusion also reached through experimental and immunological studies2. The impressive correlation also supports the need for proper clinical trials to test whether improved vitamin D nutrition (not the vitamin D hormone, 1,25(OH)2D) can reduce formation of CNS lesions and slow the progression of MS. Until definitive results are available, clinicians may want to ensure their MS patients are receiving a meaningful vitamin D intake (3000-4000 IU/day) throughout the year. Although well above current nutritional guidelines (200-400 IU), such an intake is physiological and safe and most importantly, provides a desirable target amount of circulating 25(OH)D5. Month-to-month variation of average number of active lesions in multiple sclerosis patients from southern Germany as reported by Auer et al1 and month-to-month variation in the median levels of the vitamin D metabolite, 25(OH)D, in 50-80 year olds from southern Germany as reported in Scharla4. The two, fitted, third order polynomial curves show a close correspondence when the 25(OH)D data are lagged two months as illustrated. Note that the 25(OH)D concentration decreases upward on the Y axis to allow a better comparison of the curves which correlate inversely. References Auer DP, Schumann EM, Kumpfel T et al. Seasonal fluctuations of gadolinium-enhancing magnetic resonance imaging lesions in multiple sclerosis. Ann Neurol 2000;47:276-277 CE, Cantorna MT and DeLuca HF Vitamin D and multiple sclerosis. Proc Soc Exp Biol Med 1997; 216:21-27 Maxwell JD Seasonal variation in vitamin D. Proc Nutr Soc 1994;54:533-543 Scharla SH Prevalence of subclinical vitamin D deficiency in different European countries. Osteoporo Int 1998 Suppl;8:S7-S12 Vieth R Vitamin D supplementation, 25-hydroxyvitamin D concentrations, and safety. Am J Clin Nutr 1999; 69:842-856 Quote Link to comment Share on other sites More sharing options...
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