Guest guest Posted July 1, 2002 Report Share Posted July 1, 2002 Hi, Steve! Didn't know anything about XO so did some snooping. I found a couple of interesting things. The first article says that XO apparently assists the release of iron from from ferritin stores. " The enzyme has been reported to have anti-tumor effect in mice ( Haddow et al. 1958) and to participate in the release of iron from hepatic ferritin stores in the plasma ( Mazur et al. 1958). " http://www.worthington-biochem.com/manual/X/XO.html " According to Dr. Kurt Oster, Chief of Cardiology, Emeritus, at Bridgeport Park City Hospital, Dr. Ross of Fairfield University, and Dr. Zikakis of University of Delaware, homogenization allows the enzyme xanthine oxidase to pass intact into the blood stream. There it attacks the plasmalogen tissue of the artery walls and parts of the heart muscle. This causes lesions that the body tries to heal by laying down a protective layer of cholesterol. The end result is scar tissue and calcified plaques and a buildup of cholesterol and other fatty deposits. We call these arteriosclerosis and atherosclerosis. According to these esteemed researchers, dietary cholesterol isn't the main cause of heart attacks, homogenized milk is. Consider this: Finns drink about 600 pounds of milk a year; 90 percent is homogenized. Swedes drink about 375 pounds, but only 2 percent is homogenized. The heart attack death rate in Finland is about 245 per 100,000. In Sweden it is only 75. While this doesn't prove anything, it sure gets your attention. " http://www.argee.net/Thrawn%20Rickle/Thrawn%20Rickle%2026.htm This scientific article says that NAD(P)H oxidases (XO is one) are a problem, but that XO is probably not one of them. :/ http://www.cardiov.ox.ac.uk/staff/channonpdf/guzik.pdf Quote Link to comment Share on other sites More sharing options...
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