Guest guest Posted August 28, 2002 Report Share Posted August 28, 2002 I'd suggest emailing Mark Purdey. > has anyone read Mercola's latest newsletter re CWD (chronic wasting disease) > in deer in Wisconsin? The state may kill up to 50,000 deer. This has got me > wondering about North Star Bison, located in WI. I know many of us order > from them. I thought i had read of CWD crossing species barriers, as it > began in elk in colorado then somehow deer ended up with it. does anyone > think North Star's bison may be at risk of contracting CWD now that it's hit > the state? I've read a couple of theories on the origins and transmission of > CWD but am not sure what to think or believe... > > any thoughts? anyone here have a firm belief in it's origins and > transmission? > > Suze Fisher > Web Design & Development > http://members.bellatlantic.net/~vze3shjg/ > mailto:s.fisher22@v... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 28, 2002 Report Share Posted August 28, 2002 At 11:19 AM 8/28/2002 -0400, you wrote: >has anyone read Mercola's latest newsletter re CWD (chronic wasting disease) >in deer in Wisconsin? The state may kill up to 50,000 deer. This has got me >wondering about North Star Bison, located in WI. I know many of us order >from them. I thought i had read of CWD crossing species barriers, as it >began in elk in colorado then somehow deer ended up with it. does anyone >think North Star's bison may be at risk of contracting CWD now that it's hit >the state? I've read a couple of theories on the origins and transmission of >CWD but am not sure what to think or believe... > >any thoughts? anyone here have a firm belief in it's origins and >transmission? > >Suze Fisher There is a good writup on the WAP website (I don't have the link handy though), that makes you think. There are two mutually opposing viewpoints on it: one says that the issue is a magnesium overdose combined with lack of copper, more or less. Which good evidence. The thing about the deer is, they are likely NOT eating ground up animals (though they eat an occasional baby bird, according to one researcher). I don't have firm thoughts on it. Scrapie seems to have been around for a long time before feeding animals to animals: but the prion-transmission theory has a lot of research behind it too. Heidi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 28, 2002 Report Share Posted August 28, 2002 >>>> There is a good writup on the WAP website (I don't have the link handy though), that makes you think. There are two mutually opposing viewpoints on it: one says that the issue is a magnesium overdose combined with lack of copper, more or less. Which good evidence. The thing about the deer is, they are likely NOT eating ground up animals (though they eat an occasional baby bird, according to one researcher). I don't have firm thoughts on it. Scrapie seems to have been around for a long time before feeding animals to animals: but the prion-transmission theory has a lot of research behind it too. ->thanks for your thoughts heidi - i'll take a look at the WAPF article. i just did a quick google search on Purdey and the article mentions a *manganese* excess in conjunction with other factors. Quote: Manganese in excess (and in conjunction with pour-ons such as the Pthithalidimii-organo phosphate (already used in Australia) causes the copper in the brain to be chelated to the point where the prions 'fold' and BSE or similar is the result. There is a lot of research and the results are plain alarming. http://216.239.51.100/search?q=cache:HjE-q_9yMB4C:www.bfa.com.au/news/articl es/2002_autumn_patcolbybse.htm+Mark+Purdey+CWD & hl=en & start=1 & ie=UTF-8 I'm going to have to read more of his thesis, though, to fully understand it. re deer eating meat...i read one of the theories was that wild deer were eating the commercial feed from a deer farm in CO, somehow the feed had been spread about or something. that was one of the original theories about the herbivores eating meat hypothesis. Suze Fisher Web Design & Development http://members.bellatlantic.net/~vze3shjg/ mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 28, 2002 Report Share Posted August 28, 2002 Suze, Mark Purdey has his own very interesting site dealing with -Organophosphates, BSE, CJD, Manganese, Copper, Scrapie, Prions, Chronic Fatigue Syndrome, Pesticides, Industrial Pollutants, Soils, Diet. http://www.purdeyenvironment.com/ Dedy ----- Original Message ----- From: " Suze Fisher " <s.fisher22@...> < > Sent: Wednesday, August 28, 2002 5:15 PM Subject: RE: CWD in Wisconsin? > >>>> > There is a good writup on the WAP website (I don't have the link handy > though), that makes you think. There are two mutually opposing viewpoints > on it: one says that the issue is a magnesium overdose combined with lack > of copper, more or less. Which good evidence. The thing about the deer is, > they are likely NOT eating ground up animals (though they eat an occasional > baby bird, according to one researcher). I don't have firm thoughts on it. > Scrapie seems to have been around for a long time before feeding animals to > animals: but the prion-transmission theory has a lot of research behind it > too. > > > ->thanks for your thoughts heidi - i'll take a look at the WAPF article. i > just did a quick google search on Purdey and the article mentions a > *manganese* excess in conjunction with other factors. > > Quote: > > Manganese in excess (and in conjunction with pour-ons such as the > Pthithalidimii-organo phosphate (already used in Australia) causes the > copper in the brain to be chelated to the point where the prions 'fold' and > BSE or similar is the result. There is a lot of research and the results are > plain alarming. > http://216.239.51.100/search?q=cache:HjE-q_9yMB4C:www.bfa.com.au/news/articl > es/2002_autumn_patcolbybse.htm+Mark+Purdey+CWD & hl=en & start=1 & ie=UTF-8 > > I'm going to have to read more of his thesis, though, to fully understand > it. > > re deer eating meat...i read one of the theories was that wild deer were > eating the commercial feed from a deer farm in CO, somehow the feed had been > spread about or something. that was one of the original theories about the > herbivores eating meat hypothesis. > > > Suze Fisher > Web Design & Development > http://members.bellatlantic.net/~vze3shjg/ > mailto:s.fisher22@... > > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 28, 2002 Report Share Posted August 28, 2002 At 12:15 PM 8/28/2002 -0400, you wrote: >re deer eating meat...i read one of the theories was that wild deer were >eating the commercial feed from a deer farm in CO, somehow the feed had been >spread about or something. that was one of the original theories about the >herbivores eating meat hypothesis. Purdey's idea that the hunters are putting out salt licks fits with his theory. Dueling theories! I seem to be getting manganese and magnesium mixed up. No wonder I'm confused. I wonder if both theories could be correct. Maybe the prions first appear because of excess manganese, but then they are transmissible or at least start changing other proteins in the same animal? Heidi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 29, 2002 Report Share Posted August 29, 2002 >re deer eating meat...i read one of the theories was that wild deer were >eating the commercial feed from a deer farm in CO, somehow the feed had been >spread about or something. that was one of the original theories about the >herbivores eating meat hypothesis. Purdey's idea that the hunters are putting out salt licks fits with his theory. Dueling theories! ---->is there manganese in salt? or organophosphates? I wonder if both theories could be correct. Maybe the prions first appear because of excess manganese, but then they are transmissible or at least start changing other proteins in the same animal? -------->well, you know what's interesting...manganese concentrations in soil are higher in the east and get progressively lower as you go west, according to Wharton in " Metabolic Man.. " and i think he's quoting Albrecht. Yet recent CWD outbreaks started in the west and seem to be rare in the east. if we're only discussing *wild* animals...then how are those eating plants that grow on western soil getting excess manganese while those in the east that eat plants that grow in soil richer in manganese not getting CWD? I guess the other contributing factors need to be considered. heck, i haven't even read the WAPF article...so this is just blind speculation...but it just came to mind about soil manganese concentration. i remember that there was an outbreak on a couple of mink farms in MN and WI back in the 1947. There were additional outbreaks in the 60's. (the deer CWD dates back to 1967 according to http://w3.aces.uiuc.edu/AnSci/BSE/TSE_Other_Animals.htm#tme) AND, according to a wholistic vet on another list i'm on, CWD in deer and elk started at at Colorado State University's Foothills Research Station, where they were keeping captive deer. so maybe it DID start with a captive population. (BSE is this vet's pet topic). I'll have to read more about Purdey's theories...i'm assuming they fit all the data... Suze Fisher Web Design & Development http://members.bellatlantic.net/~vze3shjg/ mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 29, 2002 Report Share Posted August 29, 2002 How about this from the WAP site: http://www.westonaprice.org/myths_truths/myths_truths_beef.html What about " Mad Cow Disease " ? Beef consumption in England plummeted recently with the 'Mad Cow Disease " scare. Mad cow disease, or bovine spongiform encephalopathy (BSE), is a wasting disease of cattle characterized by nervous disorders and weakness, said to be related to Creutzfeldt-Jakob disease (CJD) in humans. Scientists have not been able to link a virus to this disease, so they theorize that an abnormal protein particle called a prion, found in the brains of cattle with BSE and humans with CJD, is the cause. The theory is that these prions are infectious agents, passed along to cows though the practice of animal part feeding and then to humans who eat infected meat, particularly meat from the nervous system, like brain. There's a lot wrong with this theory. For one thing, BSE is nonexistent in the USA, where animal part feeding has been going on for almost one hundred years. Another is recorded cases of CJD among vegetarians; yet another is the absence of CJD in the Shetlands where scrapie, a disease similar to BSE, is common in sheep and where potted sheeps brain is a national dish. The research of Mark Purdey, a diary farmer in England, indicates that the mad cow disease epidemic in England occurred in areas where farmers were forced to treat their cattle with organophosphate pesticides in a warble fly eradication program.20 The warble fly makes holes in the cows' backs-not dangerous in itself, but it reduces the value of pelts sold to leather manufacturers. These holes are open doors to the spinal cord and organophosphate pesticides are very toxic to the nervous system. By a complex process, these compounds seem to cause certain proteins to fold in pathological ways-these are the prions that are found in the brains of animals with BSE and humans with CJD. Mineral deficiencies are also involved, particularly magnesium, which is a mineral that protects the nervous system. Finally, a similar disease occurs among wild animals living in areas of volcanic soils, whose diets are high in aluminum and manganese, minerals known to be toxic to the nervous system. Clusters of human CJD cases are also found in areas where the soil has mineral imbalances, where there are cement factories and where high levels of organophosphate insecticides have been used. So the answer to CJD and BSE is good soil management and the elimination of neurotoxic compounds in farming-but it's easier to just blame it on beef. By the way, now that animal part feeding has been outlawed, feedlot operators are turning to soy feeds as a protein substitute. Soy is very toxic to cows' livers. Does the use of soy in cattle feeding explain why beef-lean beef-has become politically correct again? After all, the other politically correct meats-chicken and salmon-use up vast quantities of soybean meal in battery feeding and fish farms. Peace, Kris , gardening in harmony with nature in northwest Ohio If you want to hear the good news about butter check out this website: http://www.westonaprice.org/know_your_fats/know_your_fats.html ----- Original Message ----- From: " Suze Fisher " <s.fisher22@...> " " < > Sent: Wednesday, August 28, 2002 11:19 AM Subject: CWD in Wisconsin? > has anyone read Mercola's latest newsletter re CWD (chronic wasting disease) > in deer in Wisconsin? The state may kill up to 50,000 deer. This has got me > wondering about North Star Bison, located in WI. I know many of us order > from them. I thought i had read of CWD crossing species barriers, as it > began in elk in colorado then somehow deer ended up with it. does anyone > think North Star's bison may be at risk of contracting CWD now that it's hit > the state? I've read a couple of theories on the origins and transmission of > CWD but am not sure what to think or believe... > > any thoughts? anyone here have a firm belief in it's origins and > transmission? > > Suze Fisher > Web Design & Development > http://members.bellatlantic.net/~vze3shjg/ > mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 29, 2002 Report Share Posted August 29, 2002 i remember that there was an outbreak on a couple of mink farms in MN and WI back in the 1947. According to a very reliable source who lives in Madison and has actively studied animals around the world... This is the third time WI is killing off deer in this area of Madison. The first time they killed off the deer herd and brought in new deer, the deer contracted CWD again. They killed off that herd and waited a while saying whatever causes it is living i the soil and introduced this herd they are killing off now. My question is why is it so concentrated in Madison area. They know deer hunters from all over the state come to this area to hunt and then bring the carcasses back home with them and are assuming people have brought back to other areas of the state CWD infested deer. They are also claiming to not have found CWD in our area at all (2 driving hours away) and claiming that the deer farms are not infested in Wisconsin. The scary thing is people are claiming it is fine to eat the meat as long as you do not eat the organs. Grace, a Augustine I wish you enough sun to keep your attitude bright. I wish you enough rain to appreciate the sun more. I wish you enough happiness to keep your spirit alive. I wish you enough pain so that the smallest joys in life appear much bigger. I wish you enough gain to satisfy your wanting. I wish you enough loss to appreciate all that you possess. I wish you enough ''Hello's " to get you through the final goodbye. --anonymous Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 29, 2002 Report Share Posted August 29, 2002 >>>>>How about this from the WAP site: http://www.westonaprice.org/myths_truths/myths_truths_beef.html What about " Mad Cow Disease " ? ----------->Kris, thanks for posting that. while i wholeheartedly agree that cattle should not be subjected to organophosphates and that soil management is vitally important, i think the authors are jumping to a conclusion based on a hypothesis that has not been demonstrated to be fact as yet. at least that's the impression i get from reading these 4 paragraphs. i'm sure sally and mary are thoroughly familiar with purdey's work and have based their conclusion on that, but it still seems somewhat speculative to me, yet they definitively state in the last paragraph that the answer is soil management and the elimination of neurotoxic compounds, as if there's no questioning that purdey is correct. this is the type of article that bugs me...jumping to a conclusion after presenting a just a few brief paragraphs of 'evidence.' I think it underestimates the intelligence of the reader and comes across as somewhat didactic. i don't care what web site i'm going to, no author is going to convince me that they know the definitie answer to something as complex as BSE/CWD by presenting 3-4 paras of 'evidence' then stating the solution as fact. I wish, in cases like this, they'd just say something like, we have read " x, y, z " literature on the subject and find Purdey's hypothesis to fit the evidence more strongly than any of the other hypothesis on BSE/CWD. As such we feel the solution is...blah, blah, blah. Suze Fisher Web Design & Development http://members.bellatlantic.net/~vze3shjg/ mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 29, 2002 Report Share Posted August 29, 2002 At 07:24 PM 8/28/2002 -0400, you wrote: >-------->well, you know what's interesting...manganese concentrations in >soil are higher in the east and get progressively lower as you go west, >according to Wharton in " Metabolic Man.. " and i think he's quoting Albrecht. >Yet recent CWD outbreaks started in the west and seem to be rare in the >east. if we're only discussing *wild* animals...then how are those eating >plants that grow on western soil getting excess manganese while those in the >east that eat plants that grow in soil richer in manganese not getting CWD? >I guess the other contributing factors need to be considered. heck, i >haven't even read the WAPF article...so this is just blind speculation...but >it just came to mind about soil manganese concentration. It seems manganese is all over but overdoses are rare -- the body gets rid of it. Purdey was talking a lot about huge concentrations (mining operations, insecticides), AND in conjunction with lack of copper and possibly high altitude. Obviously a lot more work needs to be done. What is interesting to me is that something THIS contagious and fatal (i.e. you can wipe out the deer herd and it comes back, even though no one is especially feeding them) should have wiped out all mammals on earth by now. Prions must have been in existence for millions of years: why are they cropping up now? *Something* doesn't fit here. I've always believed the " animal feed " theory but I'm beginning to wonder ... mink and the like DO eat other animals, and being a carnivore offers no protection, so why hasn't it wiped out the dingos and wolves of the world? Has it been endemic all along and no one noticed? Humans have been eating brains and organ meats (as do dogs and scavengers) all along, so you would think there would be a high level of endemic prion-damage in both populations, if it crops up in herbivores as often as now seems to be the case. Heidi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 29, 2002 Report Share Posted August 29, 2002 This is also very interesting that there is no Mad cow disease in the US. I was touring the 4-H building at the MN state fair Monday and there was a poster on Mad cow disease. According to their resources Mad Cow disease has been in Asia, Africa, South America and Europe. The only continents it is missing is North America, Australia, and of course Antarctica... why? Too many questions.. Grace, a Augustine I wish you enough sun to keep your attitude bright. I wish you enough rain to appreciate the sun more. I wish you enough happiness to keep your spirit alive. I wish you enough pain so that the smallest joys in life appear much bigger. I wish you enough gain to satisfy your wanting. I wish you enough loss to appreciate all that you possess. I wish you enough ''Hello's " to get you through the final goodbye. --anonymous ----- Original Message ----- From: Kris Sent: Wednesday, August 28, 2002 11:02 PM Subject: Re: CWD in Wisconsin? How about this from the WAP site: http://www.westonaprice.org/myths_truths/myths_truths_beef.html What about " Mad Cow Disease " ? Beef consumption in England plummeted recently with the 'Mad Cow Disease " scare. Mad cow disease, or bovine spongiform encephalopathy (BSE), is a wasting disease of cattle characterized by nervous disorders and weakness, said to be related to Creutzfeldt-Jakob disease (CJD) in humans. Scientists have not been able to link a virus to this disease, so they theorize that an abnormal protein particle called a prion, found in the brains of cattle with BSE and humans with CJD, is the cause. The theory is that these prions are infectious agents, passed along to cows though the practice of animal part feeding and then to humans who eat infected meat, particularly meat from the nervous system, like brain. There's a lot wrong with this theory. For one thing, BSE is nonexistent in the USA, where animal part feeding has been going on for almost one hundred years. Another is recorded cases of CJD among vegetarians; yet another is the absence of CJD in the Shetlands where scrapie, a disease similar to BSE, is common in sheep and where potted sheeps brain is a national dish. The research of Mark Purdey, a diary farmer in England, indicates that the mad cow disease epidemic in England occurred in areas where farmers were forced to treat their cattle with organophosphate pesticides in a warble fly eradication program.20 The warble fly makes holes in the cows' backs-not dangerous in itself, but it reduces the value of pelts sold to leather manufacturers. These holes are open doors to the spinal cord and organophosphate pesticides are very toxic to the nervous system. By a complex process, these compounds seem to cause certain proteins to fold in pathological ways-these are the prions that are found in the brains of animals with BSE and humans with CJD. Mineral deficiencies are also involved, particularly magnesium, which is a mineral that protects the nervous system. Finally, a similar disease occurs among wild animals living in areas of volcanic soils, whose diets are high in aluminum and manganese, minerals known to be toxic to the nervous system. Clusters of human CJD cases are also found in areas where the soil has mineral imbalances, where there are cement factories and where high levels of organophosphate insecticides have been used. So the answer to CJD and BSE is good soil management and the elimination of neurotoxic compounds in farming-but it's easier to just blame it on beef. By the way, now that animal part feeding has been outlawed, feedlot operators are turning to soy feeds as a protein substitute. Soy is very toxic to cows' livers. Does the use of soy in cattle feeding explain why beef-lean beef-has become politically correct again? After all, the other politically correct meats-chicken and salmon-use up vast quantities of soybean meal in battery feeding and fish farms. Peace, Kris , gardening in harmony with nature in northwest Ohio If you want to hear the good news about butter check out this website: http://www.westonaprice.org/know_your_fats/know_your_fats.html ----- Original Message ----- From: " Suze Fisher " <s.fisher22@...> " " < > Sent: Wednesday, August 28, 2002 11:19 AM Subject: CWD in Wisconsin? > has anyone read Mercola's latest newsletter re CWD (chronic wasting disease) > in deer in Wisconsin? The state may kill up to 50,000 deer. This has got me > wondering about North Star Bison, located in WI. I know many of us order > from them. I thought i had read of CWD crossing species barriers, as it > began in elk in colorado then somehow deer ended up with it. does anyone > think North Star's bison may be at risk of contracting CWD now that it's hit > the state? I've read a couple of theories on the origins and transmission of > CWD but am not sure what to think or believe... > > any thoughts? anyone here have a firm belief in it's origins and > transmission? > > Suze Fisher > Web Design & Development > http://members.bellatlantic.net/~vze3shjg/ > mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 > >-------->well, you know what's interesting...manganese concentrations in > >soil are higher in the east and get progressively lower as you go west, > >according to Wharton in " Metabolic Man.. " and i think he's quoting Albrecht. > >Yet recent CWD outbreaks started in the west and seem to be rare in the > >east. if we're only discussing *wild* animals...then how are those eating > >plants that grow on western soil getting excess manganese while those in the > >east that eat plants that grow in soil richer in manganese not getting CWD? > >I guess the other contributing factors need to be considered. heck, i > >haven't even read the WAPF article...so this is just blind speculation...but > >it just came to mind about soil manganese concentration. Purdey has identified about three factors (as you mention below) that he found wherever he went to check on an outbreak of " mad cow " . So it's more complcated that just excess manganese. > It seems manganese is all over but overdoses are rare -- the body gets > rid of it. Purdey was talking a lot about huge concentrations (mining > operations, insecticides), AND in conjunction with lack of copper and > possibly high altitude. Obviously a lot more work needs to be done. > > What is interesting to me is that something THIS contagious and fatal (i.e. > you can wipe out the deer herd and it comes back, even though > no one is especially feeding them) should have wiped out all > mammals on earth by now. Prions must have been in existence > for millions of years: why are they cropping up now? Purdey is saying that this is not " contageous " but is the result of this peculiar combination of circumstances that results in this outcome. > *Something* doesn't fit here. I've always believed the " animal feed " > theory but I'm beginning to wonder ... mink and the like DO eat > other animals, and being a carnivore offers no protection, so We heard Mark Purdey speak at the Wise Traditions conference in April. He's done an incredible job of ferreting out the combination of factors that apparently leads to this syndrome called 'Mad Cow'. But for the authorities to accept his theory and test it they would have to admit that all those hundreds of thousands of cattle, sheep, etc were killed in vain and that they caused the epidemic by requiring the use of the insecticide involved. You can imagine how likely that is to happen without a battle! You should study his website. I'm sure all the information is there. Kris Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 What about " Mad Cow Disease " ? ----------->Kris, thanks for posting that. while i wholeheartedly agree that cattle should not be subjected to organophosphates and that soil management is vitally important, i think the authors are jumping to a conclusion based on a hypothesis that has not been demonstrated to be fact as yet. at least that's the impression i get from reading these 4 paragraphs. i'm sure sally and mary are thoroughly familiar with purdey's work and have based their conclusion on that, but it still seems somewhat speculative to me, yet they definitively state in the last paragraph that the answer is soil management and the elimination of neurotoxic compounds, as if there's no questioning that purdey is correct. Suze wrote >this is the type of article that bugs me...jumping to a conclusion after presenting a just a few brief paragraphs of 'evidence.' I think it underestimates the intelligence of the reader and comes across as somewhat didactic. i don't care what web site i'm going to, no author is going to convince me that they know the definitie answer to something as complex as BSE/CWD by presenting 3-4 paras of 'evidence' then stating the solution as fact. I wish, in cases like this, they'd just say something like, we have read " x, y, z " literature on the subject and find Purdey's hypothesis to fit the evidence more strongly than any of the other hypothesis on BSE/CWD. As such we feel the solution is...blah, blah, blah.> Suze, if you had listened to Purdey lecture for an hour you might think differently. He has actually visited a lot of places where there has been an out break of BSE/CWD and found a related set of factors in each place. The problem is, as I said in my previous post, that there is great reluctance on the part of authorities to admit that he is on to something and do the research needed to prove it. Kris Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 Suze how do you know BSE/CWE is complex? Dennis > Suze wrote > >this is the type of article that bugs me...jumping to a conclusion after > presenting a just a few brief paragraphs of 'evidence.' I think it > underestimates the intelligence of the reader and comes across as somewhat > didactic. i don't care what web site i'm going to, no author is going to > convince me that they know the definitie answer to something as complex as > BSE/CWD by presenting 3-4 paras of 'evidence' then stating the solution as > fact. I wish, in cases like this, they'd just say something like, we have > read " x, y, z " literature on the subject and find Purdey's hypothesis to fit > the evidence more strongly than any of the other hypothesis on BSE/CWD. As > such we feel the solution is...blah, blah, blah.> > > Clipped by Dennis Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 Heidi wrote: > Purdey's idea that the hunters are putting out salt licks > fits with his theory. Dueling theories! > > I seem to be getting manganese and magnesium mixed up. No wonder > I'm confused. > > I wonder if both theories could be correct. Maybe the prions first appear > because > of excess manganese, but then they are transmissible or at least start > changing other proteins in the same animal? As I remember prions are always there but the manganese displaces copper in the prion leading to a misshapen prion that doesn't work right. Check out: http://www.markpurdey.com/ Kris Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 At 02:26 PM 8/29/2002 -0500, you wrote: >This is also very interesting that there is no Mad cow disease in the US. >I was touring the 4-H building at the MN state fair Monday and there was a >poster on Mad cow disease. According to their resources Mad Cow disease >has been in Asia, Africa, South America and Europe. The only continents it >is missing is North America, Australia, and of course Antarctica... why? >Too many questions.. > >Grace, >a Augustine Maybe it IS copper deficiency? American cows pretty much ALWAYS get salt licks, which have all kinds of minerals in them (maybe the wrong minerals for deer though????). Heidi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 Suze, if you had listened to Purdey lecture for an hour you might think differently. He has actually visited a lot of places where there has been an out break of BSE/CWD and found a related set of factors in each place. The problem is, as I said in my previous post, that there is great reluctance on the part of authorities to admit that he is on to something and do the research needed to prove it. ----->Hi Kris, I'm not at all saying that Purdey is wrong. whether he is correct or not has nothing to do with the point i was making. the point is simply that only 3-4 paras of 'evidence' were presented in an article that goes on to state conclusively that it's correct. most people are going to bristle at that, imo, and want to read more information on a complex subject before coming to their *own* conclusion. i have no issue with whether it's correct or not, only in the way that it was presented. i think it would've been better to offer some links to the more indepth article by Purdey right there on the WAPF website and to his own extensive site and invite the reader to draw their own conclusions rather than to conclude for them after presenting such a brief explanation. Suze Fisher Web Design & Development http://members.bellatlantic.net/~vze3shjg/ mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 30, 2002 Report Share Posted August 30, 2002 Suze how do you know BSE/CWE is complex? ------->i don't. but the information i've read about it to date, including some of Purdey's work, is. Suze Fisher Web Design & Development http://members.bellatlantic.net/~vze3shjg/ mailto:s.fisher22@... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 31, 2002 Report Share Posted August 31, 2002 I agree with you Suze, that the article on the WAPF site should have links to more in depth info. That may be true of a number of articles on the WAPF website, since crosslinks are one of the powerful things about the Internet. Kris > ----->Hi Kris, > > I'm not at all saying that Purdey is wrong. whether he is correct or not has > nothing to do with the point i was making. the point is simply that only > 3-4 paras of 'evidence' were presented in an article that goes on to state > conclusively that it's correct. most people are going to bristle at that, > imo, and want to read more information on a complex subject before coming to > their *own* conclusion. i have no issue with whether it's correct or not, > only in the way that it was presented. i think it would've been better to > offer some links to the more indepth article by Purdey right there on the > WAPF website and to his own extensive site and invite the reader to draw > their own conclusions rather than to conclude for them after presenting such > a brief explanation. > > Suze Fisher Quote Link to comment Share on other sites More sharing options...
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