6 polio outbreaks caused by the polio vaccine show how important oral polio vaccine is - say what?

[Guest guest]

From: Meryl Dorey <meryl@...>

These are the sorts of reports that make me think that most mainstream

scientists are living in some sort of alternate reality. They describe 6

outbreaks of polio that were caused by vaccine virus and say that these

outbreaks " highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks " . Well, excuse me, but wouldn't it be much

truer to say that these outbreaks highlight the importance of banning the

oral poliovirus vaccine as most developed countries have already done?

http://www.infectiousdiseasenews.com/article/85273.aspx

CDC.MMWR. 2011;60:846-850.Three new outbreaks of

circulating vaccine-derived polioviruses were identified and three

previously identified outbreaks continued through late 2010 or into 2011.

These new outbreaks highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks, according to the CDC.

The three new outbreaks of circulating VDPVs (cVDPVs) ranged in size from

six to 16 cases and were reported in Afghanistan, Ethiopia and India. The

previously reported outbreaks in Nigeria, Democratic Republic of Congo

(DRC) and Somalia continued through late 2010 or into 2011 and resulted

in a total of 405 cases. In addition, two countries experienced

importations of cVDPVs from Nigeria, and nine people living in seven

middle-income and developing countries who were newly identified as

paralyzed were found to excrete VDPVs. VDPVs were also found among people

and environmental samples in 15 countries. The emerging cVDPVs were type

2 in all but one country, according to a report in today’s Morbidity

and Mortality Weekly Report.

Story continues below↓

Vaccine virus vs. wild virus

Poliovirus isolates are grouped

into three categories, based on the extent of divergence of the major

viral surface protein (VP1) nucleotide region compared with the

corresponding OPV strain: 1) VRPVs (<1% divergent [types 1 and 3] or

<0.6% divergent [type 2]); 2) VDPVs (VRPVs that are >1% divergent

[types 1 and 3] or >0.6% divergent [type 2] from the corresponding OPV

strain); and 3) Wild polioviruses (WPV)

(no genetic evidence of derivation from any vaccine strain).

VDPVs are further categorized as 1) cVDPVs, defined as evidence of

person-to-person transmission in the community; 2)

immunodeficiency-associated VDPVs (iVDPVs), which is when the virus is

isolated from people with primary immunodeficiencies who have prolonged

VDPV infections; and 3) ambiguous VDPVs (aVDPVs), defined as either

clinical isolates from people with no known immunodeficiency or sewage

isolates whose source is unknown, according to the CDC.

VDPV

The three categories of VDPVs

differ in their public health importance, according to the CDC report,

which was partially compiled by WHO and the Global Polio Laboratory

Network.

First, cVDPVs are made of biologic properties of WPVs and have the

potential to circulate for years in settings where polio vaccination

coverage to prevent that particular type is low. In addition, for each

case detected, another 100 to 1,000 asymptomatic infections occur among

susceptible children, which is also true for WPVs.

Second, iVDPVs can be excreted for many years by people with specific

primary immunodeficiencies, and some chronic infections are latent. Many

people with prolonged iVDPV infections either spontaneously clear the

infections or die from the complications of immunodeficiency.

Nonetheless, in the absence of effective antiviral therapy, those

infected with iVDPVs without paralysis are at risk for developing

paralytic poliomyelitis and may potentially infect others with the

virus.

Third, aVDPVs are heterogeneous. Some represent the initial isolates from

cVDPV outbreaks, especially in areas with type-specific immunity gaps.

The authors of the report stress that aVDPVs isolated during cVDPV

outbreaks of the same serotype might be cVDPVs whose progenitors or

progeny were not detected.

Other aVDPVs are likely iVDPVs from latent chronic infections, while

other aVDPVs, “especially those with limited divergence, might

represent limited spread of OPV virus or the upper limit of OPV

divergence in a single normal vaccine recipient or contact,†the

authors wrote.

The increased frequency of VDPV detection compared with the previous

reporting period in 2009 is attributable partly to increased surveillance

sensitivity and improved laboratory methods. The increase in cVDPVs is

mostly a result of increasing type-specific immunity gaps in areas with

low routine vaccination, arising from the intensive use of monovalent OPV

type 1 and bivalent OPV in mass immunization campaigns, according to the

report.

Sheri Nakken, former R.N., MA, Hahnemannian

Homeopath

Vaccination Information & Choice Network, Washington State, USA

Vaccines -

http://vaccinationdangers.wordpress.com/ Homeopathy

http://homeopathycures.wordpress.com

Vaccine Dangers, Childhood Disease Classes & Homeopathy

Online/email courses - next classes start April 22

[Guest guest]

From: Meryl Dorey <meryl@...>

These are the sorts of reports that make me think that most mainstream

scientists are living in some sort of alternate reality. They describe 6

outbreaks of polio that were caused by vaccine virus and say that these

outbreaks " highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks " . Well, excuse me, but wouldn't it be much

truer to say that these outbreaks highlight the importance of banning the

oral poliovirus vaccine as most developed countries have already done?

http://www.infectiousdiseasenews.com/article/85273.aspx

CDC.MMWR. 2011;60:846-850.Three new outbreaks of

circulating vaccine-derived polioviruses were identified and three

previously identified outbreaks continued through late 2010 or into 2011.

These new outbreaks highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks, according to the CDC.

The three new outbreaks of circulating VDPVs (cVDPVs) ranged in size from

six to 16 cases and were reported in Afghanistan, Ethiopia and India. The

previously reported outbreaks in Nigeria, Democratic Republic of Congo

(DRC) and Somalia continued through late 2010 or into 2011 and resulted

in a total of 405 cases. In addition, two countries experienced

importations of cVDPVs from Nigeria, and nine people living in seven

middle-income and developing countries who were newly identified as

paralyzed were found to excrete VDPVs. VDPVs were also found among people

and environmental samples in 15 countries. The emerging cVDPVs were type

2 in all but one country, according to a report in today’s Morbidity

and Mortality Weekly Report.

Story continues below↓

Vaccine virus vs. wild virus

Poliovirus isolates are grouped

into three categories, based on the extent of divergence of the major

viral surface protein (VP1) nucleotide region compared with the

corresponding OPV strain: 1) VRPVs (<1% divergent [types 1 and 3] or

<0.6% divergent [type 2]); 2) VDPVs (VRPVs that are >1% divergent

[types 1 and 3] or >0.6% divergent [type 2] from the corresponding OPV

strain); and 3) Wild polioviruses (WPV)

(no genetic evidence of derivation from any vaccine strain).

VDPVs are further categorized as 1) cVDPVs, defined as evidence of

person-to-person transmission in the community; 2)

immunodeficiency-associated VDPVs (iVDPVs), which is when the virus is

isolated from people with primary immunodeficiencies who have prolonged

VDPV infections; and 3) ambiguous VDPVs (aVDPVs), defined as either

clinical isolates from people with no known immunodeficiency or sewage

isolates whose source is unknown, according to the CDC.

VDPV

The three categories of VDPVs

differ in their public health importance, according to the CDC report,

which was partially compiled by WHO and the Global Polio Laboratory

Network.

First, cVDPVs are made of biologic properties of WPVs and have the

potential to circulate for years in settings where polio vaccination

coverage to prevent that particular type is low. In addition, for each

case detected, another 100 to 1,000 asymptomatic infections occur among

susceptible children, which is also true for WPVs.

Second, iVDPVs can be excreted for many years by people with specific

primary immunodeficiencies, and some chronic infections are latent. Many

people with prolonged iVDPV infections either spontaneously clear the

infections or die from the complications of immunodeficiency.

Nonetheless, in the absence of effective antiviral therapy, those

infected with iVDPVs without paralysis are at risk for developing

paralytic poliomyelitis and may potentially infect others with the

virus.

Third, aVDPVs are heterogeneous. Some represent the initial isolates from

cVDPV outbreaks, especially in areas with type-specific immunity gaps.

The authors of the report stress that aVDPVs isolated during cVDPV

outbreaks of the same serotype might be cVDPVs whose progenitors or

progeny were not detected.

Other aVDPVs are likely iVDPVs from latent chronic infections, while

other aVDPVs, “especially those with limited divergence, might

represent limited spread of OPV virus or the upper limit of OPV

divergence in a single normal vaccine recipient or contact,†the

authors wrote.

The increased frequency of VDPV detection compared with the previous

reporting period in 2009 is attributable partly to increased surveillance

sensitivity and improved laboratory methods. The increase in cVDPVs is

mostly a result of increasing type-specific immunity gaps in areas with

low routine vaccination, arising from the intensive use of monovalent OPV

type 1 and bivalent OPV in mass immunization campaigns, according to the

report.

Sheri Nakken, former R.N., MA, Hahnemannian

Homeopath

Vaccination Information & Choice Network, Washington State, USA

Vaccines -

http://vaccinationdangers.wordpress.com/ Homeopathy

http://homeopathycures.wordpress.com

Vaccine Dangers, Childhood Disease Classes & Homeopathy

Online/email courses - next classes start April 22

[Guest guest]

From: Meryl Dorey <meryl@...>

These are the sorts of reports that make me think that most mainstream

scientists are living in some sort of alternate reality. They describe 6

outbreaks of polio that were caused by vaccine virus and say that these

outbreaks " highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks " . Well, excuse me, but wouldn't it be much

truer to say that these outbreaks highlight the importance of banning the

oral poliovirus vaccine as most developed countries have already done?

http://www.infectiousdiseasenews.com/article/85273.aspx

CDC.MMWR. 2011;60:846-850.Three new outbreaks of

circulating vaccine-derived polioviruses were identified and three

previously identified outbreaks continued through late 2010 or into 2011.

These new outbreaks highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks, according to the CDC.

The three new outbreaks of circulating VDPVs (cVDPVs) ranged in size from

six to 16 cases and were reported in Afghanistan, Ethiopia and India. The

previously reported outbreaks in Nigeria, Democratic Republic of Congo

(DRC) and Somalia continued through late 2010 or into 2011 and resulted

in a total of 405 cases. In addition, two countries experienced

importations of cVDPVs from Nigeria, and nine people living in seven

middle-income and developing countries who were newly identified as

paralyzed were found to excrete VDPVs. VDPVs were also found among people

and environmental samples in 15 countries. The emerging cVDPVs were type

2 in all but one country, according to a report in today’s Morbidity

and Mortality Weekly Report.

Story continues below↓

Vaccine virus vs. wild virus

Poliovirus isolates are grouped

into three categories, based on the extent of divergence of the major

viral surface protein (VP1) nucleotide region compared with the

corresponding OPV strain: 1) VRPVs (<1% divergent [types 1 and 3] or

<0.6% divergent [type 2]); 2) VDPVs (VRPVs that are >1% divergent

[types 1 and 3] or >0.6% divergent [type 2] from the corresponding OPV

strain); and 3) Wild polioviruses (WPV)

(no genetic evidence of derivation from any vaccine strain).

VDPVs are further categorized as 1) cVDPVs, defined as evidence of

person-to-person transmission in the community; 2)

immunodeficiency-associated VDPVs (iVDPVs), which is when the virus is

isolated from people with primary immunodeficiencies who have prolonged

VDPV infections; and 3) ambiguous VDPVs (aVDPVs), defined as either

clinical isolates from people with no known immunodeficiency or sewage

isolates whose source is unknown, according to the CDC.

VDPV

The three categories of VDPVs

differ in their public health importance, according to the CDC report,

which was partially compiled by WHO and the Global Polio Laboratory

Network.

First, cVDPVs are made of biologic properties of WPVs and have the

potential to circulate for years in settings where polio vaccination

coverage to prevent that particular type is low. In addition, for each

case detected, another 100 to 1,000 asymptomatic infections occur among

susceptible children, which is also true for WPVs.

Second, iVDPVs can be excreted for many years by people with specific

primary immunodeficiencies, and some chronic infections are latent. Many

people with prolonged iVDPV infections either spontaneously clear the

infections or die from the complications of immunodeficiency.

Nonetheless, in the absence of effective antiviral therapy, those

infected with iVDPVs without paralysis are at risk for developing

paralytic poliomyelitis and may potentially infect others with the

virus.

Third, aVDPVs are heterogeneous. Some represent the initial isolates from

cVDPV outbreaks, especially in areas with type-specific immunity gaps.

The authors of the report stress that aVDPVs isolated during cVDPV

outbreaks of the same serotype might be cVDPVs whose progenitors or

progeny were not detected.

Other aVDPVs are likely iVDPVs from latent chronic infections, while

other aVDPVs, “especially those with limited divergence, might

represent limited spread of OPV virus or the upper limit of OPV

divergence in a single normal vaccine recipient or contact,†the

authors wrote.

The increased frequency of VDPV detection compared with the previous

reporting period in 2009 is attributable partly to increased surveillance

sensitivity and improved laboratory methods. The increase in cVDPVs is

mostly a result of increasing type-specific immunity gaps in areas with

low routine vaccination, arising from the intensive use of monovalent OPV

type 1 and bivalent OPV in mass immunization campaigns, according to the

report.

Sheri Nakken, former R.N., MA, Hahnemannian

Homeopath

Vaccination Information & Choice Network, Washington State, USA

Vaccines -

http://vaccinationdangers.wordpress.com/ Homeopathy

http://homeopathycures.wordpress.com

Vaccine Dangers, Childhood Disease Classes & Homeopathy

Online/email courses - next classes start April 22

[Guest guest]

From: Meryl Dorey <meryl@...>

These are the sorts of reports that make me think that most mainstream

scientists are living in some sort of alternate reality. They describe 6

outbreaks of polio that were caused by vaccine virus and say that these

outbreaks " highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks " . Well, excuse me, but wouldn't it be much

truer to say that these outbreaks highlight the importance of banning the

oral poliovirus vaccine as most developed countries have already done?

http://www.infectiousdiseasenews.com/article/85273.aspx

CDC.MMWR. 2011;60:846-850.Three new outbreaks of

circulating vaccine-derived polioviruses were identified and three

previously identified outbreaks continued through late 2010 or into 2011.

These new outbreaks highlight the importance of trivalent oral poliovirus

vaccination campaigns to prevent both wild-type and vaccine-derived

poliovirus outbreaks, according to the CDC.

The three new outbreaks of circulating VDPVs (cVDPVs) ranged in size from

six to 16 cases and were reported in Afghanistan, Ethiopia and India. The

previously reported outbreaks in Nigeria, Democratic Republic of Congo

(DRC) and Somalia continued through late 2010 or into 2011 and resulted

in a total of 405 cases. In addition, two countries experienced

importations of cVDPVs from Nigeria, and nine people living in seven

middle-income and developing countries who were newly identified as

paralyzed were found to excrete VDPVs. VDPVs were also found among people

and environmental samples in 15 countries. The emerging cVDPVs were type

2 in all but one country, according to a report in today’s Morbidity

and Mortality Weekly Report.

Story continues below↓

Vaccine virus vs. wild virus

Poliovirus isolates are grouped

into three categories, based on the extent of divergence of the major

viral surface protein (VP1) nucleotide region compared with the

corresponding OPV strain: 1) VRPVs (<1% divergent [types 1 and 3] or

<0.6% divergent [type 2]); 2) VDPVs (VRPVs that are >1% divergent

[types 1 and 3] or >0.6% divergent [type 2] from the corresponding OPV

strain); and 3) Wild polioviruses (WPV)

(no genetic evidence of derivation from any vaccine strain).

VDPVs are further categorized as 1) cVDPVs, defined as evidence of

person-to-person transmission in the community; 2)

immunodeficiency-associated VDPVs (iVDPVs), which is when the virus is

isolated from people with primary immunodeficiencies who have prolonged

VDPV infections; and 3) ambiguous VDPVs (aVDPVs), defined as either

clinical isolates from people with no known immunodeficiency or sewage

isolates whose source is unknown, according to the CDC.

VDPV

The three categories of VDPVs

differ in their public health importance, according to the CDC report,

which was partially compiled by WHO and the Global Polio Laboratory

Network.

First, cVDPVs are made of biologic properties of WPVs and have the

potential to circulate for years in settings where polio vaccination

coverage to prevent that particular type is low. In addition, for each

case detected, another 100 to 1,000 asymptomatic infections occur among

susceptible children, which is also true for WPVs.

Second, iVDPVs can be excreted for many years by people with specific

primary immunodeficiencies, and some chronic infections are latent. Many

people with prolonged iVDPV infections either spontaneously clear the

infections or die from the complications of immunodeficiency.

Nonetheless, in the absence of effective antiviral therapy, those

infected with iVDPVs without paralysis are at risk for developing

paralytic poliomyelitis and may potentially infect others with the

virus.

Third, aVDPVs are heterogeneous. Some represent the initial isolates from

cVDPV outbreaks, especially in areas with type-specific immunity gaps.

The authors of the report stress that aVDPVs isolated during cVDPV

outbreaks of the same serotype might be cVDPVs whose progenitors or

progeny were not detected.

Other aVDPVs are likely iVDPVs from latent chronic infections, while

other aVDPVs, “especially those with limited divergence, might

represent limited spread of OPV virus or the upper limit of OPV

divergence in a single normal vaccine recipient or contact,†the

authors wrote.

The increased frequency of VDPV detection compared with the previous

reporting period in 2009 is attributable partly to increased surveillance

sensitivity and improved laboratory methods. The increase in cVDPVs is

mostly a result of increasing type-specific immunity gaps in areas with

low routine vaccination, arising from the intensive use of monovalent OPV

type 1 and bivalent OPV in mass immunization campaigns, according to the

report.

Sheri Nakken, former R.N., MA, Hahnemannian

Homeopath

Vaccination Information & Choice Network, Washington State, USA

Vaccines -

http://vaccinationdangers.wordpress.com/ Homeopathy

http://homeopathycures.wordpress.com

Vaccine Dangers, Childhood Disease Classes & Homeopathy

Online/email courses - next classes start April 22

[Guest guest]

" alternate reality " indeed! Or really good spin doctors are working over time!

They can take any outcome, and spin it around to justify their industry.

Sickening, but impressive.

Sylvia

>

> From: Meryl Dorey <meryl@...>

>

>

>

> These are the sorts of reports that make me think

> that most mainstream scientists are living in

> some sort of alternate reality. They describe 6

> outbreaks of polio that were caused by vaccine

> virus and say that these outbreaks " highlight the

> importance of trivalent oral poliovirus

> vaccination campaigns to prevent both wild-type

> and vaccine-derived poliovirus outbreaks " . Well,

> excuse me, but wouldn't it be much truer to say

> that these outbreaks highlight the importance of

> banning the oral poliovirus vaccine as most

> developed countries have already done?

>

>

<http://www.infectiousdiseasenews.com/article/85273.aspx>http://www.infectiousdi\

seasenews.com/article/85273.aspx

>

>

>

> CDC.MMWR. 2011;60:846-850.

>

> Three new outbreaks of circulating

> vaccine-derived polioviruses were identified and

> three previously identified outbreaks continued

> through late 2010 or into 2011. These new

> outbreaks highlight the importance of trivalent

> oral poliovirus vaccination campaigns to prevent

> both wild-type and vaccine-derived poliovirus outbreaks, according to the CDC.

>

> The three new outbreaks of circulating VDPVs

> (cVDPVs) ranged in size from six to 16 cases and

> were reported in Afghanistan, Ethiopia and India.

> The previously reported outbreaks in Nigeria,

> Democratic Republic of Congo (DRC) and Somalia

> continued through late 2010 or into 2011 and

> resulted in a total of 405 cases. In addition,

> two countries experienced importations of cVDPVs

> from Nigeria, and nine people living in seven

> middle-income and developing countries who were

> newly identified as paralyzed were found to

> excrete VDPVs. VDPVs were also found among people

> and environmental samples in 15 countries. The

> emerging cVDPVs were type 2 in all but one

> country, according to a report in today’s

> Morbidity and Mortality Weekly Report.

>

> Story continues below↠"

>

> Vaccine virus vs. wild virus

>

> Poliovirus isolates are grouped into three

> categories, based on the extent of divergence of

> the major viral surface protein (VP1) nucleotide

> region compared with the corresponding OPV

> strain: 1) VRPVs (<1% divergent [types 1 and 3]

> or <0.6% divergent [type 2]); 2) VDPVs (VRPVs

> that are >1% divergent [types 1 and 3] or >0.6%

> divergent [type 2] from the corresponding OPV

> strain); and 3) Wild polioviruses (WPV) (no

> genetic evidence of derivation from any vaccine strain).

>

> VDPVs are further categorized as 1) cVDPVs,

> defined as evidence of person-to-person

> transmission in the community; 2)

> immunodeficiency-associated VDPVs (iVDPVs), which

> is when the virus is isolated from people with

> primary immunodeficiencies who have prolonged

> VDPV infections; and 3) ambiguous VDPVs (aVDPVs),

> defined as either clinical isolates from people

> with no known immunodeficiency or sewage isolates

> whose source is unknown, according to the CDC.

>

> VDPV

>

> The three categories of VDPVs differ in their

> public health importance, according to the CDC

> report, which was partially compiled by WHO and

> the Global Polio Laboratory Network.

>

> First, cVDPVs are made of biologic properties of

> WPVs and have the potential to circulate for

> years in settings where polio vaccination

> coverage to prevent that particular type is low.

> In addition, for each case detected, another 100

> to 1,000 asymptomatic infections occur among

> susceptible children, which is also true for WPVs.

>

> Second, iVDPVs can be excreted for many years by

> people with specific primary immunodeficiencies,

> and some chronic infections are latent. Many

> people with prolonged iVDPV infections either

> spontaneously clear the infections or die from

> the complications of immunodeficiency.

> Nonetheless, in the absence of effective

> antiviral therapy, those infected with iVDPVs

> without paralysis are at risk for developing

> paralytic poliomyelitis and may potentially infect others with the virus.

>

> Third, aVDPVs are heterogeneous. Some represent

> the initial isolates from cVDPV outbreaks,

> especially in areas with type-specific immunity

> gaps. The authors of the report stress that

> aVDPVs isolated during cVDPV outbreaks of the

> same serotype might be cVDPVs whose progenitors or progeny were not detected.

>

> Other aVDPVs are likely iVDPVs from latent

> chronic infections, while other aVDPVs,

> “especially those with limited divergence,

> might represent limited spread of OPV virus or

> the upper limit of OPV divergence in a single

> normal vaccine recipient or contact,†the authors wrote.

>

> The increased frequency of VDPV detection

> compared with the previous reporting period in

> 2009 is attributable partly to increased

> surveillance sensitivity and improved laboratory

> methods. The increase in cVDPVs is mostly a

> result of increasing type-specific immunity gaps

> in areas with low routine vaccination, arising

> from the intensive use of monovalent OPV type 1

> and bivalent OPV in mass immunization campaigns, according to the report.

>

>

> Sheri Nakken, former R.N., MA, Hahnemannian Homeopath

> Vaccination Information & Choice Network, Washington State, USA

> Vaccines -

> http://vaccinationdangers.wordpress.com/

> Homeopathy http://homeopathycures.wordpress.com

> Vaccine Dangers, Childhood Disease Classes &

> Homeopathy Online/email courses - next classes start April 22

>