Re: Beta endorphins, LDN and Nausea

[Guest guest]

Can you use Lactose? If not, switch to Acidophilus as your filler. Avicel and

Calcium Carbonate can cause nausea if your body is intolerant to them.

Acidophilus is great for the digestive system. Avicel caused nausea with me,

I'm allergic to it. I use Lactose now.

Bren

[Guest guest]

Can you use Lactose? If not, switch to Acidophilus as your filler. Avicel and

Calcium Carbonate can cause nausea if your body is intolerant to them.

Acidophilus is great for the digestive system. Avicel caused nausea with me,

I'm allergic to it. I use Lactose now.

Bren

[Guest guest]

Hey Daryssa.

Have you tried eating a little something when taking it before bedtime?

-----Original Message-----

From: daryssafox [mailto:daryssafox@...]

Sent: Saturday, July 31, 2004 11:34 AM

low dose naltrexone

Subject: [low dose naltrexone] Beta endorphins, LDN and Nausea

Hi,

The article below got me to wondering. Since taking LDN, I have been

nauseous - partcularly in the morning. I have used both avicel and calcium

carbonate as filler and it doesn't seem to mater except that the avicel is

worse. The only time I can remember feeling this way was during the early

months of my pregnancies many years ago. I have been post menopausal for

quite some time. I have tried taking LDN during the day with food and that

does not cause nausea,

Has this happened to others? Is it possible that the nausea might be

related to some homonal pregnancy type changes? Any suggestions on what to

do about it? Any comments Thanks.

Daryssa

--- In low dose naltrexone , Radnai Márton <radnaim@f...>

wrote:

> Hi All,

>

> I found an interesting research article about pregnancy and MS. It

says

> that during pregancy the Th2 cells are upregulated and Th1 cells

> downregulated.

>

> The same thing happens when beta endorphine level is elevated by LDN.

>

> -----------------------------------------

>

> J Neurol Sci. 2004 Jul 15;222(1-2):21-7. Related Articles,

Links

>

>

> Th1/Th2 cytokine patterns and clinical profiles during and after

> pregnancy in women with multiple sclerosis.

>

> Al-Shammri S, Rawoot P, Azizieh F, AbuQoora A, Hanna M, Saminathan TR,

> Raghupathy R.

>

> Department of Medicine, Faculty of Medicine, Kuwait University,

P.O. Box

> 24923, Safat 13110, Kuwait.

>

> Pregnancy in multiple sclerosis (MS) patients is associated with a

lower

> risk of progression and lower rate of exacerbation. These

beneficial

> effects are reversed postpartum. Considering that the pathogenesis

of

> MS appears to involve cell-mediated immune reactivity, and that

> pregnancy is accompanied by a depressed cell-mediated immunity, it has

> been proposed that the lower relapse rate and risk of

progression

> of MS during pregnancy may be due to a pregnancy-associated down-

> regulation of cell-mediated immunity. In addition, pregnancy

results in a

> shift towards a T helper (Th) 2 cytokine profile, which is

presumably

> protective for MS. This study was aimed at investigating the

> relationship between clinical status of MS and cytokine levels in

eight

> patients with MS who were followed through pregnancy and after

> delivery. Peripheral blood lymphocytes from these women were

> stimulated with a mitogen at different time points during and

after

> gestation and the levels of Th1 cytokines (IFNgamma, TNFalpha) and

> Th2 cytokines (IL-4, IL-10) were estimated by ELISA. It was

established

> that six of the eight MS patients studied showed a distinct shift

from a

> Th2 cytokine bias during pregnancy towards a Th1 cytokine bias

after

> delivery. These results suggest a possible association between

> decreased incidence of exacerbation of MS in pregnancy and a

> pregnancy-induced shift towards Th2 cytokine bias.

[Guest guest]

LDN has been used to improve fertility (study from Australia) in

females, it would be reasonable to assume that it affects the FSH

(follicular Stimulating Hormone) and other female hormones.

A

> > Hi All,

> >

> > I found an interesting research article about pregnancy and MS.

It

> says

> > that during pregancy the Th2 cells are upregulated and Th1 cells

> > downregulated.

> >

> > The same thing happens when beta endorphine level is elevated by

LDN.

> >

> > -----------------------------------------

> >

> > J Neurol Sci. 2004 Jul 15;222(1-2):21-7. Related Articles,

> Links

> >

> >

> > Th1/Th2 cytokine patterns and clinical profiles during and after

> > pregnancy in women with multiple sclerosis.

> >

> > Al-Shammri S, Rawoot P, Azizieh F, AbuQoora A, Hanna M,

Saminathan TR,

> > Raghupathy R.

> >

> > Department of Medicine, Faculty of Medicine, Kuwait University,

> P.O. Box

> > 24923, Safat 13110, Kuwait.

> >

> > Pregnancy in multiple sclerosis (MS) patients is associated with

a

> lower

> > risk of progression and lower rate of exacerbation. These

> beneficial

> > effects are reversed postpartum. Considering that the

pathogenesis

> of

> > MS appears to involve cell-mediated immune reactivity, and that

> > pregnancy is accompanied by a depressed cell-mediated immunity,

it has

> > been proposed that the lower relapse rate and risk of

> progression

> > of MS during pregnancy may be due to a pregnancy-associated down-

> > regulation of cell-mediated immunity. In addition, pregnancy

> results in a

> > shift towards a T helper (Th) 2 cytokine profile, which is

> presumably

> > protective for MS. This study was aimed at investigating the

> > relationship between clinical status of MS and cytokine levels in

> eight

> > patients with MS who were followed through pregnancy and after

> > delivery. Peripheral blood lymphocytes from these women were

> > stimulated with a mitogen at different time points during and

> after

> > gestation and the levels of Th1 cytokines (IFNgamma, TNFalpha)

and

> > Th2 cytokines (IL-4, IL-10) were estimated by ELISA. It was

> established

> > that six of the eight MS patients studied showed a distinct shift

> from a

> > Th2 cytokine bias during pregnancy towards a Th1 cytokine bias

> after

> > delivery. These results suggest a possible association between

> > decreased incidence of exacerbation of MS in pregnancy and a

> > pregnancy-induced shift towards Th2 cytokine bias.

>

>

>

>

[Guest guest]

It makes sense that LDN could be effecting female hormones which is

why I asked. If it does, then why haven't other women taking LDN

had similar experiences? It looks like this is going to be a trial

and error experience. Dar

> > > Hi All,

> > >

> > > I found an interesting research article about pregnancy and

MS.

> It

> > says

> > > that during pregancy the Th2 cells are upregulated and Th1

cells

> > > downregulated.

> > >

> > > The same thing happens when beta endorphine level is elevated

by

> LDN.

> > >

> > > -----------------------------------------

> > >

> > > J Neurol Sci. 2004 Jul 15;222(1-2):21-7. Related Articles,

> > Links

> > >

> > >

> > > Th1/Th2 cytokine patterns and clinical profiles during and

after

> > > pregnancy in women with multiple sclerosis.

> > >

> > > Al-Shammri S, Rawoot P, Azizieh F, AbuQoora A, Hanna M,

> Saminathan TR,

> > > Raghupathy R.

> > >

> > > Department of Medicine, Faculty of Medicine, Kuwait University,

> > P.O. Box

> > > 24923, Safat 13110, Kuwait.

> > >

> > > Pregnancy in multiple sclerosis (MS) patients is associated

with

> a

> > lower

> > > risk of progression and lower rate of exacerbation. These

> > beneficial

> > > effects are reversed postpartum. Considering that the

> pathogenesis

> > of

> > > MS appears to involve cell-mediated immune reactivity, and

that

> > > pregnancy is accompanied by a depressed cell-mediated

immunity,

> it has

> > > been proposed that the lower relapse rate and risk of

> > progression

> > > of MS during pregnancy may be due to a pregnancy-associated

down-

>

> > > regulation of cell-mediated immunity. In addition, pregnancy

> > results in a

> > > shift towards a T helper (Th) 2 cytokine profile, which is

> > presumably

> > > protective for MS. This study was aimed at investigating the

> > > relationship between clinical status of MS and cytokine levels

in

> > eight

> > > patients with MS who were followed through pregnancy and after

> > > delivery. Peripheral blood lymphocytes from these women were

> > > stimulated with a mitogen at different time points during and

> > after

> > > gestation and the levels of Th1 cytokines (IFNgamma, TNFalpha)

> and

> > > Th2 cytokines (IL-4, IL-10) were estimated by ELISA. It was

> > established

> > > that six of the eight MS patients studied showed a distinct

shift

> > from a

> > > Th2 cytokine bias during pregnancy towards a Th1 cytokine bias

> > after

> > > delivery. These results suggest a possible association between

> > > decreased incidence of exacerbation of MS in pregnancy and a

> > > pregnancy-induced shift towards Th2 cytokine bias.

> >

> >

> >

> >