Tinnitus

March 22, 2010

Hi ,

Your email had me laughing (about the auditory nerve squawking back at us) and

feeling hopeful at the thought of soon being able to understand what a DJ or

weather man is saying. Thanks so much for the advice. My audiologist did give me

a softer program on my device, which I can switch to if I want to turn

everything down. However, even when I turn off the implant completely, my

tinnitus has decided to mimic the implant noise, so the screeching is almost as

loud as when the implant is on. When I wake up in the morning, I keep thinking I

fell asleep with it on because my tinnitus is so loud. I had terrible tinnitus

pre-implant which has really tired me out or made me cry due to discomfort (it's

actually painful sometimes). So I am used to dealing with it daily, it's just a

bit magnified now with the implant. But I will definitely call my audiologist

for an adjustment if it does get to a point where I can't handle it. Thanks so

much for the helpful and

informative message!

-

________________________________

From: Kinsella <jmkinsella55@...>

< >

Sent: Mon, March 22, 2010 10:45:38 AM

Subject: Tinnitus

I'm changing the subject header since so many of you have been discussing their

problems with tinnitus. This is for those of you who are recently implanted and

are finding their tinnitus to be more active. As you know, the surgery itself

effects the inner ear, and in some people, this causes the tinnitus to act up.

I was one of those people, and was also blessed to have it disappear after a

time. Then for some people, when they are activated, this also causes the

tinnitus to act up more. I imagine that the auditory nerve is suddenly told to

get back to work after a long period of being partially or totally lazy

(depending on your hearing history), and now it is squawking back, saying, " No,

I don't wanna, you can't make me, I'm going to whine (bells ringing, train

sounding, jackhammer going off, name your poison) until you let me off the

hook! " Well, maybe that isn't what is going on, but hey, just imagine it,

okay??? Anyway, whatever is

happening, it is a reaction to the changes that are taking place. Hopefully for

most of you, after a period of time the tinnitus will settle down so that

eventually the tinnitus will either disappear completely, or if that isn't the

case the sounds coming through the processor will drown out the tinnitus enough

so you can ignore it. In either case, patience will have to be the order of the

day. However...

- you said " I'm hoping mine goes down too, once the sounds coming through

my implant start sounding like normal sounds (instead of a louder version of my

tinnitus.) As far as dealing with it, I wish I had an answer. It really exhausts

me, sometimes makes me dizzy or will cause my eyes to start tearing up. " One

thing you may want to consider is that you may need to have your map adjusted.

For some people certain electrodes need to be shut down because they cause

physical problems, such as facial or eye twitching. The audiologist can

determine if this is the case, and turn off those electrodes. There is no need

to suffer with this, waiting for when sounds become more normal like. In my

case, my head vibrated when there was a high frequency sound. We turned off

those electrodes for about 4 months until my brain got accustomed to hearing all

of the other noise, and then we were able to turn those electrodes back on. So

please, don't just

suffer along with this. There are things that can be done to help you brain to

gradually adjust. Just call your audiologist, and see if they can turn down the

volume a little (you may be able to do this yourself), turn down the sensitivity

a little (again, you may be able to do this yourself), or just have the

offending electrodes turned off to allow your brain time to adjust.

When I was activated I considered my brain to be going through a training period

much like a person does when they want to run their first marathon. You don't

just go out and run 26.3 miles right off the bat. You have to gradually

condition your body to get in shape for the big day. The same is true for your

brain. You are taking it from a quiet mode into a very noisy world, and

suddenly the brain has to process all this sound, and so it is confused and

tired. While you don't want to give in to the brain by giving it quiet periods,

because that will take longer for you to reach your goal, but perhaps you can

slowly " condition " your brain to the sounds by starting with a quieter map, and

then each week turn it up a little louder. What I did was on week 1 I started

at volume 4 and sensitivity 5. Each week I increased it by 1, until I had

reached 9 for volume and 10 for sensitivity (normal range). After 6 weeks of

this I went back in for a

new map and did the same thing, gradually conditioning my brain to tolerate

sound. Eventually I was able to tolerate all sounds, and hear in the 20 dB

range for all frequencies.

So consider what might work for your individual situation. Give your brain a

chance to gradually adjust...don' t ever give up because it will eventually get

better! Another suggestion if you are usually in a quiet world ( I work in an

office with minimal sounds unless someone comes to talk to me) is to have your

processor hooked up to a MP3 player, and listen to a radio station. That has a

mix of words and music. Over time (like several months), eventually your brain

will make out what the DJ is saying, the weather man is saying, and possibly the

music that is playing. I did this, and will never forget the day when I

realized that I understood the weather man when he gave the current temperature!

If the music is too annoying at this early stage, then listen to a book while

reading along. Be active in your rehab! But only do what your brain will

tolerate. Don't make it so unbearable you don't want to wear the processor.

That isn't the

purpose of bringing you back into the hearing world.

I hope sharing my experiences will help you all!! I want you to all experience

the true joy of hearing and understanding what you hear once again!! Best of

luck!

July 1, 2010

> Anyone out there dealing with aspirin induced tinnitus? Wondering if I could

> drop down to 81 mg twice a day an stay desensitized. This would be temporary

> until the ringing goes away. I would then slowly increase the dose.

samters/surveys?id=12542588

July 2, 2010

Hi Jim,

I had to decrease my aspirin dosage because of tinnitus (also found that

long-term Zyrtec use can contribute to it). I chew five 81 mg tabs, which is 405

mg, twice per day. Easier on my stomach, and tinnitus has greatly decreased, but

not completely disappeared. Good luck to you. Uri

October 8, 2010

Diane~I read a study once that indicated that tinnitus sufferers often have low amplitude Alpha at the temporal lobes, AND, can also be connected to a Vitamin B12 deficiency, or nerve damage.~From: "Diane Stoler, Ed.D." <diane@...>brainm , neuroguide , Sent: Friday, October 8, 2010 10:43:53 AMSubject: Tinnitus

Does anyone have an idea for treating

tinnitus? I've tried T3/T4 Othmer

and it does not seem to help. C3/C4 does not seem to

help. Doing LENS and it does not seem to help.

Any ideas are truly welcomed.

Diane

Dr. Diane Stoler, Ed.D., LLC

P.O. Box 148

town, MA 01833

Toll Free in US 888.760.8730

Direct Dial 978.352.6349

For information on how to obtain books:

" Coping with Mild Traumatic Brain Injury:

A Guide to Living with the Challenges Associated with Concussion/

Brain Injury"

"Timeless- a novel:

Who Am I? "

Click the link below

<http://

www.drdiane.com

>

Neuroband:

For the Professional and Home User of

Neurofeedback equipment:

Clink the link below

http://www.drdiane.com/neuroband_order.html

Confidentiality:

This electronic message (E-mail) and any files

attached hereto contain confidential, legally

privileged and protected by copyright. If you

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E-mail is prohibited. If you have received this in error,

please notify the sender by telephone 978.352.8269 or replying by

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d/b/a-Dr. Diane for personal E-mails, or E-mails unconnected with Dr.

Diane Stoler, Ed.D, LLC patients' or client

business.

Dr. Diane ~

Catalyst for ChangeÂ® - A neuropsychologist who works with

individuals and organizations worldwide, to help them find Solutions

and ResourcesÂ® to overcome lifeâ€™s challenges and reach their

goals.

October 8, 2010

Dr. Diane,You could try referring your client to a neurotologist for testing of his/her inner ear and vestibular system. Also, sometimes hormone imbalances, metabolic and/or nutritional deficiencies can cause this as well. So, you may also try referring your client to a specialist in these areas.

Mike On Fri, Oct 8, 2010 at 8:43 AM, Diane Stoler, Ed.D. <diane@...> wrote:

Does anyone have an idea for treating

tinnitus? I've tried T3/T4 Othmer

and it does not seem to help. C3/C4 does not seem to

help. Doing LENS and it does not seem to help.

Any ideas are truly welcomed.

Diane

Dr. Diane Stoler, Ed.D., LLC

P.O. Box 148

town, MA 01833

Toll Free in US 888.760.8730

Direct Dial 978.352.6349

For information on how to obtain books:

" Coping with Mild Traumatic Brain Injury:

A Guide to Living with the Challenges Associated with Concussion/

Brain Injury "

" Timeless- a novel:

Who Am I? "

Click the link below

<http://

www.drdiane.com

>

Neuroband:

For the Professional and Home User of

Neurofeedback equipment:

Clink the link below

http://www.drdiane.com/neuroband_order.html

Confidentiality:

This electronic message (E-mail) and any files

attached hereto contain confidential, legally

privileged and protected by copyright. If you

are not the intended recipient, dissemination or copying of this

E-mail is prohibited. If you have received this in error,

please notify the sender by telephone 978.352.8269 or replying by

E-mail to info@..., then delete the E-mail completely from your

system.

This E-mail and any attachments have been scanned for viruses, but it is

the responsibility of the recipient to conduct their own security

measures and no responsibility is accepted by Dr. Diane Stoler,

Ed,D. , LLC d/b/a -Dr. Diane for loss or damage from receipt or use of

this E-mail.

No responsibility is accepted by Dr. Diane Stoler, Ed.D., LLC

d/b/a-Dr. Diane for personal E-mails, or E-mails unconnected with Dr.

Diane Stoler, Ed.D, LLC patients' or client

business.

Dr. Diane ~

Catalyst for Change® - A neuropsychologist who works with

individuals and organizations worldwide, to help them find Solutions

and Resources® to overcome life’s challenges and reach their

goals.

-- Mike

October 8, 2010

Hi Mike,

I always check out physical first. Been there, done it.

Appreciate you input.

Dr. Diane

On 10/08/2010 11:21:21 AM, Rouse (michael.hugh.rouse@...)

wrote:

October 8, 2010

Diane,

You may want to try a T5T6.

Tinnitus

Does anyone have an idea for treating tinnitus? I've tried T3/T4 Othmer and it does not seem to help. C3/C4 does not seem to help. Doing LENS and it does not seem to help. Any ideas are truly welcomed. Diane

Dr. Diane Stoler, Ed.D., LLC P.O. Box 148town, MA 01833Toll Free in US 888.760.8730Direct Dial 978.352.6349For information on how to obtain books:" Coping with Mild Traumatic Brain Injury: A Guide to Living with the Challenges Associated with Concussion/ Brain Injury" "Timeless- a novel: Who Am I? " Click the link below <http:// www.drdiane.com >Neuroband: For the Professional and Home User of Neurofeedback equipment: Clink the link belowhttp://www.drdiane.com/neuroband_order.htmlConfidentiality: This electronic message (E-mail) and any files attached hereto contain confidential, legally privileged and protected by copyright. If you are not the intended recipient, dissemination or copying of this E-mail is prohibited. If you have received this in error, please notify the sender by telephone 978.352.8269 or replying by E-mail to info@..., then delete the E-mail completely from your system. This E-mail and any attachments have been scanned for viruses, but it is the responsibility of the recipient to conduct their own security measures and no responsibility is accepted by Dr. Diane Stoler, Ed,D. , LLC d/b/a -Dr. Diane for loss or damage from receipt or use of this E-mail. No responsibility is accepted by Dr. Diane Stoler, Ed.D., LLC d/b/a-Dr. Diane for personal E-mails, or E-mails unconnected with Dr. Diane Stoler, Ed.D, LLC patients' or client business.Dr. Diane ~ Catalyst for Change® - A neuropsychologist who works with individuals and organizations worldwide, to help them find Solutions and Resources® to overcome life’s challenges and reach their goals.

October 8, 2010

Hello

Diane,

Note:

You can contact me back channel if you want copies of the articles (20-24)

specifically dealing with NFB treatment of tinnitus.

The

frequent tacit assertion that most tinnitus, especially in the elderly, is a

form of early osteoporosis is nowhere supported in the literature. This should

be obvious from the following well-established findings.

Causes

of tinnitus include blast or noise injury [3], middle ear trauma [4], genetic

disturbances [5], dietary glutamate over-exposure, [6], temporomandibular

dysfunction [7,8] , aspirin [9] and antibiotic reactions [10], Ménière's

disease [10], vestibular Schwannoma [10], electromagnetic hypersensitivity

[11,12], chronic intracranial hypotension [13], palatal myoclonus [14],

mitochondriopathies [15], anemia [16,17], immune-mediated ear disease [18],

allergy (food, perfume, dust, petrochemicals) [19], as well as the more benign

type accompanying old age (with or without osteoporosis) [10,20].

The

fact that some of these causes of tinnitus are life threatening yet reversible

(viz., aplastic anemia), should give us all cause to make certain a complete

competent diagnostic workup has been performed.

Where

does neurofeedback come in? The common final pathway most these etiologies

engage is one that might best be described as deafferentiation, similar to that

involved in phantom limb pain. That is why the few studies involving tinnitus,

qEEG and neurofeedback focus on increased delta around the auditory cortex.

[20, 21, 22, 23].

I

would like to make two additional observation, which I hope will stimulate some

discussion.

Electromagnetic

hypersensitivity and tinnitus: Literature suggests a significant relationship

between susceptibility to commercial 60 Hz electromagnetic fields and tinnitus

[11,12]. Is it not interesting that " all subjects with tinnitus showed a

unilateral localised area of high frequency (30–80 Hz, maximum 60 Hz) activity

over the temporal lobe auditory cortex. " [23]

Autism,

noise, neuroplasticity, and tinnitus: Rats have a rich set of ultrasonic vocal

calls. Investigators masked the ability of infant rats to discern these social

calls using 'white noise', which, by definition, contains no information. This

led the rats to increased nondifferentiated cortical growth similar to that

exhibited by autistic children. [25]

Conclusion:

Management of tinnitus, and client well-being, requires a comprehensive

evaluation. The two main final pathways are excessive glutamatergic activity

(approached through diet and nutriceuticals) and deafferentiation (approached

through NFB). Neurofeedback, to be effective, requires that the client be able

to sustain the stress of neural 'rewiring' without suffering rebound synaptic

recidivism. Robust neuroplasticity is also required. The roles of attention and

networks are only recently being elucidated [26,27]. The future of

neurofeedback will include direct ability to influence the long range and

default mode networks, which also play a role in tinnitus [28, 29]

I

apologize for any shortcomings of this brief post.

Dailey

Near

San Francisco

[3]

Fausti SA,, et al. Auditory and vestibular dysfunction associated with

blast-related traumatic brain injury. J Rehabil Res Dev. 2009;46(6):797-810.

[4]

Lao WW, et al. Assessment of changes in cochlear function with pneumolabyrinth

after middle ear trauma. Otol Neurotol. 2007 Dec;28(8):1013-7.

[5]

Hendricks JJ. Familial aggregation of tinnitus: a European multicentre study.

B-ENT. 2007;3 Suppl 7:51-60.

[6]

Pujol R, et al. Excitotoxicity, synaptic repair, and functional recovery in the

mammalian

cochlea:

a review of recent findings. Ann N Y Acad Sci. 1999 Nov 28;884:249-54.

[7]

de Felício CM, et al. Otologic

symptoms of temporomandibular disorder and effect of orofacial myofunctional

therapy. Cranio. 2008 Apr;26(2):118-25.

[8]

Schellhas KP. Temporomandibular joint injuries. Radiology. 1989 Oct;173(1):211-6.

[9]

Sahley TL. Endogenous dynorphins: possible role in peripheral tinnitus. Int

Tinnitus J. 1999;5(2):76-91.

[10]

Møller AR. Tinnitus: presence and future. Prog Brain Res.

2007;166:3-16.

[11]

Landgrebe M, et al. Association

of tinnitus and electromagnetic hypersensitivity: hints for a shared

pathophysiology? PLoS One. 2009;4(3):e5026. Epub 2009 Mar 27.

[12]

Mortazavi SM, et al. Prevalence of subjective poor health symptoms associated

with exposure to electromagnetic fields among university students.

Bioelectromagnetics. 2007 May;28(4):326-30.

[13]

Mackenzie RA, et al. Chronic intracranial hypotension. J Clin Neurosci. 1998

Oct;5(4):457-60.

[14]

Bryce GE, et al. Botulinum toxin treatment of essential palatal myoclonus

tinnitus. J Otolaryngol. 1998 Aug;27(4):213-6.

[15]

Finsterer J. Mitochondriopathies. Eur J Neurol. 2004 Mar;11(3):163-86.

[16]

Piltcher O, et al. Sensorineural hearing loss among sickle cell disease

patients from southern Brazil. Am J Otolaryngol. 2000 Mar-Apr;21(2):75-9.

[17]

Ogawa K, et al. Aplastic anemia and sudden sensorineural hearing loss. Acta

Otolaryngol Suppl. 1994;514:85-8.

[18]

Bovo R, et al. i A. Immune-mediated inner ear disease. Acta Otolaryngol.

2006 Oct;126(10):1012-21.

[19]

Lasisi AO , et al. The inner ear in patients with nasal allergy. J Natl Med

Assoc. 2008 Aug;100(8):903-5.

[20]

Zagólski O. Management of tinnitus in patients with presbycusis. Int Tinnitus

J. 2006;12(2):175-8.

[21]

Dohrmann K, et al. Chapter 46 Neurofeedback for treating tinnitus. Progress in

Brain Research, Vol. 166, 2007.

[22]

Lorenz I, et al. Loss of alpha power is related to increased gamma

synchronization - A marker of reduced inhibition in tinnitus. Neuroscience

Letters 453 (2009) 225–228

[23]

Ashton H, et al. High frequency localized hot spots in temporal lobes of

patients with intractable tinnitus - A QEEG study. Neuroscience Letters 426

(2007) 23–28

[24]

Weisz N, et al (2005) - Tinnitus perception and distress is related to abnormal

spontaneous brain activity as measured by magnetoencephalography. PLos

Medicine, June 2005, Vol 2 Issue 6.

[25]

Doidge N. The Brain that Changes Itself. Penguin Books 2007.

]26]

Low YF, et al. The role of attention in the tinnitus decompensation:

reinforcement of a large-scale neural decompensation measure. Conf Proc IEEE

Eng Med Biol Soc. 2007;2007:2485-8.

[27]

Lenhardt ML, et al. The role of the insula cortex in the final common pathway

for tinnitus:

experience

using ultra-high-frequency therapy. Int Tinnitus J. 2008;14(1):13-6.

[28]

Schlee W, et al. Mapping cortical hubs in tinnitus. BMC Biol. 2009 Nov 23;7:80.

[29]

Schlee W, et al. Abnormal resting-state cortical coupling in chronic tinnitus.

BMC Neurosci. 2009 Feb 19;10:11.

-----

Original Message -----

From: Diane Stoler, Ed.D.

brainm

; neuroguide

;

Sent: 10/08/2010 7:43 AM

Subject:

Tinnitus

Does anyone have an idea for treating tinnitus?

I've tried T3/T4 Othmer and it does not seem to help. C3/C4 does

not seem to help. Doing LENS and it does not seem to help.

Any ideas are truly welcomed.

Diane

Dr. Diane Stoler, Ed.D., LLC

P.O. Box 148

town, MA 01833

Toll Free in US 888.760.8730

Direct Dial 978.352.6349

__

October 8, 2010

Low amplitude alpha and too high delta.

Foxx

On Fri, 8 Oct 2010 14:50:08 +0000 (UTC), Duncan wrote

>

> > Diane~

> I read a study once that indicated that tinnitus sufferers often have low amplitude Alpha at the temporal lobes, AND, can also be connected to a Vitamin B12 deficiency, or nerve damage.

> ~

>

> >

> From: " Diane Stoler, Ed.D. " <diane@...>

> brainm , neuroguide ,

> Sent: Friday, October 8, 2010 10:43:53 AM

> Subject: Tinnitus

> >

> > Does anyone have an idea for treating tinnitus? I've tried T3/T4 Othmer and it does not seem to help. C3/C4 does not seem to help. Doing LENS and it does not seem to help. > > Any ideas are truly welcomed. > > Diane

> >

> Dr. Diane Stoler, Ed.D., LLC > P.O. Box 148

> town, MA 01833

> > Toll Free in US 888.760.8730

> Direct Dial 978.352.6349

> > For information on how to obtain books:

> > " Coping with Mild Traumatic Brain Injury: > A Guide to Living with the Challenges Associated with Concussion/ Brain Injury " > > " Timeless- a novel: > Who Am I? " > > Click the link below > <http:// www.drdiane.com >

> > Neuroband: For the Professional and Home User of Neurofeedback equipment: > Clink the link below

> http://www.drdiane.com/neuroband_order.html

> > Confidentiality: > > This electronic message (E-mail) and any files attached hereto contain confidential, legally privileged and protected by copyright. If you are not the intended recipient, dissemination or copying of this E-mail is prohibited. If you have received this in error, please notify the sender by telephone 978.352.8269 or replying by E-mail to info@..., then delete the E-mail completely from your system. > > This E-mail and any attachments have been scanned for viruses, but it is the responsibility of the recipient to conduct their own security measures and no responsibility is accepted by Dr. Diane Stoler, Ed,D. , LLC d/b/a -Dr. Diane for loss or damage from receipt or use of this E-mail. > > No responsibility is accepted by Dr. Diane Stoler, Ed.D., LLC d/b/a-Dr. Diane for personal E-mails, or E-mails unconnected with Dr. Diane Stoler, Ed.D, LLC patients' or client business.

> > Dr. Diane ~ Catalyst for Change® - A neuropsychologist who works with individuals and organizations worldwide, to help them find Solutions and Resources® to overcome [uTF-8?]lifeâ€™s challenges and reach their goals.

>

> >

--

October 8, 2010

Diane,At the 2009 iSNR conference there was a keynote that dealt with tinnitus and other "phantom" perceptions, I think that a DVD of the presentation is available from iSNR.

DIRK DeRIDDER, MD, PhDK4-09 (1 Hour)A Neuristic Model for Phantom Perceptions2009 Keynote SpeakerOn Oct 8, 2010, at 11:48 AM, Dailey wrote:Hello Diane, Note: You can contact me back channel if you want copies of the articles (20-24) specifically dealing with NFB treatment of tinnitus. The frequent tacit assertion that most tinnitus, especially in the elderly, is a form of early osteoporosis is nowhere supported in the literature. This should be obvious from the following well-established findings. Causes of tinnitus include blast or noise injury [3], middle ear trauma [4], genetic disturbances [5], dietary glutamate over-exposure, [6], temporomandibular dysfunction [7,8] , aspirin [9] and antibiotic reactions [10], Ménière's disease [10], vestibular Schwannoma [10], electromagnetic hypersensitivity [11,12], chronic intracranial hypotension [13], palatal myoclonus [14], mitochondriopathies [15], anemia [16,17], immune-mediated ear disease [18], allergy (food, perfume, dust, petrochemicals) [19], as well as the more benign type accompanying old age (with or without osteoporosis) [10,20]. The fact that some of these causes of tinnitus are life threatening yet reversible (viz., aplastic anemia), should give us all cause to make certain a complete competent diagnostic workup has been performed. Where does neurofeedback come in? The common final pathway most these etiologies engage is one that might best be described as deafferentiation, similar to that involved in phantom limb pain. That is why the few studies involving tinnitus, qEEG and neurofeedback focus on increased delta around the auditory cortex. [20, 21, 22, 23]. I would like to make two additional observation, which I hope will stimulate some discussion. Electromagnetic hypersensitivity and tinnitus: Literature suggests a significant relationship between susceptibility to commercial 60 Hz electromagnetic fields and tinnitus [11,12]. Is it not interesting that "all subjects with tinnitus showed a unilateral localised area of high frequency (30–80 Hz, maximum 60 Hz) activity over the temporal lobe auditory cortex." [23] Autism, noise, neuroplasticity, and tinnitus: Rats have a rich set of ultrasonic vocal calls. Investigators masked the ability of infant rats to discern these social calls using 'white noise', which, by definition, contains no information. This led the rats to increased nondifferentiated cortical growth similar to that exhibited by autistic children. [25] Conclusion: Management of tinnitus, and client well-being, requires a comprehensive evaluation. The two main final pathways are excessive glutamatergic activity (approached through diet and nutriceuticals) and deafferentiation (approached through NFB). Neurofeedback, to be effective, requires that the client be able to sustain the stress of neural 'rewiring' without suffering rebound synaptic recidivism. Robust neuroplasticity is also required. The roles of attention and networks are only recently being elucidated [26,27]. The future of neurofeedback will include direct ability to influence the long range and default mode networks, which also play a role in tinnitus [28, 29] I apologize for any shortcomings of this brief post. DaileyNear San Francisco [3] Fausti SA,, et al. Auditory and vestibular dysfunction associated with blast-related traumatic brain injury. J Rehabil Res Dev. 2009;46(6):797-810. [4] Lao WW, et al. Assessment of changes in cochlear function with pneumolabyrinth after middle ear trauma. Otol Neurotol. 2007 Dec;28(8):1013-7. [5] Hendricks JJ. Familial aggregation of tinnitus: a European multicentre study. B-ENT. 2007;3 Suppl 7:51-60. [6] Pujol R, et al. Excitotoxicity, synaptic repair, and functional recovery in the mammaliancochlea: a review of recent findings. Ann N Y Acad Sci. 1999 Nov 28;884:249-54. [7] de Felício CM, et al. Otologic symptoms of temporomandibular disorder and effect of orofacial myofunctional therapy. Cranio. 2008 Apr;26(2):118-25. [8] Schellhas KP. Temporomandibular joint injuries. Radiology. 1989 Oct;173(1):211-6. [9] Sahley TL. Endogenous dynorphins: possible role in peripheral tinnitus. Int Tinnitus J. 1999;5(2):76-91. [10] Møller AR. Tinnitus: presence and future. Prog Brain Res. 2007;166:3-16. [11] Landgrebe M, et al. Association of tinnitus and electromagnetic hypersensitivity: hints for a shared pathophysiology? PLoS One. 2009;4(3):e5026. Epub 2009 Mar 27. [12] Mortazavi SM, et al. Prevalence of subjective poor health symptoms associated with exposure to electromagnetic fields among university students. Bioelectromagnetics. 2007 May;28(4):326-30. [13] Mackenzie RA, et al. Chronic intracranial hypotension. J Clin Neurosci. 1998 Oct;5(4):457-60. [14] Bryce GE, et al. Botulinum toxin treatment of essential palatal myoclonus tinnitus. J Otolaryngol. 1998 Aug;27(4):213-6. [15] Finsterer J. Mitochondriopathies. Eur J Neurol. 2004 Mar;11(3):163-86. [16] Piltcher O, et al. Sensorineural hearing loss among sickle cell disease patients from southern Brazil. Am J Otolaryngol. 2000 Mar-Apr;21(2):75-9. [17] Ogawa K, et al. Aplastic anemia and sudden sensorineural hearing loss. Acta Otolaryngol Suppl. 1994;514:85-8. [18] Bovo R, et al. i A. Immune-mediated inner ear disease. Acta Otolaryngol. 2006 Oct;126(10):1012-21. [19] Lasisi AO , et al. The inner ear in patients with nasal allergy. J Natl Med Assoc. 2008 Aug;100(8):903-5. [20] Zagólski O. Management of tinnitus in patients with presbycusis. Int Tinnitus J. 2006;12(2):175-8. [21] Dohrmann K, et al. Chapter 46 Neurofeedback for treating tinnitus. Progress in Brain Research, Vol. 166, 2007. [22] Lorenz I, et al. Loss of alpha power is related to increased gamma synchronization - A marker of reduced inhibition in tinnitus. Neuroscience Letters 453 (2009) 225–228 [23] Ashton H, et al. High frequency localized hot spots in temporal lobes of patients with intractable tinnitus - A QEEG study. Neuroscience Letters 426 (2007) 23–28 [24] Weisz N, et al (2005) - Tinnitus perception and distress is related to abnormal spontaneous brain activity as measured by magnetoencephalography. PLos Medicine, June 2005, Vol 2 Issue 6. [25] Doidge N. The Brain that Changes Itself. Penguin Books 2007. ]26] Low YF, et al. The role of attention in the tinnitus decompensation: reinforcement of a large-scale neural decompensation measure. Conf Proc IEEE Eng Med Biol Soc. 2007;2007:2485-8. [27] Lenhardt ML, et al. The role of the insula cortex in the final common pathway for tinnitus:experience using ultra-high-frequency therapy. Int Tinnitus J. 2008;14(1):13-6. [28] Schlee W, et al. Mapping cortical hubs in tinnitus. BMC Biol. 2009 Nov 23;7:80. [29] Schlee W, et al. Abnormal resting-state cortical coupling in chronic tinnitus. BMC Neurosci. 2009 Feb 19;10:11. TinnitusDoes anyone have an idea for treating tinnitus? I've tried T3/T4 Othmer and it does not seem to help. C3/C4 does not seem to help. Doing LENS and it does not seem to help. Any ideas are truly welcomed. DianeDr. Diane Stoler, Ed.D., LLC P.O. Box 148town, MA 01833Toll Free in US 888.760.8730Direct Dial 978.352.6349__

October 8, 2010

Hello ,Wonderful post. I was inquiring about getting the articles you mentioned to Diane about the treatment of tinnitus. I have been suffering from tinnitus for several years now and I am always looking for new avenues to explore about the condition.Thanx for your time,MarcusFrom: Dailey <ddailey@...> Sent: Fri, October 8, 2010 10:48:24 AMSubject: RE: Tinnitus

Hello

Diane, Note:

You can contact me back channel if you want copies of the articles (20-24)

specifically dealing with NFB treatment of tinnitus. The

frequent tacit assertion that most tinnitus, especially in the elderly, is a

form of early osteoporosis is nowhere supported in the literature. This should

be obvious from the following well-established findings. Causes

of tinnitus include blast or noise injury [3], middle ear trauma [4], genetic

disturbances [5], dietary glutamate over-exposure, [6], temporomandibular

disease [10], vestibular Schwannoma [10], electromagnetic hypersensitivity

[11,12], chronic intracranial hypotension [13], palatal myoclonus [14],

mitochondriopathies [15], anemia [16,17], immune-mediated ear disease [18],

allergy (food, perfume, dust, petrochemicals) [19], as well as the more benign

type accompanying old age (with or without osteoporosis) [10,20]. The

fact that some of these causes of tinnitus are life threatening yet reversible

(viz., aplastic anemia), should give us all cause to make certain a complete

competent diagnostic workup has been performed. Where

does neurofeedback come in? The common final pathway most these etiologies

engage is one that might best be described as deafferentiation, similar to that

involved in phantom limb pain. That is why the few studies involving tinnitus,

qEEG and neurofeedback focus on increased delta around the auditory cortex.

[20, 21, 22, 23]. I

would like to make two additional observation, which I hope will stimulate some

discussion. Electromagnetic

hypersensitivity and tinnitus: Literature suggests a significant relationship

between susceptibility to commercial 60 Hz electromagnetic fields and tinnitus

[11,12]. Is it not interesting that "all subjects with tinnitus showed a

unilateral localised area of high frequency (30â€“80 Hz, maximum 60 Hz) activity

over the temporal lobe auditory cortex." [23] Autism,

noise, neuroplasticity, and tinnitus: Rats have a rich set of ultrasonic vocal

calls. Investigators masked the ability of infant rats to discern these social

calls using 'white noise', which, by definition, contains no information. This

led the rats to increased nondifferentiated cortical growth similar to that

exhibited by autistic children. [25] Conclusion: