Guest guest Posted September 24, 1999 Report Share Posted September 24, 1999 Dear : Thanks for placing Dr. Neubauer Summary Statement regarding HBO for Strokes due to Thrombosis. My wife,' Orie Lee Manson, closely parallels the case 3A 7 3B on page 2 of the post. It summarizes the results of an 85 year old patient who was able to get HBO after a CVA attack 6 months previous. My wife was refused HBO by Dr. Titus and DR. Roy Myers, both highly regarded HBO physicians. She was refused therapy on the basis there was no scientific or medical basis for allowing it, no justification. She was only 8 or 10 days into the stroke when Dr. Titus said no. Bit I knew better as I had attended the HBO stroke conference at the Sheraton in and heard Drs Neubauer, Harch, , Barrett, Van Meter and others stating they had cause to believe itHBO would help, not cure perhaps, but help. Dr. john Stauch at ndale had a monoplace chamber and had offered from the start to treat my wife if I could get her to the chamber.. She was unable until I bought a specially equipped van with an access chair. Thus on March 3rd Lee began her HBO, 11 months after her stroke. Now she has completed 92 treatments, the same as reported in cases 3A and 3B. My wife is much improved. She walks by holding her hand to help stabilize. She has taken 27 steps without any assistance. She helps me decide what to put on the Salmon to poach it and measures with her finger how much water to use. She still doesn't talk, except sometimes spontaneously a few words, but never responds by asking her to say something. She knows her home, all her surroundings and goes out on the lake with me quite often. She remembers all her CNA's and knows her family and is currently working with a speech therapist who has her practicing symbols to communicate. This is still difficult but she is getting much better with symbols and plays Solitaire with about 60% recognition of each play. I have kept a daily journal for the first 60 or so HBO's She did have a couple of falls that set her back and we are temporarily halting at 92 as we have noted a few mood shifts, late in the afternoon and she gets agitated with one of the CNA's Can't decide if its toxicity from HBO or personality problem with the CNA. But we plan to resume HBO in another 3 or 4 weeks. I'll try to keep you posted. Manson Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 2, 2000 Report Share Posted September 2, 2000 I had a similar experience 4 years ago and a portion of my left leg is still numb. Dr. Cheney said it was probably because my blood was too alkaline (later confirmed by test) which can cause arteries to spasm. I have not had any similar problems since beginning his protocol which includes whey protein, hydroxycobalamin, Somatomed..... Steve Bullock Stroke > > Ken, > > About two years ago, (five years into CFS) I was laying on a large > tank lid, with my head and upper torso inverted down in the tank. > When I got up, I realized the whole outside of my right thigh was > numb on the surface. This gradually resolved itself over the period > of about 18 months. At the time it worried me, but the best > explanation I could come up with was that I pinched a nerve somewhere > while I was leaning over. Now after reading what you said about > stroke, could that have caused it I wonder. > > Any comment? I am still waiting for a doctors appointment in December > before I can get any coagulation testing. > > Matt > > > This list is intended for patients to share personal experiences with each other, not to give medical advice. If you are interested in any treatment discussed here, please consult your doctor. > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 2, 2000 Report Share Posted September 2, 2000 Matt: I'm pretty sure 1000 mcg = 1 mg. You need to take at least 5 mg/day of hydroxycobalamin to get any benefit. I started at 1-2 mg/day with little affect. I get my hydroxy from Wellness Pharmacy via air-mail overnight shipped in ice. It comes in small vials with 10 mg. of hydrxy each in a 1 cc vial. Steve B. Re: Stroke > > That is interesting, Steve. This is not a symptom I have seen > associated with CFS before, and it is nice now to be able to find out > about someone else with the same experience AND hear DR. Chenny's > evaluation. I am working on the below mentioned protocol. If I can > just get someone to prescribe me B12. So far I have only gotten > 2cc/month of hydroxy 500mcg/ml. Does anyone know what mcg is? I guess > it means micro???????? > > Matt > > > > I had a similar experience 4 years ago and a portion of my left leg > is still > > numb. Dr. Cheney said it was probably because my blood was too > alkaline > > (later confirmed by test) which can cause arteries to spasm. I > have not had > > any similar problems since beginning his protocol which includes > whey > > protein, hydroxycobalamin, Somatomed..... Steve Bullock > > > > This list is intended for patients to share personal experiences with each other, not to give medical advice. If you are interested in any treatment discussed here, please consult your doctor. > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 2, 2000 Report Share Posted September 2, 2000 That is interesting, Steve. This is not a symptom I have seen associated with CFS before, and it is nice now to be able to find out about someone else with the same experience AND hear DR. Chenny's evaluation. I am working on the below mentioned protocol. If I can just get someone to prescribe me B12. So far I have only gotten 2cc/month of hydroxy 500mcg/ml. Does anyone know what mcg is? I guess it means micro???????? Matt > I had a similar experience 4 years ago and a portion of my left leg is still > numb. Dr. Cheney said it was probably because my blood was too alkaline > (later confirmed by test) which can cause arteries to spasm. I have not had > any similar problems since beginning his protocol which includes whey > protein, hydroxycobalamin, Somatomed..... Steve Bullock Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 2, 2000 Report Share Posted September 2, 2000 Real difficult to say -- I assume that you were likely working on the Abrams and thus many things could have been happening... for example muscle contraction around blood vessels which could have cut off/reduced blood flow (ie. " a foot felling asleep " ) and potentially a minor blockage formed which resolved over time. The simplest practical definition of a stroke is a " reduction of blood flow that produced symptoms due to coagulation, or for which coagulation is a significant factor " (thus stress producing Adrenalin, tightening blood vessels, inducing a stroke qualifies). The catch is that the usual stroke detection techniques generally do not work for PWCs because scans shows abnormal results usually.... M Lassesen, M.S. ex " Dr.Gui (MSDN) " , " Dr. VB " cv: http://www.folkarts.com/kenl/ KenL@... Phone: 360 297.4717 Cell: 360 509.8970 Fax 520 832.6836 ----- Original Message ----- From: Matt Hoppe Now after reading what you said about stroke, could that have caused it I wonder. Any comment? I am still waiting for a doctors appointment in December before I can get any coagulation testing. Matt Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 2, 2000 Report Share Posted September 2, 2000 on 9/02/00 6:02 AM, Matt Hoppe at hoppe@... wrote: > About two years ago, (five years into CFS) I was laying on a large > tank lid, with my head and upper torso inverted down in the tank. > When I got up, I realized the whole outside of my right thigh was > numb on the surface. I find this very interesting, this business about Transient Ischemic Attacks, because I believe that I've had at least one before. Now that ken has refreshed my memory as to the common signs and symptoms of TIAs, I believe it even more than I did just after the event. In fact right after the event I read a book written by a neurologist, who described TIAs in his patients, and I was convinced that that is what had happened. This is what happened to me: I had recently begun an exercise program consisting of jogging up the road of a huge hill, on top of which sat an Astro-Physical Observatory. The observatory hill was a beautiful place to walk or jog and few cars passed by. On about the 3rd day of my daily jogging routine, I quite suddenly noticed that my right jaw was painful and that my right side was also painful--very much like a 'stitch' in the side or a pinching sensation. I slowed down at once to a walk and the acute pain in my right rib cage subsided but I was left with the most uncomfortable feeling in my jaw--that's hard to explain: sorta stiff, numb and tingling. Since my father suffered from angina, I thought that's what was happening to me. As I returned home, my right arm from about the wrist up to the shoulder became excruciatingly uncomfortable. It was sorta numb and 'frozen', but I couldn't stand the feeling, nor could I identify it. And I'm familiar with what a pinched nerve feels like. I figured I was having cardiac ischemia. So I went to the hospital by cab and waited in the ER for nearly 3 hours--in major pain, laying down on my left side across 3 chairs. When the intake worker took my history, I foolishly told them that I have a history of anxiety problems--not panic disorder, but social anxiety. After that, I could tell that I had been written off as a " panic attack case " and I was seen last. When the doc saw me, he gave me an ECG and told me I was fine. told me to keep up the jogging... One thing that seemed to play into the situation is that I had been a daily marijuana smoker up until about 1 week before the incident. I now think that the cannabinoids had had a positive effect against TIAs. The recent development of Dexanibinol (a cannabinoid derivative) as a pharmaceutical to be used for stroke victims, provides support for this. Anyhow, I wouldn't want to repeat the whole experience. Hud Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 14, 2000 Report Share Posted November 14, 2000 Thanks for that snippet from the Chiropractic Review, Larry. I can't agree that rotation is fine, however. Rotation is what appears to trigger or exacerbate the intimal tear that ultimately leads to the stroke. Manipulative techniques described by Darald that don't involve rotation as well as instrument techniques that do not involve rotation are inherently less risky. As long as we continue to believe that there is nothing that can be done to prevent this kind of tragic result of a manipulation the factions in medicine that want to call chiropractic dangerous and unwarranted (although nauseatingly hypocritical, as Vern as very nicely pointed out) will have an ear in the popular media and elsewhere. We are the ones who have to recognize and address the problem. D FreemanMailing address: 2480 Liberty Street NE Suite 180Salem, Oregon 97303phone 503 763-3528fax 503 763-3530pager 888 501-7328 Stroke I found one of the articles I base my last E-mail on. I will continue to search. The Chiropractic Report by Chapman July 1999 Vol13 No.4 published an excellent review of cervical adjusting. Haldeman's article published in Spine was included. I highly recommend it. Some of the key conclusions were and I quote ... " Cerebrovascular accidents from vertebrobasilar artery dissection (CVAs) can be an unexpected and devastating case of stroke. However they are on 1.3 in 1000 cases of stroke." ... "43% of the 367 reported cases follow no known precipitating event and are thus labeled 'spontaneous. Of those with identifiable trauma 16% are trivial, involving normal sporting activities, walking, household chores, turning the head while driving, coughing, ect. "..."accordingly 6 in 10 (59%) of CVAs have a trivial cause. The other 41% of the cases have been attributed to cervical manipulation (31%) and major trauma (10%) motor vehicle accidents, significant sports injuries, lifting injuries, ect. The facts of various individual cases make it plain that manipulation can be the proximate case of CVA. Haldeman et al suggests that it may not be the underlying cause, or even the cause, in many cases where it is assumed to be. Patients may have had a spontaneous dissection from a minor trauma, and consulted a practitioner with the resulting symptoms of neck pain or headaches. Spinal manipulation is then " administered to patients who already had spontaneous dissection in progress". This conclusion is supported by a compelling argument. If the primary case of CVA was cervical manipulation, or indeed any specific head position or movement or trauma, "considerably more cases would be anticipated". There are about 250 million office visits to chiropractors in the US each year and millions of whiplash injuries, falls and other activities causing violent movements to the head and neck, but few CVA's. This suggests that there may be "some unique factor that causes certain people to be at risk." "All of this amounts to good and bad news. They good news is that in chiropractic practice you can use any cervical adjustment technique you deem appropriate, since the best evidence is that no position or technique carries additional risk. Additional studies from North America and Europe report that pre-manipulation test of vertebral artery function are invalid and unnecessary. The bad news is that rare patients are CVA's waiting to happen. No one knows why. They cannot be screened in advance, competent and skillful practice will not protect you, and a CVA can happen in your office tomorrow. Fortunately the is risk is remote. Most persons turning to reverse their car out of a driveway will not have a CVA, and most chiropractors will never experience one in a lifetime of practice. What should you be doing about this? The answer, in terms of patients rights, law and ethics, is be disciplined and responsible about getting informed consent. In the words of the current US national guidelines for chiropractic practice: "Patient consent to treatment is always necessary, it is often implied rather than expressed. However, where there is risk of significant harm from the treatment proposed, this risk must be disclosed, understood and accepted by the patient. Such informed consent is required for ethical and legal reasons. The best record of consent is one that is objectively documented (e.g. a witnessed written consent or videotape)."... "If a patient of yours were to suffer serious consequences from a CVA caused by cervical adjustment, wouldn't you sleep more soundly at night knowing you had disclosed this very rare but foreseeable risk of your treatment?" The article can be condensed into the sub title: ROTATION IS FINE, PRETESTING IS OUT, BUT GET CONSENT" Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 15, 2000 Report Share Posted November 15, 2000 In the courses I teach, when we cover this topic after covering all the info., I suggest the adjustment very much described by Les and probably taught the best by the Motion Palpation folk. That being a little long axis extension, a slight amount of lateral bending and rotation, just enough to bring the joint to tension, then an " impuse " thrust. As instead of the old rotatary break! I just love that name, " rotatary break " sounds so good in court, don't ya know! Vern Re: Stroke Thanks for that snippet from the Chiropractic Review, Larry. I can't agree that rotation is fine, however. Rotation is what appears to trigger or exacerbate the intimal tear that ultimately leads to the stroke. Manipulative techniques described by Darald that don't involve rotation as well as instrument techniques that do not involve rotation are inherently less risky. As long as we continue to believe that there is nothing that can be done to prevent this kind of tragic result of a manipulation the factions in medicine that want to call chiropractic dangerous and unwarranted (although nauseatingly hypocritical, as Vern as very nicely pointed out) will have an ear in the popular media and elsewhere. We are the ones who have to recognize and address the problem. D FreemanMailing address: 2480 Liberty Street NE Suite 180Salem, Oregon 97303phone 503 763-3528fax 503 763-3530pager 888 501-7328 Stroke I found one of the articles I base my last E-mail on. I will continue to search. The Chiropractic Report by Chapman July 1999 Vol13 No.4 published an excellent review of cervical adjusting. Haldeman's article published in Spine was included. I highly recommend it. Some of the key conclusions were and I quote ... " Cerebrovascular accidents from vertebrobasilar artery dissection (CVAs) can be an unexpected and devastating case of stroke. However they are on 1.3 in 1000 cases of stroke. " ... " 43% of the 367 reported cases follow no known precipitating event and are thus labeled 'spontaneous. Of those with identifiable trauma 16% are trivial, involving normal sporting activities, walking, household chores, turning the head while driving, coughing, ect. " ... " accordingly 6 in 10 (59%) of CVAs have a trivial cause. The other 41% of the cases have been attributed to cervical manipulation (31%) and major trauma (10%) motor vehicle accidents, significant sports injuries, lifting injuries, ect. The facts of various individual cases make it plain that manipulation can be the proximate case of CVA. Haldeman et al suggests that it may not be the underlying cause, or even the cause, in many cases where it is assumed to be. Patients may have had a spontaneous dissection from a minor trauma, and consulted a practitioner with the resulting symptoms of neck pain or headaches. Spinal manipulation is then " administered to patients who already had spontaneous dissection in progress " . This conclusion is supported by a compelling argument. If the primary case of CVA was cervical manipulation, or indeed any specific head position or movement or trauma, " considerably more cases would be anticipated " . There are about 250 million office visits to chiropractors in the US each year and millions of whiplash injuries, falls and other activities causing violent movements to the head and neck, but few CVA's. This suggests that there may be " some unique factor that causes certain people to be at risk. " " All of this amounts to good and bad news. They good news is that in chiropractic practice you can use any cervical adjustment technique you deem appropriate, since the best evidence is that no position or technique carries additional risk. Additional studies from North America and Europe report that pre-manipulation test of vertebral artery function are invalid and unnecessary. The bad news is that rare patients are CVA's waiting to happen. No one knows why. They cannot be screened in advance, competent and skillful practice will not protect you, and a CVA can happen in your office tomorrow. Fortunately the is risk is remote. Most persons turning to reverse their car out of a driveway will not have a CVA, and most chiropractors will never experience one in a lifetime of practice. What should you be doing about this? The answer, in terms of patients rights, law and ethics, is be disciplined and responsible about getting informed consent. In the words of the current US national guidelines for chiropractic practice: " Patient consent to treatment is always necessary, it is often implied rather than expressed. However, where there is risk of significant harm from the treatment proposed, this risk must be disclosed, understood and accepted by the patient. Such informed consent is required for ethical and legal reasons. The best record of consent is one that is objectively documented (e.g. a witnessed written consent or videotape). " ... " If a patient of yours were to suffer serious consequences from a CVA caused by cervical adjustment, wouldn't you sleep more soundly at night knowing you had disclosed this very rare but foreseeable risk of your treatment? " The article can be condensed into the sub title: ROTATION IS FINE, PRETESTING IS OUT, BUT GET CONSENT " Quote Link to comment Share on other sites More sharing options...
Guest guest Posted November 15, 2000 Report Share Posted November 15, 2000 The article I posted quotes from were with out any of my own editorial elaboration. My intent was only to give a snapshot of the current literature. As long as you asked for my opinion, and I'm sure you did this, I'll give it to you now. I grew up in the Mt Horb Wisconsin area and studied at the Gonstead clinic. I graduated from NWCC and at that time, Gonstead was the predominant influence on the core adjusting curriculum. We were never allowed to perform master cervicals. Rotatory moves would not get you a passing grade in technique class. Personally I found rotatory moves to be crude and painful. My formative years and technique were forged from Gonstead, , Pettibon, Meric, SOT and Activator. I do not know of any literature that is sympathetic to Gonstead or Pettibon methods producing fewer CVA's. I would like to believe that this is true and that careful application of our art with competent clinical caution would produce more expeditious outcomes. The literature however does not support this. For my patients, I'll continue do a good history, take vitals, do a good physical exam, identify the pain generator or upstream events, make a diagnosis, and arrive at a treatment plan. At this point, I'll continue to avoid rotatory maneuvers as I have for the last 21 years. However, it sounds like the suits say I need to add a standard consent form with a PARQ note. So , you see, I never disagreed with you. Stroke I found one of the articles I base my last E-mail on. I will continue to search. The Chiropractic Report by Chapman July 1999 Vol13 No.4 published an excellent review of cervical adjusting. Haldeman's article published in Spine was included. I highly recommend it. Some of the key conclusions were and I quote ... " Cerebrovascular accidents from vertebrobasilar artery dissection (CVAs) can be an unexpected and devastating case of stroke. However they are on 1.3 in 1000 cases of stroke." ... "43% of the 367 reported cases follow no known precipitating event and are thus labeled 'spontaneous. Of those with identifiable trauma 16% are trivial, involving normal sporting activities, walking, household chores, turning the head while driving, coughing, ect. "..."accordingly 6 in 10 (59%) of CVAs have a trivial cause. The other 41% of the cases have been attributed to cervical manipulation (31%) and major trauma (10%) motor vehicle accidents, significant sports injuries, lifting injuries, ect. The facts of various individual cases make it plain that manipulation can be the proximate case of CVA. Haldeman et al suggests that it may not be the underlying cause, or even the cause, in many cases where it is assumed to be. Patients may have had a spontaneous dissection from a minor trauma, and consulted a practitioner with the resulting symptoms of neck pain or headaches. Spinal manipulation is then " administered to patients who already had spontaneous dissection in progress". This conclusion is supported by a compelling argument. If the primary case of CVA was cervical manipulation, or indeed any specific head position or movement or trauma, "considerably more cases would be anticipated". There are about 250 million office visits to chiropractors in the US each year and millions of whiplash injuries, falls and other activities causing violent movements to the head and neck, but few CVA's. This suggests that there may be "some unique factor that causes certain people to be at risk." "All of this amounts to good and bad news. They good news is that in chiropractic practice you can use any cervical adjustment technique you deem appropriate, since the best evidence is that no position or technique carries additional risk. Additional studies from North America and Europe report that pre-manipulation test of vertebral artery function are invalid and unnecessary. The bad news is that rare patients are CVA's waiting to happen. No one knows why. They cannot be screened in advance, competent and skillful practice will not protect you, and a CVA can happen in your office tomorrow. Fortunately the is risk is remote. Most persons turning to reverse their car out of a driveway will not have a CVA, and most chiropractors will never experience one in a lifetime of practice. What should you be doing about this? The answer, in terms of patients rights, law and ethics, is be disciplined and responsible about getting informed consent. In the words of the current US national guidelines for chiropractic practice: "Patient consent to treatment is always necessary, it is often implied rather than expressed. However, where there is risk of significant harm from the treatment proposed, this risk must be disclosed, understood and accepted by the patient. Such informed consent is required for ethical and legal reasons. The best record of consent is one that is objectively documented (e.g. a witnessed written consent or videotape)."... "If a patient of yours were to suffer serious consequences from a CVA caused by cervical adjustment, wouldn't you sleep more soundly at night knowing you had disclosed this very rare but foreseeable risk of your treatment?" The article can be condensed into the sub title: ROTATION IS FINE, PRETESTING IS OUT, BUT GET CONSENT" Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 5, 2000 Report Share Posted December 5, 2000 Dear Corny, - Cayenne pepper to clean out the arteries. - Homozon to clean out the colon. - Liver cleanse to clean out the liver. - Black tea (decaffeinated) with lemon juice 4 times per day to strengthen veins and arteries. -Brewers yeast, IP6 and sublingual B12 to reduce homocysteine, and aid in cell repair. Best of Health! Dr. Saul Pressman, DCh stroke > My dear aunt has been having stroke symptoms for several days. Tingling > and numbness in arms, neck and face. She is approx 65 and eating poorly > and too much all her life. She may finally be ready to accept some > alternative advice. > > I am suggesting needed diet changes and am wondering what others have > experienced with supplements, therapies and techniques that will help her > avoid the serious symptoms that will likely soon follow. > > Thanks, Corny > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 5, 2000 Report Share Posted December 5, 2000 Allow me to send you information on Stroke and HBOT Were does your aunt live Please send your address stroke > My dear aunt has been having stroke symptoms for several days. Tingling > and numbness in arms, neck and face. She is approx 65 and eating poorly > and too much all her life. She may finally be ready to accept some > alternative advice. > > I am suggesting needed diet changes and am wondering what others have > experienced with supplements, therapies and techniques that will help her > avoid the serious symptoms that will likely soon follow. > > Thanks, Corny > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 5, 2000 Report Share Posted December 5, 2000 Allow me to send you information on Stroke and HBOT Were does your aunt live Please send your address stroke > My dear aunt has been having stroke symptoms for several days. Tingling > and numbness in arms, neck and face. She is approx 65 and eating poorly > and too much all her life. She may finally be ready to accept some > alternative advice. > > I am suggesting needed diet changes and am wondering what others have > experienced with supplements, therapies and techniques that will help her > avoid the serious symptoms that will likely soon follow. > > Thanks, Corny > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 5, 2000 Report Share Posted December 5, 2000 Corny, There is some other good advice being offered, but if it was me, I'd also start immediately on either piracetam, hydergine or pramiracetam to protect brain cells in case she is having minor strokes & to protect her if she has a major cerebral accident. If one should have a stroke, having one of those already in the bloodstream can make a major difference in the outcome. My personal choice would be piracetam, since it has no known drug interactions or toxicity. jim Mike E Cornwall wrote: > > My dear aunt has been having stroke symptoms for several days. Tingling > and numbness in arms, neck and face. She is approx 65 and eating poorly > and too much all her life. She may finally be ready to accept some > alternative advice. > > I am suggesting needed diet changes and am wondering what others have > experienced with supplements, therapies and techniques that will help her > avoid the serious symptoms that will likely soon follow. > > Thanks, Corny > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. -- ----- carpe diem, carpe pecunia, carpe femina. -- Jim Lambert jlambert@... http://www.entrance.to/madscience http://www.entrance.to/poetry Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 5, 2000 Report Share Posted December 5, 2000 Rapid Recovery Hyperbarics CHRONIC STROKE REHABILITATION With the injury to the brain, blood vessels are damaged or destroyed. The tissue that surrounds the area of outright necrosis has had its circulation compromised and may be only receiving a fraction of the blood flow and oxygen that it needs for optimum health. Thus a disruption in structure creates immediately a change (decrease) in function. This decrease in function remains for months or years and the neurons in these areas are said to be in " hibernation " or " sleeping " . Hyperbaric oxygen treatments when given daily stimulates a process called " angiogenesis " or the formation of new blood vessels. New blood vessels form in the vicinity of the damaged tissues as a result of certain chemical signals (e.g. angiogenin) that are produced by the newly re-energized neurons, endothelial cells and macrophages and are then secreted into the surrounding tissues. These signals stimulate new blood vessels which slowly reconnect to the damaged tissues and within 60 days of daily treatments, the " sleeping " neurons wake up and resume their normal functions as the proper structures return back in place. The hyperbaric oxygen induced blood vessel repair results in a permanent structural change in the blood vessels that re-supply the previously damaged and nonfunctioning nerve tissue which was occurring due to diminished and inadequate blood flow. These new blood vessels improve the blood flow and oxygen delivery to the damaged brain tissues and this results in permanent improvements in the stroke and traumatically brain injured person. Clinically, what you see is the return to life of a previously paralyzed and useless limb or limbs, improvement in swallowing, speech, thinking (cognition), memory, etc. Quite obviously not all of the disabilities disappear since there was a central core of dead tissue that can not be revived. However, after the two months of therapy, these people may continue to improve for at least two years after their treatment with hyperbaric oxygen especially if they continue with physical therapy. This all occurs in patients who may have not seen any improvement in their conditions for years after their stroke even with the use of any and all other therapies indicating that the brain’s milieu intérieur has been altered for the better since the neurons are able to slowly re-establish their lost connections in ways not possible before hyperbaric oxygen. Outcome in stroke may be predicted to some degree by the volume of tissue affected. Comparative functional volume obtained by single photon emission computerized tomography (SPECT) often indicates a larger region of recoverable tissue than CT.(Mountz, JM 1990) This functional volume of the infarct size can be demonstrated to decrease after one to several hyperbaric oxygen treatments (Neubauer, 1990, 1992) and this increase in blood flow to the area of infarction that occurs as a result of hyperbaric oxygen can serve as a clinical test to determine if there is salvageable neurons still present in the penumbra. Presumably, if the test (SPECT first, then HBO then repeat SPECT) is positive, the person should receive benefit from the use of a series of hyperbaric oxygen treatments because of the revitalization of the ischemic penumbral tissues. This is a good test if the test is positive since we are generally assurred that the person will experience improvement with hyperbaric oxygen. However, what if the test is negative? Since the literature and clinical experience predicts that between 80 to 90 percent of stroke victims will be helped by hyperbaric oxygen, perhaps the SPECT scan may be missing some other fundamental mechanism by which hyperbaric oxygen is helping these people improve. For example, when rat’s forebrains are made ischemic for 10 minutes and then after 1, 2, 3 weeks and 3 months their cerebral glucose utilization is measured, generalized reductions in glucose utilization is found throughout the majority of gray matter indicating that widespread alterations of functional activity prevail in postischemic brains beyond the selectively vulnerable regions. (Beck T, et al 1995) Following acute, localized lesions of the central nervous system, arising from any cause, there are immediate depressions of neuronal synaptic functions in other areas of the central nervous system remote from the lesion. These remote effects result from deafferentation, a phenomenon known as " diaschisis " . (Von Monakow C. 1914) After an interval of time, which will vary directly with the severity of the lesion, functional recovery may occur to some degree due to synaptic reactivation of neurons. This is favorably influenced by rehabilitation. Diaschisis most commonly manifests itself by such neurological signs as impaired consciosness or cognitive impairments including dementia, dyspraxias, dystaxias, dysphasias, incoordination and sensory neglect. The nature of diaschisis has been demonstrated by widespread depressions of local cerebral blood flow and metabolism extending far beyond the anatomical lesion. Von Monakow pointed out that development of diaschisis is enhanced by latent circulatory disorders in both the affected and unaffected areas of the brain. Recovery of function is associated with recovery of local perfusion and metabolism. (Meyer, JS,et al 1993) More recently PET scans have shown that diaschisis does not independently add to the clinical deficit in human cerebral infarction but represents part of the damage done by the stroke. (Bowler JV et al. 1995) " Diaschisis is a functional phenomenon that correlates with both stroke severity and infarct hypoperfusion volume " (Infeld B; et al.1995) In another PET scan study of 31 patients with infarcts involving the frontal sensorimotor cortex, 23 had persistent diaschisis up to 5 years after onset while the remaining 8 had the diaschisis recover without recovery of oxygen metabolism in the infarcted area (implying that tissue in the ischemic penumbra did recover and this is what allowed for recovery of the diaschisis). (Miuura H; et al.1994.) Thus if functionless ischemic penumbral tissue can be " re-activated " and be made to function again, a coresponding amount of the areas of diaschisis will be returned to normal with normal blood flow and function returning. In a number of studies in normal dogs, monkeys and Man, hyperbaric oxygen has been shown to diminish cerebral blood flow from 1 to 29% (average 14.7%) which some people have claimed to be detrimental to a stroke or brain injured patient. All of these studies were done in normal non-brain injured subjects while the studies that were done in brain injured patients all showed an increase in cerebral blood flow (Jain, 1996 page 239). Dr. K.K. Jain states, " Vasoconstriction and reduced cerebral blood flow do not produce any clinically observable effects in a healthy adult when pressures of 1.5 to 2 ATA are used. ..The effects of HBO are more pronounced in hypoxic/ischemic states of the brain. HBO reduces cerebral edema and improves the function of neurons rendered inactive by ischemia/hypoxia. The improvement of brain function is reflected by the improved electrical activity of the brain. " stroke >Thanks everyone for all the good advice. Now it's up to me to impress >upon auntie the importance of the advice. Her good " doctor " has >suggested a couple aspirin per day as the immediate remedy. (Hippocrates >would be spinning in his grave....) > >, please send that info to me at 5801 e cambridge ave, scottsdale, >az. 85257. Auntie lives in southern Wa, nearest city is Portland, Ore. > >Thanks, Corny > > >OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > >THERE IS NO MEDICAL ADVICE HERE! > >This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. 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Guest guest Posted December 5, 2000 Report Share Posted December 5, 2000 RESEARCH IN HYPERBARIC OXYGEN AND STROKE GENERAL ARTICLES Moon, R.E. et al. " Hyperbaric oxygen therapy: From the nineteenth to the twenty-first century. " RESPIR CARE CLIN N AM, 5(1):1-5; 1999. Hyperbaric oxygen technology now occupies a legitimate place in modern medical practice, and the number of clinically active hyperbaric facilities has grown. The authors estimate that more than 200 monoplace (single-patient) chambers and over two dozen multiplace facilities are pesently active in the United State. Nevertheless, the majority of patients suffering from syndromes amenable to HBO therapy are treated in hospitals devoid of such modalities. This situation stems in part from the significant cost of appropriate facilities, the relative scarcity of trained personnel, and the difficulties in obtaining appropriate compensation in an era of rapidly changing reimbursement paradigms. The demonstrated efficacy of HBO as an important part of the treatment of certain acute disease processes, however, justifies the facilities and skilled personnel necessary for the care of critically ill patients in a hyperbaric environment. STROKE ARTICLES Akimov, G.A. et al. " Assessment of the Efficiency of Hyperbaric Oxygenation Therapy in Early Forms of Cerebrovascular Disorders. " NEUROSCI BEHAV PHYS, l985; 15: 13 - 16. We present results of the assessment of the efficiency of hyperbaric oxygenation therapy in 104 patients with cerebrovascular diseases. Of these patients, 32 had chronic cerebrovascular insufficiency and 72 showed transient disturbances of the cerebral circulation. A good effect was noted in 74 patients, a satisfactory one in 22, and a doubtful one in 8 patients. It is concluded from clinical, electro-physiological, psychophysiological, biochemical, and ophthalmoscopic examinations that hyperbaric oxygenation therapy is quite efficient when used as part of a combined therapy and as a means of prompt therapy of acute cerebrovascular crises. Observations over three to five years of patients repeatedly receiving the hyperbaric oxygenation therapy at 6 month intervals allows us to recommend it for the prevention of cerebral strokes. Berrouschot, J. et al. " Hyperbaric oxygen therapy (HBO) after acute focal cerebral ischemia. " NERVENARZT, 1998, 69(12): 1037-44. For a large number of patients with stroke, no therapeutic option can be offered, even after approval of thrombolytic therapy for treatment of acute ischemic stroke in the U.S. In cerebral ischemia local anoxia and energy failure lead to further cellular damage and finally to complete stroke. All therapeutic concepts try to salvage structurally intact tissue which is at risk for irreversible damage (so-called penumbra). Hyperbaric oxygen (HBO) treatment has been reported in animal models of cerebral ischemia, and in a few clinical reports. In general, the results of these studies have been promising. Hart, G.B. et al. " The Treatment of Cerebral Ischemia with Hyperbaric Oxygen (OHP). " STROKE, l971; 2: 247-250. The treatment of a patient for three and one-half months, following occlusion of the right middle cerebral artery with the associated neurological sequelae, with hyperbaric oxygen combined with methyldopa and hydrochlorthazide is presented. Treatment scheduled was two and one-half atmospheres absolute. The treatment was interrupted after 15 treatments to rule out spontaneous remission for a period of 30 days, and no further improvement occurred until treatments were reinstituted. The dramatic return to a near normal state during treatment appears to indicate that he did benefit from therapy. Heyman, A. et al. " The use of Hyperbaric Oxygenation in the Treatment of Cerebral Ischemia and Infarction. " CIRCULATION, Supplement 2: May, l966; 20- 27. The therapeutic usefulness of hyperbaric oxygenation in cerebral vascular disease was evaluated in 22 persons with recent neurologic deficits caused by cerebral embolism, thrombosis, hemorrhage, or arteriographic complications. Hyperoxygenation produced a significant elevation in content and tension of oxygen in blood and increased the reservoir of oxygen available for utilization by neurons. Remarkable and dramatic improvement in neurologic function occurred in four patients. In two others the neurologic deficit recurred a few hours after removal from the hyperbaric chamber; repeated exposure to high oxygen pressures was associated with only temporary improvement. In six other patients there was some evidence of clinical recovery immediately after onset of hyperoxygenation, but the neurologic deficit returned during decompression. The remaining 12 patients did not improve during hyperbaric oxygenation. These observations indicate that in some patients neuronal structures remain viable for some hours after loss of function in acute cerebral ischemia. In such instances an increase in oxygen delivery may reverse cellular ischemia and prevent death of cerebral tissues. Hyperbaric oxygenation may provide supportive therapy in some patients with acute cerebral ischemia, thereby permitting the removal of the occlusive lesion by surgery or other methods. Holbach, K. H. et al. " Neurological and EEG Analytical Findings in the Treatment of Cerebral Infarction with Repetitive Hyperbaric Oxygenation. " PROCEEDINGS OF THE SIXTH INTERNATIONAL CONGRESS ON HYPERBARIC MEDICINE, Aug. 31 - Sept. 2, l979, University of Aberdeen, Aberdeen, Scotland, pp. 205-210; DSC, MD (Ed), Aberdeen University Press, l979. " ...These findings indicate that unilateral occlusion or stenosis of the internal carotid or middle cerebral artery can lead to distinct focal neurological deficits and EEG alterations as well as to bilateral reduction of cerebral function and EGA (electrical brain activity). It also appears that such ischemic alterations of the brain can be improved by HBO therapy not only in the acute but also in the chronic post-stroke stage. Accordingly, we feel that this mode of treatment may be considered as an additional measure in the management of stroke. " Holbach, K. H. et al. " Advantage of Using Hyperbaric Oxygenation (HO) in Combination with Extra-Intracranial Arterial Bypass (EIAB) in the Treatment of Completed Stroke. " ACTA NEUROCHIRUGICA, Suppl 28: 309; l979. " ...The evaluation of the effect of HO treatment on post-stroke alterations of the brain can be helpful in differentiating between reversible and irreversible changes, and thus response to HBO treatment may be used as a criterion for the prognosis of the cerebrovascular lesion and also for selection of patients for EIAB surgery. " Holbach, K.H. et al. " Differentiation between Reversible and Irreversible Post-Stroke Changes in Brain Tissue: Its Relevance for Cerebrovascular Surgery. " SURG. NEUROL., l977; 7: 325-331. Thirty-five selected patients with chronic stroke were studied. They had internal carotid occlusion with considerable neurological deficit persisting for an average of ten weeks. First, hyperbaric oxygen treatment was administered to each patient. Subsequently extra-intracranial anastomosis operations were performed on 20 of these patients. These patients were divided into three groups. Group 1 - 15 of the 35 patients - showed a significant improvement of cerebral function at the conclusion of the hyperbaric oxygen treatment. Subsequently an extra-intracranial anastomosis operation was carried out on each patient resulting in considerable further recovery of cerebral functions. Group II consisted of 15 patients who showed only little change in neurological deficit at the conclusion of hyperbaric oxygen therapy. Extra-intracranial anastomosis operations were not carried out in Group II. Group III consisted of five patients with little or no change at the conclusion of hyperbaric oxygen treatment. Subsequent extra-intracranial anastomosis operations were, however, performed in these five patients. Although post-operative angiography revealed considerable filling of the affected middle cerebral territory by the new collateral channel, there was little change in their status. These findings suggest that in the chronic post-stroke stage a) hyperbaric oxygen therapy can improve ischemic alterations of the brain, it may be helpful in differentiating between reversible and irreversible alterations of brain tissue, c) extra-intracranial anastomosis may result in additional recovery of impaired neurological functions in those patients who have shown significant improvement from hyperbaric oxygen therapy and d) response to hyperbaric oxygenation may be used as a criteria for selection of patients for cerebral revascularisation procedures. Holbach, K.H. et al. " Reversibility of the Chronic Post-Stroke State. " STROKE, l976; 7(3): 296-300. Forty patients with cerebral infarction associated with occlusion of the internal carotid artery (ICA) or the middle cerebral artery (MCA) were treated with hyperbaric oxygenation (HO). EEG analysis were performed regularly in order to assess the course of the cerebral lesion. Patients in an early post-stroke stage (IIIB) and patients in a chronic post-stroke stage (IV) had the changes in EEG analysis and neurological findings distributed evenly between these two groups. In 27% of the cases, the improvement was considerable, 53% had moderate improvement, and 20% showed no change of condition. The improvement mainly consisted of an increase in alpha-wave and beta-wave activity over the affected brain region. We were able to show this fact clearly by means of the EEG-analysis-system applied. The results show that (a) hyperbaric oxygenation therapy (HOT) has a very favorable influence upon the course of disease, and ( simultaneous application of HOT and EEG analysis allows for a differentiation between reversible and irreversible post-stroke changes in brain tissue. Ingvar, et al. " Treatment of Focal Cerebral Ischemia with Hyperbaric Oxygen. " ACTA NEUROL. SCANDINAV., l965; 41: 92-95. Four cases of focal ischemia were treated with inhalation of pure oxygen at a pressure of 2.0 to 2.5 atmospheres ( " hyperbaric oxygen " ) for periods of 1.5 to 2.5 hours. In three of the cases beneficient effects of the treatment were seen which in two of them could be objectively demonstrated in the EEG. In one case with progressive ischemic lesions of the brain stem, treated sub finem, very dramatic effects were seen, which were probably to a great extent due to the effects of the treatment upon the failing systemic circulation. Jain, K. K. " Chapter 17: Role of Hyperbaric Oxygen Therapy in the Management of Stroke. " pp. 227 - 252; in TEXTBOOK OF HYPERBARIC MEDICINE, Hogrefe & Huber Publishers, ton, NY, l990. " HBO therapy should be started in the acute phase of a stroke as an adjunct to conventional medical management. Rehabilitation of stroke patients should also be planned during the first few months following stroke. Long-term follow-up studies are required to determine whether such measures would reduce the chronic disability from stroke and reduce the incidence of severe spasticity in stroke patients. The use of HBO may also reduce the need for some surgical procedures…Animal experimental studies and uncontrolled human trials have shown the effectiveness and safety of HBO therapy after strokes. At the Fachklinik Klausenbach (FRG) simultaneous HBO and physical therapies were used in the rehabilitation of stroke patients. Objective evaluation of patients during the HBO session showed a 100% response rate (improvement of spasticity or motor power or both). The improvement was initially transient but could be maintained, following a course of daily treatments (1.5 ATA for 45 min.) for 6 weeks, in most of the cases " with the evidence available, it would unethical to carry out randomized double-blind studies in stroke patients to evaluate the effect of HBO therapy " . Jain, K.K. et al. " Hyperbaric Oxygen Therapy in the Rehabilitation of Stroke Patients. " 2nd EUROPEAN CONFERENCE ON HYPERBARIC MEDICINE, l990; Organized by the Foundation for Hyperbaric Medicine in Basel and the Department of Surgery of the University Clinic in Basil. A 100% response rate was demonstrated in 25 patients in sub-acute and chronic post-stroke stage. In spite of medical management and physical therapy, these patients had shown no day-to-day changes in their neurological status. Increase of motor power of the paralyzed hand was demonstrated by a dynamometer. The improvement was transient initially but was maintained following a course of daily treatments (1.5 ATA for 45 min.) for 6 weeks in most of the cases. There was also a significant reduction of spasticity during HBO treatment and this relief could be extended by instituting physical therapy in the chamber. In conclusion, we feel that HBO is a useful adjunctive treatment in the rehabilitation of stroke patients. Jain, K. K., " Effect of Hyperbaric Oxygenation on Spasticity in Stroke Patients. " J Hyperbaric Med, l989; 4(2): 55-61. The effect of hyperbaric oxygenation (HBO) at 1.5 ATA on spasticity of stroke was observed in 21 patients undergoing rehabilitation. The patients served as their own controls. HBO reduced spasticity in all the patients, an effect that was more marked than that of physical therapy, hyperbaric air, or 100% normobaric air. Initially the effect was transient and subsided within 24 h after treatment, but by conducting physical therapy simultaneously with daily, 45 min HBO sessions, lasting results were achieved after 5 wks and could be maintained by physical therapy alone during the follow-up, which varied from 6 mo. to 1 yr. The exact mechanism of relief of spasticity is not known but it is probably due to improvement of the function of neurons in the penumbra zone of the cerebral hemisphere affected by stroke. This concept is supported by documented improvement of cerebral metabolism, EEG, rCBF, and motor function in stroke patients after HBO therapy. From the available evidence, HBO is considered to be an invaluable adjunct in the rehabilitation of stroke patients with spastic hemiplegia. Although the effects were documented in the paralyzed limbs, spasticity improved in other groups of muscles as well. Kapp, . " Neurological Response to Hyperbaric Oxygen - A Criterion for Cerebral Revascularization. " SURGICAL NEUROLOGY, l981; 15(1): 43-46. Twenty-two patients with cerebral infarction secondary to occlusion of a carotid or middle cerebral artery were exposed to hyperbaric oxygen at 1.5 atmospheres absolute pressure. Ten of the patients demonstrated improved motor function during hyperbaric exposure. Seven of these patients had successful surgical revascularization and no recurrence of neurological deficit. In 3 patients who were not successfully revascularized, the neurological deficit recurred. It is concluded that response to hyperbaric oxygen may be of use in the selection of patients with neurological deficit who will benefit from surgical revascularization of the brain. Lebedev, V.V., et al. " Effect of Hyperbaric Oxygenation on the Clinical Course and Complications of the Acute Period of Ischemic Stroke. " ZHURNAL VOPR NEIROKHIRNRY, l983; 3: 37-42. Hyperbaric oxygenation (HBO) was included in the therapeutic complex for 124 patients in the acute stage of ischemic stroke. The effect of HBO on the clinical course was appraised by comparing the dynamics of changes in the clinical symptoms and the frequency of complications in patients exposed to HBO with those in the control group (patients not exposed to HBO). It was established that the depth of unconsciousness and the motor and aphasic disorders decreased during an HBO session, but the effect was usually short-lived. Aggravation of the patients' condition in the first week of the disease, evidently caused by increase of cerebral edema, occurred much less frequently when HBO was included in the complex of therapeutic measures. The number of patients with regression of the neurological symptoms was practically the same with and without the use of HBO, but the regression of the neurological defects was most evident in patients exposed to HBO. HBO prevents the development of recurrent cerebral circulatory disorders in the acute stage of ischemic stroke and reduces the incidence of some complications in this period (pneumonia, pulmonary edema, thromboembolism of the pulmonary artery, etc). Marroni, A. et al. " Hyperbaric Oxygen Therapy at 1.5 or 2.0 ATA as an Adjunct to the Rehabilitation of Stabilized Stroke Patients. A Controlled Study. " PROCEEDINGS OF THE 9th INTERNATIONAL CONGRESS ON HYPERBARIC MEDICINE, March 1-4, l987; Sydney, Australia, pp. 161-167. HBO Therapy has been studied by many authors as an adjunctive treatment for stroke patients. Satisfactory results have been reported for the use of HBO as a predictive tool for EC-IC revascularization. The questions of the appropriate treatment pressure has been debated in the literature. We studied a group of 80 well stabilized cerebral thrombosis patients not any more undergoing any form of treatment or care. Average age was 59.7 yrs., average stroke age 29.2 months. The patients were divided into 8 groups: A: control group not undergoing any care; B: in water rehabilitation, 30 sessions, no HBO; C1: 30 HBO sessions at 2.0 ATA; C2: same at 1.5 ATA; D1: HBO at 2 ATA plus rehabilitation as above; D2: same at 1.5 ATA; E1: HBO and simultaneous rehabilitation in our specially built Hyperbaric pool at 2 ATA; E2: same at 1.5 ATA. The Rehabilitation protocol was originally developed at our Center as well as a quantitized and repeatable Neuromotor Disability Evaluation Scale. Patients were controlled prior to beginning, every 10 days during treatment, then 1 and 3 months after. Obtained data show defined and similar HBO effects on the improvement of patients' performance at 1.5 and 2.0 ATA, a clear and significant potentiation of this effect being evident for the Hyperbaric Rehabilitation groups and especially for the group treated at 2.0 ATA. The obtained results were still present at the third month after treatment. Neubauer, R.A. et al. " Cerebral oxygenation and the recoverable brain. " NEUROL RES, 20 Suppl 1: S33-6, 1998. Oxygenation is the most critical function of blood flow and a sudden reduction in oxygen availability is an inevitable consequence of severe ischemia. The resulting cascade of events may result in the failure of membrane integrity of some cells and necrosis, but in the surrounding zone of tissue, less affected by hypoxia, cells survive to form the ischemic penumbra. The timing of these events is uncertain, but sufficient oxygen is available to these cells to maintain membrane ion pump mechanisms, but not enough for them to generate action potentials and therefore function as neurons. The existence of such areas has been suspected for some time based upon the natue of clinical recovery, but has now been demonstrated by SPECT imaging with a high plasma oxygen concentration under hyperbaric conditions as a tracer. A course of hyperbaric oxygen therapy frequently results in a permanent improvement in both flow and metabolism. These changes apparently represent a reversal of the changes that render neurons dormant and the activity of cells, previously undetectable by standard electrophysiological methods, can now be demonstrated. Three patients are presented in whom recoverable brain tissue has been identified using SPECT imaging and increased cerebral oxygenation under hyperbaric conditions. Improved perfusion from reoxygenation has correlated with clinical evidence of benefit especially with continued therapy. Neubauer, R.A. et al. " Hyperbaric Oxygen and Imaging Techniques in Diagnosis and Therapy of Stroke. Does the Ischemic Penumbra Alter the Outcome in Stroke? " INTERNATIONAL SYMPOSIUM: NEUROPSYCHOMOTOR, NEURO-PHARMACOLOGICAL, PSYCHOSOCIAL AND ETHICAL ASPECTS, Oct. 7-11, l992; Siracusa, Italy. pp. 1-9. Recovery from stroke (a global phenomena) and predictability of outcome may be directly related not only to tissue damage, but also the ischemic penumbra or surrounding zone of idling neurons. The local and global effects of stroke are well known. Actual recovery or evolution in the neuronal tissue may go on for months. All events related to recovery have yet to be elucidated. It is known that recovery of ischemic or hypoxic tissue is more related to the oxygen content than to blood flow. Utilization of Single Photon Emission Computerized Tomography (SPECT) with the radiotracer Iofetamine I123, aids in demonstrating ischemic penumbras (reperfusion amplitudes) in strokes, thus lending support to the work of Symon, Astrup and Holbach. SPECT analysis before and after a single exposure of hyperbaric oxygen at 1.5 ATA for 60 minutes was performed on 15 stroke patients with strokes ranging in time from 6 hours to 15 years. In all of these patients marked changes in flow and metabolism were seen after hyperbaric intervention, even in cases with neurologic defects present for up to 15 years. This causes speculation as to when stroke is really completed or fully evolved and whether the standard methods of treatment of stroke, and, by extension, all brain injury, encompass the full understanding of the hypoxic or ischemic penumbra. Five cases are presented here: 4 showed varying degrees of improvement associated with a viable halo zone. One patient demonstrated an absent ischemic penumbra. A new protocol combining HBO and surface oxygen will be suggested. Neubauer, R. et al. " Enhancing idling neurons. " letter. THE LANCET, March 3, l990; 542. " After HBO there was a sharp increase in tracer uptake in areas showing hypometabolism on the pre-HBO study...Reduced spasticity, improved ambulation and speech, and cessation of drolling were noted. " Neubauer, R. et al. " Stroke Treatment. " (letter). THE LANCET, June 29, l991; 1601. " Hyperbaric oxygen (HBO) efficiently increases the diffusional driving force for oxygen, thereby increasing tissue oxygen availability. This overcomes ischemia/hypoxia and so reduces cerebral edema, restores integrity to the blood/brain barrier and cell membranes, neutralizes toxic amines, promotes phagocytosis, scavenges free radicals, stimulates angiogenesis, and reactivates idling neurons. " Neubauer, R. et al. " Delayed Metabolism or Reperfusion in Brain Imaging after Exposure to Hyperbaric Oxygenation - A Therapeutic Indicator? " PROCEEDINGS OF THE XV ANNUAL MEETING OF THE EUROPEAN UNDERSEA BIOMEDICAL SOCIETY, Sept. 17-21, l989; Eilat, Israel, pp.1-5. Single Photon Emission Computerized Tomography (SPECT) analysis with Iofetamine I123 was performed in patients with various Central Nervous System (CNS) dysfunctions before and after a single exposure to hyperbaric oxygen (1.5 ATA for 60 minutes) as a guide to potential therapeutic intervention. In CNS disorders current measurements had precluded the identification of idling neurons or the ischemic penumbra, as most techniques involved electrophysiological computerized data. Poorly functioning, yet viable cells, if not electrically active are not identifiable. These cells, however, given the proper oxygen/glucose ratio may return to normal function with dramatic results. Increased Iofetamine I-123 tracer uptake in these ischemic areas (idling neurons) after hyperbaric oxygen therapy probably reflects reactivation of hypometabolic neuronal tissue. Unlike MRI or CT, SPECT reflects regional blood flow as well as grey matter metabolism. The similarity to PET imaging is noteworthy. A variety of patients with central nervous system dysfunction were studied. Reactivation of marginal or idling neurons was seen in many disease entities, the most dramatic being long standing hypoxic encephalopathies. Demonstrative cases will be presented including hypoxic encephalopathy and acute and chronic neurologic deficit of stroke. Reactivation of the idling neuron may be of clinical significance. It is important for the physician to differentiate between viable and non-viable tissue, both from the standpoint of treatment and prognosis. Neubauer, R.A. " Generalized small-vessel stenosis in the brain. A case History of a Patient Treated with Monoplace Hyperbaric Oxygen at 1.5 to 2 ATA. " MINERVA MEDICA, l983; 74: 2051-2055. Complete evaluation of older patients with mental changes always leaves us with a certain percentage whose condition can only be attributed to atherosclerosis. Little is being done for these patients because this generalized stenosis of the brain does not reverse with any known treatment. This writer has treated many such patients with hyperbaric oxygen (HBO), and presents this case history, along with regional cerebral blood flow (rCBF) studies, showing the type of changes which frequently occur. This case initially presented with symptoms of gross mental confusion, memory loss, both recent and remote, irrational speech and occasional violence. Although prior complete evaluations were concluded with no recommended treatment, the initial series of HBO treatment resulted in a well-functioning patient. This was maintained for four years with intermittent HBO. The patient then presented with acute stroke, total disorientation and confusion. He again became functional with HBO. A discussion of the mechanisms of HBO which might account for the changes is given. Neubauer, R.A. et al. " Hyperbaric Oxygenation as an Adjunct Therapy in Strokes Due to Thrombosis. " STROKE, l980; 11(3): 297-300. Results are reported using hyperbaric oxygenation (HBO) in 122 patients with strokes due to thrombosis, both acute and completed. HBO is used as adjunctive treatment and there appears to be justification for a controlled study to delineate the treatment further. The authors believe it is essential to treat patients with stroke at 1.5 to 2 atmospheres absolute (ATA). Nighoghossian, N. et al. " Hyperbaric oxygen in the treatment of acute ischemic stroke: an unsettle issue. " ; JOURNAL OF THE NEUROLOGICAL SCIENCES, 1997; 150(1): 27-31. Therapy for acute ischemic stroke can be approached in two basic ways: first, by an attempt to restore or improve blood flow in an occluded vascular territory and, second, via therapy directed at the cellular and metabolic targets. As local anoxia and energy failure are the initiating cellular stage in ischemia, the inhalation of oxygen at increased atmospheric pressures might be effective. Treatment of acute focal cerebral ischemia with hyperbaric oxygen (HBO) has been reported in animals and humans. In general, the results of research in animals have suggested a promising role for the use of HBO. More than 400 cases of human ischemic stroke treated with HBO have been reported. In about half of the cases, improvement in status has been claimed on clinical or electroencephalographic grounds. " It might be speculated that the patients most likely to respond favorably to HBO therapy are those who have infarcts related to large vessel thrombosis and surrounded by ischemic penumbra. In support of this are reports claiming a favorable transient or,less often,permanent response to HBO in cases selected for demonstrated carotid occlusion. A large double-blind study might be required in the future. Based on experimental data, HBO at 1.5 ATA during 1 hour might be proposed, as neurotoxicity is rare with low pressure and short duration. If HBO treatment is safe and effective, it could be added to thrombolytic therapy which has recently shown its efficiency in restoring cerebral blood flow. " Nighoghossian, N. et al. " Hyperbaric Oxygen in the Treatment of Acute Ischemic Stroke. A Double-blind Pilot Study. " STROKE, l995; 26: 1369-1372. Background and Purpose: The effects of hyperbaric oxygen (HBO) therapy on humans are uncertain. Our study aims first to outline the practical aspects and the safety of HBO treatment and then to evaluate the effect of HBO on long-term disability. Methods: Patients who experienced middle cerebral artery occlusion and were seen within 24 hours of onset were randomized to receive either active (HBO) or sham (air) treatment. The HBO patients were exposed daily to 40 minutes at 1.5 atmospheres absolute for a total of 10. We used the Orgogozo scale to establish a pretreatment functional level. Changes in the Orgogozo scale score at 6 months and 1 year after therapy were used to assess the therapeutic efficacy of HBO. In addition, we used the Rankin scale and our own 10-point scale to assess long term-disability at 6 months and 1 year. Two sample t tests and 95% confidence intervals were used to compare the mean differences between the two treatment groups. Student's two-tailed test was used to compare the differences between pre-therapeutic and post-therapeutic scores at 6 months and 1 year in the two treatment groups. Results: Over the 3 years of study enrollment, 34 patients were randomized, 17 to hyperbaric treatment with air and 17 to hyperbaric treatment with 100% oxygen. There was no significant difference at inclusion between groups regarding age, time from stroke onset to randomization and Orgogozo scale. Neurological deterioration occurred during the first week in 4 patients in the sham group, 3 of whom died; this worsening was clearly related to the ischemic damage. Treatment was also discontinued for 3 patients in the HBO group who experienced myocardial infarction, a worsening related to the ischemic process, and claustrophobia. Therefore, 27 patients (13 in the sham group and 14 in the HBO group) completed a full course of therapy. The mean score of the HBO group was significantly better on the Orogozo scale at 1 year. However, the difference at 1 year between pre-therapeutic and post-therapeutic scores was not significantly different in the two groups. Moreover, no statistically significant improvement was observed in the HBO group at 6 months and 1 year according to Rankin score and our own 10-point scale. Conclusions: Although the small number of patients in each group precludes any conclusion regarding the potential deleterious effect of HBO, we did not observe the major side effects usually related to HBO. Accordingly, it can be assumed that hyperbaric oxygen might be safe. We hypothesize that HBO might improve outcome after stroke, as we detected an outcome trend favoring HBO therapy. A large randomized trial might be required to address the efficacy of this therapy. Raju, GS, et al. " Cerebral accident during endoscopy: Consider cerebral air embolism, a rapidly reversible event with hyperbaric oxygen therapy. " GASTROINTEST ENDOSC, 47(1): 70-73, 1998. A 75 year old male could not swallow solids. During endoscopic dilatation, the patient became hypoxic and unresponsive. He was mute, could not understand speech and could not move his limbs on the left side. A CT scan of the brain showed intravascular air bubbles in the right middle cerebral artery. Within two hours of the event, he was treated with hyperbaric oxygen therapy at 3 ata for 46 minutes and at 2 ata for 150 minutes. A follow up CT scan showed resolution of the air bubbles. That evening he was able to move his left arm again. Several days later, he was able to walk with minimal assistance and his speech and comprehension returned to normal. Veltkamp, Roland. " Hyperbaric oxygen-A neuroprotective adjunct for hyperacute ischemic stroke? " Letter. J NEUROL SCI, 150, pg.1-2; 1997. The author writes that though the benefit from HBO therapy is still speculative, its neuroprotective potential may be greater just after stroke onset. He suggests that portable HBO equipment be designed for the initiation of therapy on site by trained personnel before and during emergency transportation to the hospital. He then states that as soon as the equipment is available, " the effectiveness of HBOT in combination with other new hyperactue stroke therapies can and must be(re)explored. " stroke >Thanks everyone for all the good advice. Now it's up to me to impress >upon auntie the importance of the advice. Her good " doctor " has >suggested a couple aspirin per day as the immediate remedy. (Hippocrates >would be spinning in his grave....) > >, please send that info to me at 5801 e cambridge ave, scottsdale, >az. 85257. Auntie lives in southern Wa, nearest city is Portland, Ore. > >Thanks, Corny > > >OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > >THERE IS NO MEDICAL ADVICE HERE! > >This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > >You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - >DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 19, 2000 Report Share Posted December 19, 2000 Hi a I understand there is Glen from Rife New Zealand doing some form of ozone therapy. This is their address u might want to contact them for detail. Rife New Zealand Ltd 185 Gills Road, Albany, Auckland, New Zealand 1310 Phone : 9 415 6562 Fax : 9 415 6553 Email : rifenz@... Regards Reginald At 05:15 AM 20/12/00 -0000, you wrote: >HI My first time to the list can anyone help I live in NZ .My mother >had a stroke two years ago and is institutionalised she can walk with >the aid of a walker and nurse and she does know her family but she is >sliding in health and losing interest .Short term memory is not good >but the hospital insist on feeding her diabetic drink which isnt >helping, as she is a food controlled diabetic they say she wont drink >water and has to drink due to recurring bladder infections. Is this >treatment you have been discussing available in NZ and does my mum >sound like she could maybe ? benefit from it ,she has lost some use >of her left arm and foot I would dearly like to hear from anyone who >can help IN antisipation THANKS a > > > >OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > >THERE IS NO MEDICAL ADVICE HERE! > >This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > >You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - >DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > " The problems of today cannot be solved using the same thinking that created them " . - Einstein Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 20, 2000 Report Share Posted December 20, 2000 In a message dated 12/20/00 5:36:14 AM Eastern Standard Time, saul@... writes: << Try and get her some : 3) Methylene blue; or cranberry juice if you can 4) Hyperbaric oxygen treatments >> Saul, Can an infant (who suffered a stroke at birth) take methylene blue? Dotsie Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 20, 2000 Report Share Posted December 20, 2000 Dear a, Try and get her some : 1) Chromium + vanadium 2) Gymnema sylvestre 3) Methylene blue; or cranberry juice if you can 4) Hyperbaric oxygen treatments Best of Health! Dr. Saul Pressman, DCh stroke > HI My first time to the list can anyone help I live in NZ .My mother > had a stroke two years ago and is institutionalised she can walk with > the aid of a walker and nurse and she does know her family but she is > sliding in health and losing interest .Short term memory is not good > but the hospital insist on feeding her diabetic drink which isnt > helping, as she is a food controlled diabetic they say she wont drink > water and has to drink due to recurring bladder infections. Is this > treatment you have been discussing available in NZ and does my mum > sound like she could maybe ? benefit from it ,she has lost some use > of her left arm and foot I would dearly like to hear from anyone who > can help IN antisipation THANKS a > > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 20, 2000 Report Share Posted December 20, 2000 I do not know about Methylene blue, However we have treated five children with Stroke at birth with wonderful results. CHT Rapid Recovery Hyperbarics http://www.hbot4u.com Re: stroke > In a message dated 12/20/00 5:36:14 AM Eastern Standard Time, > saul@... writes: > > << Try and get her some : > 3) Methylene blue; or cranberry juice if you can > 4) Hyperbaric oxygen treatments >> > > Saul, > Can an infant (who suffered a stroke at birth) take methylene blue? > Dotsie > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 20, 2000 Report Share Posted December 20, 2000 Dear Dotsie, Yes. 3 drops in some water and give it with an eyedropper. Try and get him/her some time in a hyperbaric chamber. It is the easiest and most effective treatment. Best of Health! Dr. Saul Pressman, DCh Re: stroke > In a message dated 12/20/00 5:36:14 AM Eastern Standard Time, > saul@... writes: > > << Try and get her some : > 3) Methylene blue; or cranberry juice if you can > 4) Hyperbaric oxygen treatments >> > > Saul, > Can an infant (who suffered a stroke at birth) take methylene blue? > Dotsie > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 24, 2000 Report Share Posted December 24, 2000 In a message dated 12/20/00 12:40:14 PM Eastern Standard Time, saul@... writes: << Try and get him/her some time in a hyperbaric chamber. It is the easiest and most effective treatment. >> Thanks Saul and . I saved your previous post on Methylene blue and I never thought I could give it to an infant. , I'm still looking into finding a hyperbaric chamber that is suitable for an infant on the East Coast with no luck. She would never tolerate sitting alone in a chamber. Dotsie Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 24, 2000 Report Share Posted December 24, 2000 I would never put an infant in a chamber alone, or any child for that matter, We have treated three month olds that go in with mom, See my website for trinity who regain the use of her arm after treatment, you will see a picture of her in the chamber, I have also treated twins at three months old with stroke. We have treated a child who had a stroke at 6 years old, all of which when into our Large chambers with the parents, Both parents and child have full phys exam before any therapy is started to rule out the contra indications for HBOT. We have treated adults who have stroked as well. Please send the details again i.e. age etc.. Call me anytime lets talk about it, 909-889-0517.. CHT EMT, DMT CHT Dr. Underwood MD, DO, JD Rapid Recovery Hyperbarics http://www.hbot4u.com Re: stroke > In a message dated 12/20/00 12:40:14 PM Eastern Standard Time, > saul@... writes: > > << Try and get him/her some time in a hyperbaric chamber. It is > the easiest and most effective treatment. >> > > Thanks Saul and . I saved your previous post on Methylene blue and I > never thought I could give it to an infant. , I'm still looking into > finding a hyperbaric chamber that is suitable for an infant on the East Coast > with no luck. She would never tolerate sitting alone in a chamber. > Dotsie > > > OxyPLUS is an unmoderated e-ring dealing with oxidative therapies, and other alternative self-help subjects. > > THERE IS NO MEDICAL ADVICE HERE! > > This list is the 1st Amendment in action. The things you will find here are for information and research purposes only. We are people sharing information we believe in. If you act on ideas found here, you do so at your own risk. Self-help requires intelligence, common sense, and the ability to take responsibility for your own actions. By joining the list you agree to hold yourself FULLY responsible FOR yourself. Do not use any ideas found here without consulting a medical professional, unless you are a researcher or health care provider. > > You can unsubscribe via e-mail by sending A NEW e-mail to the following address - NOT TO THE OXYPLUS LIST! - > DO NOT USE REPLY BUTTON & DO NOT PUT THIS IN THE SUBJECT LINE or BODY of the message! : > > oxyplus-unsubscribeegroups > > oxyplus-normalonelist - switch your subscription to normal mode. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 14, 2002 Report Share Posted May 14, 2002 I did a small scale research project in 1998 for presentation to the local hospital grand rounds on stroke incidence with CMT. I took the number of stroke cases over a two year period from the two malpractice carriers as the numerator, and the number of practicing DCs x average visits per week x 104 x 50%. the assumptions were: 1. every stroke case following manipulation will result in litigation 2. about half of DC office visits involve cervical manipulation 3. Washington DC's visit average is about the same as the national average. (I think it was about 100 office visits at the time. ) This number came out to a frequency of 1/3.5 million office visits. This was a considerably lower frequency than was published to the time, and I thought it would cause a furor with the MDs at grand rounds. Oddly, they weren't up in arms about it... This type of analysis was used by Haldeman in the 2001 Spine article, with even lower frequency; about 1:5.8 million office visits. Most of the malpractice cases I have seen really revolve around the lack of proper response to a stroke in evolution, rather than strictly causation. Dr. Roy Steinberg Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 14, 2002 Report Share Posted May 14, 2002 Your right on Roy. The cases I have reviewed always file on negligence which is always based on the DC not gaining proper informed consent from the patient. I personally recommend a written informed constent form that both the doctor and the patient signs after the patient has read the consent form. I follow this up with a note in my chart note that I discussed this informed consent and asked if the patient if they had any questions and that they did not etc. It's not enough to simply have the patient's sign a form and then place it in their file you MUST have a conversation with the patient and document this conversation in your chart. Alternatively you DO NOT have to tell them that " oh and by the way did I mention that this could KILL YOU! Its very very rare of course but just the same you could DIE! You only have to mention material risks, in my form it covers the term stroke. The statement or analogy I use is this " Mr./Ms. you have a 300 to 400 times greater chance of having a serious side effect from taking Advil than any thing I'm going to do. " If any of you colleagues would like a copy of my informed constent form and the paragraph I use in my chart note system respond to this E-mail with your mailing information and I'll get this out to you ASAP. Vern Saboe, DC ACA Delegate (Oregon) CAO Executive Board Albany Re: stroke > I did a small scale research project in 1998 for presentation to the local > hospital grand rounds on stroke incidence with CMT. I took the number of > stroke cases over a two year period from the two malpractice carriers as the > numerator, and the number of practicing DCs x average visits per week x 104 x > 50%. the assumptions were: > > 1. every stroke case following manipulation will result in litigation > 2. about half of DC office visits involve cervical manipulation > 3. Washington DC's visit average is about the same as the national average. > (I think it was about 100 office visits at the time. ) > > This number came out to a frequency of 1/3.5 million office visits. This was > a considerably lower frequency than was published to the time, and I thought > it would cause a furor with the MDs at grand rounds. Oddly, they weren't up > in arms about it... > > This type of analysis was used by Haldeman in the 2001 Spine article, with > even lower frequency; about 1:5.8 million office visits. > > Most of the malpractice cases I have seen really revolve around the lack of > proper response to a stroke in evolution, rather than strictly causation. > > Dr. Roy Steinberg > > > OregonDCs rules: > 1. Keep correspondence professional; the purpose of the listserve is to foster communication and collegiality. No personal attacks on listserve members will be tolerated. > 2. Always sign your e-mails with your first and last name. > 3. The listserve is not secure; your e-mail could end up anywhere. However, it is against the rules of the listserve to copy, print, forward, or otherwise distribute correspondence written by another member without his or her consent, unless all personal identifiers have been removed. > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 11, 2003 Report Share Posted January 11, 2003 Mark, Do an assessment. Look for the excessive slow activity, especially if you find it in a focal area Train down the slow activity where you find it. Pete Stroke Dear Group: Monday morning I will be assessing my first stroke victim. If anyone has any tips for working with this type of client, I would really appreciate them. Mark Quote Link to comment Share on other sites More sharing options...
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