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Guar Gum & colon cancer

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It seems to me that insoluble fiber (bran category) carries the most

protection from colon cancer, while soluble fiber (guar category)

provides minimal protection (if any). Do you think the following

studies are saying the same thing?

http://snipurl.com/av7v PMID: 8759370

Dietary guar gum alters colonic microbial fermentation in

azoxymethane-treated rats.

Our results show that patterns of short-chain fatty acid production may

affect the results of fiber-carcinogenesis experiments. Dietary addition

of hydrolyzed guar is associated with fecal fermentation low in

propionate and high in butyrate; short-chain fatty acid concentrations

are greater proximally than distally. These results suggest that

butyrate protects against colonic neoplasia, whereas propionate enhances

it, and demonstrate that colonic microbiota adapt to produce more

butyrate if given time and the proper substrate.

~~~~~~~~~~

http://snipurl.com/av7z PMID: 8386131

Butyrate production from dietary fibre and protection against large

bowel cancer in a rat model.

These findings indicate that fibre which is associated with high

butyrate concentrations in the distal large bowel is protective against

large bowel cancer, while soluble fibres that do not raise distal

butyrate concentrations, are not protective. Thus, butyrate production

in vivo does bear a significant relationship to suppression of tumour

formation.

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Hi Apricot:

Yes. My impression is the same. But .................

" Slow-release pellets of sodium butyrate do not modify azoxymethane

(AOM)-induced intestinal carcinogenesis in F344 rats.

Caderni G, Luceri C, De Filippo C, Salvadori M, Giannini A, Tessitore

L, Dolara P.

Department of Pharmacology of the University of Florence, Viale G.

Pieraccini, 6, 50139, Florence, Italy. gioca@...

Butyrate exerts anti-tumour effects in vitro, but not consistently in

vivo. We previously demonstrated that the administration of slow-

release gastro-resistant pellets of sodium butyrate increases

apoptosis in the colon mucosa of rats, an effect which may protect

against carcinogenesis. Therefore, we studied whether the

administration of butyrate pellets could protect rats against

experimental colon carcinogenesis. Four to 5 week old male F344 rats

were fed a high-fat (HF) diet (230 g/kg corn oil w/w) and treated

s.c. with two injections (one week apart) of azoxymethane (AOM) at a

dose rate of 15 mg/kg body weight or saline. Rats were then divided

into two groups: one group received sodium butyrate pellets mixed

into the diet (1.5% w/w) for 33 weeks (150 mg butyrate/day) and the

second group received the high-fat diet with no butyrate.

Administration of sodium butyrate pellets in the diet did not

significantly affect colon carcinogenesis: the number of intestinal

tumours/rat was 1.6 +/- 0.2 in controls and 2.1 +/- 0.2 in butyrate-

fed rats (means +/- SE; P = 0.22, by ANOVA), while the incidence of

intestinal tumours was 79 (23/29) and 90% (27/30) in controls and in

butyrate-fed rats, respectively (P = 0.29 by Fisher's exact test).

The level of apoptosis in the tumours was not affected by butyrate,

nor was the expression of p21(CIP), a cell cycle-related protein. In

conclusion, the current study indicates that butyrate does not

protect against AOM-induced colon carcinogenesis in rats.

PMID: 11238196 [PubMed - indexed for MEDLINE] "

So who knows?

Rodney.

--- In , apricot85 <apricot85@a...>

wrote:

> It seems to me that insoluble fiber (bran category) carries the

most

> protection from colon cancer, while soluble fiber (guar category)

> provides minimal protection (if any). Do you think the following

> studies are saying the same thing?

>

> http://snipurl.com/av7v PMID: 8759370

> Dietary guar gum alters colonic microbial fermentation in

> azoxymethane-treated rats.

>

> Our results show that patterns of short-chain fatty acid production

may

> affect the results of fiber-carcinogenesis experiments. Dietary

addition

> of hydrolyzed guar is associated with fecal fermentation low in

> propionate and high in butyrate; short-chain fatty acid

concentrations

> are greater proximally than distally. These results suggest that

> butyrate protects against colonic neoplasia, whereas propionate

enhances

> it, and demonstrate that colonic microbiota adapt to produce more

> butyrate if given time and the proper substrate.

> ~~~~~~~~~~

> http://snipurl.com/av7z PMID: 8386131

> Butyrate production from dietary fibre and protection against large

> bowel cancer in a rat model.

>

> These findings indicate that fibre which is associated with high

> butyrate concentrations in the distal large bowel is protective

against

> large bowel cancer, while soluble fibres that do not raise distal

> butyrate concentrations, are not protective. Thus, butyrate

production

> in vivo does bear a significant relationship to suppression of

tumour

> formation.

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Hi Apricot:

So, if bran truly is beneficial, PERHAPS it is not the butyrate that

is responsible?

But also, did not one Harvard study from about a year ago show no

relationship between 'fiber' and colon cancer?

So, whatever it is we decide to do, it is in the nature of placing

bets, I think.

Hopefully, fifty years from now we will have more consistent

evidence. By which time some of us will be 112, and still arguing

about it here.

Rodney.

> > It seems to me that insoluble fiber (bran category) carries the

> most

> > protection from colon cancer, while soluble fiber (guar category)

> > provides minimal protection (if any). Do you think the following

> > studies are saying the same thing?

> >

> > http://snipurl.com/av7v PMID: 8759370

> > Dietary guar gum alters colonic microbial fermentation in

> > azoxymethane-treated rats.

> >

> > Our results show that patterns of short-chain fatty acid

production

> may

> > affect the results of fiber-carcinogenesis experiments. Dietary

> addition

> > of hydrolyzed guar is associated with fecal fermentation low in

> > propionate and high in butyrate; short-chain fatty acid

> concentrations

> > are greater proximally than distally. These results suggest that

> > butyrate protects against colonic neoplasia, whereas propionate

> enhances

> > it, and demonstrate that colonic microbiota adapt to produce more

> > butyrate if given time and the proper substrate.

> > ~~~~~~~~~~

> > http://snipurl.com/av7z PMID: 8386131

> > Butyrate production from dietary fibre and protection against

large

> > bowel cancer in a rat model.

> >

> > These findings indicate that fibre which is associated with high

> > butyrate concentrations in the distal large bowel is protective

> against

> > large bowel cancer, while soluble fibres that do not raise distal

> > butyrate concentrations, are not protective. Thus, butyrate

> production

> > in vivo does bear a significant relationship to suppression of

> tumour

> > formation.

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Share on other sites

Hi Apricot:

Two studies on colon cancer, disputing supposed causes:

" Prospective study of bowel movement, laxative use, and risk of

colorectal cancer among women.

Dukas L, Willett WC, Colditz GA, Fuchs CS, Rosner B, Giovannucci EL.

Department of Nutrition, Harvard School of Public Health, Boston, MA,

USA.

The authors prospectively examined the association between bowel

movement frequency, laxative use, and the risk of colorectal cancer

in 84,577 women of the Nurses' Health Study living in the United

States, 36-61 years of age and free of cancer in 1982. Between 1984

and 1996, 611 incident cases of colorectal cancer were documented.

After controlling for age, body mass index, fiber intake,

postmenopausal status and hormone use, physical activity, and use of

laxatives, the relative risks associated with having bowel movements

every third day or less, compared with those with bowel movements

once daily, were 0.94 (95% confidence interval (CI): 0.69, 1.28) for

colorectal cancer, 0.88 (95% CI: 0.62, 1.26) for colon cancer, and

1.18 (95% CI: 0.63, 2.20) for rectal cancer. Compared with women who

never used laxatives, the multivariate relative risks associated with

weekly to daily laxative use were 1.00 (95% CI: 0.72, 1.40) for

colorectal cancer, 1.09 (95% CI: 0.76, 1.57) for colon cancer, and

0.68 (95% CI: 0.29, 1.57) for rectal cancer. These findings do not

support an association between infrequent bowel movement, laxative

use, and risk of colorectal cancer and indicate that simple questions

directed at bowel movement frequency are unlikely to enhance our

ability to predict colorectal cancer risk.

PMID: 10853634 [PubMed - indexed for MEDLINE] "

And ............................

" Dietary fiber and the risk of colorectal cancer and adenoma in women.

Fuchs CS, Giovannucci EL, Colditz GA, Hunter DJ, Stampfer MJ, Rosner

B, Speizer FE, Willett WC.

Department of Medicine, Brigham and Women's Hospital and Harvard

Medical School, Boston, MA, USA.

BACKGROUND: A high intake of dietary fiber has been thought to reduce

the risk of colorectal cancer and adenoma. METHODS: We conducted a

prospective study of 88,757 women, who were 34 to 59 years old and

had no history of cancer, inflammatory bowel disease, or familial

polyposis, who completed a dietary questionnaire in 1980. During a 16-

year follow-up period, 787 cases of colorectal cancer were

documented. In addition, 1012 patients with adenomas of the distal

colon and rectum were found among 27,530 participants who underwent

endoscopy during the follow-up period. RESULTS: After adjustment for

age, established risk factors, and total energy intake, we found no

association between the intake of dietary fiber and the risk of

colorectal cancer; the relative risk for the highest as compared with

the lowest quintile group with respect to fiber intake was 0.95 (95

percent confidence interval, 0.73 to 1.25). No protective effect of

dietary fiber was observed when we omitted, adjustment for total

energy intake, when events during the first six years of follow-up

were excluded, or when we excluded women who altered their fiber

intake during the follow-up period. No significant association

between fiber intake and the risk of colorectal adenoma was found.

CONCLUSIONS: Our data do not support the existence of an important

protective effect of dietary fiber against colorectal cancer or

adenoma.

PMID: 9895396 [PubMed - indexed for MEDLINE] "

Rodney.

> > > It seems to me that insoluble fiber (bran category) carries the

> > most

> > > protection from colon cancer, while soluble fiber (guar

category)

> > > provides minimal protection (if any). Do you think the

following

> > > studies are saying the same thing?

> > >

> > > http://snipurl.com/av7v PMID: 8759370

> > > Dietary guar gum alters colonic microbial fermentation in

> > > azoxymethane-treated rats.

> > >

> > > Our results show that patterns of short-chain fatty acid

> production

> > may

> > > affect the results of fiber-carcinogenesis experiments. Dietary

> > addition

> > > of hydrolyzed guar is associated with fecal fermentation low in

> > > propionate and high in butyrate; short-chain fatty acid

> > concentrations

> > > are greater proximally than distally. These results suggest

that

> > > butyrate protects against colonic neoplasia, whereas propionate

> > enhances

> > > it, and demonstrate that colonic microbiota adapt to produce

more

> > > butyrate if given time and the proper substrate.

> > > ~~~~~~~~~~

> > > http://snipurl.com/av7z PMID: 8386131

> > > Butyrate production from dietary fibre and protection against

> large

> > > bowel cancer in a rat model.

> > >

> > > These findings indicate that fibre which is associated with

high

> > > butyrate concentrations in the distal large bowel is protective

> > against

> > > large bowel cancer, while soluble fibres that do not raise

distal

> > > butyrate concentrations, are not protective. Thus, butyrate

> > production

> > > in vivo does bear a significant relationship to suppression of

> > tumour

> > > formation.

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