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Vitamin E reduces colds

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Hi All,

Does taking vitamin E reduce the incidence of

respiratory tract infections?

It seems that the supplement in this placebo/

control random trial given a standard alpha

form of vitamin E not statistically significantly

reduced the overall rate of respiratory tract

infections.

For those completing the one-year trial, the

numbers shown below in the pdf-available

paper's Table 4 are compelling.

Lower respiratory tract infections were very

slightly more and so was the corresponding

use of antibiotics.

The number of days with infections resembled

the incidence of infections and thus the infections

were prevented but not treated with the vitamin.

Alternative medicines have been suggested to

reduce infections such as these if taken early in

the course of the infection, but this apparently

does not apply for vitamin E.

The routinely used of the alpha form of the

vitamin was taken by the subjects of the trial.

Also observed was that " Post hoc analysis

found no significant effect of vitamin E on other

URIs (0.035 vs 0.050 for influenzalike infections,

0.082 vs 0.073 for pharyngitis, 0.013 vs 0.009

for otitis media, and 0.030 vs 0.009 for sinusitis

per person-year in the vitamin E and placebo

groups, respectively) " . Many more had colds,

as would be expected.

JAMA. 2004 Aug 18;292(7):828-36.

Vitamin E and respiratory tract infections in elderly nursing home

residents: a randomized controlled trial.

Meydani SN, Leka LS, Fine BC, Dallal GE, Keusch GT, Singh MF, Hamer

DH.

Objective To determine the effect of 1 year of vitamin E

supplementation on respiratory tract infections in elderly nursing

home residents.

Design, Setting, and Participants A randomized, double-blind,

placebo-controlled trial was conducted from April 1998 to August 2001

at 33 long-term care facilities in the Boston, Mass, area. A total of

617 persons aged at least 65 years and who met the study's

eligibility criteria were enrolled; 451 (73%) completed the study.

Intervention Vitamin E (200 IU) or placebo capsule administered

daily; all participants received a capsule containing half the

recommended daily allowance of essential vitamins and minerals.

Main Outcome Measures Incidence of respiratory tract infections,

number of persons and number of days with respiratory tract

infections (upper and lower), and number of new antibiotic

prescriptions for respiratory tract infections among all participants

randomized and those who completed the study.

Results Vitamin E had no significant effect on incidence or

number of days with infection for all, upper, or lower respiratory

tract infections. However, fewer participants receiving vitamin E

acquired 1 or more respiratory tract infections (60% vs 68%; risk

ratio [RR], 0.88; 95% confidence interval [CI], 0.76-1.00; P = .048

for all participants; and 65% vs 74%; RR, 0.88; 95% CI, 0.75-0.99; P

= .04 for completing participants), or upper respiratory tract

infections (44% vs 52%; RR, 0.84; 95% CI, 0.69-1.00; P = .05 for all

participants; and 50% vs 62%; RR, 0.81; 95% CI, 0.66-0.96; P = .01

for completing participants). When common colds were analyzed in a

post hoc subgroup analysis, the vitamin E group had a lower incidence

of common cold (0.67 vs 0.81 per person-year; RR, 0.83; 95% CI, 0.68-

1.01; P = .06 for all participants; and 0.66 vs 0.83 per person-year;

RR, 0.80; 95% CI, 0.64-0.98; P = .04 for completing participants) and

fewer participants in the vitamin E group acquired 1 or more colds

(40% vs 48%; RR, 0.83; 95% CI, 0.67-1.00; P = .05 for all

participants; and 46% vs 57%; RR, 0.80; 95% CI, 0.64-0.96; P = .02

for completing participants). Vitamin E had no significant effect on

antibiotic use.

Conclusions Supplementation with 200 IU per day of vitamin E did

not have a statistically significant effect on lower respiratory

tract infections in elderly nursing home residents. However, we

observed a protective effect of vitamin E supplementation on upper

respiratory tract infections, particularly the common cold, that

merits further investigation.

Publication Types:

Clinical Trial

Multicenter Study

Randomized Controlled Trial

PMID: 15315997 [PubMed - indexed for MEDLINE]

Infections, particularly respiratory tract infections, are common

in elderly individuals, resulting in decreased daily activity,

prolonged recovery times, increased health care service use, and more

frequent complications, including death.1-11 In the United States, an

estimated 43% of elderly persons will be admitted to a nursing home,

with more than 85% of them admitted to long-term (>1 year) care

facilities.12 Infections occur more frequently in nursing home

residents than among independent-living elderly,2-10,13 and

respiratory tract infections are a major cause of morbidity and

mortality.9, 14-15 Contributing to the increased incidence of

infection with age is the well-described decline in immune

response.16 For example, those who have diminished delayed-type

hypersensitivity skin test responses have higher morbidity and

mortality from cancer, pneumonia, and postoperative complications.17-

19

Nutritional status is an important determinant of immune

function.20-21 Nutritional supplementation has been shown to enhance

the immune response in older persons.22-23 In our earlier placebo-

controlled, double-blind trials in elderly persons, vitamin E

supplementation improved immune response, including delayed-type

hypersensitivity and response to vaccines.24-25 Furthermore,

participants receiving vitamin E in the 6-month trial25 had a 30%

lower incidence of infectious diseases (primarily respiratory tract

infections) compared with those receiving placebo, but this result

was not significant, perhaps because of insufficient power, and

infections were self-reported. To overcome these limitations, the

current study determined the effect of 1 year of supplementation with

vitamin E on objectively recorded respiratory tract infections in

elderly nursing home residents.

Study Design

...

The vitamin E group received a daily capsule containing 200 IU of

vitamin E (DL--tocopherol)

...

RESULTS

Participant Characteristics

The mean (SD) follow-up time was 317 (104) days for the vitamin E

group and 321 (97) days for the placebo group. Of the 617 randomized

persons, 231 (37%) and 220 (36%) in the vitamin E and placebo groups,

respectively, completed the 1-year study period (Figure 1). The 2

groups did not differ statistically in the proportion or causes of

discontinuation (Figure 1) or in mortality rates (12.5% [39/311] and

14.4% [44/306] for the vitamin E and placebo groups, respectively).

Table 1 shows participant characteristics for all who were

enrolled in the study (all) and for those who completed 1 year

(completers). The groups were well balanced with regard to baseline

characteristics. One exception was a lower percentage of completers

with diabetes mellitus in the vitamin E group compared with placebo

(P = .04) (Table 1).

All participants received influenza vaccine, and the 2 groups did

not differ statistically in the percentage of participants receiving

pneumococcal vaccine (30/311 [9.6%] vitamin E vs 23/306 [7.5%]

placebo, P = .53 for all; 29/231 [12.6%] vitamin E vs 19/220 [8.6%]

placebo, P = .18 for completers). The mean number of days during

which completers took immune-related medications during the study

period did not differ significantly (nonsteroidal anti-inflammatory

drugs [131 vs 110], antihistamines [4.5 vs 7.9], steroids [16.3 vs

9.2], or nutrient supplements [84 vs 92] for vitamin E and placebo

groups, respectively).

Biochemical and hematological measurements before and after

vitamin E supplementation indicated no difference between the 2

groups, except as otherwise specified (complete data available on

request).

Adherence

Ninety-eight percent (442/451) of those completing the study

consumed the capsules for at least 330 days (>90% of the 1-year

supplementation period). The number of missed supplements did not

differ statistically between the vitamin E and placebo groups (data

available on request). Adherence was confirmed by plasma vitamin E

measurement every 3 months.

Nutritional Status

Vitamin E and placebo groups did not differ statistically in body

mass index or serum levels of vitamins and minerals before or after

supplementation (data available on request). The vitamin E group had

small but significantly higher hemoglobin levels than the placebo

group before and after supplementation (mean [sD], 12.4 [1.4] vs 12.2

[1.3] g/dL before and 12.4 [1.3] vs 12.1 [1.5] g/dL after in the

vitamin E and placebo groups, respectively; P = .02). Significantly

fewer participants had low serum albumin levels in the vitamin E

group compared with placebo at baseline and after supplementation

(Table 2). The percentage of participants with low albumin levels

increased significantly during the 1-year period for both groups, but

the change over time in serum albumin between the 2 groups did not

differ significantly.

Except for vitamin E, the level of micronutrients did not change

significantly during the study period in either group. Plasma vitamin

E levels increased significantly in the vitamin E group, which

doubled after 3 months of supplementation with no further change

(mean [sD], 1141 [391] vs 2119 [689] µg/dL before and after

supplementation, respectively; P<.001). No significant change in

serum vitamin E levels was observed in the placebo group (1148 [429]

vs 1209 [408] µg/dL before and after supplementation, respectively).

The fraction of participants with low serum vitamin A levels

increased slightly but significantly, whereas the fraction of

participants with low vitamin D and B6 levels decreased in both

groups (Table 2), with no significant difference between treatments

in change over time.

Significantly fewer participants had low hemoglobin levels in the

vitamin E group before and after supplementation (Table 2). The

fraction of participants with low hemoglobin levels in each group did

not change significantly over time. Low serum zinc levels were

equally prevalent in both groups (Table 2).

Respiratory Tract Infections

Results generally were similar whether the data from all

participants (Table 3) or completing participants (Table 4) were

compared. Adjustment for obstructive lung diseases, current smoking

status, diabetes mellitus, dementia, year of enrollment, and baseline

albumin and hemoglobin levels did not affect the outcomes, with a few

exceptions, as noted in the text. Further adjustment for nursing home

gave essentially the same results. Thus, only the unadjusted data are

shown (Table 3 and Table 4), except as noted in the text.

Table 4. Respiratory Tract Infection Among Participants

Completing the Study*

------------------------------------------------------

Vitamin E

(n = 231)

Placebo

(n = 220)

Rate Ratio

(95% CI)

P

Value

----------------------------------------

Incidence of infection

All respiratory tract infections

Infections, No. 304 320

Infections per person per year 1.30 1.44 0.91 (0.77 to 1.06) .22

Lower respiratory tract infection**

Infections, No. 115 105

Infections per person per year 0.49 0.47 1.05 (0.80 to 1.36) .74

Upper respiratory tract infection***

Infections, No. 189 215

Infections per person per year 0.81 0.96 0.84 (0.69 to 1.02) .08

Colds

Infections, No. 155 186

Infections per person per year 0.66 0.83 0.80 (0.64 to 0.98) .04

-------------------------------------

Antibiotic prescriptions for all respiratory tract infections

Prescriptions, No. 153 125

Prescriptions per person per year 0.655 0.561 1.17 (0.92 to 1.47) .20

------------------------------------

-----------------------------------Risk Ratio

Participants with =/>1 infection, No.

All respiratory tract infections 150 163 0.88 (0.75 to 0.99) .04

Lower respiratory tract infection 76 71 1.02 (0.77 to 1.31) .89

Upper respiratory tract infection 116 136 0.81 (0.66 to 0.96) .01

Colds 106 126 0.80 (0.64 to 0.96) .02

------------------------------------

-----------------------Difference (Vitamin E & #8722;Placebo)

No. of days with infection per person per year

All respiratory tract infections 15.36 17.32 & #8722;1.97 ( & #8722;5.55 to 1.62)

..28

Lower respiratory tract infection 6.69 6.45 0.24 ( & #8722;2.11 to 2.59) .84

Upper respiratory tract infection 8.66 10.87 & #8722;2.21 ( & #8722;4.95 to 0.54)

..12

Colds 7.37 9.42 & #8722;2.05 ( & #8722;4.57 to 0.47) .11

-------------------------------------------

Abbreviation: CI, confidence interval.

*Total days in study: 85 342 vitamin E, 81 436 placebo.

**Bronchitis, pneumonia.

***Common cold, influenzalike infection, pharyngitis, otitis media,

sinusitis.

The highest incidence of respiratory tract infection occurred in

the winter and the lowest in the summer (0.41 and 0.24 episodes per

placebo participant, respectively). For all study participants, the

rate of respiratory tract infection for vitamin E and placebo groups

was 1.35 and 1.47 per person per year, respectively (Table 3), and

for completers, 1.30 and 1.44 respiratory tract infections per person

per year, respectively (Table 4). Rates of respiratory tract

infections and number of days with respiratory tract infections per

person-year (Table 3 and Table 4), although lower in the vitamin E

group, did not differ significantly in either group. However,

significantly fewer persons in the vitamin E group contracted 1 or

more respiratory tract infections (60% [186/311] vs 68% [207/306] for

all participants, 65% [150/231] vs 74% [163/220] for completing

participants in the vitamin E and placebo groups, respectively).

The incidence, proportion, or number of sick days of lower

respiratory tract infection (includes acute bronchitis and pneumonia)

did not differ significantly between the 2 treatment groups (Table 3,

Table 4).

The number of upper respiratory tract infections (URIs) per person-

year and days with URI, although lower in the vitamin E group, were

not significantly different between groups (Table 3 and Table 4).

However, significantly fewer participants in the vitamin E–treated

group contracted 1 or more URIs compared with the placebo group (44%

[137/311] vs 52% [159/306], respectively, for all participants [Table

3]; 50% [116/231] vs 62% [136/220], respectively, for completers

[Table 4]). After adjusting for obstructive lung disease, current

smoking status, diabetes mellitus, dementia, year of enrollment, and

baseline albumin and hemoglobin levels, the RR for having at least 1

URI was 0.82 (95% CI, 0.66-0.98, P = .03) among all persons

randomized to receive vitamin E.

Among the URIs, 84% [397/470] were common colds. Post hoc subgroup

analysis indicated that vitamin E–supplemented participants who

completed the study had a significantly lower incidence of common

colds (Table 4). In addition, significantly in the vitamin E group

acquired at least 1 cold (for all participants: 40% [125/311] vs 48%

[147/306] in the placebo group [Table 3]; for completers: 46%

[106/231] vs 57% [126/220] in the placebo group [Table 4]). After

adjustment for obstructive lung disease, current smoking status,

diabetes mellitus, dementia, year of enrollment, and baseline albumin

and hemoglobin levels, the risk ratio for all persons randomized to

vitamin E having at least 1 cold was 0.81 (95% CI, 0.64-0.98; P

= .03). The vitamin E group had fewer days with common cold per

person-year, but the result did not reach statistical significance

(20% for all randomized in the vitamin E group [difference of 1.59

days compared with 7.82 days in the placebo group; P = .11] and 22%

for completers [difference of 2.05 days compared with 9.42 days in

the placebo group; P = .11; Table 3 and Table 4]).

Post hoc analysis found no significant effect of vitamin E on

other URIs (0.035 vs 0.050 for influenzalike infections, 0.082 vs

0.073 for pharyngitis, 0.013 vs 0.009 for otitis media, and 0.030 vs

0.009 for sinusitis per person-year in the vitamin E and placebo

groups, respectively), although incidence was low and the study was

not powered to detect differences.

Vitamin E had no significant effect on antibiotic use for all

respiratory tract infections (Table 3 and Table 4), number of

emergency department visits (0.086 for vitamin E vs 0.058 for placebo

per person-year; RR, 1.66; 95% CI, 0.80-3.43; P = .17) or

hospitalizations for respiratory tract infection (0.060 for vitamin E

vs 0.067 for placebo per person-year; RR, 0.91; 95% CI, 0.43-1.95; P

= .81).

COMMENT

We found that vitamin E had no statistically demonstrable effect

on the incidence or duration of all respiratory tract infections, as

well as upper and lower (after adjustment for confounding factors).

However, fewer persons in the vitamin E group acquired 1 or more

respiratory tract infections or URIs. Common colds were the most

frequent URIs, and in a post hoc subgroup analysis, participants in

the vitamin E group who completed the study had significantly fewer

common colds and a 20% lower risk of acquiring a cold than those in

the placebo group. Further clinical trials of vitamin E

supplementation in elderly persons, with common cold as the primary

outcome, are warranted.

Although our data suggest that vitamin E may protect against the

common cold, the most frequently encountered form of URI in this

study, vitamin E had no effect on the incidence or duration of other

URIs or of lower respiratory tract infections, which may have been

due to the small number of such episodes or differences in the types

of pathogens responsible. Most URIs, especially the common cold, are

caused by viruses. Animal studies suggest that vitamin E protects

against viral but not bacterial infection in aged mice.42 We have

found that although vitamin E supplementation did not protect old

mice against primary pulmonary Staphylococcus aureus infection, it

was protective against secondary S aureus infection after influenza

infection.43

The respiratory tract infection definitions applied in our study

were derived by using commonly accepted criteria from the medical

literature.13, 32-37 These criteria do not allow the differentiation

of viral from bacterial etiology. Future studies should include

detailed microbiologic methods to determine whether vitamin E has an

effect on respiratory tract infections of viral vs bacterial

etiology.

Vitamin E did not affect antibiotic use. If the effects of vitamin

E were on URIs of viral etiology, this could explain the finding. In

addition, overuse of antimicrobial agents in nursing homes44 may have

impaired our ability to demonstrate an effect of vitamin E on

antibiotic use.

Previous studies of vitamin E and infection in the elderly have

demonstrated mixed results. A retrospective study showed that persons

with plasma vitamin E levels above 1670 µg/dL had significantly fewer

infections compared with those with plasma vitamin E levels below

1200 µg/dL (mean, 1.0 vs 2.3, respectively; 95% CI for difference,

0.12-2.48).45 A recent double-blind trial of Dutch elderly46 persons

living in the community reported no difference for all respiratory

tract infections among those receiving vs not receiving vitamin E

(RR, 1.12; 95% CI, 0.88-1.25). Our population and diagnostic method

differed from those of the Dutch study. In the Dutch study,46

participants self-reported their infections by telephone, and then

the infections were confirmed by nurse visits. Lack of infection was

not confirmed. In our study, the presence and type of respiratory

tract infection, or absence, was documented by infectious disease

specialists according to review of data gathered by trained research

nurses during weekly participant interviews, review of medical

records, and physical examination focused on respiratory tract

infection according to standardized case definitions.13, 32-35 Our

results indicate that vitamin E may reduce URIs, particularly common

colds, with no effect on lower respiratory tract infections or

seasonal allergies. Graat et al46 did not differentiate between types

of infections or between respiratory tract infections and allergies,

and thus might have overlooked any effect of vitamin E on URI.

Furthermore, in our study adherence was checked by nursing home

medication records and by periodic plasma vitamin E measurements,

whereas the study by Graat et al46 measured plasma vitamin E levels

only at baseline.

Several potential limitations of our study merit comment. First,

of the originally planned sample size of 640, 617 were enrolled.

However, this limitation should not influence the reported results

because the change in power to detect statistical significance was

from 80% to 78.5%. Second, 27% (166/617) of the enrolled persons did

not complete the study because of withdrawal or death. This level of

loss to follow-up was anticipated in our original study design. It

demonstrates the challenges inherent in a 1-year study of a frail

nursing home population. Because there were minimal differences in

the characteristics of those who did and did not complete the study,

this loss to follow-up did not have an impact on our overall results.

Results among completers only were more likely to show an effect of

vitamin E because attaining plasma and tissue saturation levels of

vitamin E requires several months.25 However, the analysis of all

patients randomized is the most conservative analysis and showed

fewer significant effects.

Third, the use of a half RDA multivitamin28 capsule for all

participants might have lessened the impact of vitamin E on

respiratory tract infection by improving the micronutrient status of

the placebo group. However, we found no statistically significant

differences between the vitamin E and placebo groups with change over

time in the status of any nutrients other than vitamin E. Although

our vitamin E group had a lower proportion of persons with low

albumin and hemoglobin levels at baseline and follow-up, statistical

adjustment for these potentially confounding factors did not change

our conclusion. A high percentage of participants had low plasma zinc

levels, but the 2 groups did not differ in the fraction of zinc-

deficient participants before or after treatment and thus did not

influence the reported results.

Fourth, the significant reduction in URIs with vitamin E

supplementation was not consistent in all analyses, and the common

cold analysis was post hoc. However, these results suggest that

future randomized trials of vitamin E should concentrate on these end

points. The common cold is generally less severe than influenza.

However, its much higher incidence and its recognized morbidity in

the elderly33 make it an important public health problem in this age

group. This is particularly relevant because no clinically useful

vaccine or antiviral therapy is available to combat colds.

In conclusion, we found no effect of vitamin E supplementation

on the incidence or duration of respiratory tract infections.

However, significantly fewer vitamin E participants acquired 1 or

more respiratory tract infections, which was most evident in URIs.

Post hoc subgroup analysis among individuals completing the study

revealed a significantly lower incidence of common cold and fewer

participants acquiring a cold. Common colds are frequent36 and

associated with increased morbidity33 in this age group, and if

confirmed, these findings suggest important implications for the well-

being of the elderly. Future studies in elderly individuals should

assess the effect of vitamin E supplementation on the common cold and

incorporate microbiologic methods to allow for assessment of the

impact of vitamin E on specific types of respiratory pathogens.

Al Pater.

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