Re: Re: Dietary alpha-linolenic acid ...

[Guest guest]

Thanks, Tony,

They didn't have Udo's oil either.(ha)

For thousands of years, people got along without rancid, over-processed encapsulated fish oils. They are not a necessity for health. Today, people can still get along without these damaged, fishy ‘fish oil pills’.

The other thing is if people are eating a lot of LA, it will block the ALA pathway, so EPA is necessary, not more ALA?

But that aside the ratio of a supplement is not the whole ratio, right? There's the FA's in the veggies, etc. If I look at the LA and ALA in foods, excluding oils, there's hardly an opportunity for a "correct" ratio, whatever that is, without flax or fish. Looks like the ratio we could eat approaches that of walnuts, maybe 3LA:1ALA.

Is there no n-6 in fish?

Regards.

----- Original Message -----

From: citpeks

Sent: Wednesday, July 13, 2005 4:41 PM

Subject: [ ] Re: Dietary alpha-linolenic acid ...

The Udo Erasmus web site has a lot of information about ALA. He is inthe business of selling his oil blend which has an n-3:n-6 ratio of2:1. He recommends one tablespoon (approx. 14g) per 50 lb of bodyweight. http://www.udoerasmus.com/articles/udo/fish_oil.htmThe bulk of the literature generally says that n-3:n-6 ratio of 1:4 isdesirable. This is quite different from Udo's oil blend, but Udo'sformula may try to compensate for the extra n-6 in the diet.Tony> > Hi All,> > > > The more important, I believe, total cholesterol/HDL ratio didnot > significantly> > change,> > > > See the below from the avaialbe pdf.> > > > TABLE 4 Serum lipid, lipoprotein, and apolipoproteinconcentrations > in subjects when> > they consumed the AAD, LA Diet, and ALA Diet for 6 wk1> > ..............................> > ----AAD LA Diet ALA Diet> > ..........................> > TC, mmol/L 5.59±0.16b 4.98±0.13a 4.99±0.14a > > LDL-C, mmol/L 3.74±0.14b 3.28±0.12a 3.33±0.11a > > HDL-C, mmol/L 1.18±0.06b 1.15±0.06ab 1.11±0.05a > > TG, mmol/L 1.47±0.13b 1.20±0.11a 1.20±0.11a > > TC:HDL-C 4.90±0.18b 4.52±0.18a 4.65±0.19a > > Apo AI, g/L 1.51±0.04b 1.45±0.05ab 1.43±0.04a > > Apo B, g/L 1.11±0.03b 1.01±0.03a 1.01±0.03a > > ...............................> > 1 Values are means±SEM, n = 23. Means in a row withsuperscripts > without a common> > letter differ, P < 0.05.

[Guest guest]

Thanks, Rodney,

I buy the 5# shelled at Sam's and keep them in the freezer. We can only hope the USDA is doing the testing. I worry more about the stuff they haven't found to test for yet. Turns out that verapamil inhibits delta 6 desaturase too. I now wonder how many inhibitors are out there?

Regards.

----- Original Message -----

From: Rodney

Sent: Wednesday, July 13, 2005 7:19 PM

Subject: [ ] Re: Dietary alpha-linolenic acid ...

Hi JW:Don't forget that walnuts are a prime target of that nasty Aspergillus flavius which figures that god put it there for the sole purpose of creating aflatoxin. Walnuts are probably third on the list behind corn and peanuts. Perhaps after opening them one needs to wash them?This is presumably another reason for the demonstrated link between ALA sources and prostate cancer.Rodney.> > Looking at > > http://www.benbest.com/health/essfat.html#chem> > about 11 pgdns is a diagram "production of fatty acid derivatives".> > Also pg 85/86 of MNHD, and Calder, Br J Nutr, 1Jan2002, Suppl 1,> S37.> > > > These diagrams show the Linoleic and linolenic pathways to produce> COXs and LOXs. > > The diagrams do not show a metabolite of AA, eicosatetraynoic acid> (ETYA). ETYA serves to inhibit delta 6 desaturase to limit production> of AA AND EPA.> > > > Supplementing GLA or EPA bypasses the ETYA control of delta 6> desaturase.> > So GLA and EPA can influence COXs and LOXs separately.> > > > EPA is known to control AA as well. > > So the question is what oils to eat/supplement?> > There's lot of articles that discuss the role in inflammation -not> a shoddy role since it's connected with a lot of bad stuff - athero,> cancer, arthritis, CVD, PAD and brain. > > > > It's hard for me to think ALA exerts an influence that promotes> prostate cancer, when in fact a lot of soy oil is consumed. The> COXs/LOXs from EPA, I think are necessary. That means ALA or EPA. > > If I think I'm smart enough to use Saff or Sunf oils, then I must be> able to judge how much EPA to supplement. > > I think I use walnuts and forget the oils.> > > > Pg 85 lists from LA: LTB3, LTC3,LTD3,LTA4, LTB4,LTC4,LTD4,LTE4,LTF4> from LOXs ad PGD1, PGF1alpha, PGE1, TXA2, PGE2,PGF2alpha, PGD2, and> PGD2 form COXs. (plus 5-HETE)> > From ALA: LTA5, LTB5, LTC5, LTD5,LTE5, LTF5 from LOXS, and TXA3,> PGE3,PGF3alpha, PGD3, PGI3 from COXs.> > Thas a lotta eicosanoids, right there. > > > > Regards.> > > > ----- Original Message ----- > > From: Rodney > > > > Sent: Sunday, July 10, 2005 9:44 AM> > Subject: [ ] Re: Dietary alpha-linolenic acid reduces> inflammatory and lipid cardiovascular risk factors in> hypercholesterolemic men and women> > > > > > Hi Al:> > > > That is interesting, thank you. > > > > It certainly shows that alpha-linolenic acid (ALA) improves lipid > > values. But, if I am reading it right, is also indicates that > > linoleic acid (LA) is somewhat better than ALA for all measures > > listed except for Apo AI.> > > > Which I find even more interesting, since LA is for certain > > an 'essential' nutrient. While we are unsure whether ALA is > > essential if one is getting EPA and DHA from other sources.> > > > Rodney.> > > > > TABLE 4 Serum lipid, lipoprotein, and apolipoprotein> concentrations > > in subjects when> > > they consumed the AAD, LA Diet, and ALA Diet for 6 wk1> > > Apo AI, g/L 1.51±0.04b 1.45±0.05ab 1.43±0.04a > > > Apo B, g/L 1.11±0.03b 1.01±0.03a 1.01±0.03a > > > ...............................

[Guest guest]

The toxin is not killed by killing the bacteria. Peanuts are tested, before washing, et al processing. If the levels are too high, it's dumped. Even the animal feed corn now is guaranteed human safe.

Regards.

----- Original Message -----

From: Ron Corselli

Sent: Thursday, July 14, 2005 6:08 PM

Subject: [ ] Re: Dietary alpha-linolenic acid ...

Nuts for commercial sale should be subjected to a fumigation process that kills the bacteria and mold. Now you can stay awake at night worrying about residual effects if you are so inclined.