Guest guest Posted June 18, 2008 Report Share Posted June 18, 2008 Related to this topic - are you aware that there is a group of infants who upon initial newborn screening are suspected to have congenital hypothyroidism? Upon retesting most are found to be normal. This is thought to be related to the use of iodine-provodine as a disinfectant during the labor/delivery process and clean up after. Also to prepare for caesarean or epidural. All the iodine goes right to the infant through the breast milk - so this is of greatest concern to infants who are solely breast fed in the first few days - and iodine is also used often for the umbilical cord area. If the infant is retested and found to be normal the parents are not informed. So the infant gets a huge flood of iodine - highly disproportionate to its size. It is not really known what the long term effects are although there are some who say this effects the CNS. Iodine is used in surgery situations as well - so this is something to be aware of. http://www.euthyroid.org/images/survey.htm#The%20potential On top of that - congenital hypothyroidism is on the rise apparently. (I have also read that subclinical hypothyroidism could be associated with fluoride in water, etc). Increase in congenital hypothyroidism in New York State and in the United States Katharine B. , a, and A. Passa aNew York State Department of Health, Wadsworth Center, Laboratory of Molecular Screening Technologies, Empire State Plaza, Albany, NY 12201, USA Received 28 February 2007; revised 21 March 2007; accepted 22 March 2007. Available online 23 May 2007. Abstract Mandated screening of newborns for congenital hypothyroidism (CH) in NYS was initiated in l978. Currently, every newborn screening program in the U.S. includes CH in its panel. Between 1978 and 2005, 7.4 million newborns were screened for CH in NYS. In NYS, between 1978 and 2005, the incidence of CH has increased by 138%. Nationwide (excluding NYS data), with nearly 58 million infants screened between 1987 and 2002, the incidence has increased 73% between 1987 and 2002. These data and possible reasons for the increases are discussed, though no definitive causes are identified. In a message dated 6/18/2008 8:00:40 A.M. Eastern Daylight Time, ladybugsandbees@... writes: There is a problem with this research. These children tested where tested positive for congenital hypothyroidism. That is a completely different animal than normal children w/o this defect. They did not test mothers who took in high amounts of iodine and did not have a child with this defect. You can't draw a conclusion on the toxicity of iodine when there is a pre-disposition to thyroid conditions. Where are the normal children being evaluated? Fw: Re: high anti tpo and high thyroglobulin>>> >> > can any one comment?> > Gracia> >> >> >> >> Transient Hypothyroidism or Persistent Hyperthyrotropinemia in Neonates> >> Born to Mothers with Excessive Iodine Intake> >>> >> Soroku Nishiyama, Tomohiro Mikeda, Toshihisa Okada, Kimitoshi Nakamura,> >> Tomio Kotani, Akira Hishinuma. Thyroid. December 1, 2004, 14(12):> >> 1077-1083.> >>> >> Perinatal exposure to excess iodine can lead to transient > >> hypothyroidism> >> in the newborn. In Japan, large quantities of iodine-rich seaweed such> >> as kombu (Laminaria japonica) are consumed. However, effects of iodine> >> from food consumed during the perinatal period are unknown. The> >> concentration of iodine in serum, urine, and breast milk in addition to> >> thyrotropin (TSH), free thyroxine (FT4), and thyroglobulin was measured> >> in 34 infants who were positive at congenital hypothyroidism screening.> >> Based on the concentration of iodine in the urine, 15 infants were> >> diagnosed with hyperthyrotropinemia caused by the excess ingestion of> >> iodine by their mothers during their pregnancy. According to serum> >> iodine concentrations, these infants were classified into group A (over> >> 17 µg/dL) and group B (under 17 µg/dL) of serum iodine. During their> >> pregnancies these mothers consumed kombu, other seaweeds, and instant> >> kombu soups containing a high level of iodine. It was calculated that> >> the mothers of group A infants ingested approximately 2300-3200 µg of> >> iodine, and the mothers of group B infants approximately 820-1400 µg of> >> iodine per day during their pregnancies. Twelve of 15 infants have> >> required levo-thyroxine (LT4) because hypothyroxinemia or persistent> >> hyperthyrotropinemia was present. In addition, consumption of iodine by> >> the postnatal child and susceptibility to the inhibitory effect of> >> iodine may contribute in part to the persistent hyperthyrotropinemia. > >> We> >> propose that hyperthyrotropinemia related to excessive iodine ingestion> >> by the mother during pregnancy in some cases may not be transient.> >>> >> Complete paper:> >> http://www.liebertonline.com/doi/abs/10.1089%2Fthy.2004.14.1077> >>> >> Chuck> >>> >> ------------------------------------> >>> >> Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 18, 2008 Report Share Posted June 18, 2008 In the study on these children in Japan, there was also no measure of Deiodinase enzymes, which could certainly play a role here. These mom's could be deficient in selenium and sulfur.Gas prices getting you down? Search AOL Autos for fuel-efficient used cars. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 18, 2008 Report Share Posted June 18, 2008 It raises an interesting question, which is why is iodine causing some babies/adults to become hypothyroid, when we would expect the opposite? > > > > It makes me wonder if their seaweed products are filled with toxic halides > > and arsenic like ours are. If that is the case then it would explain the > > hypoT more than high iodine levels. I don't know toxicology issues in > > Japan. Anyone else? > > > > > > > > > > Fw: Re: high anti tpo and high thyroglobulin > > > > > > > > > > can any one comment? > > > Gracia > > > > > >> > > > >> Transient Hypothyroidism or Persistent Hyperthyrotropinemi Transient Hy > > >> Born to Mothers with Excessive Iodine Intake > > >> > > >> Soroku Nishiyama, Tomohiro Mikeda, Toshihisa Okada, Kimitoshi Nakamura, > > >> Tomio Kotani, Akira Hishinuma. Thyroid. December 1, 2004, 14(12): > > >> 1077-1083. > > >> > > >> Perinatal exposure to excess iodine can lead to transient > > >> hypothyroidism > > >> in the newborn. In Japan, large quantities of iodine-rich seaweed such > > >> as kombu (Laminaria japonica) are consumed. However, effects of iodine > > >> from food consumed during the perinatal period are unknown. The > > >> concentration of iodine in serum, urine, and breast milk in addition to > > >> thyrotropin (TSH), free thyroxine (FT4), and thyroglobulin was measured > > >> in 34 infants who were positive at congenital hypothyroidism screening. > > >> Based on the concentration of iodine in the urine, 15 infants were > > >> diagnosed with hyperthyrotropinemi diagnosed with hyperthyrotropinemi > > >> iodine by their mothers during their pregnancy. According to serum > > >> iodine concentrations, these infants were classified into group A (over > > >> 17 µg/dL) and group B (under 17 µg/dL) of serum iodine. During their > > >> pregnancies these mothers consumed kombu, other seaweeds, and instant > > >> kombu soups containing a high level of iodine. It was calculated that > > >> the mothers of group A infants ingested approximately 2300-3200 µg of > > >> iodine, and the mothers of group B infants approximately 820-1400 µg of > > >> iodine per day during their pregnancies. Twelve of 15 infants have > > >> required levo-thyroxine (LT4) because hypothyroxinemia or persistent > > >> hyperthyrotropinemi hyperthyrotropinemi<WBR>a was present. In addition, > > >> the postnatal child and susceptibility to the inhibitory effect of > > >> iodine may contribute in part to the persistent hyperthyrotropinemi io > > >> We > > >> propose that hyperthyrotropinemi propose that hyperthyrotropinemi<WBR>a > rel > > >> by the mother during pregnancy in some cases may not be transient. > > >> > > >> Complete paper: > > >> _http://www.liebertohttp://wwwhttp://www.http://www.http://www.l_ > (http://www.liebertonline.com/doi/abs/10.1089/thy.2004.14.1077) > > >> > > >> Chuck > > >> > > >> ------------ ---- ---- - > > >> > > >> Quote Link to comment Share on other sites More sharing options...
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