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PCI-32765 vs CAL-101?

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I'm interested in a comparison of the effect of each of these drugs, as

understood right now in these early days. My understanding is that CAL-101 has

been around a bit longer and is now in Phase II trials combined with R and

perhaps O or other monoclonals, while PCI-32765 is Phase I or Ib by itself and

perhaps now being, or about to be, tried in combination? I know they are both

kinase inhibitors, but would love to see a discussion of where each seems to

work best (nodes, spleen, marrow, blood).

Also, I think Ramona brings up an interesting point for those of us who are on

the edge of the treatment pit, but haven't toppled in yet. (Sorry for the weird

metaphor.) Dr. Furman, do you think more trials will continue to open for the

untreated elderly for both of these drugs?

I can imagine the drug company can only afford so many trials and then it has to

get into the business of making some money. For Calistoga, clearly the paradigm

was to show remarkable results and get bought. But now the new owner needs to

start to see how to profit and the FDA is somewhere on that path. I don't see

how we go from trial to prescription for these impressive new treatments. Dr.

Furman, do you have a vision here to share?

Lots of questions in this post .. Should I have made three different posts?

Hope all will be revealed, as much as is possible in the ever changing

unpredictable world of CLL.

Lynn

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