*****BSE/CJD/Mad Cow/Sheep Dip- ALSO BEWARE LICE LOTIONS (see article)-Very significant article***

[Guest guest]

HERE is the complete article that Dave sent including URL to excellent

website.

This deserves much attention - read carefully!

Sheri

http://eionews.addr.com/organop.htm

ICI's ex-chemical

weapon

insecticide causes BSE & CJD

Cover-up

Insecticide causes

Mad Cow disease

by Fintan Dunne

Research Kathy Mc Mahon

13th December 2000

Pharmaceutical interests in the UK are ignoring new scientific research

that shows the

insecticide used in the UK government's own warble-fly campaigns triggered

the surge of 'Mad

Cow' disease.

Latest experiments by Cambridge University prion specialist, R.

Brown, have shown that

manganese bonds with prions to cause BSE. Other researchers unpublished

work shows that prions

in the bovine spine --along which insecticides are applied-- can be damaged

by ICI's Phosmet

organophosphate(OP) insecticide -causing the disease.

British scientists have led the current theory that an infectious prion in

bonemeal fed to

cattle causes bovine spongiform disease (BSE). Infectious prions are

also claimed to

cause new variant Creutzfeld-Jakob Disease (CJD) in humans -from ingesting

beef. But the

infectious prion theory serves to obscure a tragic chemical poisoning

scandal behind the

majority of BSE cases.

The new work proves that the prions can bond with manganese in animal feeds

or mineral licks.

These manganese prions cause the eurological degeneration seen in BSE. By a

similar process,

prions in human brains are damaged by lice lotions containing

organophosphate. This can result

in neurological diseases like CJD and Alzheimers -later in life.

Many might be surprised to hear that organophosphates were developed by

Nazi chemists during

the course World War Two, as a chemical weapon nerve agent. One formulation

of the insecticide

--Maneb, or Mancozeb-- actually contains manganese in addition to

organophosphate.

The marginalised research has devestating financial implications for ICI.

It would provide a

firm basis for litigants -who could include CJD sufferers, farmers across

the world, and the

spouses and families of the many British farmers who committed suicide

during this BSE

debacle.

Phosmet organophosphate has been used at high doses in British warble fly

campaigns.

In 1996, ICI subsidiary Zeneca sold the phosmet patent to a PO Box company

in Arizona called

Gowan -just one week before the UK government admitted to a link between

BSE and nvCJD.

The politically well-connected British pharmaceuticals group, ICI has the

financial and

political clout to block research into any cause other than the infective

model. Indeed no

substantive alternative research has been done. British BSE disease

management and research

bodies have taken decisions that do not seem guided by spirited scientific

enquiry. Mysterious

prions that jump species is the preferred research arena.

Scientist and organic farmer, Mark Purdey gave evidence to the UK BSE

inquiry, that

warble fly insecticide was the cause of the disease. The scientist wheeled

out to rubbish

Purdy's evidence -Dr. Ray, later turned out to have been receiving

funding from the

insecticide manufacturer ICI.

A lobby group that includes Bayer, Monsanto, Novartis, Pfizer, Roche and

Schering-Plough was

behind the effort to discredit Purdey. In December 1999, the same Ray

was appointed to

the UK Veterinary Products Committee (VPC) -a government body that licences

animal medicines.

Purdey has been consistently denied even exploratory funding to extend his

privately supported

research. Yet, the Purdey/Brown chemical poisoning model matches with the

epidermiological

spread of CJD clusters in humans. It also predicts the incidence of

BSE-type diseases in

animals. The accepted infectious model does not fit either.

The pharmaceutical industry is all the more determined to hide the chemical

source of BSE and

CJD, because a spotlight on chemicals would expose the role the

insecticides in Alzheimer's -

another neurodegenerative disease. That might lead to claims which would

dwarf those from BSE

and CJD litigants. In fact, two leading brain researchers into CJD and

Alzheimers have died in

suspicious circumstances in recent years.

In the United States, the Environmental Protection Agency is already

reviewing Phosmet's

safety. The Centers for Disease control in the US have recently conducted

experiments on mice

that confirm the organophosphate risk.

Not only is the EC beef slaughter campaign futile, because the disease is

mostly non-

infectious, but unless the underlying chemical cause is addressed, BSE will

simply reappear

from chemical causes. A new warble fly campaign is already underway in

France using the same

organophosphate insecticide.

Of greater concern is that some lotions for scabies and head lice are now

priming children and

adults, for CJD and Alzheimers in later life.

BONDING THE PRION

Cambridge University prion biochemist, R.

Brown is dismissive of the science behind the infectious model of BSE. He

terms it “a very limited amount of science by a few assumed- reputable

scientists.” He insists there is “no evidence an infectious agent is

present in either meat or milk.”

“Simple tests on udder walls of cows —which

could easily detect an infectious prion— have not been done, why I don’t

understand.”

A number of researchers have found that

organophosphate(OP) in systemic warble fly insecticide can deform the prion

molecule, rendering it ineffective at buffering free radical effects in the

body. Worse still, the prion is then partial to bond with manganese and

become a ‘rogue’ prion. A chain reaction whereby rogue prions turn others

to rogues also, can explain the bovine spongiform disease mechanism.

Brown showed how prion protein bonds benignly

with copper, but lethally with manganese. Even natural variations in

relative environmental availability of manganese versus copper can trigger

prion degradation.

The CJD and BSE symptoms mirror ‘manganese

madness’, an irreversible fatal neuro-psychiatric degenerative syndrome

that plagued manganese miners in the first half of the last century

SHINING A LIGHT ON SPONGIFORM

Organic dairy farmer and peer-review-published

independent scientist, Mark Purdey, says the accepted theory of

transmission from BSE-infected cattle to human CJD -by bonemeal or meat, is

dependent on a mutant prion that has never been isolated under the

scientific protocol called Koch’s postulates.

Purdey’s insistence on sticking to the letter of

this scientific law earned him the condemnation of UK officialdom when he

first mooted his theory. But Purdey pointed to CJD clusters downwind of a

British Phosmet production plant to back his case.

He gave evidence to the UK Government BSE

inquiry and was supported by Conservative MP, Thessa Gorman. His views were

discounted, but his subsequent research and the new Cambridge prion work

have confirmed the alternative theory. Despite this, and the backing of a

British peer, he is denied even exploratory funding.

Speaking from his rural English

Somerset farm yesterday -as plans forge ahead for the European cattle cull,

he asks: “Why does CJD degeneration in humans begin in the retina, and why

are CJD disease clusters found in high altitude locations?”

The question is rhetorical, and Purdey has an

eye-opening answer. He argues that the prion molecule has a known natural

role as a shock adsorber of damaging energy from ultraviolet rays and other

oxidizing agents.

Once this prion defence system is rendered

ineffective by organophosphates - for example in human head lice lotions,

these oxidizing effects have an unmediated impact on tissues. Eventually,

UV radiation damages the retina and oxidative stress destroys the brain

tissues of CJD patients. This theory would expect to find higher CJD

incidence in mountain regions -where UV radiation levels are elevated. That

prediction holds true.

A similar but accelerated mechanism could be

driving BSE. ICI’s Phosmet organophosphate warble fly insecticide -applied

on the backs of animals along the spinal column, similarly degrades prions.

“Systemic versions of the insecticide are designed to make the entire cow

carcass toxic to warble fly,” explains Purdey. “Unfortunately it’s toxic to

prions too -especially those prions located just millimeters from the point

of application.”

The damaged prions are then ready to react with

manganese in animal feed, or manganese sprayed on land or in mineral licks

-to become the driving force of BSE neurodegeneration. Purdey says

manganese-tipped prions set off lethal chain reactions that neurologically

burn through the animal.

Chickens notoriously excrete most of the

supplements fed to them -including manganese. And their manganese-rich

excreta have been blended into cattle feed in the UK. Natural variations in

the relative environmental availability of copper and manganese can also

spur prion degeneration says Purdey.

From this research, any prudent person would

conclude there is a significant risk attaching to the use of

organophosphate in humans. Preparations for head lice and scabies are known

to be overused in practice and might be priming users for CJ disease.

Purdey believes his bias for field work is the

key to his success. He bemoans the “reductionism” of much lab-centered

science. “I have traveled the world to investigate known clusters of

spongiform disease -something mainstream researchers don’t seem remotely

interested in doing.”

Since first postulating an environmental -rather

than infectious- theory of spongiform diseases, Purdey has built evidence

from around the world that explains and predicts the incidence in humans

and animals: a cluster of CJD in Slovakia, Eastern Europe -around a

manganese plant; Rocky Mountain deer with Chronic Wasting Disease (CWD),

who were found to be eating pine needles rich in manganese; the futile

slaughter of sheep in Cyprus -only for BSE to reemerge within years.

“The reappearance of BSE in Cyprus obviously

points to an environmental cause,” says Purdey, who is sanguine when

reflecting on the condemnation of him by mainstream scientists.

“I suppose they have mortgages and kids who need

to go to university,” he muses. “Privately, some were agreeing with me, but

then they would denounce me publicly. It was quite strange really.”

THE MONEY TRAIL

Critical scientists like Purdey are

unlikely to prevail. The pharma industry holds most research purse strings,

and would hardly energetically explore an avenue of research that could

expose them to litigation for causing BSE. The official theory is lavishly

funded, alternative theories rarely, if at all.

There are more explosive implications to his -and

other’s latest research. Purdey says similar organophosphate-induced

protein deformation could also underlie Alzheimer’s disease. If that were

true, the litigation fallout would destroy some pharmaceutical giants, and

a lot of very influential noses would be out of joint.

Disturbingly, Purdey and other brain researchers

seem to have had an undue share of unfortunate accidents. Purdey’s house

was burned down and his lawyer who was working with him on Mad Cow Disease

was driven off the road by another vehicle and subsequently died. The

veterinarian on the case also died in a car crash -locally reported as:

‘Mystery Vet Death Riddle.’

Dr. C. Bruton, a CJD specialist —who had just

produced a paper on a new strain of CJD— was killed in a car crash before

his work was announced to the public. Purdey speculates that Bruton might

have known more than what was revealed in his last scientific paper.

In 1996, leading Alzheimer’s researcher Tsunao

Saitoh, 46 and his 13 -year-old daughter were killed in La Jolla,

California, in what a Reuters report described as a “very professionally

done” shooting.

What Alzheimer’s Disease, Mad Cow Disease, and

CJ Disease have in common, is abnormal brain proteins and a putative link

to organophosphates. Even Gulf War syndrome among returning veterans has

been attributed, in part to the insecticide. But the sidelined scientists’

suspicions are still largely ignored.

In their favour at the moment, is a growing

unease on the part of the public. As BSE forges on and Governments panic,

Science may be out to lunch on BSE, compromised by bovine spongythinking

myopathy.

Discuss or plan your response to this

issue............HERE

Mark Purdey funds his own research, testing/

labs/travel to cluster sites. Donations to his research fund will help him

carry on his work. Mark Purdey Research Fund, High Barn Farm, Elworthy, Nr

Taunton, Somerset TA4 3PX, UK.

Copyright © 2000 eionews.com

--------------------------------------------------------

Sheri Nakken, R.N., MA

Vaccination Information & Choice Network, Nevada City CA

530-272-7306

http://www.nccn.net/~wwithin/vaccine.htm

" All that is necessary for the triumph of evil is that good men ( &

women) do nothing " ...Edmund Burke

ANY INFO OBTAINED HERE NOT TO BE CONSTRUED AS MEDICAL OR LEGAL ADVICE. THE

DECISION TO VACCINATE IS YOURS AND YOURS ALONE.

Well Within's Earth Mysteries & Sacred Site Tours

http://www.nccn.net/~wwithin

International Tours, Homestudy Courses, ANTHRAX & OTHER Vaccine Dangers

Education, Homeopathic Education

KVMR Broadcaster/Programmer/Investigative Reporter, Nevada City CA

CEU's for nurses, Books & Multi-Pure Water Filters