Re: Overdose

November 16, 2000

nathan does combos but his doc only increases one at a time to see which one

is or isnt working. shawna.

November 12, 2007

One day.

Val

[ ] Overdose

Can anyone please tell me how long a bad reaction lasts when you've taken too much MMS?

My husband did 15 drops this morning and cannot get out of bad now. Bad cramps and pain. He had taken 15 drops this weekend fine, but he must have had food in his stomach and not realized it.

I would appreciate any advice and responses on this.

Thank you,

November 12, 2007

Did you give him Vitamin C to counteract it? That would make the time shorter. I took 8 drops once and had a stomach ache and cramps most of night - I didnt use vitamin Cthough which might make it better faster. Valery Galperin <lordnval@...> wrote: One day. Val [ ] Overdose Can anyone please tell me how long a bad reaction lasts when you've taken too much MMS? My husband did 15 drops this morning and cannot get out of bad now. Bad cramps and pain. He had taken 15 drops this weekend fine, but he must have had food in his stomach and not realized it. I would appreciate any advice and responses on this. Thank you, Wilkins

http//:www.juiceplus.com/+lw55887

The Children's Research Foundation Children 6-15 FREE for 3 years __________________________________________________

November 12, 2007

I have Vit. C, but when once I got cramps, I decided to see what will be and did not take Vit. C.

It is always good to begin to take MMS very slow. I take certain number of the drops, starting from ONE, and repeating that number 3 days on a row.

Nothing serious if take to much but why to hurry?

Val

[ ] Overdose

Can anyone please tell me how long a bad reaction lasts when you've taken too much MMS?

My husband did 15 drops this morning and cannot get out of bad now. Bad cramps and pain. He had taken 15 drops this weekend fine, but he must have had food in his stomach and not realized it.

I would appreciate any advice and responses on this.

Thank you,

Wilkins

http//:www.juiceplus.com/+lw55887

The Children's Research Foundation Children 6-15 FREE for 3 years

__________________________________________________

November 12, 2007

Thank you for your responses. He had been going very slowly. He just decided that after he got to 12 drops he would go for the 15 on the weekend and if he got sick he would be home to sleep it off. But he was fine yesterday. He has babesia which is very similar ro malaria so we thought he had already worked his way up slowly he would go for the 15 to try to knock the remainder out him.

He took vit. C to try to stop it but I think it was too late.

Thanks for the input.

-- Re: [ ] Overdose

I have Vit. C, but when once I got cramps, I decided to see what will be and did not take Vit. C.

It is always good to begin to take MMS very slow. I take certain number of the drops, starting from ONE, and repeating that number 3 days on a row.

Nothing serious if take to much but why to hurry?

Val

[ ] Overdose

Can anyone please tell me how long a bad reaction lasts when you've taken too much MMS?

My husband did 15 drops this morning and cannot get out of bad now. Bad cramps and pain. He had taken 15 drops this weekend fine, but he must have had food in his stomach and not realized it.

I would appreciate any advice and responses on this.

Thank you,

Wilkins

http//:www.juiceplus.com/+lw55887

The Children's Research Foundation Children 6-15 FREE for 3 years

__________________________________________________

November 12, 2007

My experience is 3 days of liquid bowel movements.

Chuck

You might be a redneck computer junkie if...

Ya think www. in a url is a logo for a wrasslin' organization.

Yer mouse keeps knocking over yer spit can.

On 11/12/2007 1:04:54 PM, susan (ssiegel5@...) wrote:

> Can anyone please tell me how long a bad reaction lasts when you've taken too

much MMS?

>

> My husband did 15 drops this morning and cannot get out of bad now. Bad cramps

and pain. He had taken 15 drops this weekend fine, but he must have had food in

his stomach and not realized it.

>

> I would appreciate any advice and responses on this.

>

> Thank you,

>

> [image " Free " ] [link: www.incredimail.com/index.asp?id=101218] [image]

>

September 14, 2010

Hi there!

Just wondering is there a limit on the dosage of Omega 369 and EPA?

I read that the ratio is 2:1 - 2 Caps ProEFA (Omega 369) abd 1 cap for EPA.

DS is 60 Ibs (8.6years old). Can I dose morning and evening... Means 4 caps

Omega 369 and 2 caps for EPA?? What is the max dose for his age and weight

if there was one?

Also, I got the " EPA extra " instead of EPA. Is that OK to just go ahead?

Thanks a lot!

Sara

September 15, 2010

I don't know the difference between the " extra " and regular EPA, but general

speaking one should not go over 3 grams of fish oils per day without doctor's

approval since amounts over 3 grams can cause bleeding issues in some

individuals.

From: lokesp@...

Date: Wed, 15 Sep 2010 11:58:16 +0800

Subject: Re: [ ] Overdose

Hi there!

Just wondering is there a limit on the dosage of Omega 369 and EPA?

I read that the ratio is 2:1 - 2 Caps ProEFA (Omega 369) abd 1 cap for EPA.

DS is 60 Ibs (8.6years old). Can I dose morning and evening... Means 4 caps

Omega 369 and 2 caps for EPA?? What is the max dose for his age and weight

if there was one?

Also, I got the " EPA extra " instead of EPA. Is that OK to just go ahead?

Thanks a lot!

Sara

September 15, 2010

The EPA extra is too high and raises the EPA/O3 too much over the O6 for the

majority. It's more expensive anyway- no need for the added expense. As many

in this group learn the hard way -paying more in money, time, doesn't always

mean better or more effective therapy. Just use the regular ProEPA for the

child and you or your spouse can take the extra.

Now here's more about dosage -and if anyone doubts anything I have here again I

just posted another message and as I have to keep archiving the same info over

and over and over -even I get bored sometimes and wanted to share new info...but

be assured I have way more information to support that yes you can use more than

3 grams a fish oils a day -even with toddlers -this group has for years. As I

say below I'm not saying don't share with your child's doctor -but we are an

intelligent group -let's stop perpetuating myths!

I know this is one of the myths from reputable sources too and shame on them

-but the major issue isn't the fish oils -it is for those who are on medications

with or without fish oils. Man made in nutrition doesn't work nearly as well as

Mother Nature made!!!

It probably took forever back then to get people to stop parroting that the

Earth is flat! At one point all have to look at proof and separate fact from

fiction -those using fish oils alone vs those using fish oils and blood

thinners!!

Also I just have one study below which was with 9 grams a day -most studies I

have seen with fish oils are 6 grams a day or higher even in children. And

what about this one for ADHD with 16 grams a day which many experts say is

typical for Eskimos. " but the successful studies have used rather large doses

(up to 16 g fish oil per day). " Note they don't say the ADHD people that had

success had bleeding issues -one would think that was a slight side effect of

the therapy that WOULD have been mentioned ...don't you think?!!!

Attention deficit hyperactivity disorder (ADHD) - an omega-3 deficiency?

GUELPH, ONTARIO, CANADA. The development of an assay for EPA and DHA in red

blood cell fatty acids has made possible a number of interesting studies from

sudden cardiac death to ADHD. In a just published study from the University of

Guelph in Ontario, researchers examined the association between both dietary

intake and red blood cell fatty acid status in a group of adolescents diagnosed

with ADHD as compared to an age-matched control group. Both groups had similar

anthropometric measurements such as weight, height, % fat mass, etc. Both groups

consumed equivalent amounts of omega-3 and omega-6 fatty acids but the ADHD

group consumed more energy and fat even though they had similar anthropometry.

The ADHD children had significantly lower red blood cell levels of DHA and total

omega-3 fatty acids, higher omega-6 fatty acids and a lower omega-3 to omega-6

ratio. In addition, this lower omega-3 status correlated with scores obtained

with a standard behaviour measurement scale (the Conners' Parent Rating Scale or

CPRS). The authors point out that these abnormal fatty acid profiles are also

observed in younger ADHD children and again are distinctly different from

controls of a similar age. Given that the dietary intakes in this study were

similar, the results suggest that there are metabolic differences in fatty acid

handling between ADHD adolescents and normal controls. Finally, they provide

evidence from other studies that it may be possible to improve behaviour

patterns with omega-3 supplements, but the successful studies have used rather

large doses (up to 16 g fish oil per day).

Colter, AL, et al. Fatty acid status and behavioural symptoms of Attention

Deficit Hyperactivity Disorder in adolescents: A case-control study. Nutrition

Journal, Vol. 7, No. 1, February 14, 2008, p. 8 www.nutritionj.com/content/7/1/8

Here are some stats:

One capsule of ProEFA is 1000 mg which is one gram of fish oils. We have had

children on well over three grams of fish oils for years, and I have numerous

times shared data that there are no cases of hemorrhage in even cardiac patients

on mega dosages of PUFAs even when undergoing emergency open heart surgery.

Fish oils are blood thinners and one should be careful if combining them with

man made blood thinners (drugs -including aspirin) but again nature works

different. It's again just like those that go a biomedical route and add

supplements vs those that get those same nutrients from whole food sources. The

chance of side effects and issues goes up the further one goes from whole food

sources of supporting the internal pharmacy.

Because I have just recently even shared information that there is not yet ONE

documented case of irregular bleeding issues in our group of parents who in some

cases using up to 9 grams of fish oils a day for years (my one son BTW is on 10

grams a day) I want to share the following article- please note the dosage of

fish oils which may in research sometimes go by PUFA (polyunsaturated fatty

acids) or EFA (essential fatty acid) or O3 O6 etc.

Don't get me wrong -always check with your child's pediatrician -but as educated

individuals -we should try not to just parrot and perpetuate myths which may

prevent a dosage that is therapeutic for an individual. SO the reason we need

research to get rid of the fear of what is healthy.

Dietary Fatty Acids Essential for Mental Health

By J. Bender, Pharm.D., M.A. | December 1, 1998

Insufficient intake of essential fatty acids (EFAs) may contribute to the

pathogenesis of mental diseases, while their supplementation may relieve some

symptoms, according to researchers who attended the National Institutes of

Health (NIH) Workshop on Omega-3 Essential Fatty Acids and Psychiatric Disorders

held in Bethesda, Md., in September 1998.

*

" This is the first time researchers in this field have been brought together, "

said ph Hibbeln, M.D., National Institute on Alcohol Abuse and Alcoholism

(NIAAA), who organized the workshop. Other organizers were Jerry Cott, Ph.D.,

National Institute of Mental Health (NIMH), Sahebarao Mahadik, Ph.D., Medical

College of Georgia, Augusta, and the NIH Offices of Dietary Supplements and

Research on Women's Health.

Despite having long established that omega-3 fatty acids are highly concentrated

in neurosynaptic membranes, Hibbeln said that researchers have only recently

considered the physiologic effects of these brain-specific nutrients. These

include the pathologic effects of their deficiency on neural and mental

functioning.

The EFAs, essential components of the diet that are not synthesized in the body,

are ubiquitous in immune and anti-inflammatory response mechanisms and in

oxidative cell injury defense. It is in these roles, Cott posited, that EFA

deficiency might contribute to the well-known association of depression with

coronary artery disease. Depression can follow from the emotional stress of

myocardial injury and can exacerbate cardiac illness (Frasure- et al.,

1995). However, Cott suggested that lowered EFA levels could predispose an

individual to both the myocardial injury and the depression.

" Epidemiological studies in various countries and in the United States in the

last century suggest that decreased omega-3 fatty acid consumption correlates

with increasing rates of depression, " Cott wrote in the initial proposal of the

workshop. " There is evidence that deficiency of long-chain, omega-3

polyunsaturate may contribute to symptoms of schizophrenia, alcoholism, multiple

sclerosis and postpartum depression. Adequate long-chain polyunsaturated fatty

acid [LC-PUFA], particularly docosahexaenoic acid [DHA], may reduce the

development of depression and schizophrenia just as omega-3 polyunsaturated

fatty acids may reduce coronary artery disease. "

Mahadik has examined the possibility of oxidative cell injury underlying the

pathology of first-episode psychosis; noting such injury involves " predominately

and selectively " the neuronal membrane phospholipids that are highly enriched in

EFAs, particularly arachidonic acid [AA] and DHA, which are most sensitive to

free radical-mediated peroxidation.

" Since AA and DHA are critical for brain and behavioral development and for

maintenance and neurotransmitter function throughout life, " Mahadik reasoned,

" their levels may play critical roles in etiogenesis as well as pathogenesis of

several neurological and major mental disorders, particularly bipolar disorder

and schizophrenia, both of which seem to involve abnormal neurodevelopment. "

Maes, M.D., Ph.D., of the Clinical Research Centre for Mental

Healthcare, Antwerp, Belgium, was a presenter at this workshop. He and

, M.D., described in a Biological Psychiatry editorial how EFA deficiency

could lead to depression through effects on immune function and production of

cytokines (Maes and , 1998).

Maes and point to evidence that depression is accompanied by, and possibly

follows from, an " acute phase " response in which there is an increased secretion

of eicosanoids such as prostaglandins, and of proinflammatory cytokines.

" Inflammatory cytokines or lipopolysaccharide given to animals or humans provoke

an extensive set of symptoms and signs similar to, if not identical to, those

found in major depression, " wrote Maes and . They added, " Cytokines may be

common mediators of external stressors [that is, psychosocial stressors such as

adverse life events] and internal stressors [that is, organic disorders] that

are known to play a role in the etiology of depression. "

One of the indicators of cytokine involvement in the pathogenesis of depression

offered by Maes and is the suppression of cytokine release from human

blood monocytes by antidepressants. This suggests, they wrote, " that these drugs

have potent anti-inflammatory activities. "

A more fundamental method of countering the proinflammatory cytokine effects

proposed by Maes and others at this workshop, however, is to increase the

omega-3 PUFA from sources such as fish oil in relation to the consumption of

omega-6 PUFA from vegetable oils (Table). " The increased incidence rate of major

depression since 1913 may be explained by a sharp increase in the rate of

omega-6/omega-3 PUFAs in the diet, " wrote Maes and .

has previously suggested that the increased omega-6/omega-3 PUFA ratio of

the Western diet, replacing omega-3 fatty acids from fish, wild game and leaves

with omega-6 fatty acids from seeds, has contributed to the increased incidence

of cardiovascular and inflammatory disorders (, 1991).

Clinical Effects of Diet

Hibbeln reported investigating a possible correlation of EFA status with

occurrence of suicide in 50 patients hospitalized for suicide attempts. Thirty

of the patients had a primary diagnosis of depression, 20 had another primary

diagnosis. Hibbeln found that for those not having a primary diagnosis of

depression, a higher plasma concentration of eicosapentaenoic acid (EPA)

predicted " strikingly " lower scores, indicating less suicidal risk across six

different psychological rating scales. No similar correlation was found with

other fatty acids, including DHA. Although this was not a prospective study of

dietary intervention, Hibbeln concluded, " These data do suggest that some

subgroups of suicidal patients may reduce their suicidal risk with the

consumption of EPA. "

In a previous investigation, Hibbeln and Norman Salem Jr., Ph.D., acting

scientific director of NIAAA, had implicated the depletion of DHA as a probable

etiologic factor for major depression (Hibbeln and Salem, 1995). Their study was

the basis for Rhian , Ph.D. candidate, department of psychiatry,

University of Sheffield, England, comparing measurements of red blood cell

membrane (RBCM) fatty acid levels in 10 depressed patients and 14 matched

healthy controls. reported finding that the patients with depression had

significantly lower RBCM levels of both DHA and EPA. This correlated, he

indicated, with a lower mean dietary intake of DHA and, generally, of other

omega-3 EFAs and a higher omega-6 EFA intake than those patients without

depression.

acknowledged that the insidious onset of depression and the limitations

in assessing diet make it difficult to establish a causal relationship. He

concluded, however, " the role of diet and DHA/EPA supplementation in major

depression warrants further investigation, as the findings raise the possibility

that depressive symptoms may be alleviated by omega-3 EFA supplements. "

Yao, Ph.D., Veterans Administration Medical Center, Pittsburgh,

described recent evidence that altered membrane structure and function of red

blood cells and platelets are " unequivocally " present in patients with

schizophrenia. Specifically, according to Yao, there are decreased amounts of

linoleic acid and AA in the RBCM of patients with schizophrenia during treatment

with antipsychotic medication and in drug-free periods.

Malcolm Peet, M.D., University of Sheffield, added that epidemiologic evidence

suggests a correlation between national outcome figures for schizophrenia and

the proportion of polyunsaturated to saturated fat in the national diet. Peet

previously correlated dietary omega-3 fatty acid levels with the severity of

schizophrenic symptoms, and now related two new studies of omega-3 EFA

supplementation as an adjunct to antipsychotic medication. The dietary

supplementation, Peet said, " led to significant clinical improvement in

treatment-resistant patients. " In one of the two double-blind studies, Peet

recounted, EPA-but not DHA-was found to be a useful adjunct to antipsychotic

medication.

Stoll, M.D., department of psychiatry, Harvard University, reported

finding high-dose omega-3 fatty acid supplementation to be helpful in treating

patients with unstable bipolar disorder. Stoll previously investigated the

utility of choline to attenuate cell signaling through the phosphatidylcholine

system (Stoll et al., 1996), which he hypothesized can become heightened in

lithium-resistant patients after lithium inhibits the phosphatidylinositol

second messenger system (Berridge et al., 1982).

With evidence that omega-3 fatty acids can also inhibit hydrolysis of membrane

phospholipids such as phosphatidylinositol, and may thus inhibit receptor-linked

G-protein signal transduction, Stoll's group undertook a four-month controlled

trial comparing high-dose omega-3 fatty acids (9 gms daily) with olive oil

placebo in 30 patients with bipolar disorder.

Stoll reported that in contrast to the recurrence of symptoms in seven of 15

patients (46.7%) receiving placebo, only one of the 15 patients (6.7%) receiving

the omega-3 fatty acids supplementation had breakthrough mood symptoms. His data

analysis indicated, further, that the patients receiving the omega-3 fatty acids

had a significantly longer period of remission than those receiving placebo;

and, by the end of the study period, demonstrated greater improvement on both

the Clinical Global Impression and the Hamilton Depression Scales scores.

" Omega-3 fatty acids represent a new class of membrane-active agents capable of

altering signal transduction pathways, " Stoll concluded. " This study provides

additional evidence of aberrant postsynaptic signal transduction mechanisms in

the pathophysiology of bipolar disorder, and may herald the advent of a new

class of rationally designed mood-stabilizing drugs. "

Stoll's assessment was supported by evidence presented by Burton J. Litman,

Ph.D., NIAAA, in the workshop section examining EFAs and cellular signal

transduction. Litman described DHA-containing phospholipids potentiating

receptor activation through G-protein signaling pathways. " Many neurotransmitter

receptors implicated in the pathophysiology of affective disorders are members

of the superfamily of G-protein-coupled receptors, " he explained; and so

modulation of G-protein mediated signal transduction may underlie mechanisms of

antidepressants as well as mood stabilizers.

Additional EFA Investigations

Other presentations at the workshop included investigations of effects of EFA

supplementation on attention and cognition, and assessments of the importance of

EFAs in early development. Anton Llorente, Ph.D., of Baylor University,

administered 200 mg DHA or placebo daily to 140 women in late-stage pregnancy to

ascertain the effect on postpartum depression. This population did not present

sufficient depression to distinguish severity of symptoms between groups.

However, Llorente did demonstrate with the Stroop Interference Test, a measure

of alterations in formation processing, that the supplement improved cognitive

capacity in postpartum women without depression.

Two separate evaluations of EFA intervention for attention-deficit/hyperactivity

disorder (ADHD) failed to show therapeutic effect for this condition. Jay

Burgess, Ph.D., of Purdue University, compared LC-PUFA supplementation to an

olive oil placebo in 50 children with ADHD. He found an indication of benefit on

some behavioral assessments, but not across all measures. Voigt, M.D., of

the Mayo Clinic, provided DHA supplements (345 mg daily) or placebo to 63

children with ADHD who had been successfully treated with stimulant medication.

After four months of supplementation, the stimulant dosage was halved. Although

Voigt found the DHA to be no more useful than placebo in sustaining clinical

effect with reduced dosage of stimulants, his group is now assessing whether

there was benefit demonstrated in any subpopulation.

Martha Neuringer, Ph.D., from the Oregon Health Sciences University, in

Portland, Ore., and the Oregon Regional Primate Research Center, reported that

in rhesus monkeys as well as human infants, diets low in omega-3 EFAs result in

lowered DHA levels in the cerebral cortex and retina. Although it has been

difficult to assess whether the lower omega-3 EFA status correlates with

cognitive development, Neuringer indicated that it has been associated with

changes in visual development, including abnormalities in retinal function,

slower development of visual acuity and a longer visual fixation that relates to

slower visual processing.

Neuringer said that the effects of early omega-3 EFA status on other behavioral

domains such as sleep patterns, temperament, emotional reactivity and response

to stress remain largely unexplored. " There is a need for more comprehensive and

long-term studies in both humans and animal models to understand the influence

of dietary fatty acids on development as well as on later behavior, " she

concluded.

A note of caution was expressed by Nakamura, Ph.D., acting deputy

director, NIMH, who presented an introduction to the workshop and later

discussed the findings with Psychiatric Times. Nakamura said that while NIMH is

interested in new ideas about causes and treatments of mental disorders, " the

information being provided at this conference could not be interpreted as more

than suggestive. "

Nakamura indicated that NIMH is receptive to applications for further research

into the role of EFAs in psychiatric disorders, but is not now setting aside

funding specifically for this area. He emphasized that with only preliminary

studies, psychiatric patients should be cautioned against interrupting treatment

with proven medications; and, if they choose to take EFA supplements, should be

advised that the quality of purity in these dietary supplements is not regulated

in the same manner as medication products.

References

1. Berridge MJ, Downes CP, Hanley MR (1982), Lithium amplifies agonist-dependent

phosphatidylinositol responses in brain and salivary glands. Biochem J

206(3):587-595.

2. Frasure- N, Lesperance F, Talajic M (1995), Depression and 18-month

prognosis after myocardial infarction. Circulation 91(4):999-1005.

3. Hibbeln JR, Salem N Jr (1995), Dietary polyunsaturated fats and depression:

when cholesterol does not satisfy. Am J Clin Nutr 61(1):1-9.

4. Maes M, RS (1998), Fatty acids, cytokines and major depression. Biol

Psychiatry 43(5):313-314.

5. RS (1991), The macrophage theory of depression. (Erratum appears in Med

Hypotheses 36[2]:178.) Med Hypotheses 35(4):298-306.

6. Stoll AL, Sachs GS, Cohen BM et al. (1996), Choline in the treatment of

rapid-cycling bipolar

7. disorder: clinical and neurochemical findings in lithium-treated patients.

Biol Psychiatry 40(5):382-388.

http://www.psychiatrictimes.com/display/article/10168/49501

And in conclusion -from the ADHD study referenced above

" but the successful studies have used rather large doses (up to 16 g fish oil

per day). "

PS -that would be about 16 capsules of fish oils a day.

=====

September 15, 2010

I appreciate that others have been on higher than 3 gram doses, but all the

studies are under the direction of doctors. I was referring to the legal

quanitifier that is put on fish oil and listed on the many sites, including the

Mayo Clinic, that it is recommended going over 3 grams be done under the

direction of your doctor as there have been instances of bleeding issues, not

necessarily hemorraging. I didn't indicate 3 grams was a stopping point, just

to go forward under doctor supervision. Futhermore, when major medical colleges

and associations list it as follows, it can hardly be considered a myth.

From the University of land Medical:

Children (18 years and younger)

There is no established dose for children. Omega-3 fatty acids are used in some

infant formulas. Fish oil capsules should not be used in children except under

the direction of a health care provider. Children should avoid eating fish that

may be high in mercury, such as shark, swordfish, king mackerel, and tilefish.

(See Precautions section.)

Adults

Do not take more than 3 grams daily of omega-3 fatty acids from capsules without

the supervision of a health care provider, due to an increased risk of bleeding.

For healthy adults with no history of heart disease: The American Heart

Association recommends eating fish at least 2 times per week.

For adults with coronary heart disease: The American Heart Association

recommends an omega-3 fatty acid supplement (as fish oils), 1 gram daily of EPA

and DHA. It may take 2 - 3 weeks for benefits of fish oil supplements to be

seen.

For adults with high cholesterol levels: The American Heart Association

recommends an omega-3 fatty acid supplement (as fish oils), 2 - 4 grams daily of

EPA and DHA. It may take 2 - 3 weeks for benefits of fish oil supplements to be

seen.

From: kiddietalk@...

Date: Wed, 15 Sep 2010 13:57:54 +0000

Subject: [ ] Re: Overdose

The EPA extra is too high and raises the EPA/O3 too much over the O6 for the

majority. It's more expensive anyway- no need for the added expense. As many in

this group learn the hard way -paying more in money, time, doesn't always mean

better or more effective therapy. Just use the regular ProEPA for the child and

you or your spouse can take the extra.

Now here's more about dosage -and if anyone doubts anything I have here again I

just posted another message and as I have to keep archiving the same info over

and over and over -even I get bored sometimes and wanted to share new info...but

be assured I have way more information to support that yes you can use more than

3 grams a fish oils a day -even with toddlers -this group has for years. As I

say below I'm not saying don't share with your child's doctor -but we are an

intelligent group -let's stop perpetuating myths!

I know this is one of the myths from reputable sources too and shame on them

-but the major issue isn't the fish oils -it is for those who are on medications

with or without fish oils. Man made in nutrition doesn't work nearly as well as

Mother Nature made!!!

It probably took forever back then to get people to stop parroting that the

Earth is flat! At one point all have to look at proof and separate fact from

fiction -those using fish oils alone vs those using fish oils and blood

thinners!!

Also I just have one study below which was with 9 grams a day -most studies I

have seen with fish oils are 6 grams a day or higher even in children. And what

about this one for ADHD with 16 grams a day which many experts say is typical

for Eskimos. " but the successful studies have used rather large doses (up to 16

g fish oil per day). " Note they don't say the ADHD people that had success had

bleeding issues -one would think that was a slight side effect of the therapy

that WOULD have been mentioned ...don't you think?!!!