Guest guest Posted May 20, 2008 Report Share Posted May 20, 2008 Human T lymphotrophic Virus Associated Myelophathy (HTLV) has alot of components that fit Morgellons, as a matter of fact it is the closest fit I seen so far. It lowers the immune system and often has a co infection of worms and Lyme's. It causes the host to produce florescent lipofuscin which attracts insects. Lipofuscin are brown yellow granules which encase foreign objects to push them from the body. Ear wax is lipofuscin. HTLV causes infectious skin outbreaks. HTLV causes a breakdown and rebundling of fibers. If there is a co-infection of worms you will become more allergic. HTLV usually results in leukemia but in 4% percent of the people it becomes a type of Myelophathy and I am guessing that is because of a genetic and Lyme's factor. Here is a picture I found that looks similar to the golden head. http://www.aegis.com/topics/virus/xhiv3.jpg http://www.aegis.com/topics/virus/ HTLV-1, unlike the distantly related retrovirus HIV, has an immunostimulating effect, which, however, turns out to be immunosuppressive. The virus activates a subset of T-helper cells called Th1 cells. The result is a proliferation of Th1 cells and overproduction of Th1 related cytokines (mainly IFN-gamma and TNF-alpha). Feedback mechanisms of these cytokines cause a suppression of the Th2 lymphocytes and a reduction of Th2 cytokine production (maily IL-4, IL-5, IL-10 and IL-13). The end result is a reduction in the ability of the infected host to mount an adequate immune response to invading organisms that require a predominantly Th2 dependent response (these include parasitic infections and production of mucosal and humoral antibodies). See http://en.wikipedia.org/wiki/HTLV Quote Link to comment Share on other sites More sharing options...
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