Re: American Journal of Neuroradiology

[Guest guest]

Although, the whole question about brain problems is still a big one, and I

don't think most of us are actually talking about actual fungal infections. The

toxins alone (tricothecenes in particular) have been reported to have some of

the effects we mentioned. When I was relatively well for a while, I didn't seem

to have any problems. Other people who have experienced what I call remissions

don't seem to have any lingering problems, either. When I first got sick again

and was still in and out of that building, I got to the point where I couldn't

read or write and couldn't understand spoken words much at all. That has

improved somewhat. Maybe the problerms will go away altogether over time. I

can't say.

Anyway, the stuff doesn't seem to alter basic intelligence or anything. It

interferes with memory and attention span. (Which can make a person look really

stupid. I can sure swear to that!) learned. I can learn. It just takes a

long time, and it's hard to remember things once they've been learned.

Part of the reason I asked, , is because I find it interesting that people

with Lyme Disease or Gulf War Illness or us all seem to have the same kinds of

problems thinking and remembering. I would have thought they'd be somewhat

different because the sources for the illness aren't necessarily the same. But

not.That makes me think that the problems can have different causes, but then

they all operate according to the same mechanism.

tigerpaw2c <tigerpaw2C@...> wrote:

American Journal of Neuroradiology 26:1115-1121, May 2005

© 2005 American Society of Neuroradiology

Serena

www.freeboards.net/index.php?mforum=sickgovernmentb

---------------------------------

Mobile

Take with you! Check email on your mobile phone.

[Guest guest]

SERENA,

I Agree this is happening to me.

Elvira

Re: [] American Journal of Neuroradiology

Although, the whole question about brain problems is still a big one, and I

don't think most of us are actually talking about actual fungal infections.

The toxins alone (tricothecenes in particular) have been reported to have

some of the effects we mentioned. When I was relatively well for a while, I

didn't seem to have any problems. Other people who have experienced what I

call remissions don't seem to have any lingering problems, either. When I

first got sick again and was still in and out of that building, I got to the

point where I couldn't read or write and couldn't understand spoken words

much at all. That has improved somewhat. Maybe the problerms will go away

altogether over time. I can't say.

Anyway, the stuff doesn't seem to alter basic intelligence or anything. It

interferes with memory and attention span. (Which can make a person look

really stupid. I can sure swear to that!) learned. I can learn. It

just takes a long time, and it's hard to remember things once they've been

learned.

Part of the reason I asked, , is because I find it interesting that

people with Lyme Disease or Gulf War Illness or us all seem to have the same

kinds of problems thinking and remembering. I would have thought they'd be

somewhat different because the sources for the illness aren't necessarily

the same. But not.That makes me think that the problems can have different

causes, but then they all operate according to the same mechanism.

tigerpaw2c <tigerpaw2C@...> wrote:

American Journal of Neuroradiology 26:1115-1121, May 2005

© 2005 American Society of Neuroradiology

Serena

www.freeboards.net/index.php?mforum=sickgovernmentb

---------------------------------

Mobile

Take with you! Check email on your mobile phone.

[Guest guest]

Boy do I need This Place I went today to see a Natural Doctor I'm trying

something called BOLO take at night Chelation 2 at night green food Plus nervino

liquid Tonic Morning this is a well know Doctor big name people use him and I'm

giving him a try he help me and I can't pay his price.

Elvira

[] American Journal of Neuroradiology

American Journal of Neuroradiology 26:1115-1121, May 2005

© 2005 American Society of Neuroradiology

http://www.ajnr.org/cgi/content/abstract/26/5/1115

BRAIN

Diffusion-Weighted Imaging of Fungal Cerebral Infection

Paola Gaviania, B. Schwartze,g, E. Tessa Hedley-Whyteb,g,

L. Ligonf,g, Ari Robicsekc, Pamela Schaeferd,g and W.

Hensona,d,g

a E. and Pappas Center for Neuro-oncology Unit,

Massachusetts General Hospital, Boston

b E. and Pappas Center for Neuropathology Unit,

Massachusetts General Hospital, Boston

c E. and Pappas Center for Infectious Disease

Unit, Massachusetts General Hospital, Boston

d Division of Neuroradiology, Massachusetts General Hospital, Boston

e Division of Neuroradiology, Brigham and Women's Hospital, Boston

f Division of Neuropathology, Brigham and Women's Hospital, Boston

g Harvard Medical School, Boston

Address reprint requests to W. Henson, MD, E. and

Pappas Center for Neuro-oncology and Division of

Neuroradiology, Massachusetts General Hospital, Yawkey 9E, Fruit

Street, Boston, MA 02114

BACKGROUND AND PURPOSE: Diffusion-weighted imaging (DWI) is useful

in diagnosing bacterial brain abscesses, but DWI features of fungal

brain abscesses have not been characterized. Because fungal

abscesses are not purulent, we hypothesized that their DWI

characteristics are distinct from those of bacterial abscesses.

METHODS: We reviewed clinical, neuropathologic and neuroimaging

findings of patients with fungal brain infections due to Aspergillus

(n = 6), Rhizopus (n = 1), or Scedosporium (n = 1) species. DWI and

apparent diffusion coefficient (ADC) maps were obtained before

definitive diagnosis and antifungal therapy. ADC ratios

(lesion/contralateral white matter) were calculated.

RESULTS: Two patients had a rapidly progressive, fatal course, with

cerebritis and acute inflammation; fungal organisms were largely

restricted to vessels. Lesions were predominantly nonenhancing and

had heterogeneous foci of restricted diffusion. Six patients with

subacute neurologic presentations had acute or chronic inflammation,

capsule formation, focal necrosis, and fungal organisms disseminated

throughout the lesion. Their abscesses were ring enhancing. In five,

lesions had restricted diffusion in the central nonenhancing

portions. The sixth patient had a lesion with a peripheral rim of

restricted diffusion but elevated central diffusion; histopathology

showed early abscess formation. Mean ADC for all lesions was 0.33 ±

0.06 x 10-3 mm2/s, with an average ADC ratio of 0.43.

CONCLUSION: Fungal cerebral abscesses may have central restricted

diffusion similar to that of bacterial abscesses but with histologic

features of acute or chronic inflammation and necrosis rather than

suppuration. Altered water diffusion in these lesions likely

reflects highly proteinaceous fluid and cellular infiltration.

FAIR USE NOTICE:

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[Guest guest]

....>>Boy do I need This Place I went today to see a Natural Doctor I'm

trying something called BOLO take at night Chelation 2 at night green food

Plus nervino liquid Tonic Morning this is a well know Doctor big name people

use him and I'm giving him a try he help me and I can't pay his

price.Elvira>>

Elvira:

I'm an herbalist and never heard of BOLO and others you mentioned. Can you

give ingredients? Just curious?

Thank you,

Rosie

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