Dee Decision- Doug Haney

[Guest guest]

Posted by R. Haney on 5/18/05

http://toxlaw.com/chatboards/blackmold/topic307/5.18.05.09.35.46.html

ALL:

Is the " Dee " decision a setback or just another in a long

line of disappointments with the legal system? Believe it

or not, great progress is being made in helping the public

understand that pathogenic micro fungi exposures, esp.,

those encountered indoors can and do cause serious medical

health issues in animals and humans.

We had a little dispute a short time ago on this board as

to some of my statements about micro fungi exposures and

health and when I brought up the name of AV Costantini,

M.D., a few people wanted to know who he is and what his

credentials are. Before offering anything on this board,

because I have learned that there are a few " creepy

crawlers " who still like to dispute anything that is

offered I thought that I should confirm his credentials as

he expresses them in his book " Fungalbionic Series: Breast

Cancer, Hope At Last. " So, I set out to research both his

qualifications and background at the University of

California, School of Medicine in San Francisco. First I

found out from speaking directly with Personnel officials

that he in fact taught at this esteemed educational center

as he states and retired in the mid-1990s. Second that he

did publish his initial paper on " Fungalbionics " , there and

a search of the internet offered the following writings or

speeches by Dr. AV Constantini:

AV Costantini " Peer-Review/Journal " Writings:

COSTANTINI AV.

Fungalbionics: a new concept of the etiology of gout,

hyperuricemia and their related diseases.

Adv Exp Med Biol. 1989;253A:261-8. Review. No abstract

available.

PMID: 2696348 [PubMed - indexed for MEDLINE]

University of California School of Medicine, San Francisco.

Publication Types: Review Review, Tutorial

PETRAKIS NL, WOOD DA, METTIER SR, COSTANTINI AV, FARBER SM.

Preliminary clinical evaluation of 1, 2-bis(beta-

chloroethylthylthio) ethane (SM-1) in patients with

advanced lymphomas and neoplastic diseases.

Ann N Y Acad Sci. 1958 Apr 24;68(3):1151-63.

PMID: 13627768 [PubMed - OLDMEDLINE for Pre1966]

FARBER SM, COSTANTINI AV.

Chemotherapy of cancer in the teaching of diseases of the

chest.

Dis Chest. 1956 May;29(5):585-6.

PMID: 13305453 [PubMed - OLDMEDLINE for Pre1966]

COSTANTINI, AV. (1994). The fungal/mycotoxin etiology of

chronic and degenerative diseases. Excerpt form his

presentation at the Annual Meeting of New Horizons in

Health and Disease, Sept. 30, 1994, Toronto, Canada.

COSTANTINI, AV. (1993).

The Fungal/Mycotoxin Connections: Autoimmune Diseases,

Malignanacies, Atherosclerosis, Hyperlipidemias, and Gout,

Keynote Speaker, American Academy of Environmental

Medicine, Reno, Nevada.

COSTANTINI, AV. (1999). Wieland, H., and Qvick, Lars I.

Etiology and Prevention of Atherosclerosis. Freiburg,

Germany: Johann Friedrich Oberlin Verlag, 1999.

COSTANTINI, AV. (1994).

Costantini, A.V., Wieland, H., and Qvick, Lars I.

Fungalbionics, The Fungal/Mycotoxin Etiology of Human

Disease, Vol. 1 Atherosclerosis & Vol. II Cancer. Freiberg,

Germany:Johann Friedrich Oberlin Verlag. Available in

Canada from Fungal/Mycotoxin Conference, 12 Sifton Place,

Brampton, Ont. L6Y 2N8; 905-450-0445; FAX:905-450-0559.

His book/Google Search of " Fungalbionics " states the

following background information for Dr. Constantini and

his co-authors:

A.V. COSTANTINI, M.D.

Head (retired),

World Health Organization (WHO)

Collaborating Center For Mycotoxins In Food

Division of Clinical Chemistry

Department of Internal Medicine

School of Medicine

Albert Ludwigs University

Freiburg, Germany

Clinical Professorial Faculty (retired),

University of California

School of Medicine

San Francisco, California USA

HEINRICH WIELAND, M.D.

Head, World Health Organization (WHO)

Collaborating Center For Mycotoxins In Food

Division of Clinical Chemistry

Department of Internal Medicine

School of Medicine

Albert Ludwigs University

Freiburg, Germany

Professor and Medical Director,

Division of Clinical Chemistry

Department of Internal Medicine

School of Medicine

Albert Ludwigs University

Freiburg, Germany

LARS I. QVICK, M.D., Ph.D.

Medical Director,

World Health Organization (WHO)

Collaborating Center For Mycotoxins In Food

Division of Clinical Chemistry

Department of Internal Medicine

School of Medicine

Albert Ludwigs University

Freiburg, Germany

Medical Director,

Pharmacia & Upjohn

Switzerland

AV Costantini, M.D., as former head of the WHO has a very

distinguished medical career and was highly regarded by

research and medical experts world wide at the time he was

practicing medicine. I am going to continue to search for

as many of his articles, writings, and research

documentation, but I wanted everyone to know that when he

states that " Fungi/Mycotoxins are the Cause of Breast

Cancer " based on the prevailing research ongoing now, I

stand behind him as knowing is observations are solidly

based.

I also wanted to present a document previously written by

my infamous friend Ruth Etzel, M.D., Ph.D., formerly of

the " CDC Cleveland Infant Pulmonary Hemosiderosis

(Stachybotrys) Study " : (This is how I posted the article on

our website at http://www.Myhealthrights.com)

Author: R. Haney (---.dsl.scrm01.pacbell.net)

Date: 05-14-05 05:48

All:

This somewhat of an older article that has been in

existence for awhile. However it is more true today than

when it was originally published by my friend Dr. Ruth

Etzel, M.D., Ph.D. She is infamous for her medical research

along with Dorr Dearborn, M.D., in the 1994 Cleveland area

study of infants suffering with Pulmonary Hemosiderosis

though to be correlated with indoor inhalation of the

\ " Black Mold\ " (Stachybotrys chartarum). When her former

employer, the CDC, chose to challenge her outstanding

medical research methods which were documented on a PBS

documentary, Dr. Etzel one of the CDC\'s most respected

medical researchers, chose to leave the CDC declaring as

she departed that the \ " agency\'s review of the work is

\ " dead wrong\ " and that the CDC has sought to bury the

connection between mold and disease.\ " (Moran (July 26,

2000). Healthy House. WebMD Article. Retrieved on April 16,

2003 at:

http://www.ehw.org/Healthy_House/HH_Toxic_Mold.htm

There is a great deal of politicing and attempting to limit

the public knowledge as to the potential dangers of

inhalation of micro fungi and mycotoxins. Who do you think

stands to lose the most income if this were brought out

publicly and major research funding were to be expended on

the truth?

The tobacco industry who has had to defend its tobacco

products since the U.S. Surgeon General in 1954 ordered a

cautionary message be presented on each pack of cigarettes.

Let\'s see? If it is reported that approximately 450,000

people die each year from tobacco correlated illnesses, and

another approximately 50,000 from second-hand smoke

exposures, and the cigarette products are actually

\ " cured\ " for various mold species interactivity, could it

be that this industry would suffer the most financially?

From 1954 to 2005, that is 51 years and multiplying this

figure times the 500,000 deaths implicated annually, that

would come to about 25,500,000 deaths of fathers, mothers,

grandmothers and fathers, children, aunts and uncles,

friends and other relatives. If it were actually known that

it was the mycotoxins from micro fungi that actually

\ " decomposed\ " them from inhalation directly into areas of

the mouth, nose, lungs...

Would it be the Alcohol industry that might stand to lose

from all of the folks who drink this known mycotoxin

\ " alcohol\ " or if you prefer \ " ethanol\ " created by yeasts

(with a mixture of other mycotoxins to boot). Alcohol

accounts for up to 100,000 deaths per year. Using the 1954

figure 5,100,000 people have died from everything

implicatecd from heart disease to breast cancer, and from

brain tumors, to alcoholic cirrhosis. Who says a little

wine is good for you! Perhaps the same industry that

produces it? Ask yourself this question. How does the human

brain distinguish an \ " illegal\ " drug from a \ " legal\ "

drug? Would the liquor industry stand to lose the most

income if the public became more informed about the true

dangers of micro fungi exposure?

How about the HMO industry that focuses on \ " preventive

medicine\ " than major medical health care? Is there a

reason that they keep telling their patients that \ " mold

can\'t hurt anybody- its been around for ages!\ " Where is

the medical scientific research that indicates that micro

fungi inhaled does not directly influence severe/deadly

diseases? Everything researched from medical hospitals

relating to mycotoxins and nosocomial (hosptial

caused/related) diseases strongly implicates that even

small amounts of pathogenic spores can lead to deadly

consequences. How about all of those patients who are

released to go home early from the hospital to environments

that are totally uncontrolled environmentally? Would the

medical profession suffer the most financially if it were

known publicly that very often patients with mycotoxicosis

are diagnosed wrong and never treated properly, especially

those over the age of 50? I personally have witnessed this

kind of treatment in the patients I walked through the

medical process during my research on mycotic diseases.

How about the agricultural field and the fact that food

products, especally made from corn, wheat, barley, and

other harvested products contain pathogenic micro fungi

that could not possibly be completely monitored by the EPA

or the FDA? Go to \ " Google\ " search and type in

\ " Agriculture fungal disease\ " and see what I am talking

about. (495,000 articles/sites) Would this industry stand

to lose the most financially?

Would the U.S. Government agencies and State agencies who

are intrusted with our health and safety be the big

financial losers? Ever wonder why bacteria and viruses are

given megabucks for research study? How much is funded for

research into micro fungi and mycotoxins or mycotoxicosis

in comparison? Aren\'t micro fungi, even though they appear

as plant life, eukaryotic cells more structurally oriented

to animal and human cells? By the way, the answer to this

is, why \ " YES\ " they are as a matter of fact! Why haven\'t

many politicians jumped at the chance to support the \ " U.S.

Toxic Mold Health and Safety Act\ " (HR 1268)? Would this

open much exposure to the medical and health consequences

of indoor mold exposures? Would this call into question the

\ " birth/death ratio\ " motivation for keeping micro fungi

and illness quiet. Would they be \ " shooting the American

economy in the foot?\ " Or, is it healthier to let hundreds

of thousands of Americans die each year of \ " unknown cause,

unknown cure\ " diseases and allow doctors to feel that they

are actually doing something constructive?

Are there are other industries that could possibly be

injured greatly if the truth were known about our homes and

the severity of micro fungi infestations/mycotoxin

contaminations?. Would industries such as the building

manufacturers, realty industry, rental industry, and

others? Why yes, I have actually heard attorneys tell them

at their Association meetings not to learn too much for

sake of personal liability? How about the education

industry with schools across the nation facing problems

with micro fungi infestations/ mycotoxin infestations?

Would they be faced with a major financial dilema should

parents become informed about their children being kept

under such conditions? I have personally dared to inspect

schools and I can tell you from personal experience they do

not want anyone to bring up the subject of environmental

safety to them- they are scared to death that the school-

attending public will gain this knowledge!

Without further comment, Dr. Etzel\'s excellent article:

LINKING EVIDENCE AND EXPERIENCE

Mycotoxins

Ruth A. Etzel, MD, PhD

Mycotoxins, chemicals produced by fungi, may have developed

to serve as a chemical defense system against insects,

microorganisms, nematodes, grazing animals, and humans.

Approximately 400 known mycotoxins exist. This article

describes the major mycotoxins that affect human health and

highlights the mycotoxins with potential bioterrorist use.

Mycotoxins can benefit humans by their use as antibiotics

(penicillins), immunosuppressants (cyclosporine), and in

control of postpartum hemorrhage and migraine headaches

(ergot alkaloids). Mycotoxins are also capable of producing

illness and death in humans and animals.

Exposure to mycotoxins may occur through ingestion,

inhalation, and dermal exposure. The mycotoxins were

discovered when epidemics of illness were traced to

ingestion of moldy food. Massive mycotoxin contamination of

food resulting in outbreaks of illness occurs only rarely

today in developing countries. The primary concern in

developed countries is the long-term effects of ingesting

food contaminated with low levels of mycotoxins. Although

ergot alkaloids are described here because of their

historical importance, today the most commonly encountered

mycotoxins in animal feed and human foods are aflatoxins,

fumonisins, and deoxynivalenol (vomitoxin).

Aflatoxins

Aflatoxins, produced by Aspergillus flavus and A

parasiticus, are common contaminants of peanuts, soybeans,

grains, and cassava (a root), especially in tropical areas.

In the 1960s, aflatoxins were found to be potent

carcinogens in animals, the most potent of which is

aflatoxin B1. Epidemiologic studies have demonstrated that

aflatoxin B1 ingestion is an important risk factor for

hepatocellular cancer in humans. Persons with both

hepatitis B infection and aflatoxin B1 exposure have a

higher risk for hepatocellular cancer than those with only

hepatitis B infection or only aflatoxin exposure. In

Qidong, Jiangsu Province, China, hepatocellular carcinoma

is the leading cause of cancer deaths and exposure to

dietary aflatoxins is widespread. Ongoing clinical trials

there indicate that oltipraz, an antischistosomal drug, can

decrease the metabolism of aflatoxin B1 to its carcinogenic

form and increase the detoxification pathways of its

metabolites. Intervention with drugs such as oltipraz and

improved storage conditions of staple foods are measures

under investigation to reduce the incidence of

hepatocellular cancer in regions of higher risk.

In addition to chronic effects, aflatoxin exposure can

sometimes result in acute aflatoxicosis with vomiting,

abdominal pain, hepatitis, and death. Although acute

toxicity is rare, epidemics have been reported following

ingestion of food heavily contaminated with A flavus.8 The

acute lethal dose for adults is 10 to 20 mg of aflatoxin.

Ergot Alkaloids

The ergot alkaloids, produced by Claviceps purpura, were

the first mycotoxins recognized to cause epidemic disease

in humans. Persons who ingested these mycotoxins, found

primarily on moldy rye grain, developed ergotism. A

gangrenous form of ergotism was common in central Europe

from the 9th to the 14th century. The first symptom was a

prickly sensation in the limbs, which then became swollen,

inflamed, and subject to sensations of intense heat and

cold. Peripheral vasoconstriction resulted in gangrene and

limb loss. In the Middle Ages, this was known as St

\'s fire because it was often cured by a visit to

the shrine of St , which happened to be in an ergot-

free region of France. A convulsive form of ergotism

involving the nervous system occurred in Europe from the

late 16th to the late 19th century. It was also reported in

the United States and historians have hypothesized that it

may have been a factor in the Salem witchcraft trials of

1692. The vasoconstrictive properties of ergot alkaloids

have made them useful in treating migraine headaches

(ergotamine tartrate) and postpartum hemorrhage (methyl

ergonovine). Ergotism following ingestion of contaminated

food is very rare today; it is more commonly reported

following therapeutic administration of ergot alkaloids.

Fumonisins

The fumonisins are a group of mycotoxins isolated from corn

contaminated with Fusarium moniliforme, F proliferatum, and

A ochraceus. The fumonisins were discovered in 1988

following the 1970 outbreak of equine leukoencephalomalacia

in South Africa. Fumonisins seem to be universally present

in corn and corn-based products. Extensive investigations

have documented that consumption of corn and corn-based

products contaminated with fumonisin B1 causes equine

leukoencephalomalacia and porcine pulmonary edema, fatal

diseases in farm animals. In 1989 and 1990, fatal outbreaks

of equine leukomalacia, porcine prenatal and neonatal

mortality, and porcine pulmonary edema occurred in the

United States. Evidence of human health effects from

ingestion of fumonisin-contaminated foods primarily derives

from studies in South Africa, China, and northern Italy.

These studies suggest a link between fumonisin exposure and

esophageal cancer.

The fumonisins have been shown to disrupt sphingolipid

metabolism. Sphingolipids play a role in membrane and

lipoprotein structure and in cell regulation as second

messengers for growth factors, differentiation factors, and

cytokines. Disruption of sphingolipid metabolism and its

effect on human development is under study.

Fumonisin exposure may play a role in birth defects. A 1990

cluster of neural tube defects in south Texas generated the

hypothesis that ingestion of high levels of fumonisins in

corn-based products might be linked to human birth defects,

such as anencephaly and spina bifida. Mexican Americans\'

risks of neural tube defects are much higher than those of

non-Hispanic whites.

When the cluster of affected pregnancies occurred, US corn-

based products had relatively high levels of fumonisins, 2

to 3 times higher than normal. Mexican American women in

Texas, unlike their non-Hispanic counterparts, eat a lot of

corn in the form of tortillas (90 g/d vs 17 g/d). Fumonisin

has been shown to interfere with cellular folate uptake and

it is possible that exposure to dietary fumonisins may help

explain the lack of effectiveness of folic acid in reducing

neural tube defects in Mexican Americans.

Trichothecenes

Fusarium and Stachybotrys species produce mycotoxins called

trichothecenes. When ingested by humans, these mycotoxins

produce alimentary toxic aleukia. This disease first

appeared in 1913 in far eastern Siberia and was reportedly

responsible for the death of at least 100 000 Russian

people between 1942 and 1948. Affected persons developed

necrotic ulcers in the nose, mouth, throat, stomach, and

intestines, complicated by hemorrhage from the nose, mouth,

gastrointestinal tract, and kidneys. Alimentary toxic

aleukia was associated with eating wheat and corn that had

been under snow during the winter and contaminated with

Fusarium and Stachybotrys molds.

Dermal exposure to the Stachybotrys fungus may cause a

severe skin reaction. The dermatitis was first described

among workers handling fodder, using infected straw for

fuel, or sleeping on mattresses made of infected straw and

is characterized by hyperemia, encrustations, and necrosis.

The acute toxicosis resulting from the inhalation of the

Stachybotrys mycotoxin, first described by Soviet

scientists in the 1940s, has been termed

stachybotryotoxicosis. The symptoms include sore throat,

bloody discharge from the nose, dyspnea, cough, low-grade

fever, and chest tightness.

Vomitoxin

Another trichothecene mycotoxin is deoxynivalenol, also

known as vomitoxin, frequently a contaminant of wheat and

corn. In China from 1961 to 1985, multiple outbreaks of

vomiting illness were attributed to consumption of

vomitoxin-contaminated grain. In India in 1987, nearly 100

persons became ill after they consumed wheat products from

which vomitoxin and other trichothecene mycotoxins were

recovered. In 1997 to 1998, approximately 1700 US children

became ill with vomiting, nausea, headache, and abdominal

cramps linked to eating burritos. Although levels of

vomitoxin in the burritos were less than 1 ppm, the Food

and Drug Administration (FDA) advisory level, vomitoxin

could not be eliminated as the causal agent because this

advisory level is set for adults and may not be applicable

to children. Ingestion of mycotoxin-contaminated food is

the most important route of exposure; 2 other routes should

be recognized, as both dermal absorption and inhalation of

macrocyclic trichothecene mycotoxins have been associated

with human illnesses.

Satratoxin

Satratoxin is produced by Stachybotrys atra (also known as

S chartarum). This fungus can grow on any cellulose product

in the presence of water. Dissemination of spores into

indoor air occurs when the fungus is disturbed. An

epidemiologic study in 1994 found that 10 infants with life-

threatening acute pulmonary bleeding were more likely than

a matched group of 30 comparison infants to live in homes

with S atra and other molds in the air. The findings

linking S atra and other fungi to infant pulmonary

hemorrhage are controversial and have undergone careful

scrutiny. Additional research is needed to determine

whether the reported association between infant pulmonary

hemorrhage and exposure to toxigenic S atra is causal.

Exposure to S atra has subsequently been associated with

acute pulmonary hemorrhage in an infant in Kansas City, Mo,

and with pulmonary hemosiderosis in a 7-year-old in

Houston, Tex. The Texas investigators cultured S atra from

the patient\'s bronchoalveolar fluid. Trichothecenes

suppress the immune system, leading to increased

susceptibility to a variety of infectious diseases.

Prevention

Both drought and flooding contribute to problems with

mycotoxins. Fungi are usually unable to penetrate intact

seed kernels; drought may weaken the plant, allowing

penetration of the fungus. Mycotoxin problems in food may

be greater during years of extreme drought. Intense rain

and flooding can also increase mycotoxin problems; intense

rain events have increased by 20% since 1900.

Massive contamination with mold is detectable and problems

can be avoided by not eating visibly moldy foods. The

consumer cannot tell that processed products have elevated

levels of aflatoxins, vomitoxin, or fumonisins;

furthermore, these mycotoxins are not destroyed by heating.

The FDA has set action levels, informal nonbinding

guidelines, for aflatoxins in food. The FDA has advisory

levels for vomitoxin but no established action levels and

has recently released a draft guidance document for

industry on fumonisin levels in human foods and animal feed.

Mycotoxins and Biological Warfare

One of the earliest uses of mycotoxins in warfare occurred

in sixth century BC when the Assyrians poisoned enemy wells

with rye ergot. By the late 1990s, several countries had

weaponized aflatoxin and there was suspicion that

trichothecenes were also under investigation for use in

biological warfare. Controversy exists about the purported

use of T2 (a trichothecene mycotoxin) in aerosol form

(yellow rain) in Laos, Kampuchea, and Afghanistan in the

1970s and 1980s. The only effective methods to prevent

exposure are physical protection of the skin and the

airway; treatment is limited to supportive care.

Clinicians should be alert for cases of unusual illness and

report them to the local health department. Historically,

every discovery of the acute health effects of mycotoxins

has been prompted by reports of unusual illnesses from

alert clinicians; vigilance and early reporting are the

most promising lines of defense against the potential

bioterrorist use of mycotoxins.

Author/Article Information

Author Affiliation: Division of Environmental and

Occupational Health, Washington University, School

of Public Health and Health Services, Washington, DC.

Corresponding Author and Reprints: Ruth A. Etzel, MD, PhD,

US Public Health Service, Alaska Native Medical Center,

4320 Diplomacy Dr, Anchorage, AK 99508 (e-mail:

retzel@...).

Contempo Updates Section Editor: Janet M. Torpy, MD,

Fishbein Fellow.

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ABSTRACT | FULL TEXT | PDF | MEDLINE

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2000;3:109-143.

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Creasia DA, Lambert RJ.

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1989:161-170.

Now, with this article written by someone of Dr. Etzel's

level of experience and distinguishing credentials I would

pose that it would be literally stupid for someone argue

that she does not know of what she speaks.

This is all that I feel like sharing, though I have far

greater ammunition about micro fungi and health that I

could offer. When I said I do not want to give it away

freely to someone on this board who does not deserve

information that he has not researched as hard as I have

over the last 19 years, that is what I meant. I had a

highly placed and well respected California judge, two

attorneys, and a major insurance company test me in a San

Francisco mediation/settlement conference over a month ago

about what I know about microbial behavior and human health

(esp., cancer) and within 90 minutes a low six-figure

monetary settlement that was unable to be settled in over

three years of litigation was settled for what the

plaintiff's attorney stated was a " subtantial seven digit

monetary figure " as was offered in the " Dee " case, based on

the fear of future severe illness and/or cancer.

Still don't believe what I have related here? Test me in a

court of law in front of a jury, without all the publicity

and promotion that the Dee case attempted to create. And,

do not tell me that an " immunocompetent person " is not

susceptible to micro fungi exposed severe disease, because

that is easily challenged as well. Come on all you

insurance types and defense attorneys that are challenging

the facts on technicalities in court, let's have an all-

out " face off " in front of the jury. Let me explain what

pathogenic microbes are all about and how they go about

challenging the human body for territory and decomposition.

Let's see if I am bragging or just plain trying to bring

out the direct and factual truth in this matter so that

victims can finally start getting medical assistance that

might save their lives.

I told you also that I would be willing to debate this on a

national talk show that would pose a threat to my

credibility with any medical doctor or scientist in the

world that " believes " micro fungi does not influence

serious human disease, even in " immunecompetent " people. To

date you " chickens " who would rather hide behind the court

system have not had the courage to challenge my offer.

Well, it still stands, let's see who has the gonads!

Better yet, challenge me and my friends like doctors Jack

Thrasher, Marinkovich, Ritchie Shoemaker, Eckardt

Johanning, Gray, Ordog, , Doris

Rapp, Dorr Dearborn, and the many others who have been

chastised over the past few years for having the courage to

treat or present the health issues relating to micro fungi

exposures.

Like I said, it is time to challenge of fade away into

medicrity

Doug Haney

Re: Dee Decision- Doug Haney

Posted by Greg Weatherman on 5/18/05

Doug,

Leave my scotch, bourbon, rum, merlot and beer alone. Yes,

they contain mycotoxins by definition but, I'll take your

share if it bothers you too much.

However, I don't know why people can't grasp the

neurological damage done by chronic exposure from mold when

they could use extreme alcohol consumption as a model. Look

at metabolism and dose when comparing a tee-totaller and a

whino. Do the same with groups to show genetic diversity.

Who are the government bums who can't figure it out?

Like the add (with PET scan or SPECT scan image) says, " This

is a normal brain; This is an alcoholic's brain " ! We had a

display at the sonian Castle in Washington DC a few

years ago on brains that was enlightening. Toxicology 101

can be had at the corner liquor store if you pay attention.

Regards,

Greg Weatherman

aerobioLogical Solutions Inc.

Arlington VA 22202

gw@...