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J Nutr. 2005 Sept;135(9)

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J Nutr. 2005 Sept;135(9)

Hi All.

A new issue of Journal of Nutrition has been made available, and the not

pdf-available papers for which Medline has no records and abstracts were

available

and of interest, were:

1 September 2005; Vol. 135, No. 9

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Recent Advances in Nutritional Sciences

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Mechanisms by which Dietary Fatty Acids Modulate Plasma Lipids

Luz Fernandez and Kristy L. West

J. Nutr. 2005;135 2075-2078

Dietary fatty acids have a considerable effect on plasma LDL cholesterol (LDL-C)

concentrations and therefore on the risk for coronary heart disease. Numerous

studies have been conducted in animal models to elucidate the mechanisms by

which

different types of fatty acids modulate plasma cholesterol concentrations. In

addition, multiple clinical trials and epidemiological data have demonstrated

the

effects of fatty acids in determining the concentrations of circulating LDL.

SFAs

and trans fatty acids have a detrimental effect on plasma lipids, whereas PUFAs

of

the (n-6) family and monounsaturated fatty acids decrease plasma LDL-C

concentrations. Among the SFAs, stearic acid (18:0) appears to have a neutral

effect

on LDL-C, while lauric (12:0), myristic (14:0), and palmitic (16:0) acids are

considered to be hypercholesterolemic. SFAs increase plasma LDL-C by increasing

the

formation of LDL in the plasma compartment and by decreasing LDL turnover.

Although

unsaturated fatty acids increase cholesterol synthesis, they also increase

hepatic

LDL receptor number and LDL turnover in vivo. Fatty acids are also ligands of

important regulatory elements, which can play a role in determining plasma

cholesterol. This article presents a summary of the major effects of various

types

of fatty acids on plasma lipid concentrations and the mechanisms involved.

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Critical Reviews

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A Systematic Review of the Effects of Nuts on Blood Lipid Profiles in Humans

Janine Mukuddem-sen, Welma Oosthuizen, and Johann C. Jerling

J. Nutr. 2005;135 2082-2089

The inverse association of nut consumption and risk markers of coronary heart

disease (lipids) has sparked the interest of the scientific and lay community.

The

objective of this study was to conduct a systematic review to investigate the

effects of nuts on the lipid profile. Medline and Web of Science databases were

searched from the start of the database to August 2004 and supplemented by

cross-checking reference lists of relevant publications. Human intervention

trials

with the objective of investigating independent effects of nuts on lipid

concentrations were included. From the literature search, 415 publications were

screened and 23 studies were included. These papers received a rating based upon

the

methodology as it appeared in the publication. No formal statistical analysis

was

performed due to the large differences in study designs of the dietary

intervention

trials. The results of 3 almond (50–100 g/d), 2 peanut (35–68 g/d), 1 pecan nut

(72

g/d), and 4 walnut (40–84 g/d) studies showed decreases in total cholesterol

between

2 and 16% and LDL cholesterol between 2 and 19% compared with subjects consuming

control diets. Consumption of macadamia nuts (50–100 g/d) produced less

convincing

results. In conclusion, consumption of 50–100 g (1.5–3.5 servings) of nuts 5

times/wk as part of a heart-healthy diet with total fat content (high in mono-

and/or polyunsaturated fatty acids) of 35% of energy may significantly decrease

total cholesterol and LDL cholesterol in normo- and hyperlipidemic individuals.

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Biochemical and Molecular Actions of Nutrients

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Isoleucine, a Blood Glucose-Lowering Amino Acid, Increases Glucose Uptake in Rat

Skeletal Muscle in the Absence of Increases in AMP-Activated Protein Kinase

Activity

Masako Doi, Ippei Yamaoka, Mitsuo Nakayama, Shinji Mochizuki, Kunio

Sugahara, and Fumiaki Yoshizawa

J. Nutr. 2005;135 2103-2108

Leucine and isoleucine were shown to stimulate insulin-independent glucose

uptake in

skeletal muscle cells in vitro. In this study, we examined the effects of

leucine

and isoleucine on blood glucose in food-deprived rats and on glucose metabolism

in

skeletal muscle in vivo. Furthermore, we investigated the possible involvement

of

the energy sensor, 5'-AMP-activated protein kinase (AMPK), in the modulation of

glucose uptake in skeletal muscle, which is independent of insulin, and also in

leucine- or isoleucine-stimulated glucose uptake. Oral administration of

isoleucine,

but not leucine, significantly decreased the plasma glucose concentration. An

i.v.

bolus of 2-[1,2-3H]-deoxyglucose (2-[3H]DG) was administered to calculate

glucose

uptake. Glucose uptake in the skeletal muscle did not differ after leucine

administration, but glucose uptake in the muscles of rats administered

isoleucine

was 73% greater than in controls, suggesting that isoleucine increases skeletal

muscle glucose uptake in vivo. On the contrary, in the skeletal muscles,

administration of leucine but not isoleucine significantly increased

[u-14C]-glucose

incorporation into glycogen compared with controls. AMPK 1 activity in skeletal

muscle was not affected by leucine or isoleucine administration. However,

isoleucine, but not leucine, significantly decreased AMPK 2 activity. The

decrease

in AMPK 2 activity was thought to be due to decreases in AMP content and the

AMP:ATP

ratio, which were related to the isoleucine administration. This is the first

report

of isoleucine stimulating glucose uptake in rat skeletal muscle in vivo, and

these

results indicate that there might be a relation between the reduction in blood

glucose and the increase in skeletal muscle glucose uptake that occur with

isoleucine administration in rats. The alterations in glucose metabolism caused

by

isoleucine may result in an improvement of the availability of ATP in the

absence of

increases in AMP-activated protein kinase activity in skeletal muscle.

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Nutrition and Cancer

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Lycopene Inhibits Cell Migration and Invasion and Upregulates Nm23-H1 in a

Highly

Invasive Hepatocarcinoma, SK-Hep-1 Cells

Chin-Shiu Huang, Ming-Kuei Shih, Cheng-Hung Chuang, and Miao-Lin Hu

J. Nutr. 2005;135 2119-2123

The carotenoid lycopene has been associated with decreased risks of several

types of

cancer, such as prostate cancer and hepatoma. Tumor metastasis is the most

important

cause of cancer death. Although lycopene was shown to inhibit metastasis, the

mechanism underlying this action is not well understood. Here, we tested the

possibility that lycopene may inhibit cancer cell metastasis by upregulating the

expression of nm23-H1, a metastasis suppressor gene, in SK-Hep-1 cells, a highly

invasive hepatoma cell line, and we determined migration and invasion activities

and

the expression of nm23-H1 protein and mRNA. We showed that lycopene inhibited

SK-Hep-1 migration and invasion in a bell-shaped manner, with the highest effect

at

5 µmol/L (91 and 63% inhibition for migration and invasion, respectively; P <

0.05).

At the same test level (10 µmol/L), lycopene was much more effective than

ß-carotene

in reducing cell invasion (by 870%). In contrast to the effects on migration and

invasion, lycopene enhanced nm23-H1 expression at both the protein and mRNA

levels;

the effects were also bell shaped, and at 5 µmol/L, lycopene enhanced nm23-H1

protein and mRNA expressions by 220 ± 33 and 153 ± 22% (P < 0.01), respectively.

These bell-shaped effects of lycopene may be related to autoxidation of lycopene

at

elevated concentrations (10 µmol/L). Significant correlations existed between

nm23-H1 protein expression and migration (r2 = 0.78, P < 0.001) and between

nm23-H1

protein expression and invasion (r2 = 0.84, P < 0.001) in lycopene-treated

SK-Hep-1

cells. We conclude that lycopene has significant antimigration and anti-invasion

activity, and that this effect is associated with its induction of nm23-H1

expression.

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Human Nutrition and Metabolism

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Ingested Fat Oxidation Contributes 8% of 24-h Total Energy Expenditure in

Moderately

Obese Subjects

Bakary J. Sonko, V. Fennessey, ph E. Donnelly, Bessesen,

A. Sharp, Dennis J. sen, H. , and O. Hill

J. Nutr. 2005;135 2159-2165

.... The aims of this study were to determine the contributions of ingested fat

oxidation to: 1) 24-h total energy expenditure (TEE), and 2) substrate oxidation

during acute stationary cycle exercises in adult humans. Healthy, moderately

obese

(n = 18; BMI = 31 ± 1 kg/m2) subjects (8 men; 10 women) were each studied in a

whole-room calorimeter for 24 h. They were fed mixed meals (55, 30, and 15% as

energy from carbohydrate, fat and protein, respectively) to maintain energy

balance.

Each subject performed 1255-kJ cycle exercises at 50% VO2max in the calorimeter.

Study test meal fat was labeled with carbon-13 (13C). Ingested fat oxidation was

estimated from breath 13CO2 excretion and the subject’s chamber CO2 production.

Total fat and carbohydrate oxidations were estimated from nonprotein respiratory

quotient (NP-RQ) values. Endogenous fat oxidation was estimated as the

difference

between total fat and ingested fat oxidations. TEE was estimated from gas

exchanges;

28 ± 3% of ingested fat was oxidized and it provided 8 ± 1% of 24-h TEE. During

cycle exercises, ingested fat provided 50% of total fat oxidized and 13.0 ± 2%

of

energy expended. Endogenous fat oxidation contributed 10.4 ± 3% of energy

expenditure during cycle exercises. This study extended to 24-h observations of

previous studies that lasted 6–9 h on ingested fat oxidation in humans. ...

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Nutrient Metabolism

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The Biodistribution of a Single Oral Dose of [14C]-Lycopene in Rats Prefed

Either a

Control or Lycopene-Enriched Diet

Zaripheh and W. Erdman, Jr.

J. Nutr. 2005;135 2212-2218

Lycopene (lyc) has emerged as a primary candidate for dietary interventions of

prostate cancer; however, research regarding its absorption, tissue

distribution,

and metabolism is limited. Previously, we evaluated the biodistribution (3–168

h) of

a single oral dose of 14C-lyc in rats prefed lyc for 30 d. The liver was the

primary

depot for lyc, and the 14C and 14C-polar products appeared in tissues as early

as 3

h after dosing. In the current study, F344 rats (n = 48) were randomly assigned

to 1

of 4 groups prefed either a control or lyc-enriched diet (0.25 g lyc/kg diet)

for 30

d and killed at 5 or 24 h after receiving a single oral dose of 14C-lyc. The

percentage of the 14C dose absorbed at 24 h was lower (5.5 ± 0.5%) in lyc-prefed

(LP) rats than in control-prefed (CP) rats (6.9 ± 0.4%, P < 0.04). Hepatic total

14C

and 14C-lyc in CP rats was greater than in LP rats at 24 h (P < 0.005). A

portion of

14C was delivered to extrahepatic tissues as early as 5 h, irrespective of diet.

Of

the tissues analyzed, an increase in the percentage in 14C-polar products

occurred

between 5 and 24 h only in the prostate and seminal vesicles, suggesting

increased

accumulation of 14C-polar products in these tissues, irrespective of prior

dietary

treatment. These data suggest that lyc absorption, tissue uptake, and catabolism

were affected by prefeeding and that lyc can be partially taken up by

extrahepatic

tissues from the postprandial triglyceride-rich fraction.

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Nutritional Epidemiology

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Elevated Serum Fluoride Concentrations in Women Are Not Related to Fractures and

Bone Mineral Density

Fran Sowers, M. Whitford, M. Kathleen , and L. Jannausch

J. Nutr. 2005;135 2247-2252

Epidemiologic studies of the relations between drinking-water fluoride levels

and

bone mineral density (BMD) and fracture are characterized by disparate

conclusions

and an absence of information about individual circulating fluoride levels. This

study relates serum fluoride concentrations, which reflect individual fluoride

exposures, to BMD and bone fractures. Data are from 1300 female residents of 3

small

communities in which the water fluoride concentrations were 52.6 or 210.4

µmol/L.

Circulating serum fluoride concentrations were assessed by ion-specific

electrode.

Fluoride intake was estimated from interviews describing water and water-based

beverage consumption and duration of residence in the community. BMD was

measured by

dual-energy X-ray densitometry and single-photon densitometry. Self-reported

fractures were confirmed by medical record abstraction. The mean serum fluoride

concentration in the high-fluoride community, 2.11 ± 0.05 µmol/L, was

significantly

higher than serum fluoride concentrations in the control and high-calcium

communities with water fluoridation to 52.6 µmol/L. The mean serum fluoride

concentrations in these latter 2 communities were 1.6 ± 0.04 and 1.22 ± 0.05

µmol/L,

respectively. Serum fluoride was not significantly related to BMD after

adjusting

for covariates including age and body size. The mean distal radius BMD, however,

was

significantly higher in the high-fluoride community. Serum fluoride

concentrations

were not related to incident osteoporotic fractures with 4 y of observation.

Serum

fluoride concentrations were not associated with BMD or osteoporotic fractures

among

female residents of communities with water fluoride concentrations of 52.6 or

210.4 µmol/L.

Al Pater, PhD; email: old542000@...

____________________________________________________

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