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Most interesting recommendations. Some fellow Asian researchers(India

is after all in Asia)may be allowed to disagree.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=9\

316363 & dopt=Citation

>

> FWIW,

> J Cardiovasc Risk. 1996 Dec;3(6):489-94.

>

>

> Recommendations for the prevention of coronary artery disease in

Asians: a scientific statement of the International College of Nutrition.

>

> Singh RB, Mori H, Chen J, Mendis S, Moshiri M, Zhu S, Kim SH, Sy RG,

Faruqui AM.

>

> Medical Hospital and Research Centre, Moradabad, India.

>

> There has been a rapid increase in coronary artery disease (CAD) in

most Asian countries in association with rapid economic development;

however, there is no consensus of opinion on diet and lifestyle

guidelines and desirable levels of risk factors for prevention of CAD

in these countries. The proportion of deaths due to cardiovascular

diseases in Asians may be about 15% but there are wide variations. In

view of the lower fat intake of the low-risk rural populations of

India, the People's Republic of China, Indonesia, Korea, Thailand and

Japan compared with that of urban subjects, the limit for total energy

from fat intake in an average should be 21% (7% each from saturated,

polyunsaturated and mono-unsaturated fatty acids). The n-6: n-3 fatty

acids ration should be < 5.0. The carbohydrates intake should be > 65%

and mainly from complex carbohydrates (> 55%). A body mass index of 21

kg/m2 may be safe but the range may be 18.5-23.0 kg/m2 and someone

with a body mass index > 23 kg/m2 should be considered overweight. A

waist: waist:hip ratio > 0.88 for men and > 0.85 for women should be

considered to define central obesity. The desirable limit for serum

total cholesterol may be 170 mg/dl, the borderline high level may be

170-199 mg/dl and the high level 200 mg/dl or above. The corresponding

values for low-density lipoprotein cholesterol may be 90, 90-109 and

110 mg/dl or above. Fasting serum triglycerides may be < 150 mg/dl and

high-density lipoprotein cholesterol > 35 mg/dl, which are close to

the levels in low-risk rural populations. Fasting blood glucose > 140

mg/dl and postprandial blood glucose > 200 mg/dl may be considered

conditions for diabetes, and 140-200 mg/dl, glucose intolerance. An

intake of 400 g/day fruit, vegetables and legumes, mustard or soybean

oil (25 g/day) instead of hydrogenated fat, coconut oil or butter in

conjunction with moderate physical activity (1255 kJ/day), cessation

of tobacco consumption and moderation of alcohol intake may be an

effective package of remedies for prevention of CAD in Asians. PMID:

9100083

>

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Share on other sites

Most interesting recommendations. Some fellow Asian researchers(India

is after all in Asia)may be allowed to disagree.

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=9\

316363 & dopt=Citation

>

> FWIW,

> J Cardiovasc Risk. 1996 Dec;3(6):489-94.

>

>

> Recommendations for the prevention of coronary artery disease in

Asians: a scientific statement of the International College of Nutrition.

>

> Singh RB, Mori H, Chen J, Mendis S, Moshiri M, Zhu S, Kim SH, Sy RG,

Faruqui AM.

>

> Medical Hospital and Research Centre, Moradabad, India.

>

> There has been a rapid increase in coronary artery disease (CAD) in

most Asian countries in association with rapid economic development;

however, there is no consensus of opinion on diet and lifestyle

guidelines and desirable levels of risk factors for prevention of CAD

in these countries. The proportion of deaths due to cardiovascular

diseases in Asians may be about 15% but there are wide variations. In

view of the lower fat intake of the low-risk rural populations of

India, the People's Republic of China, Indonesia, Korea, Thailand and

Japan compared with that of urban subjects, the limit for total energy

from fat intake in an average should be 21% (7% each from saturated,

polyunsaturated and mono-unsaturated fatty acids). The n-6: n-3 fatty

acids ration should be < 5.0. The carbohydrates intake should be > 65%

and mainly from complex carbohydrates (> 55%). A body mass index of 21

kg/m2 may be safe but the range may be 18.5-23.0 kg/m2 and someone

with a body mass index > 23 kg/m2 should be considered overweight. A

waist: waist:hip ratio > 0.88 for men and > 0.85 for women should be

considered to define central obesity. The desirable limit for serum

total cholesterol may be 170 mg/dl, the borderline high level may be

170-199 mg/dl and the high level 200 mg/dl or above. The corresponding

values for low-density lipoprotein cholesterol may be 90, 90-109 and

110 mg/dl or above. Fasting serum triglycerides may be < 150 mg/dl and

high-density lipoprotein cholesterol > 35 mg/dl, which are close to

the levels in low-risk rural populations. Fasting blood glucose > 140

mg/dl and postprandial blood glucose > 200 mg/dl may be considered

conditions for diabetes, and 140-200 mg/dl, glucose intolerance. An

intake of 400 g/day fruit, vegetables and legumes, mustard or soybean

oil (25 g/day) instead of hydrogenated fat, coconut oil or butter in

conjunction with moderate physical activity (1255 kJ/day), cessation

of tobacco consumption and moderation of alcohol intake may be an

effective package of remedies for prevention of CAD in Asians. PMID:

9100083

>

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Share on other sites

, What we fail to realize is that humans can eat almost anything. It's eating too much of it that usually is the problem. That's what CR is about - not preaching about a particular oil, protein, mineral, vitamin, whatever, but figuring out what our bodies need.

My choice is NO oil after reading many articles, most of which praised olive oil, until I got them to compare to NO oil, and that turned out to be better.

We need very little oil, and the fatty acids that I DO need are in soy - 1 tbls, that's it. My body can make all the fat it needs except for 2 essential FA's.

So if I rely on foods I prepare myself, I can get the equiv of olive oil naturally in avocadoes, eg. IMO, I don't need to add any oils/fats. A no fat diet is not possible.

There are some CRONies who use a high fat diet, I think, and they are adamant about no carbos, but that I think, is not the main. Their concern is for the glycation theory of aging. AFAIK, there is no diet defined yet to avoid aging, just those for lower mortality. The Ornish diet is for those with CAD, tested and accepted, and if it works on CADers, it might be good to prevent CAD - who knows?

You have noticed that for every article stating one thing another can be found to oppose it. That only means to me that no one really knows for sure.

Regards.

[ ] Re: coconut oil and CAD

Most interesting recommendations. Some fellow Asian researchers(Indiais after all in Asia)may be allowed to disagree.http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=9316363 & dopt=Citation--- In , "jwwright" <jwwright@e...> wrote:>> FWIW,> J Cardiovasc Risk. 1996 Dec;3(6):489-94. > > > Recommendations for the prevention of coronary artery disease inAsians: a scientific statement of the International College of Nutrition.> > Singh RB, Mori H, Chen J, Mendis S, Moshiri M, Zhu S, Kim SH, Sy RG,Faruqui AM.> > Medical Hospital and Research Centre, Moradabad, India.> > There has been a rapid increase in coronary artery disease (CAD) inmost Asian countries in association with rapid economic development;however, there is no consensus of opinion on diet and lifestyleguidelines and desirable levels of risk factors for prevention of CADin these countries. The proportion of deaths due to cardiovasculardiseases in Asians may be about 15% but there are wide variations. Inview of the lower fat intake of the low-risk rural populations ofIndia, the People's Republic of China, Indonesia, Korea, Thailand andJapan compared with that of urban subjects, the limit for total energyfrom fat intake in an average should be 21% (7% each from saturated,polyunsaturated and mono-unsaturated fatty acids). The n-6: n-3 fattyacids ration should be < 5.0. The carbohydrates intake should be > 65%and mainly from complex carbohydrates (> 55%). A body mass index of 21kg/m2 may be safe but the range may be 18.5-23.0 kg/m2 and someonewith a body mass index > 23 kg/m2 should be considered overweight. Awaist: waist:hip ratio > 0.88 for men and > 0.85 for women should beconsidered to define central obesity. The desirable limit for serumtotal cholesterol may be 170 mg/dl, the borderline high level may be170-199 mg/dl and the high level 200 mg/dl or above. The correspondingvalues for low-density lipoprotein cholesterol may be 90, 90-109 and110 mg/dl or above. Fasting serum triglycerides may be < 150 mg/dl andhigh-density lipoprotein cholesterol > 35 mg/dl, which are close tothe levels in low-risk rural populations. Fasting blood glucose > 140mg/dl and postprandial blood glucose > 200 mg/dl may be consideredconditions for diabetes, and 140-200 mg/dl, glucose intolerance. Anintake of 400 g/day fruit, vegetables and legumes, mustard or soybeanoil (25 g/day) instead of hydrogenated fat, coconut oil or butter inconjunction with moderate physical activity (1255 kJ/day), cessationof tobacco consumption and moderation of alcohol intake may be aneffective package of remedies for prevention of CAD in Asians. PMID:9100083>

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Share on other sites

, What we fail to realize is that humans can eat almost anything. It's eating too much of it that usually is the problem. That's what CR is about - not preaching about a particular oil, protein, mineral, vitamin, whatever, but figuring out what our bodies need.

My choice is NO oil after reading many articles, most of which praised olive oil, until I got them to compare to NO oil, and that turned out to be better.

We need very little oil, and the fatty acids that I DO need are in soy - 1 tbls, that's it. My body can make all the fat it needs except for 2 essential FA's.

So if I rely on foods I prepare myself, I can get the equiv of olive oil naturally in avocadoes, eg. IMO, I don't need to add any oils/fats. A no fat diet is not possible.

There are some CRONies who use a high fat diet, I think, and they are adamant about no carbos, but that I think, is not the main. Their concern is for the glycation theory of aging. AFAIK, there is no diet defined yet to avoid aging, just those for lower mortality. The Ornish diet is for those with CAD, tested and accepted, and if it works on CADers, it might be good to prevent CAD - who knows?

You have noticed that for every article stating one thing another can be found to oppose it. That only means to me that no one really knows for sure.

Regards.

[ ] Re: coconut oil and CAD

Most interesting recommendations. Some fellow Asian researchers(Indiais after all in Asia)may be allowed to disagree.http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=9316363 & dopt=Citation--- In , "jwwright" <jwwright@e...> wrote:>> FWIW,> J Cardiovasc Risk. 1996 Dec;3(6):489-94. > > > Recommendations for the prevention of coronary artery disease inAsians: a scientific statement of the International College of Nutrition.> > Singh RB, Mori H, Chen J, Mendis S, Moshiri M, Zhu S, Kim SH, Sy RG,Faruqui AM.> > Medical Hospital and Research Centre, Moradabad, India.> > There has been a rapid increase in coronary artery disease (CAD) inmost Asian countries in association with rapid economic development;however, there is no consensus of opinion on diet and lifestyleguidelines and desirable levels of risk factors for prevention of CADin these countries. The proportion of deaths due to cardiovasculardiseases in Asians may be about 15% but there are wide variations. Inview of the lower fat intake of the low-risk rural populations ofIndia, the People's Republic of China, Indonesia, Korea, Thailand andJapan compared with that of urban subjects, the limit for total energyfrom fat intake in an average should be 21% (7% each from saturated,polyunsaturated and mono-unsaturated fatty acids). The n-6: n-3 fattyacids ration should be < 5.0. The carbohydrates intake should be > 65%and mainly from complex carbohydrates (> 55%). A body mass index of 21kg/m2 may be safe but the range may be 18.5-23.0 kg/m2 and someonewith a body mass index > 23 kg/m2 should be considered overweight. Awaist: waist:hip ratio > 0.88 for men and > 0.85 for women should beconsidered to define central obesity. The desirable limit for serumtotal cholesterol may be 170 mg/dl, the borderline high level may be170-199 mg/dl and the high level 200 mg/dl or above. The correspondingvalues for low-density lipoprotein cholesterol may be 90, 90-109 and110 mg/dl or above. Fasting serum triglycerides may be < 150 mg/dl andhigh-density lipoprotein cholesterol > 35 mg/dl, which are close tothe levels in low-risk rural populations. Fasting blood glucose > 140mg/dl and postprandial blood glucose > 200 mg/dl may be consideredconditions for diabetes, and 140-200 mg/dl, glucose intolerance. Anintake of 400 g/day fruit, vegetables and legumes, mustard or soybeanoil (25 g/day) instead of hydrogenated fat, coconut oil or butter inconjunction with moderate physical activity (1255 kJ/day), cessationof tobacco consumption and moderation of alcohol intake may be aneffective package of remedies for prevention of CAD in Asians. PMID:9100083>

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Share on other sites

More disagreement among asian researchers on the reported role played

by coconut oil (and saturated

fats)in the development of CHD in different asian populations. A

fragile scientific concensus behind the recommendations

posted below?

Dietary intake and the risk of coronary heart disease among the

coconut-consuming Minangkabau in West Sumatra, Indonesia.

" The Case groups had significantly higher intakes of meats, eggs,

sugar, tea, coffee and fruits, but lower intakes of soy products, rice

and cereals compared to the controls. Coconut consumption as flesh or

milk was not different between cases and controls. The cases had

significantly higher intakes of protein and cholesterol, but lower

intake of carbohydrate. Similar intakes of saturated and unsaturated

fatty acids between the cases and controls indicated that the

consumption of total fat or saturated fat, including that from

coconut, was not a predictor for CHD in this food culture. However,

the intakes of animal foods, total protein, dietary cholesterol and

less plant derived carbohydrates were predictors of CHD. "

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=15563444 & itool=iconabstr & query_hl=16

Contrary to popular belief, CHD incidence is high amongst Asian

Indians. Yet it seems that those risk factors which have been

identified as playing a significant role in north american incidence

of heart disease are absent. One etiology in Asia, another in the US?

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=9770863 & query_hl=16

Could it be high homocysteine?

" Plasma homocysteine is a novel and independent risk factor for CHD in

Indian Asians, and may contribute to their increased CHD risk. Raised

homocysteine concentrations in Indian Asians may be related to their

reduced vitamin B12 and folate levels, implying that the increased CHD

risk in this group may be reduced by dietary vitamin supplementation. "

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=10683001 & query_hl=16

> >

> > FWIW,

> > J Cardiovasc Risk. 1996 Dec;3(6):489-94.

> >

> >

> > Recommendations for the prevention of coronary artery disease in

> Asians: a scientific statement of the International College of

Nutrition.

> >

> > Singh RB, Mori H, Chen J, Mendis S, Moshiri M, Zhu S, Kim SH, Sy RG,

> Faruqui AM.

> >

> > Medical Hospital and Research Centre, Moradabad, India.

> >

> > There has been a rapid increase in coronary artery disease (CAD) in

> most Asian countries in association with rapid economic development;

> however, there is no consensus of opinion on diet and lifestyle

> guidelines and desirable levels of risk factors for prevention of CAD

> in these countries. The proportion of deaths due to cardiovascular

> diseases in Asians may be about 15% but there are wide variations. In

> view of the lower fat intake of the low-risk rural populations of

> India, the People's Republic of China, Indonesia, Korea, Thailand and

> Japan compared with that of urban subjects, the limit for total energy

> from fat intake in an average should be 21% (7% each from saturated,

> polyunsaturated and mono-unsaturated fatty acids). The n-6: n-3 fatty

> acids ration should be < 5.0. The carbohydrates intake should be > 65%

> and mainly from complex carbohydrates (> 55%). A body mass index of 21

> kg/m2 may be safe but the range may be 18.5-23.0 kg/m2 and someone

> with a body mass index > 23 kg/m2 should be considered overweight. A

> waist: waist:hip ratio > 0.88 for men and > 0.85 for women should be

> considered to define central obesity. The desirable limit for serum

> total cholesterol may be 170 mg/dl, the borderline high level may be

> 170-199 mg/dl and the high level 200 mg/dl or above. The corresponding

> values for low-density lipoprotein cholesterol may be 90, 90-109 and

> 110 mg/dl or above. Fasting serum triglycerides may be < 150 mg/dl and

> high-density lipoprotein cholesterol > 35 mg/dl, which are close to

> the levels in low-risk rural populations. Fasting blood glucose > 140

> mg/dl and postprandial blood glucose > 200 mg/dl may be considered

> conditions for diabetes, and 140-200 mg/dl, glucose intolerance. An

> intake of 400 g/day fruit, vegetables and legumes, mustard or soybean

> oil (25 g/day) instead of hydrogenated fat, coconut oil or butter in

> conjunction with moderate physical activity (1255 kJ/day), cessation

> of tobacco consumption and moderation of alcohol intake may be an

> effective package of remedies for prevention of CAD in Asians. PMID:

> 9100083

> >

>

Link to comment
Share on other sites

More disagreement among asian researchers on the reported role played

by coconut oil (and saturated

fats)in the development of CHD in different asian populations. A

fragile scientific concensus behind the recommendations

posted below?

Dietary intake and the risk of coronary heart disease among the

coconut-consuming Minangkabau in West Sumatra, Indonesia.

" The Case groups had significantly higher intakes of meats, eggs,

sugar, tea, coffee and fruits, but lower intakes of soy products, rice

and cereals compared to the controls. Coconut consumption as flesh or

milk was not different between cases and controls. The cases had

significantly higher intakes of protein and cholesterol, but lower

intake of carbohydrate. Similar intakes of saturated and unsaturated

fatty acids between the cases and controls indicated that the

consumption of total fat or saturated fat, including that from

coconut, was not a predictor for CHD in this food culture. However,

the intakes of animal foods, total protein, dietary cholesterol and

less plant derived carbohydrates were predictors of CHD. "

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=15563444 & itool=iconabstr & query_hl=16

Contrary to popular belief, CHD incidence is high amongst Asian

Indians. Yet it seems that those risk factors which have been

identified as playing a significant role in north american incidence

of heart disease are absent. One etiology in Asia, another in the US?

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=9770863 & query_hl=16

Could it be high homocysteine?

" Plasma homocysteine is a novel and independent risk factor for CHD in

Indian Asians, and may contribute to their increased CHD risk. Raised

homocysteine concentrations in Indian Asians may be related to their

reduced vitamin B12 and folate levels, implying that the increased CHD

risk in this group may be reduced by dietary vitamin supplementation. "

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstra\

ct & list_uids=10683001 & query_hl=16

> >

> > FWIW,

> > J Cardiovasc Risk. 1996 Dec;3(6):489-94.

> >

> >

> > Recommendations for the prevention of coronary artery disease in

> Asians: a scientific statement of the International College of

Nutrition.

> >

> > Singh RB, Mori H, Chen J, Mendis S, Moshiri M, Zhu S, Kim SH, Sy RG,

> Faruqui AM.

> >

> > Medical Hospital and Research Centre, Moradabad, India.

> >

> > There has been a rapid increase in coronary artery disease (CAD) in

> most Asian countries in association with rapid economic development;

> however, there is no consensus of opinion on diet and lifestyle

> guidelines and desirable levels of risk factors for prevention of CAD

> in these countries. The proportion of deaths due to cardiovascular

> diseases in Asians may be about 15% but there are wide variations. In

> view of the lower fat intake of the low-risk rural populations of

> India, the People's Republic of China, Indonesia, Korea, Thailand and

> Japan compared with that of urban subjects, the limit for total energy

> from fat intake in an average should be 21% (7% each from saturated,

> polyunsaturated and mono-unsaturated fatty acids). The n-6: n-3 fatty

> acids ration should be < 5.0. The carbohydrates intake should be > 65%

> and mainly from complex carbohydrates (> 55%). A body mass index of 21

> kg/m2 may be safe but the range may be 18.5-23.0 kg/m2 and someone

> with a body mass index > 23 kg/m2 should be considered overweight. A

> waist: waist:hip ratio > 0.88 for men and > 0.85 for women should be

> considered to define central obesity. The desirable limit for serum

> total cholesterol may be 170 mg/dl, the borderline high level may be

> 170-199 mg/dl and the high level 200 mg/dl or above. The corresponding

> values for low-density lipoprotein cholesterol may be 90, 90-109 and

> 110 mg/dl or above. Fasting serum triglycerides may be < 150 mg/dl and

> high-density lipoprotein cholesterol > 35 mg/dl, which are close to

> the levels in low-risk rural populations. Fasting blood glucose > 140

> mg/dl and postprandial blood glucose > 200 mg/dl may be considered

> conditions for diabetes, and 140-200 mg/dl, glucose intolerance. An

> intake of 400 g/day fruit, vegetables and legumes, mustard or soybean

> oil (25 g/day) instead of hydrogenated fat, coconut oil or butter in

> conjunction with moderate physical activity (1255 kJ/day), cessation

> of tobacco consumption and moderation of alcohol intake may be an

> effective package of remedies for prevention of CAD in Asians. PMID:

> 9100083

> >

>

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Share on other sites

The interesting thing is, after reading an enormous number of articles, I can walk down the street and pretty well guess who has HTN, CAD, type II.

It doesn't take a rocket scientist to see it.

They are not overweight, they are HUGE.

They eat too much.

Regards.

[ ] Re: coconut oil and CAD

More disagreement among asian researchers on the reported role playedby coconut oil (and saturatedfats)in the development of CHD in different asian populations. Afragile scientific concensus behind the recommendationsposted below? Dietary intake and the risk of coronary heart disease among thecoconut-consuming Minangkabau in West Sumatra, Indonesia."The Case groups had significantly higher intakes of meats, eggs,sugar, tea, coffee and fruits, but lower intakes of soy products, riceand cereals compared to the controls. Coconut consumption as flesh ormilk was not different between cases and controls. The cases hadsignificantly higher intakes of protein and cholesterol, but lowerintake of carbohydrate. Similar intakes of saturated and unsaturatedfatty acids between the cases and controls indicated that theconsumption of total fat or saturated fat, including that fromcoconut, was not a predictor for CHD in this food culture. However,the intakes of animal foods, total protein, dietary cholesterol andless plant derived carbohydrates were predictors of CHD."http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstract & list_uids=15563444 & itool=iconabstr & query_hl=16"A total of 93 eligible cases (62 men and 31 women) in the Case group and 189 subjects (113 men and 76 women) in the Control group were recruited. "

Too small a group to draw conclusions.

Contrary to popular belief, CHD incidence is high amongst AsianIndians. Yet it seems that those risk factors which have beenidentified as playing a significant role in north american incidenceof heart disease are absent. One etiology in Asia, another in the US?http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstract & list_uids=9770863 & query_hl=16

"etiologies" are diff in Finland, the Mediterranean, France, Blacks, American Indians as well. I refrain from drawing conclusions about the diff in ethnic groups. Especially, since I found out how many centenarians we have in the U.S. In 2000 we have a system with a very high weighting for accuracy since SS recipients receive money and must have (had) birth certificates. Along with a census every 10 years.

Someone tells me they're 100 yo they better be in the 1910 census, and the 1920 and 1930. They would have entered the SS in 1935 in all probablity.

In 5 yrs we get the 1940 data, and you'll find me in it.

Could it be high homocysteine?"Plasma homocysteine is a novel and independent risk factor for CHD inIndian Asians, and may contribute to their increased CHD risk. Raisedhomocysteine concentrations in Indian Asians may be related to theirreduced vitamin B12 and folate levels, implying that the increased CHDrisk in this group may be reduced by dietary vitamin supplementation."http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstract & list_uids=10683001 & query_hl=16IMO, plasma homocysteine is a result not a cause of CHD. That is based on the fact there is a methionine, cysteine, homocysteine biochem loop. I haven't yet seen the explanation of why the body chooses to maximize one component of that loop. Just an association. >> Most interesting recommendations. Some fellow Asian researchers(India> is after all in Asia)may be allowed to disagree.> > >http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=9316363 & dopt=Citation>

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Share on other sites

The interesting thing is, after reading an enormous number of articles, I can walk down the street and pretty well guess who has HTN, CAD, type II.

It doesn't take a rocket scientist to see it.

They are not overweight, they are HUGE.

They eat too much.

Regards.

[ ] Re: coconut oil and CAD

More disagreement among asian researchers on the reported role playedby coconut oil (and saturatedfats)in the development of CHD in different asian populations. Afragile scientific concensus behind the recommendationsposted below? Dietary intake and the risk of coronary heart disease among thecoconut-consuming Minangkabau in West Sumatra, Indonesia."The Case groups had significantly higher intakes of meats, eggs,sugar, tea, coffee and fruits, but lower intakes of soy products, riceand cereals compared to the controls. Coconut consumption as flesh ormilk was not different between cases and controls. The cases hadsignificantly higher intakes of protein and cholesterol, but lowerintake of carbohydrate. Similar intakes of saturated and unsaturatedfatty acids between the cases and controls indicated that theconsumption of total fat or saturated fat, including that fromcoconut, was not a predictor for CHD in this food culture. However,the intakes of animal foods, total protein, dietary cholesterol andless plant derived carbohydrates were predictors of CHD."http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstract & list_uids=15563444 & itool=iconabstr & query_hl=16"A total of 93 eligible cases (62 men and 31 women) in the Case group and 189 subjects (113 men and 76 women) in the Control group were recruited. "

Too small a group to draw conclusions.

Contrary to popular belief, CHD incidence is high amongst AsianIndians. Yet it seems that those risk factors which have beenidentified as playing a significant role in north american incidenceof heart disease are absent. One etiology in Asia, another in the US?http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstract & list_uids=9770863 & query_hl=16

"etiologies" are diff in Finland, the Mediterranean, France, Blacks, American Indians as well. I refrain from drawing conclusions about the diff in ethnic groups. Especially, since I found out how many centenarians we have in the U.S. In 2000 we have a system with a very high weighting for accuracy since SS recipients receive money and must have (had) birth certificates. Along with a census every 10 years.

Someone tells me they're 100 yo they better be in the 1910 census, and the 1920 and 1930. They would have entered the SS in 1935 in all probablity.

In 5 yrs we get the 1940 data, and you'll find me in it.

Could it be high homocysteine?"Plasma homocysteine is a novel and independent risk factor for CHD inIndian Asians, and may contribute to their increased CHD risk. Raisedhomocysteine concentrations in Indian Asians may be related to theirreduced vitamin B12 and folate levels, implying that the increased CHDrisk in this group may be reduced by dietary vitamin supplementation."http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=pubmed & dopt=Abstract & list_uids=10683001 & query_hl=16IMO, plasma homocysteine is a result not a cause of CHD. That is based on the fact there is a methionine, cysteine, homocysteine biochem loop. I haven't yet seen the explanation of why the body chooses to maximize one component of that loop. Just an association. >> Most interesting recommendations. Some fellow Asian researchers(India> is after all in Asia)may be allowed to disagree.> > >http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve & db=PubMed & list_uids=9316363 & dopt=Citation>

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