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How Bush administration lied in a cancer case so advisor made money

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Hello,

Please pray for me because I am showing how the Bush

administration lied to the American people in court about

methylmercury causing cancer when we knew it did and how it did it.

The Bush administration places money over the lives of the

American people it has the duty to defend under the U S constitution.

It will even practice perjury in order for members of its

administration to make money. Steve Burd was one of the Bush

administration Home Land Security advisors. The company he is CEO of

is Safeway and the state of Ca. wanted to put warning labels on some

of its food products so it went to court because Safeway refused to do

so. The federal government wrote a letter to defend Safeway and in

it the federal government stated that what the state of Ca. claimed

had no scientific basis to it and also that the federal government did

not want fish sells to go down. The claim that there is no scientific

basis is not true. We have known for a long time that methylmercury

and mercury binds to DNA. We have various scientific methods to show

that this occurs. For example in circular dichroism studies one uses

how items absorb beams of polarized light in order to determine the

structure of an item. Different items absorb light in different ways

and at different wave lenghts. Another way is to use DNA

sedimentation rates. Different items have different rates of moving

through sediments and that is the reason for using this way to look at

things. The next one I will mention is nuclear magnetic spectroscopy

(nmr). Nmr spectroscopy looks at how items absorb radio waves in a

strong magnetic field. The last one I will mention is thermal

analyses. Heat is used to look at things because things behave

differently at different temperatures. The point I want to make here

is that all these different ways of looking at things show that

mercury binds to the DNA and anything that binds to the DNA can

potentially cause problems to the DNA. We have known that mercury and

methylmercury mutate genes years before the court case. Safeway and

the Bush administration do not care if your DNA gets messed up due to

methylmercury and mercury. If you do not believe what I state look up

the Ca. court case the People of the State of California v Tr-Union

Seafoods, LLC, et al. (case no. CGC-04-432394) since Safeway was in

that court case and claimed that DNA damage does not happen by

methylmercury.

Over two hundred years ago we knew that mercury reacted with

oxygen. Studies by two of the most famous scientists in the history

of science found this to be important in their studies of oxygen. J.

Priestley who is best known for the discoverer of oxygen and doing

some of the first studies of photosynthesis used this fact in his

studies of oxygen. A. L. Lavoisier who is known for his work in

animal respiration also found mercury's ability to react with oxygen

useful in his studies. One can find these people's names in high

school science textbooks. The reason I state this is the fact that

this reaction is one of the reasons why mercury causes birth defects,

cancer and miscarriages. Lets look at the periodic table because

things are grouped together by the fact they have common traits.

Mercury is found in the transitional metal group. A common reaction

to this group is the Fenton reaction. It is the name for the reaction

of these metals with oxygen and it creates reactive oxygen species.

Reactive oxygen species is what causes the destruction of DNA and thus

causes what the state of Ca. claimed in court. Right next to mercury

is cadmium which is a known cancer agent and it causes cancer by doing

this reaction. Mercury does the Fenton reaction at the temperature

which living things live at but at a slow rate according to a

chemistry book. Also various science papers that I have read state

reactive oxygen species levels go up due to methylmercury.

Mercury mutates various genes found mutated in cancer patients.

Some of these genes are p53, Fos, Jun and L1. The mutation of either

Fos or Jun genes can cause tumor growth. L1 mutaions are due to

retrotransposition of DNA which causes lots of problems to the DNA due

to the misreading of DNA and this can be found in breast and blood

cancers.

We have population studies out of Japan that show areas in Japan

that had methylmercury problems have a higher rate of cancer and

prebirth deaths. I have listed a couple of them below in the listing

of science papers to back up my statements.

Now I should state some textbooks state that a change in a

single gene is not important but that is not true. The last time I

checked there was around six thousand human diseases caused by a

single bases change of a gene. For example there are osteogenesis

imperfecta (brittle bone disease) and sickle cell anemia diseases

caused by a single base changed.

Mercury also causes nondisjunction of the chromosomes. This has

been known for decades and it is because it impairs spindle function

thus the cell cannot divide correctly. Various human diseases are

caused by the nondisjunction of the chromosomes such as Down's

syndrome, 's syndrome and Patau's syndrome.

I should also state that methylmercury is found in cow's milk

and that is one of the reasons why cow's milk causes cancer and we

have known for decades (since the 1980's) that cow's milk causes

cancer. Cow's milk has low rates of lots of pollutions in it that are

known to cause cancer plus excess calcium in a cell causes tumor

growth so it is a great chemical stew for causing cancer. Last year I

sent out an email with 20 abstracts of science studies showing that

milk causes cancer. For example in 1984 DA Snowdon and etc. wrote

Diet, Obesity and Risk Of Fatal Prostate Cancer which was published in

AM. J. Epidermiol.. A good one showing that intake of calcium is

associated gene mutations is by E Kampman and etc. published by

Carcinogenesis is Animal Products and K-Ras Codon 12 and 13 Mutations

In Colon Carcinomas (2000 Feb. 21(2):307-9. To show you that over

twenty years of studies linking milk intake to cancer I will mention

Giovannucci and etc. study published in Cancer Epidemiol. Biomarkers

Prev. in 2006 (Feb. 15(2):203-10) entitled A Prospective Study Of

Calcium Intake and Incident and Fatal Prostate Cancer which was done

in the US.

Marcus Horton

P O Box 84

Salinas, Ca 93902

I should also state that methylmercury loses its methyl

functional group in the human body and becomes just mercury so one has

to look at studies that have to do with both when looking at this

issue. Also mercury also binds to other functional groups once inside

the human body.

List of some studies to back up the statements in the above

article. I have a longer list of studies showing what I stated is true.

Mercury binding to DNA

A. Casadevall and L. A. Day Silver and mercury probing of

deoxyribonucleic acid structures in the filamentous viruses fd, If1,

IKe, Xf, Pf1, and Pf3.

1983 Biochemistry Sep 27;22(20):4831-42.

D. Ding and F. S. A circular dichroism study on the structure

of DNA and the nucleosomal core particle using Hg (ll) and Ag (l)

1980 Biochim. Biophys. Acta. Nov 14; 610(1):72-80.

D. Ding and F. S. Electric dichroism and sedimentation velocity

studies of DNA-Hg(II) and DNA-Ag(I) complexes.

1980 Biochim. Biophys. Acta. Nov.14;610(1):64-71.

H. Torigoe, A. Ono and K. Kawahashi Thermodynamic analyses of the

specific interaction between T:T mismatch base pair and mercury (II)

cation: toward the efficient detection of single nucleotide

polymorphism (1).

2004 Nucleic Acids Symp. Ser. (Oxf.) (48):275-6.

P. R. Young, U. S. Nandi and N. R. Kallenbach Binding of mercury(II)

to poly (dA-dT) studied by proton nuclear magnetic resonance.

1982 Biochemistry Jan. 5;21(1):62-6.

Early studies showing mercury causes nondisjunction of the

chromosomes.

J. Magnusson and C. Ramel Genetic variation in the susceptibility to

mercury and other metal compounds in Drosophila melanogaster.

1986 Teratog. Carcinog. Mutagen 6(4);289-305.

C. Ramel and J. Magnusson Chemical induction of nondisjunction in

drosopila.

1979 Environ. Health Perspect. Aug.;31:59-66.

Some studies showing chromosome problems due to mercury.

H. C. Wulf and others. Sister chromatid exchange (SCE) in Greenlandic

Eskimos. Dose-response relationship between SCE and seal diet,

smoking and blood cadmium and mercury concentrations.

1986 Sci. Total Environ. Jan; 48(1-2):81-94.

L. Verschaeve, M. Kirsch-Volders and C. ne Mercury-induced

segregational errors of chromosomes in human lymphocytes and in Indian

muntjac cells.

1984 Toxicol. Lett. Jun;21(3):247-53.

A. Renzoni, F. Zino and E. Franchi Mercury levels along the food chain

and risk for exposed populations.

1998 Environ. Res. May;77(2):68-72.

J. R. Lazutka and others. Chromosomal aberrations and sister-chromatid

exchanges in Lithuanian populations: effects of occupational and

environmental exposures.

1999 Mutat. Res. Sep. 30;445(2):225-39.

One of the population cancer studies we have dealing with

methylmercury. Since methylmercury goes after microtubles and causes

microtubular damage one should find chronic liver damage as well and

this study also shows that.

H. Tamashiro, M. Arakaki, M. Futatsuka and E. S. Lee Methylmercury

exposure and mortality in southern Japan: a close look at causes of death.

1986 J. Epidemiol. Community Health Jun:40(2):181-5.

One of the population studies showing methylmercury causes fetuses to

die before being born.

M. Sakamoto, A. Nakano and H. Akagi Declining Minamata Male Birth

Ratio Associated With Increased Male Fetal Death Due To Heavy

Methylmercury Pollution.

2001 Environmental Research.

A couple of studies showing that methylmercury causes an increase in

the reactive oxygen species in the cell. It does this by various ways.

T. Sarafian and M. A. Verity Oxidative mechanisms underlying methyl

mercury neurotoxicity.

1991 Int. J. Dev. Neurosci. 9(2) :147-53.

S. Gasso and others. Antioxidant compounds and Ca(2+) pathway blockers

differentially protect against methylmercury and mercuric chloride

neurotoxicity.

2001 J. Neurosci. Res. Oct 1:66(1):135-45

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