Guest guest Posted January 9, 2008 Report Share Posted January 9, 2008 Very interesting read and all of the symptoms described are the same as my self and my co-workers. I wonder why I was turned down flat when I asked for a NIOSH eval. > > 1997- > Cowlitz County Health Department > http://www.cdc.gov/niosh/hhe/reports/pdfs/1997-0048-2641.pdf > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 10, 2008 Report Share Posted January 10, 2008 Thank you! This is very good. But, here we are, more than ten years later, in a different administration. Have we made progress? No, if anything, we have gone backwards, in the sense that NIOSH probably would not have published that paper today, given the political environment. They have that new rule requiring upstream approval before saying anything that " might have an economic impact " , reagrdless of its safety implications. They also have influential and well funded business organizations like the ACOEM and the Manhattan Institute breathing down their necks. > 1997- > Cowlitz County Health Department > http://www.cdc.gov/niosh/hhe/reports/pdfs/1997-0048-2641.pdf > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 10, 2008 Report Share Posted January 10, 2008 your welcome Live, thought I posted it before but dont remember. it amazes me just how much info. there is pre-acoem-ratstudy. the first toxic mold case claiming brain injury sure brought worms out of their holes. I dont think it's ever been about weither brain injury can accur or not, isn't that hard to see how that happens, it was all about the money. --- In , LiveSimply <quackadillian@...> wrote: > > Thank you! This is very good. > > But, here we are, more than ten years later, in a different > administration. Have we made progress? > > No, if anything, we have gone backwards, in the sense that > NIOSH probably would not have published that paper today, given the > political environment. > > They have that new rule requiring upstream approval before saying anything > that " might have an economic impact " , reagrdless of its safety implications. > > They also have influential and well funded business organizations like > the ACOEM and the Manhattan Institute breathing down their necks. > > > 1997- > > Cowlitz County Health Department > > http://www.cdc.gov/niosh/hhe/reports/pdfs/1997-0048-2641.pdf > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 10, 2008 Report Share Posted January 10, 2008 Jeanine, Why do you say that? There have been numerous studies on neuro effects of mold. Both human and animal. Animal studies can often be exterpolated to people. We have the same chemistry. And remember, if a person was an animal, they would have lost their job and probably everything else LONG before they would start showing the kinds of visually obvious effects that scientists monitor for toxicity. Animals can't talk! The kinds of criteria that they have at each point in time to get your case into court keeps getting raised, as science keeps discovering more. The whole point of that seems to be KILLING HOPE OF JUSTICE FOR PEOPLE. On Jan 10, 2008 11:06 AM, who <jeaninem660@...> wrote: > your welcome Live, thought I posted it before but dont remember. it > amazes me just how much info. there is pre-acoem-ratstudy. > the first toxic mold case claiming brain injury sure brought worms > out of their holes. I dont think it's ever been about weither brain > injury can accur or not, isn't that hard to see how that happens, it > was all about the money. > --- In , LiveSimply <quackadillian@...> > Mycotoxins: Can Low Level, Non-lethal Exposure Result in Parkinsonism? ABSTRACT OF INVITED LECTURE PRESENTED AT WORLD FEDERATION OF NEUROLOGY, SYMPOSIUM ON NEUROTOXICOLOGY, SYDNEY, AUSTRALIA (11/2005) -Ramos, J1,2, Sava, V1,2 1 Dr. -Ramos, Director, University of South Florida Huntington's Disease Center of Excellence, Tampa, Fl., University of South Florida, Tampa FL, USA 2 Haley VA Hospital, Tampa, FL, USA Mycotoxins are fungal metabolites with pharmacological activities that have been utilized in production of antibiotics, growth promoters, and other kinds of drugs; still others have been developed as biological and chemical warfare agents. Bombs and ballistic missiles loaded with mycotoxins were believed to be deployed by Iraq during Operation Desert Storm. In light of the excess incidence of amyotrophic lateral sclerosis in young Gulf War veterans, it is important not to forget the potential neurotoxic effects of low doses of mycotoxins. Ochratoxin A (OTA) is a common mycotoxin with a long biological half-life and mitochondrial toxicity. It is found in damp environments and moldy foods (contaminated with Aspergillus and Penicillium). As part of a program to study the brain's DNA repair response to a range of neurotoxicants, we measured the effects of acute and chronic administration of OTA on the activity of the DNA repair enzyme, oxyguanosine glycosylase (OGG1), in cerebellum (CB), cortex (CX), hippocampus (HP), midbrain (MB), caudate/putamen (CP) and pons/medulla (PM) of Swiss ICR mice. Based on the observation of pronounced alterations of OGGI in the CP, we then measured DA and its metabolites, homovanillic acid and (HVA) dihydroxyphenylacetic (DOPAC) by HPLC in the same regions. Acute administration of OTA (0.37 to 6 mg/kg ip) resulted in a dose-dependent depletion of DA with the greatest effects in CP and frontal cortex. The highest dose caused a 6 fold decrease of DA concentration in CP. The ED50 for DA depletion ranged between 3 and 4 mg/kg. OGG1 activities, which served as an index of repair of oxidative DNA damage, were associated with changes in DA, HVA and DOPAC concentration. Ongoing studies of chronic exposure to OTA and its effects on DA content may provide new insights into the pathogenesis of Parkinson's disease. Supported by: Dept of Defense Grant #DAMD17-03-1-0501 and VA Merit Review Grant Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 10, 2008 Report Share Posted January 10, 2008 Live, what? I was talking about all the people that lost their mold injury cases because of the acoem study being used by defence. > > > your welcome Live, thought I posted it before but dont remember. it > > amazes me just how much info. there is pre-acoem-ratstudy. > > the first toxic mold case claiming brain injury sure brought worms > > out of their holes. I dont think it's ever been about weither brain > > injury can accur or not, isn't that hard to see how that happens, it > > was all about the money. > > --- In , LiveSimply <quackadillian@> > > > > > > > > Mycotoxins: Can Low Level, Non-lethal Exposure Result in Parkinsonism? > > ABSTRACT OF INVITED LECTURE PRESENTED AT WORLD FEDERATION OF > NEUROLOGY, SYMPOSIUM ON NEUROTOXICOLOGY, SYDNEY, AUSTRALIA (11/2005) > > > -Ramos, J1,2, Sava, V1,2 > > 1 Dr. -Ramos, Director, University of South Florida > Huntington's Disease Center of Excellence, Tampa, Fl., University of > South Florida, Tampa FL, USA > 2 Haley VA Hospital, Tampa, FL, USA > > > Mycotoxins are fungal metabolites with pharmacological activities that > have been utilized in > production of antibiotics, growth promoters, and other kinds of drugs; > still others have been > developed as biological and chemical warfare agents. Bombs and > ballistic missiles loaded with > mycotoxins were believed to be deployed by Iraq during Operation > Desert Storm. In light of the > excess incidence of amyotrophic lateral sclerosis in young Gulf War > veterans, it is important not > to forget the potential neurotoxic effects of low doses of mycotoxins. > > Ochratoxin A (OTA) is a > common mycotoxin with a long biological half-life and mitochondrial > toxicity. It is found in damp > environments and moldy foods (contaminated with Aspergillus and > Penicillium). As part of a > program to study the brain's DNA repair response to a range of > neurotoxicants, we measured the > effects of acute and chronic administration of OTA on the activity of > the DNA repair enzyme, > oxyguanosine glycosylase (OGG1), in cerebellum (CB), cortex (CX), > hippocampus (HP), midbrain > (MB), caudate/putamen (CP) and pons/medulla (PM) of Swiss ICR mice. Based on the > observation of pronounced alterations of OGGI in the CP, we then > measured DA and its > metabolites, homovanillic acid and (HVA) dihydroxyphenylacetic (DOPAC) > by HPLC in the same > regions. Acute administration of OTA (0.37 to 6 mg/kg ip) resulted in > a dose-dependent depletion > of DA with the greatest effects in CP and frontal cortex. > > The highest dose caused a 6 fold > decrease of DA concentration in CP. The ED50 for DA depletion ranged > between 3 and 4 mg/kg. > OGG1 activities, which served as an index of repair of oxidative DNA > damage, were associated > with changes in DA, HVA and DOPAC concentration. Ongoing studies of > chronic exposure to > OTA and its effects on DA content may provide new insights into the > pathogenesis of Parkinson's > disease. > > Supported by: Dept of Defense Grant #DAMD17-03-1-0501 and VA Merit Review Grant > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 11, 2008 Report Share Posted January 11, 2008 Live, I hope you weren't implying that towards me. if so, you need to go back in the archives and refresh your memory on who worked their ass off trying to bring answers to this group while hardly being able to even remember how to spell. now eberybody a expert aren't they.I dont even know how or why I servived what happened to my head and I'm not sure I well. I have mcs to mold and chemicals, the paths the csf leaks created through my head to my schull,sinuses and ears have no barriorsm let alone the real brain barriors being broke down. and the emergency surgery that I should of had when this happened, didn't. and guess what, more csf leaks could be caused by the surgery, antifungals even if they would be of help wont stop the chemical exposures I may face while going through and getting through the sergery, than we have the little problem of fungi and bacteria in the hospitals, and gee, besides the other damage to my eyes from brain damage, I have fricken floresent green cateracts that can't be removed until the csf leaks are patched,mucoceles are dealt with. and the infections in my body from all sources are under control, because they keep going to my head weither it's from what in my body or what I breath into my body. now I dont know if the csf paths to the ears can even be foxed and my nasal roof probably needs a tissue graft cause my olfactory bulbs+ are gone, but a graft might not take there can be alot more brain injury to this than just cognitive issues. ----------- surgery http://archotol.ama-assn.org/cgi/content/full/129/5/576 csf rhinorrhea,brain injury http://www.bcm.edu/oto/grand/120398.html --------------------------------------- http://www.thefreelibrary.com/Spontaneous+bilateral+intrasphenoidal+la teral+encephaloceles:+CT+and+...-a0127352608 http://archotol.ama-assn.org/cgi/content/full/129/5/576 otolaryngology http://www.audio- digest.org/pages/htmlos/00207.1.10516480180621415858/OT3923 > > > > > your welcome Live, thought I posted it before but dont remember. > it > > > amazes me just how much info. there is pre-acoem-ratstudy. > > > the first toxic mold case claiming brain injury sure brought > worms > > > out of their holes. I dont think it's ever been about weither > brain > > > injury can accur or not, isn't that hard to see how that > happens, it > > > was all about the money. > > > --- In , LiveSimply <quackadillian@> > > > > > > > > > > > > > > > Mycotoxins: Can Low Level, Non-lethal Exposure Result in > Parkinsonism? > > > > ABSTRACT OF INVITED LECTURE PRESENTED AT WORLD FEDERATION OF > > NEUROLOGY, SYMPOSIUM ON NEUROTOXICOLOGY, SYDNEY, AUSTRALIA (11/2005) > > > > > > -Ramos, J1,2, Sava, V1,2 > > > > 1 Dr. -Ramos, Director, University of South Florida > > Huntington's Disease Center of Excellence, Tampa, Fl., University of > > South Florida, Tampa FL, USA > > 2 Haley VA Hospital, Tampa, FL, USA > > > > > > Mycotoxins are fungal metabolites with pharmacological activities > that > > have been utilized in > > production of antibiotics, growth promoters, and other kinds of > drugs; > > still others have been > > developed as biological and chemical warfare agents. Bombs and > > ballistic missiles loaded with > > mycotoxins were believed to be deployed by Iraq during Operation > > Desert Storm. In light of the > > excess incidence of amyotrophic lateral sclerosis in young Gulf War > > veterans, it is important not > > to forget the potential neurotoxic effects of low doses of > mycotoxins. > > > > Ochratoxin A (OTA) is a > > common mycotoxin with a long biological half-life and mitochondrial > > toxicity. It is found in damp > > environments and moldy foods (contaminated with Aspergillus and > > Penicillium). As part of a > > program to study the brain's DNA repair response to a range of > > neurotoxicants, we measured the > > effects of acute and chronic administration of OTA on the activity > of > > the DNA repair enzyme, > > oxyguanosine glycosylase (OGG1), in cerebellum (CB), cortex (CX), > > hippocampus (HP), midbrain > > (MB), caudate/putamen (CP) and pons/medulla (PM) of Swiss ICR mice. > Based on the > > observation of pronounced alterations of OGGI in the CP, we then > > measured DA and its > > metabolites, homovanillic acid and (HVA) dihydroxyphenylacetic > (DOPAC) > > by HPLC in the same > > regions. Acute administration of OTA (0.37 to 6 mg/kg ip) resulted > in > > a dose-dependent depletion > > of DA with the greatest effects in CP and frontal cortex. > > > > The highest dose caused a 6 fold > > decrease of DA concentration in CP. The ED50 for DA depletion ranged > > between 3 and 4 mg/kg. > > OGG1 activities, which served as an index of repair of oxidative DNA > > damage, were associated > > with changes in DA, HVA and DOPAC concentration. Ongoing studies of > > chronic exposure to > > OTA and its effects on DA content may provide new insights into the > > pathogenesis of Parkinson's > > disease. > > > > Supported by: Dept of Defense Grant #DAMD17-03-1-0501 and VA Merit > Review Grant > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted January 11, 2008 Report Share Posted January 11, 2008 Jeanine, I was just trying to say that the people who claim that there has not been any (not enough, not a lot of, etc.) research done, are wrong, Not you. There has not been human research done for obvious reasons. Its worthwhile to watch the NTP panel discussion and you can see how complicated and (IMO) needlessly drawn-out, a process this is. For example, they want to do animal studies with dried out mold BECAUSE THEY DON " T WANT TO EXPOSE THE ANIMALS TO HARMFUL HUMIDITY LEVELS!!!!! even though many scientists have said that they will miss out on an important aspect of the experiment, MVOCs, because the mold will be necessity, not be alive.. Words don't describe how stupid that is. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 7, 2010 Report Share Posted June 7, 2010 Scar Eraser Life happens -- and, as we all know, it often leaves scars. We can see them on our knees, elbows and elsewhere, but did you know that you can get scars inside your body, too? These sometimes occur after surgery or an injury such as a broken bone, but other times in response to less obvious insults, including simple inflammation. Heart attack and stroke leave scars -- but in fact, these are internal injuries. Inflammatory conditions, including asthma, sinusitis, Alzheimer’s disease, multiple sclerosis, arthritis and others also leave destructive scar tissue in their wake. I recently learned more about these internal scars and some of the surprising ways that they can affect our health -- and about a therapy that can speed healing while smoothing scars on both the inside and outside of the body. The Purpose of Scars Scars are the body’s typical and appropriate response to injury -- which is to generate healing fibers (called fibrin) where damage has been wrought. These eventually weave together to heal the damage done, but the results won’t be seamless -- remember how it looked when your grandmother used to darn socks? In the case of your body, however, cells are continually regenerating, and over time such scars should disappear until there’s little if any evidence left. Our bodies accomplish this by producing enzymes (called fibrinolases) that dissolve scar tissue and replace it with healthy tissue. Sometimes, though, our natural healing powers aren’t up to par. This is where the intriguing therapy comes into play -- they can be bolstered by using an enzyme isolated from the silkworm. It’s called serrapeptase -- this is the substance that allows the worm to dissolve its carapace so that it can become a butterfly. Since it comes from the same class of enzymes as those produced to heal the human body, supplements have been formulated from it, I was told by Daily Health News contributing editor L. Rubman, ND. While the transformative powers of serrapeptase aren’t quite so lyrical for we human beings, they can improve our health. clean-up crew Serrapeptase seems to help healing along. As a way to visualize how, Dr. Rubman suggests picturing serrapeptase as an internal carpet-cleaning formula. With repeated applications and some scrubbing, a stain -- or, in this case, scar tissue -- will begin to dissolve and dissipate. In the wake of all that activity, however, serrapeptase is apt to leave a mess behind. This detritus has a tendency to accumulate and can end up thickening your blood. That’s why Dr. Rubman routinely prescribes a second enzyme to be taken along with serrapeptase -- nattokinase. Nattokinase is made from the traditional Japanese fermented soy product, natto. It’s like a vacuum cleaner, he says -- along with plasmin, the body’s own natural anticoagulant or blood thinner, nattokinase serves as a clean-up crew to dissolve errant fibrin and other tissue particles. This completes serrapeptase’s job and encourages good blood flow throughout the body. Who Needs Enzymes? At certain times -- typically with illness, stress, when you’re eating poorly or just with age -- your body’s natural production of enzymes such as serrapeptase and nattokinase may decline, but taking supplements can help you pick up the slack. Dr. Rubman and I discussed how these enzymes can be used to improve various health problems... Respiratory disease: Serrapeptase thins the dense mucus often present in people with chronic asthma, bronchitis, sinusitis and other pulmonary diseases. Studies show that it helps repair damage to the structure and function of delicate mucous linings. Pain and inflammation: Together, serrapeptase and nattokinase have antiinflammatory properties that serve as natural analgesics. They ease pain by relieving swelling, fluid accumulation and pressure. Serrapeptase also speeds tissue repair and blocks the release of pain-inducing chemicals called amines from inflamed tissue. Cardiovascular health: Dr. Rubman prescribes a combination of serrapeptase and nattokinase to reduce arterial plaque and break up small blood clots that clog arteries in the cardiovascular system. Together these enzymes thin the blood, promote circulation and bring down levels of LDL cholesterol (the "bad" cholesterol) and C-reactive protein (CRP, a marker of inflammation and heart disease). (Note: When prescribing these two enzymes together for his patients, Dr. Rubman likes a product called Neprinol, available from the manufacturer at www.Arthur.com and at The Vitamin Shoppe stores.) Women’s wellness: Some women experience an imbalance between estrogen and progesterone that triggers inflammatory responses such as the development of scar tissue. This abnormal tissue may accumulate in the breasts or uterus, where it can lead to fibrous breasts or uterine fibroids. To control these conditions along with the pain and pressure that many women experience with menstruation, Dr. Rubman prescribes FibroVera (also from www.Arthur.com and The Vitamin Shoppe), a product he helped create, that combines serrapeptase, nattokinase and the enzymes bromelain (extracted from pineapple) and papain (from papaya). Use Under Medical Supervision While serrapeptase and nattokinase are safe and effective supplements for most people, Dr. Rubman says that they should be avoided by those with a bleeding disorder or who are taking blood-thinning drugs such as warfarin (Coumadin). Other interactions are sometimes (though rarely) problematic, including those with aspirin, fish oil and vitamin E. According to Dr. Rubman, the most effective enzymes are "enteric-coated," which means that the active ingredients are covered with a protective layer that lets them survive exposure to stomach acid and pass into the intestine intact. Enzymes should be taken 30 minutes before or 90 minutes or so after eating. Supplementation with enzymes can be complicated, Dr. Rubman said, so they should be taken only under the supervision of a trained professional... that said, these powerful enzymes have the potential to improve your health in a way that you may, indeed, find transformative. Source(s): L. Rubman, ND, founder and director, Southbury Clinic for Traditional Medicines, Southbury, Connecticut. www.southburyclinic.com. Suzi List Owner health What is a weed? A plant whose virtues have not yet been discovered. Quote Link to comment Share on other sites More sharing options...
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