I have direct phone number to Dr. 1-512-220-2434 ext. 9

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He must have called me. However, my caller id picked it up, I returned the call and it was the hospital. I went to his mailbox number 9 and left a message saying there are a lot of women who would like to be involved in this study. Patty Marriot is are moderator. Etc. Anyone interested get on it. I feel its our duty to the world. Love, also check out his website at www.immunotoxicology.com Maybe by participating in the research we'll get free tests run. Who knows, I just want to save a life. anita kessler <AnitaK001@...> wrote:

I just sent an e-mail to the Human Adjuvant Disease Corporation and asked them for a contact number. It is located in Florida. Maybe you have already tried this. If you have let me know. If not I will call them when they respond and try to get a number for Dr. . Any other questions anyone wants me to ask about the study or anything? Love, Anita

Re: Re: artical found on another group 2nd one

I went on line about 2 yrs. ago and tried to track down Dr. . No luck.LyndaAt 09:21 AM 7/15/2004, you wrote:>> > >Human Adjuvant Disease Corp. , M.D.> > >> > >Human Adjuvant Disease Corp. Update> > >We are in the process of establishing Human Adjuvant Disease Corp.> > >chapters in all 50 states and in Canada. If you would like

to>become> > >involved in this project,please contact us.> > >> > >The Human Adjuvant Disease Corp. is now accepting saline and>silicone> > >implant recipients to participate in a 5 year research study. If>you> > >are> > >a healthy patient with implants, a patient who is ill with>implants,> > >or> > >if you had your implants removed, please contact us for more> > >information. Enrollment in this study will continue until July>2004> > >> > >INFORMATIVE LINKS> > >> > >SILICONE/SALINE NEWS SPRING 2001> > >SILICONE/SALINE NEWS FALL 2001> > >SILICONE/SALINE NEWS 2002> > >DR. MYHILL- SILICONE DISEASES- UNITED KINGDOM> > >IMPLANT~INFO~NET> > >IMPLANT~INFO~NET FORUM> > >MILLENNIUM HEALTHCARE> >

>TEMPLE OF HEALTH> > >INFO-IMPLANTS MAMMAIRES INC.> > >MEDWATCH-REPORT MEDICAL PROBLEMS FROM IMPLANTS> > >IMPLANT INFORMATION PROJECT> > >HUMANTICS FOUNDATION> > >TOXIC DISCOVERY NETWORK> > >BOOKS RELATING TO BREAST IMPLANTS> > >AMERICAN AUTOIMMUNE RELATED DISEASES ORGANIZATION> > >SURVIVORS OF SALINES> > >CHIN AUGMENTATION OPTIONS> > >CHEEK AUGMENTATION OPTIONS> > >> > >> > >SILICONE-RELATED DISEASES> > >> > >What are silicone-related diseases?> > >> > >Silicone-related disease is a term used to describe all illnesses> > >caused> > >by different types of silicone implants. Silicone Induced Immune> > >Dysfunction Syndrome and Human Adjuvant Disease are two disorders> > >that> > >occur in implant

recipients.The following articles contain> > >information> > >on these illnesses.> > >> > >SILICONE INDUCED IMMUNE DYSFUNCTION SYNDROME> > > W. , M.D. , Medical Director, Center for Immune,> > >Environmental, and Toxic Disorders, Houston, TX> > >> > >Excerpt> > >Numerous patient case reports and controlled clinical trials have> > >discussed some of the many disorders and clinical syndromes> > >associated> > >with silicone implants, some of which resemble systemic lupus> > >erythematosus, rheumatoid arthritis, scleroderma, Sjogren's>disease,> > >polymyositis, mixed connective tissue disease, multiple> > >sclerosis ,and> > >other neurological disorders. However, it is important to note that> > >silicone induced immune dysfunction syndrome is a

multisystem> > >disorder,> > >which can affect any and all systems of the body.> > >> > >The following is a list of the most common patient complaints> > >compiled> > >by us from over 4,000 patients with silicone prosthetic devices:> > >> > >> > >breast pain or tenderness> > >> > >fatigue, usually made worse by exercise> > >> > >cognitive function problems, such as> > >attention deficit disorder, calculation difficulties, memory> > >disturbance, spatial disorientation, frequently saying the wrong>word> > >> > >psychological problems such as depression, anxiety, personality> > >changes,> > >mood swings> > >> > >sleep disturbance and non-restorative sleep> > >> > >headaches of a

greater intensity than before implantation> > >> > >changes in vision> > >> > >seizures> > >> > >loss of balance> > >> > >numbness and tingling> > >> > >lightheadedness> > >> > >paralysis> > >> > >joint and muscle aches and pains> > >> > >shortness of breath> > >> > >lymph node enlargement> > >> > >weight gain> > >> > >low grade fevers> > >> > >abnormal heart rhythm> > >> > >hair loss> > >> > >dry eyes and mouth> > >> > >frequent canker sores in the mouth> > >> > >low back pain> > >> > >skin changes and/or rashes> > >> >

>severe muscular weakness> > >> > >intolerance of bright lights> > >> > >intolerance of alcohol> > >> > >decreased libido> > >> > >ringing in ears> > >> > >muscle tremors> > >> > >recurrent flu-like illnesses> > >> > >severe allergies> > >> > >irritable bowel syndrome> > >> > >night sweats> > >> > >uncomfortable urination> > >> > >chest pain> > >> > >cough> > >> > >Raynaud's phenomenon> > >> > >enlarged thyroid.> > >> > >On physical examination, the common findings are:> > >> > >hair loss> > >> > >canker sores in the mouth> >

>> > >breast pain and tenderness> > >> > >low back pain> > >> > >thickening of the skin ,fingers, and hand> > >> > >optic neuritis> > >> > >enlarged thyroid> > >> > >upper back pain> > >> > >abdominal pain on palpation> > >> > >muscle pain> > >> > >photosensitive dermatitis :the skin is affected by exposure to the> > >sun> > >> > >lymphadenopathy: enlarged lymph glands in the neck, under arm, and> > >groin> > >areas> > >> > >diffuse petechiae on torso: small red spots on the chest and>abdomen> > >> > >positive schirmer's test, indicating deficiency of tear formation> > >> > >reduced range of movement of extremities

due to joint pain and> > >stiffness> > >> > >Raynaud's phenomenon, with cold fingers or toes that can turn white> > >and/or ulcerate> > >> > >malar or discoid rash :a rash over the cheeks of the face and upper> > >back> > >and chest> > >> > >migration of the implant, usually laterally, and superiorly,> > >unilateral> > >or bilateral> > >capsule formation, unilateral or bilateral> > >asymmetrical breasts from unilateral rupture ,migration of implant,> > >capsule formation, post-surgical complications such as hematoma or> > >infection> > >> > >livido reticularis :a lace-like pattern on the arms or legs caused>by> > >abnormalities of the blood vessels> > >> > >abnormal neurological examination with increased or

decreased deep> > >tendon reflexes, and signs of nerve damage.> > >> > >> > >LABORATORY TESTING FOR SILICONE INDUCED IMMUNE DYSFUNCTION SYNDROME> > >> > >SILICONE ANTIBODIES> > >> > >The effects of silicone have been described in several studies.>That> > >these effects are linked to Silicone Induced Immune Dysfunction> > >Syndrome> > >(S.I.I.D.S.) has also been established. Silicone antibodies in>serum> > >confirms the exposure to silicone. We studied 520 women and>published> > >these findings recently showing the significant relationship.It is> > >important to look for four types of silicone antibodies, IgA,> > >IgG ,IgM,> > >and IgE.> > >> > >AUTOIMMUNE ANTIBODIES> > >Autoimmune like disease caused by silicone

implanted medical>devices> > >has> > >been described extensively in literature. Breast implants are not>the> > >only implants that may lead to immune and autoimmune disease. All> > >silicone implants can cause autoimmune-like disease states .> > >> > >IMMUNE SYSTEM> > >Certain tests,a subpopulation of lymphocytes and their function,>have> > >been shown to be abnormal in patients with silicone implants.> > >Controlled> > >studies have shown a decrease in the function of Natural Killer>Cell> > >Activity in patients with silicone implants and a return to normal> > >levels after explantation Significant abnormalities in> > >T-Helper/T-Suppressor ratios have also been demonstrated indicating> > >an> > >immune dysregulation. All of the above contribute to confirming

the> > >diagnosis of silicone induced immune dysfunction syndrome.> > >> > >TREATMENT> > >Anyone suffering from Silicone Induced Immune Dysfunction Syndrome> > >should have their implant(s) removed.The surgery should also>include> > >the> > >removal of the entire capsule surrounding the implant(s). This may> > >alleviate some symptoms, especially breast pain and tenderness.>Care> > >should be taken from then on to allow the immune system to> > >recuperate by> > >avoiding the ingestions, absorptions, and inhalations of chemicals.> > >This> > >includes nicotine, caffeine, alcohol, artificial preservatives,> > >artificial food colorings, artificial sweeteners, artificial> > >flavorings,> > >household cleaning solvents, sprays, etc. Eating fresh foods,> >

>preferably> > >organically grown, avoids pesticide contamination. In short, live>and> > >eat like grandma and grandpa did. The medical treatment should>focus> > >on> > >immune restoration.It is preferable to repair the damage done to>the> > >immune system than to prescribe medication for every symptom. As> > >every> > >person's immune system is unique and different, there is no "boiler> > >plate" method.> > >> > >HUMAN ADJUVANT DISEASE (H.A.D.)> > >"Disquisition on human adjuvant disease."> > >Perspect, Biol. Med. 38(2): 274-290.> > >> > >Human Adjuvant Disease (HAD) is an autoimmune condition associated> > >with> > >foreign materials in contact with the human body HAD means the> > >disease> > >produced by the immune stimulation

is caused by the effect of'> > >foreign> > >material(s). Therefore, this definition excludes all naturally> > >occurring> > >disorders merely worsened by the immune stimulation of the foreign> > >material. Such disorders should be classified as "worsened by> > >adjuvant> > >activity of." For example system lupus that is believed to be> > >worsened> > >by adjuvant activity of' polyurethane and free siloxane in tissue> > >should> > >be classified as systemic lupus worsened by those adjuvants-and not> > >as> > >HAD.> > >> > >DEFINITION BY DIVISION> > >> > >Most cases of HAD would be expected to exhibit the following> > >characteristics.> > >> > >1. Foreign material or materials in the body at some time prior to> >

>the> > >development of the autoimmune disease.> > >> > >2. Local reaction to the implanted material with any one or all of> > >these> > >things: encapsulation, pain, tenderness, heat, swelling, redness.> > >> > >3. Some signs and symptoms generally associated with known>autoimmune> > >conditions, including but not necessarily restricted to aches and> > >pains> > >in muscles and joints, stiffness, weakness, and easy fatigue.> > >> > >4. At least one circulating autodirected antibody at some time>after> > >installation of the foreign material. Such antibodies must be> > >detected> > >in amounts elevated above normal.> > >> > >5. No other condition or disease explains the patient's illness,> > >including infection, malignancy, or

naturally occurring autoimmune> > >disease.> > >> > >6. Evidence is found for local immunological activation. This> > >commonly> > >consists of foreign body giant cells, but can be simply a chronic> > >inflammation with plasma cells, some lymphocytes, but mainly lots>of> > >macrophages marginated on or near the foreign material or>surrounding> > >the foreign material.> > >> > >7. Improvement follows explanation when most if not all the foreign> > >material is removed Time to improvement depends on the age of the> > >patient, the duration of illness, the severity of the autoimmunity,> > >and> > >many other influences and conditions not fully understood. In> > >general,> > >significant clinical and laboratory improvement in HAD occurs>within> >

>two> > >years of explantation. If improvement fails to occur within this>time> > >frame, HAD probably was not the cause of the patient's condition.> > >> > >These empiric defining criteria, like the criteria for rheumatic> > >diseases in general, derive from a dynamic area of research which>at> > >present has an incomplete concept of the disease and an imperfect> > >diagnostic technology. In the years ahead, improved knowledge and> > >techniques will likely change refine and more accurately describe> > >these> > >characteristics.> > >> > >Meanwhile, the seven divisions offer a standard against which> > >individual> > >cases or groups of patients can be identified, measured, and> > >compared.> > >> > >COMPARISON> > >> >

>Similarities and differences among cases define HAD and help> > >distinguish> > >it from other conditions. Degree also indicates the type of illness> > >and> > >cause.> > >> > >SIMILARITIES> > >> > >Most HAD patients can be identified by the pattern distribution of> > >abnormalities in their cases and their similarity to others who>have> > >had> > >comparable problems. The usual patient has had foreign material> > >installed within body tissues, followed sometime later by the> > >development of local difficulties around the site of implantation> > >and,> > >after that, systemic signs and symptoms of reaction to the>material.> > >Local complications include pain, aching, heat, contracture, and> > >tenderness around the foreign material.> >

>> > >Prominent systemic symptoms include morning stiffness, aches and> > >pains> > >in muscles and joints, gel phenomena, memory loss for recent>events,> > >dry> > >eyes, weakness and easy fatigue of skeletal muscle, regional> > >lymphadenopathy, and skin rash. Signs include local heat, redness,> > >induration, and irregularity around the implant, and generalized> > >weakness, glove and stocking sensory loss, and evidence of other> > >peripheral or central nervous system dysfunction or both. This> > >pattern> > >distribution of local complications near the foreign material> > >associated> > >constitutes a combination unique to HAD. Future studies may show it> > >the> > >sine qua non of this disease, a stereotype of abnormalities usually> > >not> > >seen

in conditions other than HAD.> > >> > >Incidentally, HAD does not occur exclusively in women. It happens>in> > >men> > >in cases associated with ruptured pectoralis and testicular>implants> > >[and facial implants].> > >> > >Laboratory reports show a variety of abnormalities not unique to>HAD> > >but> > >occurring in other autoimmune conditions.> > >All of the abnormalities probably reflect diffuse rather than focal> > >activation of the immune system. The ANA, rheumatoid factors,> > >positive> > >antibodies against human antigens, the antibodies against the major> > >ganglioside in the central nervous system, anti-GM, antibodies,> > >myelin> > >associated glycoprotein antibodies (anti-MAG), antisulfatide, and> > >anti-beta-tubulin antibodies do

not mean the patient has lupus or> > >rheumatoid arthritis or the other relatively specific diseases> > >associated with those positive blood tests, and certainly do not>mean> > >those diseases are present in the absence of clinical disease.> > >They are immune markers suggesting immune system activation by the> > >foreign material. Other tests show what organ systems are adversely> > >affected by the immune activation and by how much. So, when> > >indicated,> > >MRI, SPECT scan, EMG, and so forth can help define the extent of'> > >damage> > >already present in an individual patient and can help organize the> > >approach to treatment.> > >> > >DIFFERENCES> > >> > >Patients with HAD differ from other patients in a variety of ways,> > >including the obvious presence of

foreign material in their bodies> > >and> > >also in the multiplicity of signs and symptoms. They usually have>at> > >least 20 complaints and commonly circle over 50 percent of the>lists> > >of> > >common symptoms of disease. Other patients who do not have HAD>have a> > >much more circumscribed list of complaints. The large number of> > >complaints in HAD makes sense if one considers the disease is>likely> > >due> > >to a diffuse activation of the immune system and not a>circumscribed> > >focal activation of one part of the immune system. If the immune> > >system> > >is diffusely and globally activated, then there should be diffuse>and> > >global complaints referable to virtually every level of the nervous> > >system, to the muscles and nerves, connective tissue,

blood>vessels,> > >and> > >skin.> > >> > >DEGREE> > >> > >As the severity of local problems increases, the systemic problems>do> > >too, especially if the implant has ruptured or if the immunogenic> > >foreign material directly contacts the immune system by regional or> > >systemic spread. The presence of different time courses and>clinical> > >degrees of affliction associated with different foreign materials> > >strongly suggests that the foreign material plays an etiological>role> > >and may actually cause the disease.> > >> > >With silicone gel implants, six years usually elapse from the time>of> > >implantation to development of first systemic symptoms; silicone> > >implants surrounded by polyurethane have a shorter latency. Taken>

> >together, the similarity of disease among patients exposed to the> > >same> > >agent, the differences that the induced disease bears to other> > >naturally> > >occurring diseases, and the fact that degrees of adversity depend>on> > >degrees of exposure, as well as the time-linked nature of the> > >exposure> > >to the subsequent development of the disease, all argue that the> > >agents> > >named actually cause the condition.> > >> > >RELATIONSHIP> > >> > >The topic of relationship divides into three parts: cause and>effect,> > >antecedent and consequent, and contraries and contradictories.> > >> > >CAUSE AND EFFECT> > >> > >To assign a cause and make it stick requires four elements. The>cause> > >must be

sufficient to produce the effect; it must be the most>likely> > >among several possible causes; conditions must not inhibit the>cause> > >from exerting its effect; and the cause must invariably produce the> > >effect. In the muddy waters of statecraft, real-life situations,>the> > >legal arena, and medicine "invariably" has given way to "more>likely> > >than not" or "with a reasonable degree of medical certainty" In>HAD,> > >all four elements apply to foreign materials that are associated>with> > >local complications, such as the rupture of an implant and spill of> > >free> > >silicone into tissue. The rupture of the implant alone is>sufficient> > >to> > >produce the spill of silicone into tissue and to provoke a local> > >inflammatory reaction.> > >> >

>Systemic complications require no small degree of circumspection,>and> > >cases must be considered individually. It is not enough to show>that> > >the> > >disease followed implantation. These things considered, most people> > >will> > >associate a ruptured implant with the systemic disease that>follows,> > >because most individual cases have strong evidence for unique> > >activation> > >of the immune system.> > >> > >ANTECEDENT AND CONSEQUENT> > >> > >This less rigorous form of' cause and effect argument generally> > >applies> > >to implants and can be reduced to a rephrased syllogism:> > >> > >(1) Foreign materials produce local and systemic reactions;> > >> > >(2) an implant is a foreign material; therefore,> >

>> > >(3) an implant causes local and systemic reactions. Whether the> > >implant> > >has in fact done so in an individual case depends on the individual> > >evidence adduced.> > >> > >One can make the same argument for the causation of systemic> > >complications.> > >> > >Either implants cause systemic complications or they don't.> > >> > >Silicone directly injected into tissue causes local and systemic> > >reactions; therefore, silicone indirectly injected into tissue>after> > >a> > >silicone implant ruptures causes local and systemic complications.> > >> > >Hence, if the implant has been associated with Sjogren's syndrome,> > >Raynaud's phenomena, scleroderma, memory loss, muscle weakness, and> > >what> > >have you in

individual cases, then a patient who exhibits all of> > >those> > >afflictions can have a possible complication too.> > >> > >--PATTEN,B.M. and OSTERMEYER SHOAIB, B. (1995).> > >> > >This article has been condensed from the original publication.> > >> > >Note- Intact silicone implants have also been associated with Human> > >Adjuvant> > >Disease. These devices do not have to rupture to elicit an>autoimmune> > >response.> > >> > >Certainly, patients with ruptured implants are more likely to> > >develop a> > >systemic disease than those without a rupture.> > >> > >© 2004,Human Adjuvant Disease Corp.> > >The information on this website is presented for educational> > >purposes> > >> > >> > >Do you

?> > >Read only the mail you want -> >> ><<http://us.rd./mail_us/taglines/spamguard/*http://promotions>ht > tp://us.rd./mail_us/taglines/spamguard/*http://promotions>./new_mail/static/protection.html>> > >Mail SpamGuard.> > >> > >