Re: Memory Loss begins at 45 [2 Attachments]

[Guest guest]

Thanks for the post, Dr. Feinberg. I'm interested in how NMT "calls up" or "reactivates" the qualitatively impaired memory in order for the therapeutic intervention to address it (See quote below):"Before testing cellular reconsolidation in the hippocampus, professors Nader and Ledoux showed that intra- hippocampal infusions of the protein synthesis inhibitor anisomycin caused amnesia for a consolidated hippocampal-dependant contextual fear memory, but only if the memory was reactivated prior to infusion...."http://xa.yimg.com/kq/groups/1405920/1033633896/name/McGill%20U%20Memory%20Studies%20Project%2EpdfChiropractically, in mechanically-based subluxation, the vertebral level consistent with physical differential diagnosis and/or patient symptom picture is stimulated (adjusted) in order to "call-up" the involved pathways, mechanical and neurologically. In yoga, movement (mechanical and/or vibratory) is introduced into the involved dysfunctional organ or mechanical lesion, accessing it's extended pathways. In both cases, centralized (CNS-involved) lesions (including the "memory" of the lesion) are able to be activated. I can see how such mechanical and organic dysfunctional pathways correlate to "fear memories," as described in the articles you posted.But, how is the involved memory reactivated by NMT prior to therapeutic intervention? Is it accomplished neurologically (in the patient) by the similarity of the differential diagnosis enquiry dialogue to the memory pathway? Or, is it dependent upon the intention of the protocol as it addresses a dysfunctional complaint important to the patient? How does NMT meet the apparent therapeutic demand that the involved memory be reactivated prior to effective therapeutic intervention?I realize this may be too much to fully explain here, but any insight you could provide would be appreciated. Thanks again for the post. Sears, DC, IAYT1218 NW 21st AvePortland, Oregon 97209v: 503-225-0255f: 503-525-6902www.docbones.comOn Jan 8, 2012, at 8:15 PM, S Feinberg, DC wrote: Attached is a cool article on current research in short term/long term memory. Turns out the truth it isn’t as simple as has been thought, according to Canadian researcher Karim Nader. Instead of short term memory being converted to long term and that’s the end of it, it turns out that every time we access memory we convert it into volatile short term form during which time it is subject to present time influences that modify it. Much like the way long term data storage in files on a hard drive is subject to modification when the file is opened to RAM, subject to change during use, and re-stored on the hard drive, often modified in some small or large way. I present this research in my NMT seminars because it explains why accessing memory related to physiological function as we do in the NMT protocol and then inducing the mind-body to reconsider the situation results in a change in memory patterns when they are again converted to long term memory, with a resultant change in physiology. Pretty solid support for how the NMT protocol works neurologically. Also, explains why lawyers, judges, and law enforcement find such malleability in the memory of witnesses. Turns out just accessing memory makes it vulnerable to change. Oh yeah, the next Portland NMT seminar is March 16-18 at the Red Lion Convention Center. Want to learn something really cool to take your practice to a whole different level? Join us. S. Feinberg, D.C. From: [mailto: ] On Behalf Of BRIAN SEITZSent: Sunday, January 08, 2012 5:41 PM Subject: RE: Memory Loss begins at 45 Coconut oil is an excellent source of medium chain triglycerides (MCT), MCT's are used as fuel. "Burning " MCT's results in mild ketosis. This article is an example of trying to create a prescriptive medication where a natural source (of real food) exists. Seitz, DC Tuality Physicians 730-D SE Oak St Hillsboro, OR 97123 (503)640-3724Date: Sun, 8 Jan 2012 15:27:49 -0800From: docwiese7@...Subject: RE: Memory Loss begins at 45dcdocbrian@...How does coconut oil help the Hippocampus, where short term memory is stored? I read the article and it discusses how the brain is not getting enough glucose therefore fasting creates Keto bodies which for some reason will help Hippocampus maintain, however this futile for long term as well. For some reason the Hippocampus begins to give out due to lack of food, which it seems they do not know why there is a lack of glucose to the brain. So, this brings me back to your statement on Coconut oil, where does this fit in with this article?Walt From: BRIAN SEITZ <dcdocbrian@...>Subject: RE: Memory Loss begins at 45 Date: Saturday, January 7, 2012, 11:58 AM "At present, only lifestyle and nutrition are preventive; no pharmacological relief is forthcoming (duh!)"...eat more coconut oil!! https://www.secureinfoarea.com/literature/Reger%202004.pdf Seitz, DC Tuality Physicians 730-D SE Oak St Hillsboro, OR 97123 (503)640-3724Oregondcs From: dm.bones@...Date: Sat, 7 Jan 2012 11:38:46 -0800Subject: Memory Loss begins at 45 A new study from the British Medical Journal describes Subjective Cognitive Impairment (SCI) ("Memory not working as well as I think it should....") starting much earlier than previously thought. It also reveals that SCI progresses to Moderate Cognitive Impairment (MCI) much faster than previously thought. MCI is significant for severe short-term memory loss and progresses to Alzheimer's Disease (AD) in a high percentage of cases. Many involved scientists believe that MCI is an early form of AD itself. At present, only lifestyle and nutrition are preventive; no pharmacological relief is forthcoming (duh!)British Medical Journal: Brain drain starts at age 45 — not age 60Younger men and women had 3.6% drop in mental reasoningRead more: http://www.nydailynews.com/life-style/health/british-medical-journal-brain-drain-starts-age-45-age-60-article-1.1001921#ixzz1inqaJfaf Sears, DC, IAYT1218 NW 21st AvePortland, Oregon 97209v: 503-225-0255f: 503-525-6902www.docbones.com