Guest guest Posted May 21, 2010 Report Share Posted May 21, 2010 Hello Rose, When studying articles on chlorine dioxide you must keep in mind that they are referring to chlorine dioxide technology. MMS is acidified sodium chlorite technology, and is different. If you add chlorine dioxide to oils, you can purify the oil. If you add chlorous acid to oil, it breaks down the oil. In order to use MMS to make chlorine dioxide, you first need to generate the free chlorine dioxide. Next you bubble air through the solution to strip the chlorine dioxide out of solution. Next you capture the chlorine dioxide that you just stripped out, and introduce it into the oil. In water treatment, the chlorine dioxide gas is introduced into the water. Industry is fully aware of the differences between chlorine dioxide and acidified sodium chlorite technologies. When reading publications geared toward industrial use and safety, it is assumed that the reader also understands the difference. Tom --- In , " palulukon " <palulukon@...> wrote: > > Evening Tom, > > I have a question for you. while researching a cancer protocol for one of > our blacksalve list pooches I came across information on the now extinct > GrouppeK site (I made a mirror of the whole site) about oil vs water soluble > for cancer treatment. Dr. Steve passed on last August and his sister > removed the blog about a week ago. > > From the below, and I'm no scientist, chemist et all.but I wondered. Since > chlorine dioxide according to this site. > > http://www.btcproducts.co.za/generators_applications.asp > > Chlorine Dioxide is hugely more soluble in oil (hydrocarbons) > > My question would be would there be benefits to putting mms in something > like say coconut milk or any oil for that matter. See part in bold below to > explain my 'I wonder'. > > Xxx > > rose > > YUCKKOT AND QUERCURT TREATMENT PROTOCOLS > > There is no perfect treatment protocol for any disease, least of all cancer. > Some cancer respond very well to gentle treatment protocols that emphasize > death by programmed cell death, while other, more virulent, aggressive forms > of cancer can only be killed by necrosis. Radiation and chemotherapy kill > primarily by inducing massive necrosis in tumor masses, which makes them, to > date, the most effective forms of cancer treatment. Unfortunately, > chemotherapy and radiation are non-selective killers. They kill ALL > routinely growing cells, such as those in the bone marrow, skin, > gastrointestinal tract, hair follicle, and the cells attempting to > repopulate tissue wounds. > > The natural medicines in YUCKKOT and QUERCURT have no or very low toxicity > towards normal cells, yet, under certain circumstances, they interfere > significantly with the growth pathways of cancer cells, resulting in their > death by programmed cell death or necrosis. When some of these natural > medicines are additionally combined with commonly available supplements, > they become extremely powerful treatment modalities. The secret, which we > are now publicly disclosing to the world, is how we get these substances > into the body in sufficient concentrations to have a therapeutic effect. > > Coconut milk!!! > > We know.sounds stupid, simplistic and ridiculous. Coconut milk is the > " magic " delivery vehicle for introduction of these extremely powerful > natural medicines into the body? In a word, YES! Coconut milk is a > combination of coconut water and coconut, homogenized into a milk of sorts. > It is 50% saturated fat, but the saturated fat contains a very high > concentration of lauric acid, a medium chain fatty acid that is a wonderful > food and an enhancer of programmed cell death in cancer cells. Coconuts are > the only dietary source of lauric acid; it doesn't exist in other foods to > any meaningful extent. Coconut oil was used by theatres in the US to flavor > popcorn until the US cereal grain industry lobbied to have this " dangerous " > saturated fat replaced with cereal grain oils. Coconut oil is very healthy, > because the lauric acid, a 12 carbon fatty acid, is too short to be > incorporated into arteries. In fact, the reverse is true. Lauric acid is a > wonderful source of energy, as are all fatty acids, at least in comparison > to sugars and proteins, and is an anti-cancer natural molecule as well. So > you decide.politics and corporate greed or scientific reality. It shouldn't > be a difficult decision. > > Many natural medicines, such as quercetin and curcumin, exist in two basic > biochemical forms. One form, called a glycoside, has a sugar attached to it. > This molecular modification makes the compound soluble in water and > theoretically bioavailable. Unfortunately, glycosides have little biological > activity. The non-glycoside versions of natural molecules like quercetin and > curcumin have full biological activity but poor solubility in water. They > are, in fact, lipophilic or lipid (fat) loving. So we dissolved them in a > fat, coconut milk, and introduced them into the body via the lymphatic > system, which is how fats enter the body. > > Orally ingested molecules enter the body in two ways. Peptides, sugars, and > other molecules, such as natural medicines (flavonoids, flavones, etc.) > enter the body via the portal system. This is the classic method of entry > into the body via the blood. Entry into the blood via the intestines is a > complicated process. First, the molecules have to be actually absorbed by > the cells in the intestines. If they enter the intestinal cells, they can be > immediately modified by the cells and released back into the lumen of the > intestines in a " swinging door " fashion and excreted. Assuming a molecule > like quercetin makes it past this first step, it can be further modified or > destroyed by the liver. This is called the first pass phenomenon and it's > the " pox on the butt " of the entire pharmaceutical and biotech industry. The > liver is a wonderful organ that protects us against strange foreign > molecules, including therapeutically beneficial molecules like modern drugs > and natural medicines, such as quercetin and curcumin. Sometimes, the liver, > in its " ignorance " , works against us. > > There is a way around this problem, which the pharmaceutical industry has > been busy trying to exploit for years. Fats, as in oils of any kind, do not > enter the blood from the intestines. They directly enter the lymphatic > system, the system of ducts that bathes all the tissues of the body. The > lymph fluid originates from the blood but it is a completely different > circulatory system in the body. The clear liquid that moves from the blood > to the tissues bathes all the tissues in the body with nutrients, drugs, and > oxygen. It also removes waste products. This liquid is collected in > lymphatic ducts and returned to the blood where waste products are removed > by the liver and kidneys. If you can target the lymphatic system with a drug > or natural medicine, thereby bypassing initial contact with the blood > circulatory system, you can substantially enhance the introduction of > biologically active substances into the body. > > Introducing medicines directly into the lymphatic system is especially > important if you are attempting to treat an immunological disease, such as > lymphoma, or a viral disease such as HIV. These diseases exist, so to speak, > in the lymphatic system. But this same argument applies to carcinoma and > sarcoma cancers as well. Growth factors for endothelial cells (blood > vessels) such as VEGF (vascular endothelial growth factor) are secreted from > established tumors under a variety of conditions, including reduced oxygen > tension. VEGF not only stimulates the growth of new blood vessels into > tumors, it also stimulates established blood vessels to " leak " , as in to > release enhanced amounts of lymphatic fluid, protein, nutrients, and oxygen > from the blood into the tissue spaces. Contrary to current medical dogma and > scientific models, the requirement for blood vessel growth into tumors > depends largely on the geometry of the tumor mass. Tumors with a large > surface area, like 's open chest tumor, " wept " massive amounts of lymph > fluid, but didn't bleed. There were no or few blood vessels feeding the > cancer growth. The growth of the tumors and the spread of metastatic cells > were promoted by lymph fluid and its vascular system. Therefore, introducing > medicines directly into the lymphatic system is an attractive alternative to > IV injections or blood targeted oral formulations. > > GLUCOCORTICOIDS AND CANCER > > Glucocorticoids do not cause cancer, but they can protect cancer cells from > dying of suicide or programmed cell death, thereby perpetuating their > existence and spread throughout the body. Although glucocorticoids, such as > prednisone and dexamethasone, are frequently used in the treatment of > lymphomas and leukemias, they should never be used to treat non-lymphoid > cancers. Presently, dexamethasone is used in conjunction with chemotherapy > to prevent chemo-induced nausea, edema and other problems. Unfortunately, > the use of glucocorticoids like dexamethasone in the treatment of > non-lymphoid cancers, for whatever reason, is seriously counterproductive. > > During a stress response, glucocorticoids induce programmed cell death in > unnecessary or extraneous lymphoid cells. Immune cells that have been > activated by antigen and immune hormones are resistant to the apoptosis > inducing effects of elevated glucocorticoid concentrations. However, and > this is a point largely forgotten or ignored by many in the medical > profession, glucocorticoids stabilize virtually all other cells in the body > against damage during an acute stress response. Glucocorticoids accomplish > this by inhibiting the ability of cells, whether they are brain, liver, > kidney, skin, breast, prostate, or whatever, from committing suicide via > programmed cell death in the face of an equilibrium disruptive event, such > as a temporary lack of oxygen. The ability of glucocorticoids to stabilize > the cells of the body is absolutely critical to survival and the maintenance > of homeostasis. > > The ability of glucocorticoids to stabilize the cells of the body during > normal stress has certain drawbacks when cancer is involved. One of the > first biochemical events that occur during a cells progression to becoming > fully malignant is an increase in the number of glucocorticoid receptors > within the cell. In addition to an increased number of hormone receptors, > glucocorticoid receptors can be activated inappropriately by a process > called nitration. The nitration of proteins occurs when a particular amino > acid, such as tyrosine, is covalently modified by a nitrate group. In most > cases, this form of modification inhibits the activity of the protein. > Glucocorticoid receptors are actually activated by this process. The major > donor of nitrate groups is a highly chemically reactive molecule called > peroxynitrite. Peroxynitrite is involved in many diseases processes and is > almost always associated with inflammation. > > In addition to genetic changes to the glucocorticoid receptor and chemical > modifications to the receptor, psychological stress, characterized by an > increase in the level of glucocorticoids and epinephrine in the blood, also > contributes to the growth and spread of cancer cells throughout the body. It > is well established that the five year survival rate after successful cancer > therapy is heavily influenced by the psychological status of the patient. > Patients who experience severe anxiety or feelings of helplessness have a > greater incidence of cancer reoccurrence. In a very real sense, the fear of > cancer reoccurrence is a participating factor in the actual rate of > reoccurrence. > > In addition to inhibiting programmed cell death in cancer cells, > glucocorticoids are well established anti-inflammatory agents. Although > most, if not all cancers begin at sites of chronic inflammation, the > inflammation associated with cancer is chronic, and weak, rather than acute > and strong. The presence of a weak inflammatory environment may be due to > excessive binding of glucocorticoids within the tumor mass. Stress, > resulting in an increase in glucocorticoid synthesis and release, is also a > factor in protecting cancer cells from PCD. > > A good example of the difference between chronic and acute inflammation is > the analogy of bacteria growing in nutrient rich warm water. As long as the > temperature is not too high, the bacteria will grow rapidly. However, if the > temperature is increased, the bacteria rapidly die. This is very similar to > what happens in a cancer lesion. The inflammatory hormones are growth > promoting in low concentrations, but deadly when elevated beyond a certain > concentration. In a very real sense, it you want to kill cancer cells, > especially those associated with a tumor mass, you MUST induce an acute, > deadly inflammatory response against the tumor. No other treatment protocol > will work. This is exactly what occurs when tumors are treated with > chemotherapy and radiation. Radiation and chemotherapy drugs induce a > massive inflammatory response within the tumor mass, resulting in their > death from both PCD and necrosis, primarily the latter. Although a radiation > beam can be targeted, chemotherapeutic drugs cannot. They will kill any > growing cells, normal or carcinogenic, hence the death of hair follicles, > bone marrow failure, damage to the gastrointestinal tract resulting in > vomiting, and skin lesions. Chemotherapy drugs also cause what some people > called " brain freeze " , a state of confusion that takes a long time to go > away. In stark contrast to radiation and chemotherapy, the natural medicines > in the Kurosawa kocktail protocols kill only cancer cells with very > discomfort to the patient. > > After an acute inflammatory response is induced against the tumor, resulting > in necrosis and increased inflammation, the inflammation should be allowed > to " ride " so the lesion heals properly by the migration of new tissues into > the wound. There are two fundamental phases of inflammation at work here. > The first is destruction of the cancer cells and the second, equally > important, is the healing of the wound. Both phases of inflammation are > dependent on the systematic release of pro-inflammatory immune hormones, > hormones whose synthesis and/or release is inhibited by glucocorticoids. > > Another factor worthy of mention is the immune response against cancer > cells. Although some scientists have attempted to activate the immune > response against tumor masses, these attempts have always failed. Cancer > cells secrete a number of factors, such as prostaglandins, histamine and > reactive molecules like peroxynitrite that inhibit the ability of white > blood cells to recognize and destroy cancer cells. Immunotherapy is an > interesting concept, but it fails to recognize the harsh conditions under > which most advanced cancers survive in. Cancer cells have adapted to these > conditions but normal white blood cells have not. Even if a T lymphocyte > could recognize a particular cancer cell, the factors secreted by the cancer > cells would prevent the activation of the T lymphocyte and subsequent death > of the cancer. > > In summary, glucocorticoids affect non-lymphoid tumors in four fundamental > ways. First and foremost, they protect, as in prevent, cancer cells from > dying of programmed cell death. Second, they inhibit the biochemical > pathways necessary for mounting an acute, destructive inflammatory response > against the tumor mass. Third, they inhibit the biochemical pathways > involved in new cell migration into the lesion and the secretion of > collagen, the polymer or scaffold upon which most cells rest. Fourth, they > inhibit the activities of both specific (activated T lymphocytes) and > non-specific (natural killer cells and macrophages) immune responses against > the cancer cells. > > In the Grouppe Kurosawa model of cancer treatment, it is necessary to > partially block the synthesis of hydrocortisone in the adrenal glands, and > to curtail hydrocortisone binding to cancer cells. This will release cancer > cells from the protective shield provided by enhanced hydrocortisone > binding. This is step one in our treatment protocol and it can be > accomplished with the simple, inexpensive natural sleep hormone melatonin. > We like to call this step " releasing the parking brake " . Melatonin is one of > God's most remarkable natural drugs. More about melatonin later. > > THE PI3K/AKT BIOCHEMICAL PATHWAY > > The PI3K (phosphatidylinositol 3-kinase)/ AKT (protein kinase biochemical > pathway is fundamental to the survival of cells after activation by growth > factors (epidermal growth factor, platelet derived growth factor, insulin, > insulin-like growth factor, vascular endothelial growth factor, HER growth > factors, etc) and cytokines (immune hormones such as interleukin-3 and > interleukin-6, etc.). If activation of the PI3K/AKT (henceforth PIA) pathway > is blocked, the cellular activation signal initiated by a growth factor or > cytokine will result in the death of the cell by PCD. > > As we previously discussed, cell growth is a precarious process. The fragile > nature of cell growth was built into " the nature " of a cell as a fail-safe > mechanism. When a growth factor/cytokine stimulates biochemical pathways > that activate a cell to divide, they also activate the PIA pathway to > protect the cells from self-destruction along the way. The PIA pathway > accomplishes this critical task in numerous different ways. > > First, an abbreviated overview of the complicated PIA pathway is in order. > > When a growth factor/cytokine (henceforth GFC) stimulates its receptor on > the outer membrane of a cell, it causes the receptor to undergo a > conformational change. This conformational change allows the receptor > protein to bind the PIA enzyme and localize it to the inner membrane of the > cell. PIA is an enzyme that phosphorylates, or adds a phosphate group to the > lipid phosphatidylinositol. These newly phosphorylated lipids draw other > molecules to the inner surface of the membrane, thereby setting off a chain > reaction of biochemical activity. The phosphorylation reaction mediated by > PIA can be reversed by a molecule called PTEN, a phosphatase that removes > the phosphate associated with the 3 position on the phosphatidylinositol > molecule. If the PTEN molecule is inactive, as it is in the majority of > cancers, the PIA biochemical pathway can proceed largely unabated by > feedback inhibition. > > One of the molecules that bind PIP3, the phosphorylated phosphatidylinositol > lipid, is PDK1, a kinase. This enzyme adds a phosphate group to the AKT > enzyme, among others, thereby activating it. AKT (protein kinase , in > turn, activates and deactivates a host of other proteins involved in cell > proliferation and cell survival. PDK1 is a very important enzyme because it > activates numerous other protein kinases from the AGC group (PKA, PKG and > PKC). > > PTEN is considered a tumor suppressor gene, because the introduction of an > active PTEN gene into almost any cancer will cause it to stop growing and > die. Although PTEN can inhibit other biochemical pathways, such as > RAS/RAF/MEK/ERK and FAK, the pathway involved in cytoskeletal rearrangement, > its inhibition of the biochemical pathways " downstream " of PI3K activation > are of paramount importance. > > In the laboratory, we can inhibit the activation of specific biochemical > pathways with pin point precision. This isn't possible in the real world of > the body. Fortunately, nature has smiled on us and provided us with natural, > non-toxic inhibitors of the PI3K pathway. Although these natural inhibitors > kill tumor cells, they do not kill normal cells, quite unlike radiation and > chemotherapy. > > HIF-1 AND CANCER > > All oxygen dependent organisms, from bacteria to humans, must be able to > sequester oxygen in their environment in order to live. Hypoxia refers to a > reduction in the supply of adequate amounts of oxygen, resulting in a > failure to generate enough ATP to survive. Hypoxia can occur for many > reasons. Climbing a tall mountain can cause hypoxia and so-called oxygen > sickness. Disease states such as arteriosclerosis restrict blood flow hereby > depriving cells of oxygen, and predisposing people to heart attacks. Massive > bleeding, whether due to an operation or injury, can also cause hypoxia. > Hypoxia is a stress-inducing situation which, under certain circumstances, > can induce programmed cell death (PCD) in cells. Obviously, it would not be > in our survival interests to have a large number of our cells die, and our > organs fail, if they were temporarily deprived of oxygen. This is where the > protein HIF-1, hypoxia-inducible factor 1, plays an important role in > maintaining the homeostatic balance of the body. > > HIF-1 is induced by a reduction in the level of oxygen experienced by the > body. When expressed, HIF-1 blocks PCD, and regulates the activity of > literally hundreds of enzymes and proteins in the body in an effort to cope > with the reduced oxygen tension. While some enzymes and proteins are turned > on, others are turned off. Genes that regulate the uptake and metabolism of > glucose are powerfully activated by HIF-1. This makes perfect sense since > enhanced glucose uptake and its metabolism via glycolysis is the fastest way > to generate ATP and to correct the metabolic imbalance caused by a reduction > in available oxygen. Although the metabolism of sugars, fats and proteins > via respiration requires oxygen, the rapid metabolism of glucose via > glycolysis does not. The rapid metabolism of glucose in the presence of > oxygen is called aerobic glycolysis and it is a fundamental characteristic > of the metabolism of cancer cells. > > , Ph.D > Chief Scientist, Grouppe Kurosawa > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 21, 2010 Report Share Posted May 21, 2010 Industry is fully aware of the differences between chlorine dioxide and acidified sodium chlorite technologies. When reading publications geared toward industrial use and safety, it is assumed that the reader also understands the difference. Tom Well sure Tom...that's why I have you <wink> It was just one of those wild hairs. I wondered if there was any way to blend MMS with coconut milk, activated or not??? had not a clue, where it would enter the lymphatic system in a more direct route. Apparently not but worth a shot J Thanks so much, rose Quote Link to comment Share on other sites More sharing options...
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