Re: JOURNAL ...write on google gaston naessens

September 9, 2010

and able to see on the gaston microscope all somatid after 2 minute start to

speaking /need to 2 minutes/FANTASTIC

the microscope made in germany and 120000.00 dollar 3 DR can buy they told me

DVD is $65 on somatids Gb i have the adress

http://grayfieldoptical.com

--- In , " go2y47or " <go2y47or@...>

wrote:

>

> this is exactly the 13-14 step of our blueprint nano size cell,what is

actually the 13-14 atep become yeast fungus and mold.

> book on; Appleton PH She is the only one cooperated all other

discoverer and then I regignazed what it is..I going to send her dr russel She

not recorded that name.FEnomanal book as I did know all listed dr-s separatly

,but I newer connected them as NAncy appleton did/blue book rethink Pasteirer

gbThank So much

> researche in google gaston naessens /somatid/ all 16 step listed there gb

tnx

>

> --- In , Stauffer <gsgkill@>

wrote:

> >

> > http://www.joimr.org/phorum/list.php?f=2

> >

> > Pleomorphic Bacteria as a Cause of Hodgkin's Disease (Hodgkin's lymphoma):

> > A

> > Review of the Literature. The Forgotten Clue to the Bacterial Cause of

> > Cancer.The large size of some of these bodies suggested a fungal or

> > yeast-like parasite

> >

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> >

> > The Body - The Forgotten Clue to the Bacterial Cause of Cancer

> >

> > Author: Alan R. Cantwell, Jr (---.vnnyca.adelphia.net)

> >

> > Date: 11-30-03 09:14

> >

> > THE RUSSELL BODY: THE FORGOTTEN CLUE TO THE BACTERIAL CAUSE OF CANCER

> >

> > Author: Alan R Cantwell, Jr., M.D.

> >

> > Los Angeles, California,

> >

> > email: alancantwell@sbc global.net

> >

> > Paper Type: Review

> >

> > Please cite as: Cantwell AR Jr. The Body - The Forgotten Clue to the

Bacterial Cause of Cancer. JOIMR 2003;1(6):1

> >

> > Published: 30 November 2003

> >

> > Abstract

> >

> > In 1890 the pathologist reported spherical forms in

> > histopathologic sections from cancer which he interpreted as " the

> > parasite of cancer. " These forms were subsequently discredited as

> > microbial forms but have became known to every pathologist as "

> > bodies. " Identified in a wide variety of disease states, these forms

> > are now widely considered to be immunoglobulins. This paper reviews the

> > case for 's original belief that these forms are microbial in

> > nature and in origin. It is theorized that bodies are derived

> > from bacterial-sized intracellular organisms that have been reported in

> > cancer, proliferative, and inflammatory diseases by various authors over

> > the past century. It is also suggested that some larger-sized

> > bodies could represent large L-forms (so-called " large bodies " ) that

> > develop from the small coccal-sized intracellular and extracellular

> > microbes described in cancer. Obviously 's idea of a cancer

> > parasite is heretical. However, newer findings of the universal

> > presence of cell wall-deficient bacteria in the blood of all human

> > beings should lead to a reconsideration of the idea that such bacteria

> > might be implicated in the pathogenesis of cancer. Furthermore,

> > bodies might represent cell wall-deficient growth forms of these

> > universal bacteria in histopathologic sections and support 's

> > nineteenth century view of an infectious agent in cancer.

> >

> > Introduction

> >

> > The twentieth century was indeed the century of Modern Medicine with

> > tremendous strides made in the understanding and control of infectious

> > diseases, as well as the introduction of life-saving antibiotics and

> > vaccines. Unfortunately, along with these advances came the perils of

> > genetic engineering, the increasing threat of newly emerging viruses,

> > biowarfare, and bioterrorism

> >

> > Despite these scientific achievements, the cause of cancer remains a

> > mystery. Scientists suspect genetic susceptibility, possible

> > cancer-causing viruses, and environmental factors might play a role in

> > some cancers, but none of these factors explain why millions of people

> > die yearly from a variety of malignancies.

> >

> > How could scientists put men on the moon, but remain so ignorant about

> > cancer and its origin? How can the infectious causes of tuberculosis,

> > leprosy, syphilis, smallpox, polio, malaria, and other viral and

> > bacterial and parasitic diseases be understood, but the cause of cancer

> > be unknown? Could the cause of cancer conceivably be an infectious agent

> > that has been overlooked, ignored, or unrecognized by medical doctors

> > in the twentieth century? Could the germ of cancer be hidden in the

> > body? — a large microscopic form known to every pathologist for

> > over a century!

> >

> > (1852-1940) and " the parasite of cancer "

> >

> > On December 3, 1890 , a pathologist in the School of

> > Medicine at the Royal Infirmary in Edinburgh, gave an address to the

> > Pathological Society of London in which he outlined his histopathologic

> > findings of " a characteristic organism of cancer " that he observed

> > microscopically in fuchsine-stained tissue sections from all forms of

> > cancer that he examined, as well as in certain cases of tuberculosis,

> > syphilis and skin infection.

> >

> > The parasite was seen within the tissue cells (intracellular) and

> > outside the cells (extracellular). The size of 's parasite

> > ranged from barely visible, up to " half again as large as a red blood

> > corpuscle. " The largest round forms were easily seen microscopically.

> > The large size of some of these bodies suggested a fungal or yeast-like

> > parasite. provisionally classified the parasite as a

> > possible " blastomycete " (a type of fungus); and called the forms

> > " fuchsine bodies " because of their bluish-red staining qualities.

> >

> > Microbiology was still in its infancy in 's era, and it was

> > generally thought that each microbe could only give rise to a single

> > disease. Thus, the idea of a cancer germ (especially one that could also

> > be identified in TB and syphilis) was received cautiously. Nine years

> > later in 1899, in yet another report on " The parasite of cancer "

> > appearing in The Lancet (April 29), admitted that finding cancer

> > parasites in diseases other than cancer was indeed a " stumbling

> > block. " By this time a considerable number of scientists concluded that

> > bodies were merely the result of cellular degeneration of one

> > kind or another. Furthermore, no consistent microbe was cultured from

> > tumors; and the inoculation of these microbes into animals produced

> > conflicting and often negative results.

> >

> > was trained as a pathologist, not as a microbiologist, and he

> > avoided getting into the bacteriologic controversies regarding various

> > microbes grown from cancer. He simply concluded, " It seems almost

> > needless to add that there remains abundant work to be done in this

> > important and attractive field. "

> >

> > After three years' work at the New York State Pathological Laboratory

> > of the University of Buffalo, Harvey Gaylord confirmed 's

> > research in a 36 page report titled " The protozoon of cancer " , published

> > in May, 1901, in the American Journal of the Medical Sciences. Gaylord

> > found the small forms and the large sacs characteristic of

> > bodies in every cancer he examined. Some large spherical bodies were

> > four times the diameter of a leukocyte (white blood cell). Red blood

> > cells measure about 7 micron in diameter and leukocytes are 2 to 3 times

> > larger than red blood cells. Thus, some of the bodies that Gaylord

> > observed attained the amazing size of around 50 micron in diameter. In

> > addition, he found evidence of internal segmentation within the larger

> > bodies " after the manner recognized in malarial parasites. " The tiniest

> > forms appeared the size of ordinary staphylococci.

> >

> > 's 1899 paper ended his writings of a cancer parasite, but his

> > discovery quickly became known to pathologists as bodies. These

> > bodies continue to fascinate researchers and physicians (like myself)

> > up to the present time.

> >

> > When died at the age of 89 in 1940, the British Medical Journal

> > published a large obituary noting that he was universally respected

> > and embued with the dignity and highest ideals of his profession, and

> > that he had served at one time as President of the Royal College of

> > Physicians. No mention was made of his " parasites " or his " bodies " ,

> > except to remark that " in his earlier years devoted much time

> > to the study of the cancer cell. " Similarly, a large obituary appeared

> > in the Edinburgh Medical Journal along with a full-page photo. His

> > published books on Clinical Methods and widely read texts on

> > circulation and gastro-intestinal diseases were cited, but not a word

> > about his discovery in cancer.

> >

> > The heresy of " the cancer microbe "

> >

> > By the early part of the twentieth century the top cancer experts had

> > all rejected so-called " cancer parasites " as the cause of cancer. The

> > most influential physician to speak against it was Ewing, an

> > American pathologist and author of the widely-read textbook, Neoplastic

> > Diseases. In 1919 Ewing wrote that " few competent observers consider

> > it (the parasitic theory) as a possible explanation in cancer. "

> > According to Ewing and other authorities, cancer did not act like an

> > infection. Therefore, microbes could not possibly cause cancer. He

> > concluded, " The general facts of the genesis of tumors are strongly

> > against the possibility of a parasitic origin. "

> >

> > As a result, the parasitic theory was totally discarded and few doctors

> > dared to contradict Ewing's dogma by continuing to search for an

> > infectious agent in cancer. Nevertheless, a few die-hard physicians

> > remained convinced microbes were at the root cause of cancer and wrote

> > about it convincingly in medical journals. The long history of this

> > research is recorded in my book, The Cancer Microbe (1990) and anyone

> > with internet access can do a Google search (type in " cancer microbe " )

> > and obtain a wealth of information on the microbiology of cancer.

> > Another excellent history of cancer microbiology and the suppression of

> > this controversial research is contained in Hess' Can Bacteria

> > Cause Cancer? (1997).

> >

> > In the 1920s Young, an obstetrician from Scotland, repeatedly

> > grew pleomorphic (having many forms) bacteria from various cancers. The

> > microbes had a " specific life cycle " and " spore stages " comprised of

> > exceedingly tiny and barely visible spores. In the laboratory these tiny

> > spores transformed into larger coccoid (round) forms, rod-forms and

> > yeast-like forms (similar in size to bodies). Nuzum, a

> > Chicago physician, reported a pleomorphic coccus he repeatedly isolated

> > from breast cancer. The tiniest forms were virus-like and passed

> > through a filter designed to hold back bacteria.

> >

> > In 1925 Northwest Medicine published two papers by , a

> > Montana surgeon who learned about the cancer microbe in TJ Glover's lab

> > in 1921. 's microbe was similar to Young's. The parasite had a life

> > cycle composed of three stages: a coccus, a rod, and a " spore sac "

> > stage. believed cancer was an infection like tuberculosis and

> > attempted a vaccine treatment, but his treatment methods were quickly

> > suppressed by the medical establishment.

> >

> > In the 1930s in Germany the controversial Wilhelm von Brehmer described

> > microbes in the blood of cancer patients, evoking the wrath of his

> > scientific colleagues and prompting an intervention by Adolf Hitler.

> > (See Proctor's The Nazi War on Cancer [1999]) s Mazet, a French

> > physician, also found pleomorphic bacteria in Hodgkin's disease in 1941.

> > Hodgkin's is a type of lymphoma cancer involving the lymphatic

> > system. Mazet later reported similar acid-fast (red staining)

> > bacteria in many different kinds of cancer, including leukemia.

> >

> > In the 1950s, 60s, and 70s, a quartet of women further refined the

> > microbiology of cancer, emphasizing the extreme pleomorphism of the

> > organism and its detection in tissue with the acid-fast stain. The

> > published research of Virginia Livingston, Eleanor -,

> > Irene Diller and Florence Seibert, is essential reading for the most

> > updated understanding of the microbiology of cancer.

> >

> > In the late 1970s Guido Tedeschi and other Italian microbiologists at

> > the University of Camerino discovered " granules " in the red blood cells

> > of healthy and ill people that turned out to be bacteria that could be

> > cultured in the laboratory. Some of the staphylococcal and

> > corynebacteria-like bacteria cultured from the red blood cells were

> > acid-fast and cell wall-deficient, a staining and growth characteristic

> > shared with the cancer microbe. This research has been confirmed by

> > newer studies suggesting that bacteria reside in blood from healthy as

> > well as sick individuals. These findings of tiny blood bacteria

> > (nanobacteria) provide further evidence to support the theory that

> > microbes can cause cancer.

> >

> > Some other well-known scientists in the field of cancer microbiology

> > include Gunther Enderlein, Royal Rife, Gaston Naessens and

> > Wilhelm Reich. All have web sites devoted to their cancer research.

> >

> > bodies and their Origin

> >

> > More than a century has passed since 's discovery and although

> > electron microscopes (which have been used since the 1950s) have the

> > ability to magnify objects tens of thousands of times, the significance

> > and function of his bodies still remains unknown.

> >

> > What is well-known is that bodies can be found, not only in

> > cancer, but in the majority of inflamed tissues throughout the body.

> > Distinguishing large bodies from actual fungal forms of

> > Blastomyces can still be difficult, particularly when a pathologist

> > encounters a true case of fungal infection due to Blastomyces.

> >

> > In 1954 RG White, in " Observations on the formation and nature of

> > bodies " , produced bodies in animals by injecting them

> > with different species of bacteria. He then studied the ensuing

> > development of these bodies in the spleen, lymph nodes and plasma cells

> > of the injected animals. Plasma cells are specialized forms of white

> > blood cells that normally produce antibodies.

> >

> > EM Schleicher, in his 1965 paper on " Giant bodies " , discusses

> > the various theories of origin. Possibilities include origin from the

> > lymphocyte, origin in plasma cells with later degeneration, origin from

> > the mitochondria of cells, and even an origin from a red blood cell

> > (erythrocyte) swallowed up by a plasma cell.

> >

> > Most researchers currently believe bodies are essentially

> > immunoglobulins (proteins that acts as antibodies), but an electron

> > microscopic study by SM Hsu et al. in 1981 has cast some doubt on this

> > belief.

> >

> > None of these studies mention the possibility that bodies might

> > represent unusual large growth forms of bacteria. However, if

> > bodies prove to be tiny intracellular microbes that grow and enlarge

> > within leukocytes, it would be natural to expect these white blood

> > cells (especially the plasma cell) to produce an antibody attack against

> > these invading organisms, resulting in the production of

> > immunoglobulin-coated cells and organisms.

> >

> > Bacterial transformation into Giant forms (L-form " large bodies')

> >

> > There are many different kinds of bacteria but only one type that has

> > been consistently observed and studied in cancer for over a century. The

> > cancer microbe has many forms, some of which appear as ordinary

> > staphylococci or larger yeast-like forms that further enlarge to the

> > size of bodies. As mentioned, some bodies enlarge to

> > truly gigantic proportions, one hundred times the diameter of small

> > cocci. One can liken this growth potential to an empty balloon that is

> > then blown up to full-size. In addition, the microbe has exceedingly

> > small filterable submicroscopic forms approaching the size of viruses,

> > visible only by use of the electron microscope.

> >

> > Scientists who have extensively studied the cancer microbe claim it

> > most closely resembles the type bacteria that cause tuberculosis and

> > leprosy— the so-called mycobacteria. Mycobacteria are closely related

> > to fungi; and some microbiologists claim mycobacteria are essentially

> > derived from the " higher " fungi. " Myco " in Greek means fungus. Ergo,

> > mycobacteria are considered fungus-like bacteria.

> >

> > During the 1960s microbiologist Louis Dienes popularized the terms

> > " cell wall-deficient " and " L form " to encompass bacterial growth stages

> > that exist at one extreme as small filterable virus-sized forms, and

> > at the opposite extreme as large (50 micron or larger) spherical forms

> > that he termed " large bodies. " These so-called large bodies are what I

> > believe bodies represent.

> >

> > It must be understood that microbes are partially " classified " in

> > microbiology according to size. Viruses are submicroscopic and cannot be

> > visualized with an ordinary light microscope. Unlike bacteria, viruses

> > can only replicate inside a cell. Bacteria can be seen microscopically,

> > but smaller submicroscopic and filterable bacterial forms (now known as

> > nanobacteria) are also known. Fungi and yeast forms are much larger

> > than bacteria, and " mold " can obviously be seen with the naked eye.

> >

> > Larger bodies are indeed similar in size to certain spore forms

> > of fungi. However, what is generally not appreciated is that bacteria

> > can grow into fungal-sized large bodies, depending on certain laboratory

> > conditions. Thus, bacteria in this form can easily be mistaken for

> > fungi and yeast organisms.

> >

> > Giant-sized L-forms greatly resemble large-sized bodies. The

> > century-old history of research into atypical growth forms of bacteria

> > is reviewed in Lida Mattman's seminal text, Cell Wall Deficient Forms:

> > Stealth Pathogens (1993). A knowledge of this somewhat esoteric branch

> > of microbiology is essential to understand the proposed microbiology of

> > cancer.

> >

> > The most impressive electron microscopic photographs I have ever

> > observed of cell wall-deficient L-forms of mycobacteria were taken by

> > the late C Xalabarder of Barcelona. In a series of papers and books

> > (1953-1976) published in Spanish (with English-language summaries) by

> > the Publicaciones del Instituto Antituberculoso " Francisco Moragas " ,

> > Xalabarder totally transformed my concept about how tuberculosis-causing

> > mycobacteria reproduce and grow and drastically change their

> > appearance. In medical school we were taught that " simple " bacteria

> > simply divide in two equal halves by " binary fission " . However, nothing

> > could be further from the truth, and it is only by a refutation of

> > this simplistic concept that a serious study of the microbiology of

> > cancer can be undertaken.

> >

> > Tuberculosis and Cancer

> >

> > Because cancer is produced by a microbe similar to the bacteria that

> > cause TB, much can be learned from experiments like those performed by

> > Xalabarder in 1967. Using " atypical mycobacteria " grown from TB patients

> > who had taken long courses of drug therapy, Xalabarder then injected

> > these bacteria into guinea-pigs and rabbits. Amazingly, he was able to

> > experimentally produce lesions which microscopically resembled cancer!

> > He also produced experimental lesions characteristic of so called

> > " collagen disease " — a type of lesion seemingly unrelated to cancer.

> >

> > During the 1960s I discovered unusual pleomorphic acid-fast bacteria

> > in a collagen disease called scleroderma, and later in another collagen

> > disease called lupus erythematosus. The germs I grew from these patients

> > closely resembled scleroderma microbes that were reported by Virginia

> > Livingston in 1947, and which subsequently led to her discovery of

> > similar acid-fast microbes in cancer.

> >

> > In 1969 Xalabarder manipulated different developmental stages of TB

> > bacteria and inoculated them into one thousand guinea pigs. In the

> > process, he produced the microscopic picture of sarcoidosis in the

> > animals. Sarcoidosis is a human disease closely related to TB but one

> > in which TB germs cannot be found. Xalabarder's most impressive

> > sarcoid lesions were produced by inoculating sputum specimens from TB

> > patients who " converted " , meaning that their TB bacteria could no

> > longer be cultured from their sputum. Controversy over the cause of

> > sarcoidosis is still not settled, although I reported bacteria similar

> > to cancer microbes in this disease in the 1980s.

> >

> > The most spectacular electron microphotographs of cell wall-deficient

> > mycobacteria are presented in Xalabarder's L-forms of mycobacteria and

> > chronic nephritis (1970). In the earliest growth stages of mycobacteria

> > in culture the smallest elements appear as tiny submicroscopic forms

> > visualized only with the electron microscope. These filterable forms of

> > tuberculosis bacteria — the so-called " tuberculosis virus " — have been

> > known to cause cancer in animals since the 1920s. By adding antibiotics

> > to the lab culture media Xalabarder was able to induce many unusual

> > growth forms of tuberculosis bacteria. Using serial images, he was able

> > to trace the development of these tiny submicroscopic forms up to the

> > size of ordinary cocci — and then up to the size of " large body " forms

> > reaching and even surpassing the size of red blood cells. Some of the

> > large bodies of mycobacteria also exhibit internal structure, similar to

> > what Gaylord noted in his body research.

> >

> > Cancer and Bacteria

> >

> > Although the idea of a cancer microbe is medical heresy, there is ample

> > data to show that cancer patients are highly prone to bacterial

> > infection. A search of the PubMed database for " bacteria cancer " elicits

> > 49,345 citations. According to a 2003 article by Vento and Cainelli,

> > patients with cancer who are undergoing chemotherapy are highly

> > susceptible to almost any type of bacterial or fungal infection.

> >

> > Why are physicians, and especially pathologists and bacteriologists, so

> > unaware, so disinterested, or so antagonistic to credible cancer

> > microbe research? Why have pathologists failed to consider

> > bodies as large forms of bacteria?

> >

> > For over 30 years I studied various forms of cancer and skin diseases

> > " of unknown origin " , as well as autopsy cases of cancer, lupus,

> > scleroderma, and AIDS. In all these diseases I was able to detect

> > bacteria, although pathologists would never mention bacteria in any of

> > their official biopsy reports. In my experience, they simply could not

> > conceive of cancer and collagen disease (and AIDS) as a bacterial

> > infection, nor did they seem to be aware of bacteriology reports

> > pertaining to " large bodies " and pathologic effects produced by the

> > " tuberculosis virus. " In short, they were trained to see and report only

> > the typical rod-shaped acid-fast (red-stained) " typical " form of

> > mycobacteria, , but they were not trained to look for or to recognize

> > other growth forms of the same bacteria that might be hidden in their

> > pathologic tissue specimens.

> >

> > When objects like bodies are observed in a wide variety of

> > diseases and in " normal " tissue, the significance is lessened. Doctors

> > expect " normal " tissue to be free of microbes. I suppose they also

> > conclude that bodies cannot be an infectious agent because it

> > would be impossible for an infectious agent to appear in so many

> > different kinds of diseases and in so many different forms of cancer.

> >

> > For most of the last century stomach ulcers were thought to be

> > non-infectious because pathologists could not identify bacteria in the

> > ulcers and because doctors believed bacteria could not live in the acid

> > environment of the stomach. This thinking all changed gradually after

> > 1982 when Barry Marshall, an Australian physician, proved most stomach

> > ulcers were caused by a microbe called Helicobacter pylori, which could

> > be identified microscopically with special tissue staining techniques

> > in ulcer tissue. On the other hand, many people normally carry this

> > stomach microbe without any ill effects. Not surprisingly, pathologists

> > are now reporting numerous bodies in plasma cells in some ulcer

> > patients, giving rise to a previously unrecognized tissue reaction

> > called " cell gastritis. "

> >

> > bodies and bacteria

> >

> > When bacteria are threatened by the immune system or by antibiotics

> > they may lose their cell-wall and assume a different growth form that

> > renders them less susceptible to attack by the immune system. Some

> > bodies elicit little or no inflammatory cell response. This lack

> > of cellular response is yet another reason why physicians have a hard

> > time believing bodies could be microbes.

> >

> > I have observed the largest and most complex bodies in tissue

> > where there was almost a total lack of inflammation. My photographs of

> > such " large bodies " , some with obvious internal structure, that I

> > observed in patients with scleroderma and pseudoscleroderma, were

> > published in the American Journal of Dermatopathology in 1980. The first

> > case of fatal scleroderma I studied in 1963 had numerous " large

> > bodies " in the fat layer of the diseased skin that were unlike anything

> > ever seen in dermatology. The patient had been hospitalized for

> > pulmonary tuberculosis 7 years before developing scleroderma. The

> > mystery of these " yeast-like " bodies deep in his skin was solved years

> > later when I first learned about the existence of " large body " forms of

> > Mycobacterium tuberculosis. When this patient died, Mycobacterium

> > fortuitum, an " atypical " form of mycobacteria was cultured from his

> > scleroderma tissue.

> >

> > Bacteria are vital for our survival. They are hardy and the bacteria we

> > carry will surely outlive us. The bacteria that cause cancer are the

> > " simple " bacteria we carry with us. The cancer microbe is not an exotic

> > microbe nor a rare one. However, bacteria can change form as the

> > environment in our bodies changes. There is indeed a delicate balance

> > between our bacteria and our immune system which allows these bacteria

> > to live in harmony with us.

> >

> > But when dis-ease occurs these microbes become aggressive, giving

> > rise to a host of diseases, some of which are cancerous, and others that

> > are inflammatory, degenerative, or simply transitory. Another reason

> > for physicians to doubt that a single type of germ could cause such a

> > variety of pathologic effects.

> >

> > Bacteria are ubiquitous and so are bodies. And if

> > bodies prove to be bacteria, the reason for this becomes obvious.

> >

> > The body and the origin of cancer

> >

> > In 1981 King and Eisenberg's article on " 's fuchsin body: `The

> > characteristic organism of cancer' " appeared in the American Journal of

> > Dermatopathology. They reconfirmed that " bodies have now been

> > shown to be immunoglobulins. " They remarked that was not the

> > first to describe them; and that similar bodies were reported by Cornil

> > and Alvarez in rhinoscleroma five years earlier in a French journal in

> > 1885. Declaring it ironic that these " bodies should bear the name of a

> > man who so thoroughly misunderstood them " , the authors ended by stating:

> > " Hence, when the term body is used today, one should be aware

> > that the eponym is as inaccurate as was 's perception of their

> > significance. "

> >

> > Unlike King and Eisenberg, I believe was right on the mark.

> > There is a parasite in cancer. It has been studied and reported by

> > various scientists throughout the world for many decades, and a wealth

> > of scientific information on the cancer microbe is available in medical

> > libraries. For those with Internet capability, the words " cancer

> > microbe " typed into Google.com will give instant access to a treasure

> > trove of information on the subject.

> >

> > There is no secret to cancer. In my view, the cause is staring us right

> > in the face in the form of the body. understood

> > very well in the nineteenth century what medical science in the

> > twenty-first century has yet to discover.

> >

> > ---------------------------------

> >

> > Alan Cantwell, M.D. is a retired dermatologist and cancer researcher.

> > His book, The Cancer Microbe, is available through Internet sources. A

> > number of his full-length papers on the microbiology of cancer have been

> > published by the Journal of Independent Medical Research

> > (www.JOIMR.org/)

> >

> > List of Figures.

> >

> > Figure1: 1852-1940, as pictured in The British Medical

Journal, August 24, 1940.

> >

> > Figure 2: bodies in a lymph node of Hodgkin's disease. Gram's stain,

magnified 1000 times, (in oil). (click here to enlarge image).

> >

> > Figure 3: Solitary " giant " body in a lymph node of Hodgkin's disease

(cancer), Gram's stain, magnified 1000 times (click here to enlarge image).

> >

> > Figure 4: Larger bodies (RB) in a lymph node showing

> > non-cancerous " reactive lymph node hyperplasia " from a fatal case of

> > AIDS. The arrow points to nearby bacterial-sized intracellular coccoid

> > smaller forms from which the bodies are derived. Fite

> > (acid-fast) stain, magnified 1000 times (click here to enlarge image).

> >

> > Figure 5: Extremely large " super-giant-sized " solitary body in

> > the skin of " cutaneous lupus erythematosus " , a so-called " collagen

> > disease. " The perfectly round shape, except for one area, suggests this

> > large body is developing inside a cell that is readly to burst.

> > Kinyoun's (acid-fast) stain, magnification x 1000 (click here to enlarge

image).

> >

> > Figure 6: Pleomorphic growth forms (L-forms) of tuberculosis

> > mycobacteria photographed with an electron microscope. Note the darker

> > staining tiny coccal forms (similar in size to ordinary staphylococci)

> > and the larger clear balloon-sized " ghost " forms similar in size and

> > shape to bodies found in tissue. These forms are all

> > characteristic of " cell wall-deficient bacteria " and totally unlike the

> > well-known " typical " acid-fast rod forms of Mycobacterium tuberculosis.

> > Reproduced from L-forms of Mycobacteria and Chronic Nephritis (1970), by

> > Dr. C. Xalabarder P., page 51 (click here to enlarge image).

> >

> > BIBLIOGRAPHY:

> >

> > 1. - E. A specific type of microorganism isolated from

> > animal and human cancer: bacteriology of the organism. Growth. 1954

> > Mar;18(1):37-51.

> >

> > 2. Brown ST, Brett I, Almenoff PL, Lesser M, Terrin M, Teirstein AS.

> > Recovery of cell wall-deficient organisms from blood does not

> > distinguish between patients with sarcoidosis and control subjects.

> > Chest. 2003 Feb;123(2):413-417.

> >

> > 3. Cantwell AR, Craggs E, JW, Swatek F. Acid-fast bacteria as a

> > possible cause of scleroderma. Dermatologica. 1968: 136:141-150.

> >

> > 4. Cantwell AR. Histologic forms resembling " large bodies " in

> > scleroderma and pseudoscleroderma. Amer J Dermatopathol. 1980;

> > 2:273-276.

> >

> > 5. Cantwell AR, Rowe L. African " eosinophilic bodies " in vivo in two

> > American men with Kaposi's sarcoma and AIDS. J Dermatol Surg Oncol. 1985

> > Apr;11(4):408-12.

> >

> > 6. Cantwell AR, Kelso DW, JE. Histologic observations of coccoid

> > forms suggestive of cell wall deficient bacteria in cutaneous and

> > systemic lupus erythematosus.

> >

> > Int J Dermatol. 1982 Nov;21(9):526-37.

> >

> > 7. Cantwell AR. Histologic observations of variably acid-fast

> > pleomorphic bacteria in systemic sarcoidosis: a report of 3 cases.

> > Growth. 1982 Summer;46(2):113-25.

> >

> > 8. Cantwell AR. Variably acid-fast cell wall-deficient bacteria as a

> > possible cause of dermatologic disease. In, Domingue GJ (Ed). Cell Wall

> > Deficient Bacteria. Reading: -Wesley Publishing Co; 1982. Pp.

> > 321-360.

> >

> > 9. Cantwell A. The Cancer Microbe. Los Angeles: Aries Rising Press; 1990.

> >

> > 10. Dienes L. Morphology and reproductive processes of bacteria with

> > defective cell walls. In, Guze LB (Ed). Microbial Protoplasts,

> > Spheroplasts and L-Forms. Baltimore: & Wilkins Company;

> > 1968, Pp 74-93.

> >

> > 11. Diller IC, Diller WF. Intracellular acid-fast organisms isolated

> > from malignant tissues. Trans Amer Micr Soc. 1965; 84:138-148.

> >

> > 12. Ewing J. The parasitic theory. In, Ewing J (Ed), Neoplastic Diseases (Ed

1); Philadelphia: Saunders; 1919. Pp 114-126.

> >

> > 13. Gaylord HR. The protozoon of cancer. Amer J Med Sci. 1901;121:501-539.

> >

> > 14. Gebbers JO, Otto HF. Plasma cell alterations in ulcerative colitis.

> > An electron microscopic study. Pathol Eur. 1976;11(4):271-9.

> >

> > 15. Glover TJ. The bacteriology of cancer. Canada Lancet Pract. 1930;

75:92-111.

> >

> > 16. Haensch R, Seeliger H. Problems of differential diagnosis of

> > blastomyces and bodies. Arch Dermatol Res. 1981;270(4):381-5.

> >

> > 17. Hess D. Can Bacteria Cause Cancer? New York:New York University Press;

1997.

> >

> > 18. Jetha N, Priddy RW. Exact nature of bodies still an enigma. Am J

Clin Pathol. 1984 Apr;81(4):545.

> >

> > 19. King DF, Eisenberg D. 's fuchsine body. " The characteristic

> > organism of cancer " . Am J Dermatopathol. 1981 Spring;3(1):55-8.

> >

> > 20. Mattman LH. Cell Wall Deficient Forms (Ed 2). Boca Raton:CRC Press;

1993.

> >

> > 21. Mazet G. Corynebacterium, tubercle bacillus and cancer. Growth. 1974;

38:

> >

> > 22. McLaughlin RW, Vali H, Lau PC, Palfree RG, De Ciccio A, Sirois M,

> > Ahmad D, Villemur R, Desrosiers M, Chan E. Are there naturally occurring

> > pleomorphic bacteria in the blood of healthy humans? J Clin Microbiol.

> > 2002 Dec;40(12):4771-5.

> >

> > 23. Nuzum JW. The experimental production of metastasizing carcinoma of

> > the breast of the dog and primary epithelioma in man by repeated

> > inoculation of a micrococcus isolated from human breast cancer. Surg

> > Gynecol Obstet. 1925; 11;343-352.

> >

> > 24. W. An address on a characteristic organism of cancer. Br Med J.

1890; 2:1356-1360.

> >

> > 25. W. The parasite of cancer. Lancet. 1899;1:1138-1141.

> >

> > 26. Schleicher EM. Giant bodies in neoplastic cells in a case of

> > leukemic lymphosarcomatosis. Minnesota Medicine. 1965; 48:1125-1130.

> >

> > 27. MJ. The parasitic origin of carcinoma. Northwest Med.

1925;24:162-166.

> >

> > 28. Seibert FB, Feldmann FM, RL, Richmond IS. Morphological,

> > biological, and immunological studies on isolates from tumors and

> > leukemic bloods. Ann N Y Acad Sci. 1970 Oct 30;174(2):690-728.

> >

> > 29. Tedeschi GG, Amici D. Mycoplasma-like microorganisms probably

> > related to L forms of bacteria in the blood of healthy persons.

> > Cultural, morphological and histochemical data. Ann Sclavo. 1972

> > Jul-Aug;14(4):430-42.

> >

> > 30. Tedeschi GG, Bondi A, Paparelli M, Sprovieri G. Electron

> > microscopical evidence of the evolution of corynebacteria-like

> > microorganisms within human erythrocytes. Experientia. 1978 Apr

> > 15;34(4):458-60.

> >

> > 31. Vento S, Cainelli F. Infections in patients with cancer undergoing

> > chemotherapy: aetiology, prevention, and treatment. Lancet Oncol. 2003

> > Oct;4(10):595-604.

> >

> > 32. Von Brehmer W. " Siphonospora polymorphia " n. sp., neuer

> > Mikroorganismus des Blutes und seine Beziehung zur Tumororigenese. Med

> > Welt. 1934; 8:1179-1185.

> >

> > 33. White RG. Observations on the formation and nature of bodies. Br

J Exp Pathol. 1954; 35:365-376.

> >

> > 34. Wuerthele Caspe-Livingston V, - E, JA, et

> > al. Cultural properties and pathogenicity of certain microorganisms

> > obtained from various proliferative and neoplastic diseases. Amer J Med

> > Sci. 1950; 220;628-646.

> >

> > 35. Wuerthele-Caspe Livingston V, Livingston AM. Demonstration of

> > Progenitor cryptocides in the blood of patients with collagen and

> > neoplastic diseases. Trans NY Acad Sci. 1972; 174 (2):636-654.

> >

> > 36. Xalabarder C. La desconocida patologia provocada por micobacterias. Publ

Inst Antituberc. 1967; 17:35-52.

> >

> > 37. Xalabarder C: Formas L de micobacterias y nefritis cronicas. Publ Inst

Antituberc (Barcelona). 1970; Supple 7:7-83.

> >

> > 38. Xalabarder C. Sarcoidosis experimental. Publ Inst Antituberc

(Barcelona). 1969; 8:51-76.

> >

> > 39. Young J. Description of an oganism obtained from carcinomatous growths.

Edinburgh Med J. 1921; 27:212-221.

> >

> > KEY WORDS

> >

> > Bacteria, pleomorphic

> >

> > cancer-associated bacteria

> >

> > cancer, origin

> >

> > cell wall-deficient bacteria

> >

> > Hodgkin's disease

> >

> > L-forms

> >

> > lupus erythematosus

> >

> > mycobacteria

> >

> > body

> >

> > sarcoidosis

> >

> > scleroderma

> >

> > tuberculosis

> >

> > Go back to Main Menu

> >

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