Re: JOURNAL OF INDEPENDENT MEDICAL RESEARCH

September 9, 2010

this is exactly the 13-14 step of our blueprint nano size cell,what is actually

the 13-14 atep become yeast fungus and mold.

book on; Appleton PH She is the only one cooperated all other

discoverer and then I regignazed what it is..I going to send her dr russel She

not recorded that name.FEnomanal book as I did know all listed dr-s separatly

,but I newer connected them as NAncy appleton did/blue book rethink Pasteirer

gbThank So much

researche in google gaston naessens /somatid/ all 16 step listed there gb tnx

--- In , Stauffer <gsgkill@...>

wrote:

>

> http://www.joimr.org/phorum/list.php?f=2

>

> Pleomorphic Bacteria as a Cause of Hodgkin's Disease (Hodgkin's lymphoma):

> A

> Review of the Literature. The Forgotten Clue to the Bacterial Cause of

> Cancer.The large size of some of these bodies suggested a fungal or

> yeast-like parasite

>

> JOURNAL OF INDEPENDENT

> MEDICAL RESEARCH

>

> COLLABORATIVE PUBLISHING FOR THE

> 21stCENTURY

>

> HOME

> Peer Review

> These

> Papers

> Papers Sent

> To

> PubMed

> Peer

> Review

> Rules

> Advisory

> Board

> Contact Us

>

> Go back to Main Menu | SEARCH | Log In

>

> The Body - The Forgotten Clue to the Bacterial Cause of Cancer

>

> Author: Alan R. Cantwell, Jr (---.vnnyca.adelphia.net)

>

> Date: 11-30-03 09:14

>

> THE RUSSELL BODY: THE FORGOTTEN CLUE TO THE BACTERIAL CAUSE OF CANCER

>

> Author: Alan R Cantwell, Jr., M.D.

>

> Los Angeles, California,

>

> email: alancantwell@sbc global.net

>

> Paper Type: Review

>

> Please cite as: Cantwell AR Jr. The Body - The Forgotten Clue to the

Bacterial Cause of Cancer. JOIMR 2003;1(6):1

>

> Published: 30 November 2003

>

> Abstract

>

> In 1890 the pathologist reported spherical forms in

> histopathologic sections from cancer which he interpreted as " the

> parasite of cancer. " These forms were subsequently discredited as

> microbial forms but have became known to every pathologist as "

> bodies. " Identified in a wide variety of disease states, these forms

> are now widely considered to be immunoglobulins. This paper reviews the

> case for 's original belief that these forms are microbial in

> nature and in origin. It is theorized that bodies are derived

> from bacterial-sized intracellular organisms that have been reported in

> cancer, proliferative, and inflammatory diseases by various authors over

> the past century. It is also suggested that some larger-sized

> bodies could represent large L-forms (so-called " large bodies " ) that

> develop from the small coccal-sized intracellular and extracellular

> microbes described in cancer. Obviously 's idea of a cancer

> parasite is heretical. However, newer findings of the universal

> presence of cell wall-deficient bacteria in the blood of all human

> beings should lead to a reconsideration of the idea that such bacteria

> might be implicated in the pathogenesis of cancer. Furthermore,

> bodies might represent cell wall-deficient growth forms of these

> universal bacteria in histopathologic sections and support 's

> nineteenth century view of an infectious agent in cancer.

>

> Introduction

>

> The twentieth century was indeed the century of Modern Medicine with

> tremendous strides made in the understanding and control of infectious

> diseases, as well as the introduction of life-saving antibiotics and

> vaccines. Unfortunately, along with these advances came the perils of

> genetic engineering, the increasing threat of newly emerging viruses,

> biowarfare, and bioterrorism

>

> Despite these scientific achievements, the cause of cancer remains a

> mystery. Scientists suspect genetic susceptibility, possible

> cancer-causing viruses, and environmental factors might play a role in

> some cancers, but none of these factors explain why millions of people

> die yearly from a variety of malignancies.

>

> How could scientists put men on the moon, but remain so ignorant about

> cancer and its origin? How can the infectious causes of tuberculosis,

> leprosy, syphilis, smallpox, polio, malaria, and other viral and

> bacterial and parasitic diseases be understood, but the cause of cancer

> be unknown? Could the cause of cancer conceivably be an infectious agent

> that has been overlooked, ignored, or unrecognized by medical doctors

> in the twentieth century? Could the germ of cancer be hidden in the

> body? — a large microscopic form known to every pathologist for

> over a century!

>

> (1852-1940) and " the parasite of cancer "

>

> On December 3, 1890 , a pathologist in the School of

> Medicine at the Royal Infirmary in Edinburgh, gave an address to the

> Pathological Society of London in which he outlined his histopathologic

> findings of " a characteristic organism of cancer " that he observed

> microscopically in fuchsine-stained tissue sections from all forms of

> cancer that he examined, as well as in certain cases of tuberculosis,

> syphilis and skin infection.

>

> The parasite was seen within the tissue cells (intracellular) and

> outside the cells (extracellular). The size of 's parasite

> ranged from barely visible, up to " half again as large as a red blood

> corpuscle. " The largest round forms were easily seen microscopically.

> The large size of some of these bodies suggested a fungal or yeast-like

> parasite. provisionally classified the parasite as a

> possible " blastomycete " (a type of fungus); and called the forms

> " fuchsine bodies " because of their bluish-red staining qualities.

>

> Microbiology was still in its infancy in 's era, and it was

> generally thought that each microbe could only give rise to a single

> disease. Thus, the idea of a cancer germ (especially one that could also

> be identified in TB and syphilis) was received cautiously. Nine years

> later in 1899, in yet another report on " The parasite of cancer "

> appearing in The Lancet (April 29), admitted that finding cancer

> parasites in diseases other than cancer was indeed a " stumbling

> block. " By this time a considerable number of scientists concluded that

> bodies were merely the result of cellular degeneration of one

> kind or another. Furthermore, no consistent microbe was cultured from

> tumors; and the inoculation of these microbes into animals produced

> conflicting and often negative results.

>

> was trained as a pathologist, not as a microbiologist, and he

> avoided getting into the bacteriologic controversies regarding various

> microbes grown from cancer. He simply concluded, " It seems almost

> needless to add that there remains abundant work to be done in this

> important and attractive field. "

>

> After three years' work at the New York State Pathological Laboratory

> of the University of Buffalo, Harvey Gaylord confirmed 's

> research in a 36 page report titled " The protozoon of cancer " , published

> in May, 1901, in the American Journal of the Medical Sciences. Gaylord

> found the small forms and the large sacs characteristic of

> bodies in every cancer he examined. Some large spherical bodies were

> four times the diameter of a leukocyte (white blood cell). Red blood

> cells measure about 7 micron in diameter and leukocytes are 2 to 3 times

> larger than red blood cells. Thus, some of the bodies that Gaylord

> observed attained the amazing size of around 50 micron in diameter. In

> addition, he found evidence of internal segmentation within the larger

> bodies " after the manner recognized in malarial parasites. " The tiniest

> forms appeared the size of ordinary staphylococci.

>

> 's 1899 paper ended his writings of a cancer parasite, but his

> discovery quickly became known to pathologists as bodies. These

> bodies continue to fascinate researchers and physicians (like myself)

> up to the present time.

>

> When died at the age of 89 in 1940, the British Medical Journal

> published a large obituary noting that he was universally respected

> and embued with the dignity and highest ideals of his profession, and

> that he had served at one time as President of the Royal College of

> Physicians. No mention was made of his " parasites " or his " bodies " ,

> except to remark that " in his earlier years devoted much time

> to the study of the cancer cell. " Similarly, a large obituary appeared

> in the Edinburgh Medical Journal along with a full-page photo. His

> published books on Clinical Methods and widely read texts on

> circulation and gastro-intestinal diseases were cited, but not a word

> about his discovery in cancer.

>

> The heresy of " the cancer microbe "

>

> By the early part of the twentieth century the top cancer experts had

> all rejected so-called " cancer parasites " as the cause of cancer. The

> most influential physician to speak against it was Ewing, an

> American pathologist and author of the widely-read textbook, Neoplastic

> Diseases. In 1919 Ewing wrote that " few competent observers consider

> it (the parasitic theory) as a possible explanation in cancer. "

> According to Ewing and other authorities, cancer did not act like an

> infection. Therefore, microbes could not possibly cause cancer. He

> concluded, " The general facts of the genesis of tumors are strongly

> against the possibility of a parasitic origin. "

>

> As a result, the parasitic theory was totally discarded and few doctors

> dared to contradict Ewing's dogma by continuing to search for an

> infectious agent in cancer. Nevertheless, a few die-hard physicians

> remained convinced microbes were at the root cause of cancer and wrote

> about it convincingly in medical journals. The long history of this

> research is recorded in my book, The Cancer Microbe (1990) and anyone

> with internet access can do a Google search (type in " cancer microbe " )

> and obtain a wealth of information on the microbiology of cancer.

> Another excellent history of cancer microbiology and the suppression of

> this controversial research is contained in Hess' Can Bacteria

> Cause Cancer? (1997).

>

> In the 1920s Young, an obstetrician from Scotland, repeatedly

> grew pleomorphic (having many forms) bacteria from various cancers. The

> microbes had a " specific life cycle " and " spore stages " comprised of

> exceedingly tiny and barely visible spores. In the laboratory these tiny

> spores transformed into larger coccoid (round) forms, rod-forms and

> yeast-like forms (similar in size to bodies). Nuzum, a

> Chicago physician, reported a pleomorphic coccus he repeatedly isolated

> from breast cancer. The tiniest forms were virus-like and passed

> through a filter designed to hold back bacteria.

>

> In 1925 Northwest Medicine published two papers by , a

> Montana surgeon who learned about the cancer microbe in TJ Glover's lab

> in 1921. 's microbe was similar to Young's. The parasite had a life

> cycle composed of three stages: a coccus, a rod, and a " spore sac "

> stage. believed cancer was an infection like tuberculosis and

> attempted a vaccine treatment, but his treatment methods were quickly

> suppressed by the medical establishment.

>

> In the 1930s in Germany the controversial Wilhelm von Brehmer described

> microbes in the blood of cancer patients, evoking the wrath of his

> scientific colleagues and prompting an intervention by Adolf Hitler.

> (See Proctor's The Nazi War on Cancer [1999]) s Mazet, a French

> physician, also found pleomorphic bacteria in Hodgkin's disease in 1941.

> Hodgkin's is a type of lymphoma cancer involving the lymphatic

> system. Mazet later reported similar acid-fast (red staining)

> bacteria in many different kinds of cancer, including leukemia.

>

> In the 1950s, 60s, and 70s, a quartet of women further refined the

> microbiology of cancer, emphasizing the extreme pleomorphism of the

> organism and its detection in tissue with the acid-fast stain. The

> published research of Virginia Livingston, Eleanor -,

> Irene Diller and Florence Seibert, is essential reading for the most

> updated understanding of the microbiology of cancer.

>

> In the late 1970s Guido Tedeschi and other Italian microbiologists at

> the University of Camerino discovered " granules " in the red blood cells

> of healthy and ill people that turned out to be bacteria that could be

> cultured in the laboratory. Some of the staphylococcal and

> corynebacteria-like bacteria cultured from the red blood cells were

> acid-fast and cell wall-deficient, a staining and growth characteristic

> shared with the cancer microbe. This research has been confirmed by

> newer studies suggesting that bacteria reside in blood from healthy as

> well as sick individuals. These findings of tiny blood bacteria

> (nanobacteria) provide further evidence to support the theory that

> microbes can cause cancer.

>

> Some other well-known scientists in the field of cancer microbiology

> include Gunther Enderlein, Royal Rife, Gaston Naessens and

> Wilhelm Reich. All have web sites devoted to their cancer research.

>

> bodies and their Origin

>

> More than a century has passed since 's discovery and although

> electron microscopes (which have been used since the 1950s) have the

> ability to magnify objects tens of thousands of times, the significance

> and function of his bodies still remains unknown.

>

> What is well-known is that bodies can be found, not only in

> cancer, but in the majority of inflamed tissues throughout the body.

> Distinguishing large bodies from actual fungal forms of

> Blastomyces can still be difficult, particularly when a pathologist

> encounters a true case of fungal infection due to Blastomyces.

>

> In 1954 RG White, in " Observations on the formation and nature of

> bodies " , produced bodies in animals by injecting them

> with different species of bacteria. He then studied the ensuing

> development of these bodies in the spleen, lymph nodes and plasma cells

> of the injected animals. Plasma cells are specialized forms of white

> blood cells that normally produce antibodies.

>

> EM Schleicher, in his 1965 paper on " Giant bodies " , discusses

> the various theories of origin. Possibilities include origin from the

> lymphocyte, origin in plasma cells with later degeneration, origin from

> the mitochondria of cells, and even an origin from a red blood cell

> (erythrocyte) swallowed up by a plasma cell.

>

> Most researchers currently believe bodies are essentially

> immunoglobulins (proteins that acts as antibodies), but an electron

> microscopic study by SM Hsu et al. in 1981 has cast some doubt on this

> belief.

>

> None of these studies mention the possibility that bodies might

> represent unusual large growth forms of bacteria. However, if

> bodies prove to be tiny intracellular microbes that grow and enlarge

> within leukocytes, it would be natural to expect these white blood

> cells (especially the plasma cell) to produce an antibody attack against

> these invading organisms, resulting in the production of

> immunoglobulin-coated cells and organisms.

>

> Bacterial transformation into Giant forms (L-form " large bodies')

>

> There are many different kinds of bacteria but only one type that has

> been consistently observed and studied in cancer for over a century. The

> cancer microbe has many forms, some of which appear as ordinary

> staphylococci or larger yeast-like forms that further enlarge to the

> size of bodies. As mentioned, some bodies enlarge to

> truly gigantic proportions, one hundred times the diameter of small

> cocci. One can liken this growth potential to an empty balloon that is

> then blown up to full-size. In addition, the microbe has exceedingly

> small filterable submicroscopic forms approaching the size of viruses,

> visible only by use of the electron microscope.

>

> Scientists who have extensively studied the cancer microbe claim it

> most closely resembles the type bacteria that cause tuberculosis and

> leprosy— the so-called mycobacteria. Mycobacteria are closely related

> to fungi; and some microbiologists claim mycobacteria are essentially

> derived from the " higher " fungi. " Myco " in Greek means fungus. Ergo,

> mycobacteria are considered fungus-like bacteria.

>

> During the 1960s microbiologist Louis Dienes popularized the terms

> " cell wall-deficient " and " L form " to encompass bacterial growth stages

> that exist at one extreme as small filterable virus-sized forms, and

> at the opposite extreme as large (50 micron or larger) spherical forms

> that he termed " large bodies. " These so-called large bodies are what I

> believe bodies represent.

>

> It must be understood that microbes are partially " classified " in

> microbiology according to size. Viruses are submicroscopic and cannot be

> visualized with an ordinary light microscope. Unlike bacteria, viruses

> can only replicate inside a cell. Bacteria can be seen microscopically,

> but smaller submicroscopic and filterable bacterial forms (now known as

> nanobacteria) are also known. Fungi and yeast forms are much larger

> than bacteria, and " mold " can obviously be seen with the naked eye.

>

> Larger bodies are indeed similar in size to certain spore forms

> of fungi. However, what is generally not appreciated is that bacteria

> can grow into fungal-sized large bodies, depending on certain laboratory

> conditions. Thus, bacteria in this form can easily be mistaken for

> fungi and yeast organisms.

>

> Giant-sized L-forms greatly resemble large-sized bodies. The

> century-old history of research into atypical growth forms of bacteria

> is reviewed in Lida Mattman's seminal text, Cell Wall Deficient Forms:

> Stealth Pathogens (1993). A knowledge of this somewhat esoteric branch

> of microbiology is essential to understand the proposed microbiology of

> cancer.

>

> The most impressive electron microscopic photographs I have ever

> observed of cell wall-deficient L-forms of mycobacteria were taken by

> the late C Xalabarder of Barcelona. In a series of papers and books

> (1953-1976) published in Spanish (with English-language summaries) by

> the Publicaciones del Instituto Antituberculoso " Francisco Moragas " ,

> Xalabarder totally transformed my concept about how tuberculosis-causing

> mycobacteria reproduce and grow and drastically change their

> appearance. In medical school we were taught that " simple " bacteria

> simply divide in two equal halves by " binary fission " . However, nothing

> could be further from the truth, and it is only by a refutation of

> this simplistic concept that a serious study of the microbiology of

> cancer can be undertaken.

>

> Tuberculosis and Cancer

>

> Because cancer is produced by a microbe similar to the bacteria that

> cause TB, much can be learned from experiments like those performed by

> Xalabarder in 1967. Using " atypical mycobacteria " grown from TB patients

> who had taken long courses of drug therapy, Xalabarder then injected

> these bacteria into guinea-pigs and rabbits. Amazingly, he was able to

> experimentally produce lesions which microscopically resembled cancer!

> He also produced experimental lesions characteristic of so called

> " collagen disease " — a type of lesion seemingly unrelated to cancer.

>

> During the 1960s I discovered unusual pleomorphic acid-fast bacteria

> in a collagen disease called scleroderma, and later in another collagen

> disease called lupus erythematosus. The germs I grew from these patients

> closely resembled scleroderma microbes that were reported by Virginia

> Livingston in 1947, and which subsequently led to her discovery of

> similar acid-fast microbes in cancer.

>

> In 1969 Xalabarder manipulated different developmental stages of TB

> bacteria and inoculated them into one thousand guinea pigs. In the

> process, he produced the microscopic picture of sarcoidosis in the

> animals. Sarcoidosis is a human disease closely related to TB but one

> in which TB germs cannot be found. Xalabarder's most impressive

> sarcoid lesions were produced by inoculating sputum specimens from TB

> patients who " converted " , meaning that their TB bacteria could no

> longer be cultured from their sputum. Controversy over the cause of

> sarcoidosis is still not settled, although I reported bacteria similar

> to cancer microbes in this disease in the 1980s.

>

> The most spectacular electron microphotographs of cell wall-deficient

> mycobacteria are presented in Xalabarder's L-forms of mycobacteria and

> chronic nephritis (1970). In the earliest growth stages of mycobacteria

> in culture the smallest elements appear as tiny submicroscopic forms

> visualized only with the electron microscope. These filterable forms of

> tuberculosis bacteria — the so-called " tuberculosis virus " — have been

> known to cause cancer in animals since the 1920s. By adding antibiotics

> to the lab culture media Xalabarder was able to induce many unusual

> growth forms of tuberculosis bacteria. Using serial images, he was able

> to trace the development of these tiny submicroscopic forms up to the

> size of ordinary cocci — and then up to the size of " large body " forms

> reaching and even surpassing the size of red blood cells. Some of the

> large bodies of mycobacteria also exhibit internal structure, similar to

> what Gaylord noted in his body research.

>

> Cancer and Bacteria

>

> Although the idea of a cancer microbe is medical heresy, there is ample

> data to show that cancer patients are highly prone to bacterial

> infection. A search of the PubMed database for " bacteria cancer " elicits

> 49,345 citations. According to a 2003 article by Vento and Cainelli,

> patients with cancer who are undergoing chemotherapy are highly

> susceptible to almost any type of bacterial or fungal infection.

>

> Why are physicians, and especially pathologists and bacteriologists, so

> unaware, so disinterested, or so antagonistic to credible cancer

> microbe research? Why have pathologists failed to consider

> bodies as large forms of bacteria?

>

> For over 30 years I studied various forms of cancer and skin diseases

> " of unknown origin " , as well as autopsy cases of cancer, lupus,

> scleroderma, and AIDS. In all these diseases I was able to detect

> bacteria, although pathologists would never mention bacteria in any of

> their official biopsy reports. In my experience, they simply could not

> conceive of cancer and collagen disease (and AIDS) as a bacterial

> infection, nor did they seem to be aware of bacteriology reports

> pertaining to " large bodies " and pathologic effects produced by the

> " tuberculosis virus. " In short, they were trained to see and report only

> the typical rod-shaped acid-fast (red-stained) " typical " form of

> mycobacteria, , but they were not trained to look for or to recognize

> other growth forms of the same bacteria that might be hidden in their

> pathologic tissue specimens.

>

> When objects like bodies are observed in a wide variety of

> diseases and in " normal " tissue, the significance is lessened. Doctors

> expect " normal " tissue to be free of microbes. I suppose they also

> conclude that bodies cannot be an infectious agent because it

> would be impossible for an infectious agent to appear in so many

> different kinds of diseases and in so many different forms of cancer.

>

> For most of the last century stomach ulcers were thought to be

> non-infectious because pathologists could not identify bacteria in the

> ulcers and because doctors believed bacteria could not live in the acid

> environment of the stomach. This thinking all changed gradually after

> 1982 when Barry Marshall, an Australian physician, proved most stomach

> ulcers were caused by a microbe called Helicobacter pylori, which could

> be identified microscopically with special tissue staining techniques

> in ulcer tissue. On the other hand, many people normally carry this

> stomach microbe without any ill effects. Not surprisingly, pathologists

> are now reporting numerous bodies in plasma cells in some ulcer

> patients, giving rise to a previously unrecognized tissue reaction

> called " cell gastritis. "

>

> bodies and bacteria

>

> When bacteria are threatened by the immune system or by antibiotics

> they may lose their cell-wall and assume a different growth form that

> renders them less susceptible to attack by the immune system. Some

> bodies elicit little or no inflammatory cell response. This lack

> of cellular response is yet another reason why physicians have a hard

> time believing bodies could be microbes.

>

> I have observed the largest and most complex bodies in tissue

> where there was almost a total lack of inflammation. My photographs of

> such " large bodies " , some with obvious internal structure, that I

> observed in patients with scleroderma and pseudoscleroderma, were

> published in the American Journal of Dermatopathology in 1980. The first

> case of fatal scleroderma I studied in 1963 had numerous " large

> bodies " in the fat layer of the diseased skin that were unlike anything

> ever seen in dermatology. The patient had been hospitalized for

> pulmonary tuberculosis 7 years before developing scleroderma. The

> mystery of these " yeast-like " bodies deep in his skin was solved years

> later when I first learned about the existence of " large body " forms of

> Mycobacterium tuberculosis. When this patient died, Mycobacterium

> fortuitum, an " atypical " form of mycobacteria was cultured from his

> scleroderma tissue.

>

> Bacteria are vital for our survival. They are hardy and the bacteria we

> carry will surely outlive us. The bacteria that cause cancer are the

> " simple " bacteria we carry with us. The cancer microbe is not an exotic

> microbe nor a rare one. However, bacteria can change form as the

> environment in our bodies changes. There is indeed a delicate balance

> between our bacteria and our immune system which allows these bacteria

> to live in harmony with us.

>

> But when dis-ease occurs these microbes become aggressive, giving

> rise to a host of diseases, some of which are cancerous, and others that

> are inflammatory, degenerative, or simply transitory. Another reason

> for physicians to doubt that a single type of germ could cause such a

> variety of pathologic effects.

>

> Bacteria are ubiquitous and so are bodies. And if

> bodies prove to be bacteria, the reason for this becomes obvious.

>

> The body and the origin of cancer

>

> In 1981 King and Eisenberg's article on " 's fuchsin body: `The

> characteristic organism of cancer' " appeared in the American Journal of

> Dermatopathology. They reconfirmed that " bodies have now been

> shown to be immunoglobulins. " They remarked that was not the

> first to describe them; and that similar bodies were reported by Cornil

> and Alvarez in rhinoscleroma five years earlier in a French journal in

> 1885. Declaring it ironic that these " bodies should bear the name of a

> man who so thoroughly misunderstood them " , the authors ended by stating:

> " Hence, when the term body is used today, one should be aware

> that the eponym is as inaccurate as was 's perception of their

> significance. "

>

> Unlike King and Eisenberg, I believe was right on the mark.

> There is a parasite in cancer. It has been studied and reported by

> various scientists throughout the world for many decades, and a wealth

> of scientific information on the cancer microbe is available in medical

> libraries. For those with Internet capability, the words " cancer

> microbe " typed into Google.com will give instant access to a treasure

> trove of information on the subject.

>

> There is no secret to cancer. In my view, the cause is staring us right

> in the face in the form of the body. understood

> very well in the nineteenth century what medical science in the

> twenty-first century has yet to discover.

>

> ---------------------------------

>

> Alan Cantwell, M.D. is a retired dermatologist and cancer researcher.

> His book, The Cancer Microbe, is available through Internet sources. A

> number of his full-length papers on the microbiology of cancer have been

> published by the Journal of Independent Medical Research

> (www.JOIMR.org/)

>

> List of Figures.

>

> Figure1: 1852-1940, as pictured in The British Medical

Journal, August 24, 1940.

>

> Figure 2: bodies in a lymph node of Hodgkin's disease. Gram's stain,

magnified 1000 times, (in oil). (click here to enlarge image).

>

> Figure 3: Solitary " giant " body in a lymph node of Hodgkin's disease

(cancer), Gram's stain, magnified 1000 times (click here to enlarge image).

>

> Figure 4: Larger bodies (RB) in a lymph node showing

> non-cancerous " reactive lymph node hyperplasia " from a fatal case of

> AIDS. The arrow points to nearby bacterial-sized intracellular coccoid

> smaller forms from which the bodies are derived. Fite

> (acid-fast) stain, magnified 1000 times (click here to enlarge image).

>

> Figure 5: Extremely large " super-giant-sized " solitary body in

> the skin of " cutaneous lupus erythematosus " , a so-called " collagen

> disease. " The perfectly round shape, except for one area, suggests this

> large body is developing inside a cell that is readly to burst.

> Kinyoun's (acid-fast) stain, magnification x 1000 (click here to enlarge

image).

>

> Figure 6: Pleomorphic growth forms (L-forms) of tuberculosis

> mycobacteria photographed with an electron microscope. Note the darker

> staining tiny coccal forms (similar in size to ordinary staphylococci)

> and the larger clear balloon-sized " ghost " forms similar in size and

> shape to bodies found in tissue. These forms are all

> characteristic of " cell wall-deficient bacteria " and totally unlike the

> well-known " typical " acid-fast rod forms of Mycobacterium tuberculosis.

> Reproduced from L-forms of Mycobacteria and Chronic Nephritis (1970), by

> Dr. C. Xalabarder P., page 51 (click here to enlarge image).

>

> BIBLIOGRAPHY:

>

> 1. - E. A specific type of microorganism isolated from

> animal and human cancer: bacteriology of the organism. Growth. 1954

> Mar;18(1):37-51.

>

> 2. Brown ST, Brett I, Almenoff PL, Lesser M, Terrin M, Teirstein AS.

> Recovery of cell wall-deficient organisms from blood does not

> distinguish between patients with sarcoidosis and control subjects.

> Chest. 2003 Feb;123(2):413-417.

>

> 3. Cantwell AR, Craggs E, JW, Swatek F. Acid-fast bacteria as a

> possible cause of scleroderma. Dermatologica. 1968: 136:141-150.

>

> 4. Cantwell AR. Histologic forms resembling " large bodies " in

> scleroderma and pseudoscleroderma. Amer J Dermatopathol. 1980;

> 2:273-276.

>

> 5. Cantwell AR, Rowe L. African " eosinophilic bodies " in vivo in two

> American men with Kaposi's sarcoma and AIDS. J Dermatol Surg Oncol. 1985

> Apr;11(4):408-12.

>

> 6. Cantwell AR, Kelso DW, JE. Histologic observations of coccoid

> forms suggestive of cell wall deficient bacteria in cutaneous and

> systemic lupus erythematosus.

>

> Int J Dermatol. 1982 Nov;21(9):526-37.

>

> 7. Cantwell AR. Histologic observations of variably acid-fast

> pleomorphic bacteria in systemic sarcoidosis: a report of 3 cases.

> Growth. 1982 Summer;46(2):113-25.

>

> 8. Cantwell AR. Variably acid-fast cell wall-deficient bacteria as a

> possible cause of dermatologic disease. In, Domingue GJ (Ed). Cell Wall

> Deficient Bacteria. Reading: -Wesley Publishing Co; 1982. Pp.

> 321-360.

>

> 9. Cantwell A. The Cancer Microbe. Los Angeles: Aries Rising Press; 1990.

>

> 10. Dienes L. Morphology and reproductive processes of bacteria with

> defective cell walls. In, Guze LB (Ed). Microbial Protoplasts,

> Spheroplasts and L-Forms. Baltimore: & Wilkins Company;

> 1968, Pp 74-93.

>

> 11. Diller IC, Diller WF. Intracellular acid-fast organisms isolated

> from malignant tissues. Trans Amer Micr Soc. 1965; 84:138-148.

>

> 12. Ewing J. The parasitic theory. In, Ewing J (Ed), Neoplastic Diseases (Ed

1); Philadelphia: Saunders; 1919. Pp 114-126.

>

> 13. Gaylord HR. The protozoon of cancer. Amer J Med Sci. 1901;121:501-539.

>

> 14. Gebbers JO, Otto HF. Plasma cell alterations in ulcerative colitis.

> An electron microscopic study. Pathol Eur. 1976;11(4):271-9.

>

> 15. Glover TJ. The bacteriology of cancer. Canada Lancet Pract. 1930;

75:92-111.

>

> 16. Haensch R, Seeliger H. Problems of differential diagnosis of

> blastomyces and bodies. Arch Dermatol Res. 1981;270(4):381-5.

>

> 17. Hess D. Can Bacteria Cause Cancer? New York:New York University Press;

1997.

>

> 18. Jetha N, Priddy RW. Exact nature of bodies still an enigma. Am J

Clin Pathol. 1984 Apr;81(4):545.

>

> 19. King DF, Eisenberg D. 's fuchsine body. " The characteristic

> organism of cancer " . Am J Dermatopathol. 1981 Spring;3(1):55-8.

>

> 20. Mattman LH. Cell Wall Deficient Forms (Ed 2). Boca Raton:CRC Press; 1993.

>

> 21. Mazet G. Corynebacterium, tubercle bacillus and cancer. Growth. 1974; 38:

>

> 22. McLaughlin RW, Vali H, Lau PC, Palfree RG, De Ciccio A, Sirois M,

> Ahmad D, Villemur R, Desrosiers M, Chan E. Are there naturally occurring

> pleomorphic bacteria in the blood of healthy humans? J Clin Microbiol.

> 2002 Dec;40(12):4771-5.

>

> 23. Nuzum JW. The experimental production of metastasizing carcinoma of

> the breast of the dog and primary epithelioma in man by repeated

> inoculation of a micrococcus isolated from human breast cancer. Surg

> Gynecol Obstet. 1925; 11;343-352.

>

> 24. W. An address on a characteristic organism of cancer. Br Med J.

1890; 2:1356-1360.

>

> 25. W. The parasite of cancer. Lancet. 1899;1:1138-1141.

>

> 26. Schleicher EM. Giant bodies in neoplastic cells in a case of

> leukemic lymphosarcomatosis. Minnesota Medicine. 1965; 48:1125-1130.

>

> 27. MJ. The parasitic origin of carcinoma. Northwest Med.

1925;24:162-166.

>

> 28. Seibert FB, Feldmann FM, RL, Richmond IS. Morphological,

> biological, and immunological studies on isolates from tumors and

> leukemic bloods. Ann N Y Acad Sci. 1970 Oct 30;174(2):690-728.

>

> 29. Tedeschi GG, Amici D. Mycoplasma-like microorganisms probably

> related to L forms of bacteria in the blood of healthy persons.

> Cultural, morphological and histochemical data. Ann Sclavo. 1972

> Jul-Aug;14(4):430-42.

>

> 30. Tedeschi GG, Bondi A, Paparelli M, Sprovieri G. Electron

> microscopical evidence of the evolution of corynebacteria-like

> microorganisms within human erythrocytes. Experientia. 1978 Apr

> 15;34(4):458-60.

>

> 31. Vento S, Cainelli F. Infections in patients with cancer undergoing

> chemotherapy: aetiology, prevention, and treatment. Lancet Oncol. 2003

> Oct;4(10):595-604.

>

> 32. Von Brehmer W. " Siphonospora polymorphia " n. sp., neuer

> Mikroorganismus des Blutes und seine Beziehung zur Tumororigenese. Med

> Welt. 1934; 8:1179-1185.

>

> 33. White RG. Observations on the formation and nature of bodies. Br J

Exp Pathol. 1954; 35:365-376.

>

> 34. Wuerthele Caspe-Livingston V, - E, JA, et

> al. Cultural properties and pathogenicity of certain microorganisms

> obtained from various proliferative and neoplastic diseases. Amer J Med

> Sci. 1950; 220;628-646.

>

> 35. Wuerthele-Caspe Livingston V, Livingston AM. Demonstration of

> Progenitor cryptocides in the blood of patients with collagen and

> neoplastic diseases. Trans NY Acad Sci. 1972; 174 (2):636-654.

>

> 36. Xalabarder C. La desconocida patologia provocada por micobacterias. Publ

Inst Antituberc. 1967; 17:35-52.

>

> 37. Xalabarder C: Formas L de micobacterias y nefritis cronicas. Publ Inst

Antituberc (Barcelona). 1970; Supple 7:7-83.

>

> 38. Xalabarder C. Sarcoidosis experimental. Publ Inst Antituberc (Barcelona).

1969; 8:51-76.

>

> 39. Young J. Description of an oganism obtained from carcinomatous growths.

Edinburgh Med J. 1921; 27:212-221.

>

> KEY WORDS

>

> Bacteria, pleomorphic

>

> cancer-associated bacteria

>

> cancer, origin

>

> cell wall-deficient bacteria

>

> Hodgkin's disease

>

> L-forms

>

> lupus erythematosus

>

> mycobacteria

>

> body

>

> sarcoidosis

>

> scleroderma

>

> tuberculosis

>

> Go back to Main Menu

>

> Want to learn how to make your papers look better, with bold text and embedded

images?

> Here is the info...

>

> Papers belong to the

> There is no

> expectation of

> Privacy for

> any material posted on this site. See our Privacy Policy.

> All

> rights reserved. Click to email webmaster. Powered by Phorum, Apache, MySQL,

> and Linux

>

Sign In

Re: JOURNAL OF INDEPENDENT MEDICAL RESEARCH

Recommended Posts

Guest guest

Link to comment

Share on other sites

Join the conversation

Activity