Guest guest Posted March 29, 2008 Report Share Posted March 29, 2008 Ha! See if you read into this what I am reading into it, concerning a balance between extra thyroid hormones in the body. They've been trying to say all these decades that thyroid hormone can cause osteoporosis. What if it's a big balancing act that keeps this from happening? We don't know, do we?! Of course, that is not the main thrust of Shomon's article or the point of the doctor study. After reading the short article, if you scroll down to the bottom, you can go read the study abstract. It is in tiny wee letters right after the article. Maybe someone else was up on this way before me, but I'm nonetheless very excited. But would it stimulate the thyroid into action, when it shouldn't be (low thyroid with a damaged Hashi's gland or Grave's condition, etc..), or is that only the pathway from the pituitary TSH, instead of by injection, as they did? Wondering. My TSH has been down below a .1 for a few yrs now. http://thyroid.about.com/b/2008/03/24/thyroid-stimulating-hormone-may-be-future-osteoporosis-treatment.htm Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 29, 2008 Report Share Posted March 29, 2008 and All, I’ve always thought that my bone loss was due to me being Hypo. I get a bone density test done yearly. When I was undermedicated,it showed decreasing bone density. Now that my TSH is suppressed, and I am on T4/T3 combo. I have shown no bone loss. I think that the Pharmaceutical’s have started a nasty rumor stating that a suppressed TSH causes bone loss, so that no one will take a combination of T4/T3 medication…….My theory and I’m sticking to it! Ha ha Lynn Ha! See if you read into this what I am reading into it, concerning a balance between extra thyroid hormones in the body. They've been trying to say all these decades that thyroid hormone can cause osteoporosis. What if it's a big balancing act that keeps this from happening? We don't know, do we?! Of course, that is not the main thrust of Shomon's article or the point of the doctor study. After reading the short article, if you scroll down to the bottom, you can go read the study abstract. It is in tiny wee letters right after the article. Maybe someone else was up on this way before me, but I'm nonetheless very excited. But would it stimulate the thyroid into action, when it shouldn't be (low thyroid with a damaged Hashi's gland or Grave's condition, etc..), or is that only the pathway from the pituitary TSH, instead of by injection, as they did? Wondering. My TSH has been down below a .1 for a few yrs now. http://thyroid.about.com/b/2008/03/24/thyroid-stimulating-hormone-may-be-future-osteoporosis-treatment.htm Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 30, 2008 Report Share Posted March 30, 2008 What I think is that when the pituitary and the thyroid were working *together*, that balance was there, but when the thyroid itself gets sick, there goes the balance, as in we need more thyroid hormone, and with Hashi's, the thyroid can no longer be haggled like it always was in a normal fashion. That's the sad part, is that what was meant in nature to operate one way, has to be changed to another way. Hashi's patients' TSHs have to be either suppressed (if tolerable to that person) or near suppression, if they feel good with it suppressed, so there goes *nature's balance* down the tubes. This is why I was putting forth the supposition of injected TSH possibly bypassing that route to the thyroid gland, so it is not stimulated at all. If TSH really does feed into bone building, then I would wish that they could figure out a way to do this. Did you read the study? I think, from the readings I've done, that thyroid hormones actually help to remodel the bone. It says so in many places. So I'm thinking that it was actually designed to work hand in hand with it's stimulant, but that's what happens when the feedback system is totally destroyed. Part of the disease is to screw up everything that was a family at one time. I shudder at the thought of my TSH getting over what it is now, as it's been suppressed for several yrs now (Hashi's), but it could be that what they are studying is true. I try to keep an open mind on this, as it comes along, but am I going to let my TSH get much over what it is now? No way! Please don't shoot the messenger, as this is one study done here: http://www.tbiomed.com/content/4/1/23 And this is quite interesting, as I've never heard the flat-out statement made by lab people and creators about the untrue efficacy of our present-day TSH testing, sensitive or not, quote from this link, but bah humbug on the .4-5.0 thing. Notice what I've highlighted in red, it's fascinating, considering that almost the whole medical community goes by this TSH reading, ha: http://www.thyroidmanager.org/Chapter4/4-frame.htm Normal Physiology The concentration of TSH can now be measured with exquisite sensitivity using immunometric techniques (see below). In euthyroid humans, this concentration is 0.4-0.5 to 4.0-5.0 mU/L. This normal range is to some extent method-dependent in that the various assays use reference preparations of slightly varying biological potency. There is no crystalline human TSH preparation, so it is not possible to provide a precise molar equivalent for TSH concentrations. Chapter 4. Normal Physiology of the Hypothalmic-Pituitary-Thyroidal System and Relation to the Neural System and Other Endocrine Gland by Stefano tti, M.D. Last Revised: January 5, 2006 RE: TSH Has SOME Use! and All, I’ve always thought that my bone loss was due to me being Hypo. I get a bone density test done yearly. When I was undermedicated,it showed decreasing bone density. Now that my TSH is suppressed, and I am on T4/T3 combo. I have shown no bone loss. I think that the Pharmaceutical’s have started a nasty rumor stating that a suppressed TSH causes bone loss, so that no one will take a combination of T4/T3 medication…….My theory and I’m sticking to it! Ha ha Lynn Quote Link to comment Share on other sites More sharing options...
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