Re: Re: Lerner's response to glutathione depletion.-methylation cycle block hypothesis

[Guest guest]

lol! I kinda know how to get to the file section here, but perhaps others don't so it's always helpful to post a link, especially for those who read through email. Just to clear it up, I did look there before and didn't see it, hence I asked for the link. Either I looked too soon or it was a case of temporary blindness. I do see it now, though. Maybe it's a miracle. I CAN SEE!!!! thanks, pennyrvankonynen <richvank@...> wrote: Hi, Penny.Sorry, I don't have one yet. Dr. Schaller said he will put it on his website, but it isn't there yet. I don't maintain a website myself.Here's a direct link to the files section of your list, if that helps:infections/files/Rich--- In infections , "penny " <pennyhoule@...> wrote:>> Hi Rich,> > Would you mind posting a direct link? Thanks.> > penny> > > --- In infections , "rvankonynen" > <richvank@>

wrote:> >> > Hi, all.> > > > One of the people I met for the first time at the IACFS meeting was > > Dr. A. Lerner. As you may know, he is the doctor from > > Michigan who has specialized in treating viral infections of the > > heart, producing cardiomyopathy in CFS. He had this condition > > himself some years ago. > > > > At the meeting, I gave him copies of my poster papers, and in > > particular told him that I would like to have his comments on my > > pathogenesis paper (posted in the files of this list at GD-MCB > etc.) > > I saw him again later in the conference, and he told me that he had > > read my paper. His comment was, "It makes sense." I was really > > happy to hear this from a person who has focused on the viral > > infection aspect of CFS, since I expected that he would believe

> that > > the viruses were the "first cause." But he was willing to accept > the > > chain of events as I presented them. > > > > In the paper, I discuss viral infections, including why the viral > > infections that are present initially in CFS are the ones that are > > observed, what causes latent viruses to reactivate, how the immune > > system responds to them, including the low activity of the NK > cells, > > the activation of the RNase-L system, the shift to Th2 immune > > response, why the infections aren't knocked out, and why more > > infections accumulate over time in a PWC's body. I discuss why > > sackie seems to be more prevalent in PWCs in the UK. I also > > discuss intracellular bacterial infections, and why Chlamydia > > reactivates. Furthermore, I discuss why inflammation is more of a >

> problem later in the course of the illness. > > > > I invite everyone to take a look at this paper. There really is > > something in there for everyone, since I pulled together > essentially > > all the observed features of CFS into a common cause-effect tree. > I > > would appreciate your comments. > > > > Rich> >>

[Guest guest]

Rich, what’s your opinion of women

taking Indole 3 Carbinol for CFS or autoimmune disease?  Patrice

From: infections [mailto:infections ] On Behalf Of rvankonynen

Sent: Thursday, January 18, 2007

2:38 PM

infections

Subject:

[infections] Re: Lerner's response to glutathione

depletion.-methylation cycle block hypothesis

Hi, Penny.

My papers just came up on Co-Cure. For those who aren't signed on to

Co-Cure, you can find them toward the bottom of this page of the

archives:

http://listserv.nodak.edu/cgi-bin/wa.exe?A1=ind0701c & L=co-cure

Rich

> > >

> > > Hi, all.

> > >

> > > One of the people I met for the first time at the IACFS meeting

> was

> > > Dr. A. Lerner. As you may know, he is the doctor from

> > > Michigan

who has specialized in treating viral infections of

the

> > > heart, producing cardiomyopathy in CFS. He had this condition

> > > himself some years ago.

> > >

> > > At the meeting, I gave him copies of my poster papers, and in

> > > particular told him that I would like to have his comments on

my

> > > pathogenesis paper (posted in the files of this list at GD-MCB

> > etc.)

> > > I saw him again later in the conference, and he told me that he

> had

> > > read my paper. His comment was, " It makes sense. " I

was

really

> > > happy to hear this from a person who has focused on the viral

> > > infection aspect of CFS, since I expected that he would believe

> > that

> > > the viruses were the " first cause. " But he was willing

to

accept

> > the

> > > chain of events as I presented them.

> > >

> > > In the paper, I discuss viral infections, including why the

viral

> > > infections that are present initially in CFS are the ones that

> are

> > > observed, what causes latent viruses to reactivate, how the

> immune

> > > system responds to them, including the low activity of the NK

> > cells,

> > > the activation of the RNase-L system, the shift to Th2 immune

> > > response, why the infections aren't knocked out, and why more

> > > infections accumulate over time in a PWC's body. I discuss why

> > > sackie seems to be more prevalent in PWCs in the UK. I also

> > > discuss intracellular bacterial infections, and why Chlamydia

> > > reactivates. Furthermore, I discuss why inflammation is more

of

> a

> > > problem later in the course of the illness.

> > >

> > > I invite everyone to take a look at this paper. There really

is

> > > something in there for everyone, since I pulled together

> > essentially

> > > all the observed features of CFS into a common cause-effect

> tree.

> > I

> > > would appreciate your comments.

> > >

> > > Rich

> > >

> >

>