Re: Lerner's response to glutathione depletion.-methylation cycle block hypothesis

[Guest guest]

Hi Rich,

Would you mind posting a direct link? Thanks.

penny

>

> Hi, all.

>

> One of the people I met for the first time at the IACFS meeting was

> Dr. A. Lerner. As you may know, he is the doctor from

> Michigan who has specialized in treating viral infections of the

> heart, producing cardiomyopathy in CFS. He had this condition

> himself some years ago.

>

> At the meeting, I gave him copies of my poster papers, and in

> particular told him that I would like to have his comments on my

> pathogenesis paper (posted in the files of this list at GD-MCB

etc.)

> I saw him again later in the conference, and he told me that he had

> read my paper. His comment was, " It makes sense. " I was really

> happy to hear this from a person who has focused on the viral

> infection aspect of CFS, since I expected that he would believe

that

> the viruses were the " first cause. " But he was willing to accept

the

> chain of events as I presented them.

>

> In the paper, I discuss viral infections, including why the viral

> infections that are present initially in CFS are the ones that are

> observed, what causes latent viruses to reactivate, how the immune

> system responds to them, including the low activity of the NK

cells,

> the activation of the RNase-L system, the shift to Th2 immune

> response, why the infections aren't knocked out, and why more

> infections accumulate over time in a PWC's body. I discuss why

> sackie seems to be more prevalent in PWCs in the UK. I also

> discuss intracellular bacterial infections, and why Chlamydia

> reactivates. Furthermore, I discuss why inflammation is more of a

> problem later in the course of the illness.

>

> I invite everyone to take a look at this paper. There really is

> something in there for everyone, since I pulled together

essentially

> all the observed features of CFS into a common cause-effect tree.

I

> would appreciate your comments.

>

> Rich

>

[Guest guest]

Hi, Penny.

Sorry, I don't have one yet. Dr. Schaller said he will put it on his

website, but it isn't there yet. I don't maintain a website myself.

Here's a direct link to the files section of your list, if that helps:

infections/files/

Rich

> >

> > Hi, all.

> >

> > One of the people I met for the first time at the IACFS meeting

was

> > Dr. A. Lerner. As you may know, he is the doctor from

> > Michigan who has specialized in treating viral infections of the

> > heart, producing cardiomyopathy in CFS. He had this condition

> > himself some years ago.

> >

> > At the meeting, I gave him copies of my poster papers, and in

> > particular told him that I would like to have his comments on my

> > pathogenesis paper (posted in the files of this list at GD-MCB

> etc.)

> > I saw him again later in the conference, and he told me that he

had

> > read my paper. His comment was, " It makes sense. " I was really

> > happy to hear this from a person who has focused on the viral

> > infection aspect of CFS, since I expected that he would believe

> that

> > the viruses were the " first cause. " But he was willing to accept

> the

> > chain of events as I presented them.

> >

> > In the paper, I discuss viral infections, including why the viral

> > infections that are present initially in CFS are the ones that

are

> > observed, what causes latent viruses to reactivate, how the

immune

> > system responds to them, including the low activity of the NK

> cells,

> > the activation of the RNase-L system, the shift to Th2 immune

> > response, why the infections aren't knocked out, and why more

> > infections accumulate over time in a PWC's body. I discuss why

> > sackie seems to be more prevalent in PWCs in the UK. I also

> > discuss intracellular bacterial infections, and why Chlamydia

> > reactivates. Furthermore, I discuss why inflammation is more of

a

> > problem later in the course of the illness.

> >

> > I invite everyone to take a look at this paper. There really is

> > something in there for everyone, since I pulled together

> essentially

> > all the observed features of CFS into a common cause-effect

tree.

> I

> > would appreciate your comments.

> >

> > Rich

> >

>

[Guest guest]

Hi, Penny.

My papers just came up on Co-Cure. For those who aren't signed on to

Co-Cure, you can find them toward the bottom of this page of the

archives:

http://listserv.nodak.edu/cgi-bin/wa.exe?A1=ind0701c & L=co-cure

Rich

> > >

> > > Hi, all.

> > >

> > > One of the people I met for the first time at the IACFS meeting

> was

> > > Dr. A. Lerner. As you may know, he is the doctor from

> > > Michigan who has specialized in treating viral infections of

the

> > > heart, producing cardiomyopathy in CFS. He had this condition

> > > himself some years ago.

> > >

> > > At the meeting, I gave him copies of my poster papers, and in

> > > particular told him that I would like to have his comments on

my

> > > pathogenesis paper (posted in the files of this list at GD-MCB

> > etc.)

> > > I saw him again later in the conference, and he told me that he

> had

> > > read my paper. His comment was, " It makes sense. " I was

really

> > > happy to hear this from a person who has focused on the viral

> > > infection aspect of CFS, since I expected that he would believe

> > that

> > > the viruses were the " first cause. " But he was willing to

accept

> > the

> > > chain of events as I presented them.

> > >

> > > In the paper, I discuss viral infections, including why the

viral

> > > infections that are present initially in CFS are the ones that

> are

> > > observed, what causes latent viruses to reactivate, how the

> immune

> > > system responds to them, including the low activity of the NK

> > cells,

> > > the activation of the RNase-L system, the shift to Th2 immune

> > > response, why the infections aren't knocked out, and why more

> > > infections accumulate over time in a PWC's body. I discuss why

> > > sackie seems to be more prevalent in PWCs in the UK. I also

> > > discuss intracellular bacterial infections, and why Chlamydia

> > > reactivates. Furthermore, I discuss why inflammation is more

of

> a

> > > problem later in the course of the illness.

> > >

> > > I invite everyone to take a look at this paper. There really

is

> > > something in there for everyone, since I pulled together

> > essentially

> > > all the observed features of CFS into a common cause-effect

> tree.

> > I

> > > would appreciate your comments.

> > >

> > > Rich

> > >

> >

>