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has pointed out a number of times that autoantibodies are

common, and are not generated only in patients experiencing

autoimmune diseases.

Various proteins are capable of putting the brakes on inflammation.

IL-10 is the obvious example, but there are others. So what if the

autoantibody that some people generate happens to neutralize one of

these " brakes " ? The inflammation might persist unabated, and might

perpetuate the production of the original offending autoantibody.

Today's offering by Nature suggests that this indeed happens with MS:

http://tinyurl.com/yofjda

They present data indicating that MS patients, but not control

patients, produce antibody against CRYAB, a molecule that inhibits

inflammation. They present other data, in mice, indicating the

negative impact that this has.

If true, this is spectacular.

Matt

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