Y Shoenfeld

[Guest guest]

> Possibly a

> good guy to have a pubmed alert on in order to keep track of trends

> and developments in orthodox AI research.

Eg:

" Within the last year, a growing body of evidence for a distinct role

of antineutrophil cytoplasmic antibodies (ANCA) in the pathogenesis of

ANCA-associated vasculitides (AAV) has developed. An experimental

model of myeloperoxidase (MPO)-ANCA-associated vasculitis provided

direct [Ahem, " direct " ?] and convincing in vivo evidence that MPO-ANCA

are primary pathogenic factors in small-vessel vasculitis by

augmenting of leukocyte-vessel wall interaction and leukocyte-mediated

vascular injury. Determination of bacterial lipopolysaccharide (LPS)

effects on disease severity in a mouse model of anti-MPO-induced

glomerulonephritis showed that ANCA and other proinflammatory stimuli

of infectious origin acted in synergism in the development of

destructive inflammation. " PMID: 16839508

> I think he seemed pretty

> thoughtful to me when I read one of his papers, but I haven't read

> much of him.

At any rate, there's no doubt he picks a lot of interesting topics to

review. He also has written on CFS, and seems to be particularly

interested in infection's role in AI.

[Guest guest]

I love the guy... :-) penny <usenethod@...> wrote: > Possibly a> good guy to have a pubmed alert on in order to keep track of trends> and developments in orthodox AI research. Eg:"Within the last year, a growing body of evidence for a distinct roleof antineutrophil cytoplasmic antibodies (ANCA) in the pathogenesis ofANCA-associated vasculitides (AAV) has developed. An experimentalmodel of

myeloperoxidase (MPO)-ANCA-associated vasculitis provideddirect [Ahem, "direct"?] and convincing in vivo evidence that MPO-ANCAare primary pathogenic factors in small-vessel vasculitis byaugmenting of leukocyte-vessel wall interaction and leukocyte-mediatedvascular injury. Determination of bacterial lipopolysaccharide (LPS)effects on disease severity in a mouse model of anti-MPO-inducedglomerulonephritis showed that ANCA and other proinflammatory stimuliof infectious origin acted in synergism in the development ofdestructive inflammation." PMID: 16839508> I think he seemed pretty> thoughtful to me when I read one of his papers, but I haven't read> much of him.At any rate, there's no doubt he picks a lot of interesting topics toreview. He also has written on CFS, and seems to be particularlyinterested in infection's role in AI.