Re: Re: Teminalia - even more antimicrobial properties

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More good news. According to this study, Teminalia bellerica was highly effective (in vitro) against Staph Aureus and other organisms, including yeast. It mentions it being even more potent when converted to ethanol than in it's crude form (with the pseudomonas being the only exception). How that's done, I have no idea, but even in it's crude form, it looks very effective against quite a few organisms. Also, I'm not sure how or if this Teminalia bellerica differs from T. Chebula. http://ijcb.co.in/iaft05i2p150.pdf I still haven't figured out how or where to buy it. penny Penny Houle <pennyhoule@...> wrote: Also, Dan, where do you buy your Terminalia chebula? This sounds promising as well. Kudos to the remarkable girl and her research not to mention her desire to create a spray delivery form. What an inspiration. I think I'll dissolve some and put it in my nasal irrigator. penny kdrbrill <kdrbrill > wrote: Interesting

you should mention horses Penny. I found these links last week when I was diagnosed. This guy Eby (the man who came up with the zinc lozenge for colds) -- found that treating lame horses with the trace mineral "gallium" restored their energy, etc., after a week or so of external rubs and soaks.But the interesting part was that the people treating horses found that THEIR pains went away as well.http://george-eby-research.com/html/arthritis.htmlAnd even more interesting is this PubMed study that found that gallium inhibits pseudomonas growth:http://www.jci.org/cgi/content/abstract/117/4/877INTERESTING...???Dan--- In infections , Penny Houle <pennyhoule@...> wrote:>> Yes, definitely will consider trying it. That would be great if it can eradicate pseudomonas. Pseudomonas is a big problem for horses so it could have a lot of applications. Even if people are a bit too short sighted to recognize it as a threat, at least horses could possibly benefit. Barb, have you heard of this?> > penny> > kdrbrill <kdrbrill@...> wrote:> I encourage you to look at the link on the east indian herbal remedy again...it killed it, > destroyed the (usually difficult to destroy) outer membrane that it uses to protect itself.> > http://cfvancouver.ca/cms/index.php?option=com_content & task=view & id=63 & Itemid=38> > d. > > > > > > > > > >> > > > > > > > Hi, all.> > > > > > > > > > > > > > > > I know that some folks here would prefer that I not post > > > things > > > > > > > about> > > > > > > > the methylation cycle block treatment on this list, but > > > please> > > > > > > > forgive me for this one. I think it's a biggie, and I think> > > > > > > > everybody here should hear about it.> > > > > > > > > > > > > > > > A woman on the ImmuneSuppport CFS

discussion board who is > > > on the> > > > > > > > simplified treatment approach for lifting the methylation > > > cycle > > > > > > > block> > > > > > > > just reported that she was able to stop> > > > > > > > her use of desmopressin (which she had been using since last> > > > > > > > September to control her heavy urine volume), and her urine > > > volume> > > > > > > > did not jump back up, as it formerly did when she stopped > > > the> > > > > > > > desmopressin. I think this agrees with Hall's report > > > some > > > > > > time> > > > > > > > ago that restoring his glutathione level corrected his > > > diabetes> > > > > >

> > insipidus, too. Here is the response I wrote to this woman. > > > I took> > > > > > > > her name off to protect her privacy, but she posted to a > > > public> > > > > > > > discussion board, and you can read her post there.> > > > > > > > > > > > > > > > Rich> > > > > > > > > > > > > > > > > > > > > > > > Hi, _______.> > > > > > > > > > > > > > > > This is wonderful! It's wonderful both for you and for me > > > and for > > > > > > > the> > > > > > > > whole CFS community, because it provides more observational > > > > > > support> > > > > > > > for the GD-MCB

hypothesis.> > > > > > > > > > > > > > > > As you probably know (but I want to make sure other readers > > > are > > > > > > > aware> > > > > > > > of it, too), part of this hypothesis says that the low > > > production > > > > > > of> > > > > > > > antidiuretic hormone (also called arginine vasopressin) in > > > CFS> > > > > > > > results from low glutathione in the hypothalamus. This > > > results in > > > > > > a> > > > > > > > (usually mild) case of diabetes insipidus, not to be > > > confused with> > > > > > > > diabetes mellitus, which involves high blood sugar and low > > > > > > insulin.> > > > > >

> > > > > > > > > > "Diabetes" means you have a lot of urine. "Mellitus" means > > > your > > > > > > > urine> > > > > > > > tastes sweet, because it has elevated blood sugar or > > > glucose in> > > > > > > > it. "Insipidus" means that your urine tastes insipid, i.e. > > > it > > > > > > isn't> > > > > > > > sweet. Not many people like to diagnose these by tasting > > > the urine> > > > > > > > these days, but it's much quicker than doing the lab tests! > > > (:-)).> > > > > > > > > > > > > > > > Diabetes insipidus produces high urine volume and low total > > > blood> > > > > > > > volume, as well as constant thirst. This

is the phenomenon > > > in CFS> > > > > > > > that Dr. Teitelbaum refers to as "Pee like a racehorse, > > > drink > > > > > > like a> > > > > > > > fish."> > > > > > > > > > > > > > > > The simplified treatment approach, among other things, is > > > > > > designed > > > > > > > to> > > > > > > > allow glutathione levels to come up to normal. When this > > > happens, > > > > > > we> > > > > > > > should expect that the diabetes insipidus will disappear, > > > and you> > > > > > > > have verified that it did in your case.> > > > > > > > > > > > > > > > I should make a small correction to

what you wrote, in that > > > while> > > > > > > > this does involve the hypothalamus, it doesn't actually say > > > that > > > > > > the> > > > > > > > HPA (hypothalamus-pituitary-adrenal) axis has been restored > > > to > > > > > > > normal> > > > > > > > operation. I expect that that will occur as well, but the> > > > > > > > disappearance of the diabetes insipidus does not prove > > > that. > > > > > > > Evidence> > > > > > > > for improvement in the HPA axis would include things like > > > blood> > > > > > > > pressure coming up to normal, decrease in symptoms of > > > > > > hypoglycemia,> > > > > > > >

cortisol and DHEA levels restored to normal, disappearance > > > of> > > > > > > > orthostatic problems such as problems with blood pressure > > > or heart> > > > > > > > rate when standing, better ability to cope with stress of > > > all > > > > > > sorts,> > > > > > > > and other cortisol-related things. If you are observing > > > those > > > > > > things> > > > > > > > as well, then I would agree that your HPA axis is doing > > > better, > > > > > > too.> > > > > > > > I fully expect that to happen for you, too, if it hasn't > > > already,> > > > > > > > because the same basic mechanism in the biochemistry that > > > restored> > > >

> > > > ADH should also restore ACTH, which I think will bring the > > > HPA > > > > > > axis> > > > > > > > back to normal operation.> > > > > > > > > > > > > > > > At the biochemical level, I think this observation also > > > supports > > > > > > my> > > > > > > > more fundamental suggestion that secretory proteins that > > > contain> > > > > > > > cysteine double bonds are not being made well in CFS > > > because of> > > > > > > > glutathione depletion in the cells in which they are made. > > > If > > > > > > this > > > > > > > is> > > > > > > > true, it also provides support for my hypotheses to explain >

> > low> > > > > > > > levels of some of the other secretory proteins in CFS, > > > including> > > > > > > > human growth hormone, ACTH, oxytocin, perforin, and > > > probably some> > > > > > > > others as well. So this is big, from my point of view!> > > > > > > > > > > > > > > > Thank you so much for posting this, and keep on keeping on!> > > > > > > > > > > > > > > > Rich> > > > > > > >> > > > > > >> > > > > >> > > > >> > > >> > >> >>