Guest guest Posted May 31, 2008 Report Share Posted May 31, 2008 Because Dr. Hoffer, Md that helped my son so much with his info regarding niacin and vitamin C long before Amy, and thanks God i did it and yes it did work for us witout paying so much for testing. my son i guess was high in kynurenine but I still continued to give him until he went over the exitatory period and the infection I guess was over because then he just come down and overall improve his mental and body health as dr. Hoffer predicted. Or maybe the introduction of lumbrokinase was the key, overall it worked fine with my extra idea of introducing lumbrokinase to the soup. Dr. Hoffer told me that treatment plan with b3 and vit C for free! and I shared with everyone else for free as well. . Quinolinic acid was found to be substantially elevated in patients with Borrelia burgdorferi (Lyme) infection aid has been postulated to play a role in contributing to neurological and cognitive defects associated with Lyme disease. It is of importance to try to look at why you may have elevated activity in the tryptophan breakdown pathway in the first place. Decreased methylation, increased ID0 (an enzyme that is also effected by methylation and other factors), and chronic streptococcal or even B. burgdorferi (lyme) infection can lead to stimulation of this pathway. The final breakdown product of the tryptophan pathway is niacinamide. This B vitamin has been reported to have antimicrobial effects. It may be that the body is trying to address bacterial infection by breaking down tryptophan into niacinamide to help with infection. In some cases I have not found that the use of high dose B6 or P5P is always helpful. It can actually cause more overstimulatory or OCD type behaviors. While kynurenic acid is calming for neurotransmitters, the product that kynurenines are converted to by B6 is quinolinic acid. Quinolinic acid is an excitotoxin. So if you have high kynurenine and add B6 you can generate quinolinic acid which acts as an excitotoxin and can aggravate the nervous system. Increased levels of quinolinic acid have been implicated in Alzheimerds disease as well as with respect to excitotoxin damage of nerves. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted May 31, 2008 Report Share Posted May 31, 2008 Lyme infection in the body. Lyme disease has been implicated in a number of neurological conditions. Cat's claw is reported to be helpful for viral issues as well as for Lyme. Artemisia (wormwood) has also shown activity against Lyme. Wormwood is a component of Paradex that was already mentioned with respect to other parasitic infections. In addition, tick support RNAs and IMF 2 should help to support the body. Chronic issues with Lyme may overlap with thyroid dysfunction. From: Sent: Saturday, May 31, 2008 6:36 PMTo: 'BorreliaMultipleInfectionsAndAutism 'Subject: some amy yasko comments i encounter interesting... Because Dr. Hoffer, Md that helped my son so much with his info regarding niacin and vitamin C long before Amy, and thanks God i did it and yes it did work for us witout paying so much for testing. my son i guess was high in kynurenine but I still continued to give him until he went over the exitatory period and the infection I guess was over because then he just come down and overall improve his mental and body health as dr. Hoffer predicted. Or maybe the introduction of lumbrokinase was the key, overall it worked fine with my extra idea of introducing lumbrokinase to the soup. Dr. Hoffer told me that treatment plan with b3 and vit C for free! and I shared with everyone else for free as well. . Quinolinic acid was found to be substantially elevated in patients with Borrelia burgdorferi (Lyme) infection aid has been postulated to play a role in contributing to neurological and cognitive defects associated with Lyme disease. It is of importance to try to look at why you may have elevated activity in the tryptophan breakdown pathway in the first place. Decreased methylation, increased ID0 (an enzyme that is also effected by methylation and other factors), and chronic streptococcal or even B. burgdorferi (lyme) infection can lead to stimulation of this pathway. The final breakdown product of the tryptophan pathway is niacinamide. This B vitamin has been reported to have antimicrobial effects. It may be that the body is trying to address bacterial infection by breaking down tryptophan into niacinamide to help with infection. In some cases I have not found that the use of high dose B6 or P5P is always helpful. It can actually cause more overstimulatory or OCD type behaviors. While kynurenic acid is calming for neurotransmitters, the product that kynurenines are converted to by B6 is quinolinic acid. Quinolinic acid is an excitotoxin. So if you have high kynurenine and add B6 you can generate quinolinic acid which acts as an excitotoxin and can aggravate the nervous system. Increased levels of quinolinic acid have been implicated in Alzheimerds disease as well as with respect to excitotoxin damage of nerves. Quote Link to comment Share on other sites More sharing options...
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