Newbie

[Guest guest]

Yes, I know that feeling too. Our neighborhood is also very culturally diverse. We are caucasion but actually we're the minority here. Which I see as a good thing. I want my kids to see people for who they are not what color they are. We've had some kids that are hard to deal with, as in any area, but we've also had some kids that are just awesome with . One boy told him that he was glad had autism. When questioned him on this, said, "Well, that's part of what makes you who you are, and you are really cool. If you didn't have autism, you might not be the person you are right now. I like that person." What a neat thing for a seventh grader to say to another one. I was so touched I wanted to hunt that kid down and kiss him. lol

Re: Newbie

Oh no not at all. I'm just never sure if I express myslef correctly. I find I often have to restate things more than once because I get distracted by the baby and forget important info. I think it's harder for the Grandmas becasue A) they don't live with them and it's hard to except your childs seed is imperfect. At least in MILs case that's true. She is the favorite child, DH is her favorite child and the favorite grandchild and Corbin is her Favorite Grandchild. Dhs side of the family seems to think the favirites can do no wrong and are "of course" perfect. It would be to much a blow to the ego to admit Corbin has a rpobleme that can't be fixed by a surgery or pill. Plus she finds it impossible to except Holistic health care can work. She is the type of person that thinks a "pill" will fix any probleme. But it's been Dhs and my expereince that holistic health care works better and is less invassive. Of coarse not always, but in most cases from our experience. So we're also at odds as to how to treat him. I printed up ALOT of good info off the web as well as a gently worded letter explaining his diapgnosis, our plan for treatment and a request tha! t only po Yes, seeing him come out of his shell is heart warming. He's been in that fog for so long and so withdrawn that it was starting to feel like he was entirly lost. I feel so relieved that he is starting to feel like he's apart of this world. I am just giddy with anticipation at the prospect that I may get to have a conversation with him some day. He was jabbering a mile a minute at those kids today. One politly told me he couldn't understand him and I quietly told him he wasn't talking yet but is learning. He smiled and said "cool". My neihborhood is so culterally and racially divers that these kids aren't at all put off by his "diffrent" ways. They have been trying to get him to play with them for 6 mos. So I'm sure they were also happy to see him except them. It's nice for him to have non judgemental peers.Angie~

Oh I hope I didn't come across like I thought you were looking for someone to blame. It's not that. I meant to express that that's why my mom does that. She's in denial too I think. For a long time she wouldn't admit there was anything wrong either. Woohoo on the playing with the other kids. It just about makes you want to cry when they make such huge strides like that doesn't it? I'm so happy for you. I know when reaches out I just get all gushy. It sounds like you definately have a good understanding of what caused Corbin's problems. I'm glad that you've researched that so thoroughly and it does sound like in his case that he was definately responding to the vaccine. I think in 's case, it's just always been this way for him. You know? I do agree to that knowing what the cause is can definately help to make the changes needed to make things better. I'm blessed that is so highly functioning I think. A lot of kids have more severe issues than he does. I think that just the few days I've been in this group have shown me that there is such a huge vast difference in the behaviors that our kids display and for that I thank you all. I hope I didn't come across sounding judgemental, that was never my intention. It's so hard sometimes to express yourself in an email and really have it come out like you want it too. I'm really sorry if I gave the wrong impression there.

[Guest guest]

" One boy told him that he was glad had autism. When

questioned him on this, said, " Well, that's part of what makes

you who you are, and you are really cool. If you didn't have autism,

you might not be the person you are right now. I like that person. "

What a neat thing for a seventh grader to say to another one. I was

so touched I wanted to hunt that kid down and kiss him. lol "

awwwwwww. That makes me teary eyed. That child was raised wonderfully

to be so open, honest and kindhearted. I so prey both my boys have

friends like that, I prey " they " are like that. What an awsome world

this would be if more kids were like that.

Angie~

[Guest guest]

I agree completely.

Re: Newbie

"One boy told him that he was glad had autism. When questioned him on this, said, "Well, that's part of what makes you who you are, and you are really cool. If you didn't have autism, you might not be the person you are right now. I like that person." What a neat thing for a seventh grader to say to another one. I was so touched I wanted to hunt that kid down and kiss him. lol"awwwwwww. That makes me teary eyed. That child was raised wonderfully to be so open, honest and kindhearted. I so prey both my boys have friends like that, I prey "they" are like that. What an awsome world this would be if more kids were like that. Angie~

[Guest guest]

Gene Linked to Autism: A Newsmaker Interview With Margaret Pericak-

Vance, PhD

if thats so, then why is he improveing with BRT which is clearing vaccines from his system??

Angie~

[Guest guest]

http://909shot.com/Diseases/autismsp.htm

http://66.70.140.217/a/thrower.html

http://www.nccn.net/~wwithin/mmr.htm

Just a few link showing the other side.

Angie~

[Guest guest]

Dr Megson - link -2nd URL, does not support NAET or BRT ...

If your read her testimony to congress, she does state genetic pre-disposition to autism.

She does believe the vaccines are like straws, which along with food intolerance, leaky gut, antibiotics will eventually break the camel's back per se- but the amount of damage is

dependent on how healthy the camel was in the first place.

She is my son's Dr, so, yes, I do know 1st hand exactly what her position/theories are.

I was just shareing links that I found helpful in understanding the wide veriety of reasons kids are autistic aside from genetics. I don't deny that genetics are the cause for amny kids, just not all. I don't necissarily carry the belief that vaccines "cause" autism in everyone. Some kids are predisposed for any number of reasons and the vaccine is like you said, the straw that breaks the camles back. So it's resonable to think that without the vaccine autism may not have developed, or perhaps not as severly. Rather she supports NAET or BRT isn't the point I wanted to make. BRT is simpley a tool (in our case the key it seems) to restore his body to what it would have been without the vaccines to break him. Does that make since? I can never tell if I am clear with the written word.

Angie~

[Guest guest]

>> I would be interested.

Debi<<

Okies! :-)

I'll post the articles in a short series - sorry to spam everyone's e-

mail box, but this really is some good information. Most of the

articles will be regarding genetic information as it relates to ASD,

but I'll start with one dedicated specifically to the issue of

causation between MMR vaccines and ASD:

The New England Journal of Medicine

November 7, 2002 (Volume 347, Number 19)

A Population-Based Study of Measles, Mumps, and Rubella Vaccination

and Autism

Madsen KM, Hviid A, Vestergaard M, et al.

The New England Journal of Medicine. 2002;347(19):1477-1482

The debate over the relationship between measles-mumps-rubella (MMR)

vaccine and autism goes on. Widespread use of the vaccine has

coincided with an increase in incidence of autism, though most

experts suggest that the increase in detection of autism and autism-

spectrum disorders is the result of better diagnosis.

Multiple studies in the last 4 years do not support this association,

but many of these studies have been methodologically flawed. None

have had sufficient statistical power to detect an association and

none were population based. The current study was conducted in

Denmark between January 1991 and December 1998 and included all

children born in Denmark as reported to the Danish Civil Registration

system. Vaccine status was determined by a review of data reported to

the National Board of Health; practitioners in Denmark are required

to report this information and are reimbursed by the state on the

basis of these reports. The Danish vaccination program recommends

that first MMR dose be administered at 15 months of age and provides

it free of charge. Children were followed until a maximum of 9 years

of life.

Information about diagnoses of autism was obtained from the Danish

Psychiatric Central Register, which contains information on patients

diagnosed in hospitals, outpatient psychiatric departments, and

outpatient clinics through Denmark. In Denmark, only specialists in

child psychiatry diagnose autism and assign a diagnostic code for

this disorder. An extensive record review for 40 children with this

diagnosis was conducted, 13% of all children with the diagnosis, to

determine if the diagnosis was consistent with the International

Statistical Classification of Diseases and Related Health Problems,

10th revision.

A total of 537,303 children were included in the cohort and followed

for a total of 2,129,864 person-years; approximately 82% of these

children were vaccinated. The study provided strong evidence to

finally lay this issue to rest. Not only was the risk of autism

similar in vaccinated and unvaccinated children, but there was no

temporal clustering of cases of autism around the time of

vaccination. These results were derived from a nationwide study with

almost complete follow-up. In addition, these data were collected

prospectively, were independent of parental recall, and were prior to

the diagnosis of autism.

The overall incidence of autism and autism-spectrum disorders found

in Danish children in this study was similar to studies on incidence

in French children and US children, thus making it unlikely that

Danish children are statistically less likely to develop autism.

Editor's Comment: This very strong, methodologically sound,

population-based, large study should finally put to rest parental

fears about MMR vaccine, and nurse practitioners (NPs) can

confidently point to it when discussing this issue with their

patients.

[Guest guest]

Regressive Autism May Be Linked to Autoimmune Enteropathy

NEW YORK (MedscapeWire) Apr 30 — A regressive type of autism

described in the April issue of Molecular Psychiatry may have an

autoimmune basis, either directly or indirectly from an autoimmune

enteropathy.

" We report findings of a novel form of enteropathy in children with

autism, characterized by lymphocytic infiltration, increased crypt

cell proliferation and enterocyte numbers, with co-localization of

IgG and complement C1q on the enterocyte basolateral membrane, " write

F. Torrente, from Royal Free and University College Medical School in

London, United Kingdom, and colleagues.

This comparative histologic study examined children with a form of

autism characterized by regression in the second year of life after

apparently normal early development. Earlier reports of immunologic

abnormalities and unexpected bowel pathology in autistic children

have come from this subgroup of affected patients. In this study, the

researchers compared duodenal biopsies from 25 autistic children of

this type with those from 11 children with celiac disease, 5 with

cerebral palsy and mental retardation, and 18 histologically normal

controls.

Compared with the normal and cerebral palsy control patients, the

autistic children had increased numbers of enterocytes and Paneth

cells, increased lymphocyte infiltration in epithelium and lamina

propria, and upregulated crypt cell proliferation. Compared with

those with celiac disease, the autistic children had fewer

intraepithelial lymphocytes and lamina propria cells and more lamina

propria T-cell populations. In 23 of 25 autistic children, but in

none of the other subjects, there was IgG deposition on the

basolateral epithelial surface, co-localizing with complement C1q.

Although these findings support an autoimmune basis for the

unexpected bowel abnormalities in children with autism, the authors

question the relevance of these findings to the general autistic

population, because these children had more obvious bowel symptoms

than are typically reported.

Interestingly, however, some children with regressive autism respond

to enteric therapy. The bowel changes could also reflect a genetic

condition affecting several systems, with brain symptoms more obvious

than gastrointestinal symptoms. Although further research is needed

to clarify the role of the " gut-brain axis " in autism, autoimmune

mechanisms may suggest avenues for future treatment.

" It is possible that in the future there will be such a concept

as 'autoimmune autism' within the autism spectrum, " J. Licinio and

colleagues from the University of California, Los Angeles, write in

an accompanying editorial. " Other biological alterations may be the

hallmarks of distinct disorders that may emerge from within our

current classification of autism. "

[Guest guest]

Gene Linked to Autism: A Newsmaker Interview With Margaret Pericak-

Vance, PhD

Laurie Barclay, MD

Feb. 7, 2003 — Editor's Note: Researchers have identified at least

one gene that may be responsible for autism, according to a report in

the March issue of the American Journal of Human Genetics. Using a

new statistical method combined with careful observational

classification, investigators have determined that a gene on

chromosome 15 coding for the gamma-aminobutyric acid (GABA) receptor

beta3-subunit (GABRB3) is involved in a distinctive autistic symptom

of " insistence on sameness " (IS).

Autistic children with IS exhibit repetitive compulsions and extreme

resistance to even trivial changes in their daily routine. The

statistical technique of ordered subset analysis revealed that those

families whose children had high scores in the IS category had a

strong link to the GABRB3 gene on chromosome 15q. Rather than lumping

all autistic children together for the purposes of analysis,

separating them into clinically meaningful subgroups allowed

validation of this genetic link.

To determine the potential of this discovery, Medscape's Laurie

Barclay interviewed the lead investigator of the study, Margaret A.

Pericak-Vance, PhD, director of the Duke Center for Human Genetics

and a professor and chief of medical genetics at Duke University

Medical Center in Durham, North Carolina.

Medscape: In simplified terms, how does this new statistical method

work, and how does it facilitate genetic analysis?

Dr. Pericak-Vance: First, we identify families with two or more

affected siblings. The statistical technique allows us to quantitate

similarities between families across a number of different variables.

We distinguish single traits, look for genetic similarities for each

trait, and use the statistical method to pull them together. If we

can identify a group of patients with similar characteristics, that

enhances our ability to determine which gene is responsible. What's

nice about this is that we can test the statistical significance of

the link, so it's not just a fishing expedition.

Now that we've done all the sophisticated genome technology and we've

looked at thousands of markers, we have to apply this technology to

clinical problems like diabetes, cardiovascular disease, and

dementia. But we haven't yet exploited all the clinical information.

In the past, we thought that you either have a disease like diabetes

or you don't, but that's too simplistic. None of these complex

diseases is just a single condition — each of the subgroups is

different clinically and most likely genetically. The clinical

subgroups help define the genetics.

Medscape: Have you applied this method to other genetic diseases yet?

Dr. Pericak-Vance: Any quantitative trait in any inherited disease

can be evaluated for its genetic significance. We've looked at age of

onset as a marker in familial Alzheimer's disease. We found a nucleus

of families with early onset and have subjected them to this type of

factor analysis, and that work is now in press.

Medscape: What is the function of the gene linked to " insistence on

sameness? "

Dr. Pericak-Vance: As you would suspect from a gene coding for a GABA

receptor, it is probably involved in neurotransmission, but it's too

early to know exactly how. So far we've found a cluster of genes in

the same region of chromosome 15 as GABRB3, and they all are somehow

involved with GABA. For example, one codes for the alpha-subunit.

Autism is a very complex condition, so we'll be looking at how all

three genes interact to affect clinical presentation.

Medscape: Is the GABRB3 gene also implicated in other

neuropsychiatric disorders?

Dr. Pericak-Vance: Genetic links to Angelman syndrome and Prader-

Willi syndrome are also clustered in the same region as GABRB3. Some

children with these genetic disorders also have autistic-like

symptoms, especially repetitive behavior.

Medscape: Do you suspect that other genes identifed as being related

to autism or to other neurobehavioral disorders will also involve

GABA or other neurotransmitters?

Dr. Pericak-Vance: Possibly. We're looking at other areas and other

chromosomes. Five or six groups around the world have also been

looking at other genetic markers. Although autism is a complex

disease with a variety of symptoms, there has been reasonably good

concordance in genetic findings around the world. Chromosomes 7 and 2

may be involved in different clinical aspects of autism. But the

function of these implicated genes is still unknown.

Everyone is madly testing candidate genes — it's a wide-open area. It

sounds like we're only taking baby steps, but every little step

helps. It's like we're stripping away tiny pieces of orange peel and

eventually the whole fruit will come into view. Finding even a single

gene marker leads to exponential progress, as it gives us a clue as

to the mechanism.

Medscape: What are the other distinctive features in autism that

could help define subgroups, and are these subgroups also being

analyzed genetically?

Dr. Pericak-Vance: A group at Mt. Sinai Hospital in New York has

linked phrase speech delay to chromosome 2, and we've confirmed that

those children in whom ability to speak in phrases is delayed beyond

36 months of age have a distinctive genetic marker on chromosome 2.

Medscape: Do the findings of this study suggest that heredity is more

important than environment in autism, or do these findings still

allow for interactions between genetic predisposition and

environmental factors?

Dr. Pericak-Vance: It's difficult to look at either heredity or

environment in isolation. If there is a genetic predisposition

interacting with environmental factors, we need to know the

responsible gene or genes before we can pinpoint which environmental

risks are involved. It's quite possible that subgroups differing

genetically may each be susceptible to different environmental risk

factors.

Medscape: Ultimately, will identification of these genes lead to

development of new treatments?

Dr. Pericak-Vance: Eventually. We may find a gene that is not a good

target, but it may lead us into a pathway that does provide a good

target for therapeutic intervention.

[Guest guest]

A genomewide screen for autism-spectrum disorders: evidence for a

major susceptibility locus on chromosome 3q25-27.

Am J Hum Genet 2002 Oct;71(4):777-90 (ISSN: 0002-9297)

Auranen M; Vanhala R; Varilo T; Ayers K; Kempas E; Ylisaukko-Oja T;

Sinsheimer JS; Peltonen L; Jarvela I

Department of Molecular Medicine, National Public Health Institute,

University of Helsinki, Finland.

To identify genetic loci for autism-spectrum disorders, we have

performed a two-stage genomewide scan in 38 Finnish families. The

detailed clinical examination of all family members revealed

infantile autism, but also Asperger syndrome (AS) and developmental

dysphasia, in the same set of families.

The most significant evidence for linkage was found on chromosome

3q25-27, with a maximum two-point LOD score of 4.31 (Z(max )(dom))

for D3S3037, using infantile autism and AS as an affection status.

Six markers flanking over a 5-cM region on 3q gave Z(max dom) >3, and

a maximum parametric multipoint LOD score (MLS) of 4.81 was obtained

in the vicinity of D3S3715 and D3S3037.

Association, linkage disequilibrium, and haplotype analyses provided

some evidence for shared ancestor alleles on this chromosomal region

among affected individuals, especially in the regional subisolate.

Additional potential susceptibility loci with two-point LOD scores >2

were observed on chromosomes 1q21-22 and 7q. The region on 1q21-22

overlaps with the previously reported candidate region for infantile

autism and schizophrenia, whereas the region on chromosome 7q

provided evidence for linkage 58 cM distally from the previously

described autism susceptibility locus (AUTS1).

[Guest guest]

Evidence for a language quantitative trait locus on chromosome 7q in

multiplex autism families.

Am J Hum Genet 2002 Jan;70(1):60-71 (ISSN: 0002-9297)

Alarcon M; Cantor RM; Liu J; Gilliam TC; Geschwind DH

Center for Neurobehavioral Genetics and Neuropsychiatric Research

Institute, and Department of Neurology, UCLA School of Medicine, Los

Angeles, CA, USA; Collective Name: Autism Genetic Research Exchange

Consortium.

Autism is a syndrome characterized by deficits in language and social

skills and by repetitive behaviors. We hypothesized that potential

quantitative trait loci (QTLs) related to component autism

endophenotypes might underlie putative or significant regions of

autism linkage.

We performed nonparametric multipoint linkage analyses, in 152

families from the Autism Genetic Resource Exchange, focusing on three

traits derived from the Autism Diagnostic Interview: " age at first

word, " " age at first phrase, " and a composite measure of " repetitive

and stereotyped behavior. " Families were genotyped for 335 markers,

and multipoint sib pair linkage analyses were conducted.

Using nonparametric multipoint linkage analysis, we found the

strongest QTL evidence for age at first word on chromosome 7q

(nonparametric test statistic [Z] 2.98; P=.001), and subsequent

linkage analyses of additional markers and association analyses in

the same region supported the initial result (Z=2.85, P=.002; chi(2)

=18.84, df 8, P=.016). Moreover, the peak fine-mapping result for

repetitive behavior (Z=2.48; P=.007) localized to a region

overlapping this language QTL. The putative autism-susceptibility

locus on chromosome 7 may be the result of separate QTLs for the

language and repetitive or stereotyped behavior deficits that are

associated with the disorder.

[Guest guest]

Dr Megson - link -2nd URL, does not support NAET or BRT ...

If your read her testimony to congress, she does state genetic pre-disposition to autism.

She does believe the vaccines are like straws, which along with food intolerance, leaky gut, antibiotics will eventually break the camel's back per se- but the amount of damage is

dependent on how healthy the camel was in the first place.

She is my son's Dr, so, yes, I do know 1st hand exactly what her position/theories are.

Re: Re: Newbie

http://909shot.com/Diseases/autismsp.htmhttp://66.70.140.217/a/thrower.htmlhttp://www.nccn.net/~wwithin/mmr.htmJust a few link showing the other side. :)Angie~

[Guest guest]

I am undiagnosed, but positive that I also have

AS.

-Eva

What sorts of things make you positive. Do you plan to obtain an

official dx?

[Guest guest]

Hi C. .

You've come to the right place. We understand. I have a 14 year old aspie/social anxiety daughter who sounds like your son; sweet one minute, nasty, mean and sometimes physically and verbally agressive/abusive the next! It is not easy. Just vent to us anytime, and you'll most likely get replies and suggestions to any questions you raise, as well. There are some very helpful people on this list!

Toodles!>^..^<Maralee

Newbie

Hi, I am the mother of a 12 year old boy with aspergers. I really need support. I asked my daughter to set this up for me. Please help. My son is socially challenged and it affects every aspect of our lives. He's very short tempered and it can be frustrating. When he has a good day, he's VERY sweet, polite and enthusiastic. But generally speaking he require extreme structure and routine in his schedule. He is unable to handle change easily. Please help. -c. davis

[Guest guest]

Hi, Welcome to the Group.

Could you go into a little bit more detail about things youve tried and implemented to control his behavior. Does he get intervention at school? What types of therapy is he in privately, if any? When he gets frustrated what exactly does he do? Are there certain things he gets frustrated about more than others? Is he getting frustrated because he cant do something or because of no one else understands his point of view? Is there some type of Sensory problem that is causing him to get frustrated?

My son, although younger, is the same way. When he is having an "on" day, hes great. Those off days are killers...

TK

Newbie

Hi, I am the mother of a 12 year old boy with aspergers. I really need support. I asked my daughter to set this up for me. Please help. My son is socially challenged and it affects every aspect of our lives. He's very short tempered and it can be frustrating. When he has a good day, he's VERY sweet, polite and enthusiastic. But generally speaking he require extreme structure and routine in his schedule. He is unable to handle change easily. Please help. -c. davis

[Guest guest]

C. ,

If you answered some basic questions, it might get us started with some suggestions.

- When was he diagnosed?

- How have you coped for 12 years, but seem less prepared to cope now? That is, what has changed?

- What medical services and/or drugs has he received?

- What special services or therapies has he received?

- Are you using any diet modifications?

My son recently turned 12, also. He has all the same symptoms you described below. We operated under a diagnosis of dyspraxia for many years. Quite " happily, " I might add, since we " knew what caused our son's difficulties. " But things got worse in school the past year. The teachers pushed for some IQ testing to see if we were bumping up against an IQ ceiling or whether he should be able to do the work but couldn't (because of some other learning disability) or was resisting. We had him tested and thereafter received the diagnosis of high-functioning autism. And then he had his first seizure (very mild). And then we saw an autism specialist, who ordered a blood test that resulted in a diagnosis of the chromosomal abnormality " 47xxy. " And he has gotten a lot more short-tempered this year. This particular symptom is common to both high-functioning autism and 47xxy.

The book " A Parent's Guide to Asperger Syndrome & High-Functioning Autism, " by Sally Ozonoff, PhD, et. al., has some strategies for dealing with strong emotions. There's plenty of Internet resources on Asperger's, too. But you've probably found many of those if you found this group.

Tony

in Wyoming

Newbie

Hi, I am the mother of a 12 year old boy with aspergers. I really

need support. I asked my daughter to set this up for me. Please

help. My son is socially challenged and it affects every aspect of

our lives. He's very short tempered and it can be frustrating. When

he has a good day, he's VERY sweet, polite and enthusiastic. But

generally speaking he require extreme structure and routine in his

schedule. He is unable to handle change easily. Please help.

-c. davis

[Guest guest]

Welcome to the group! This is the greatest place to be for support,

a place to bounce ideas around and overall friendships with common

bonds. We all have children with different struggles and different

ages; but find that talking here about this.....sometimes 'sparks'

that idea that may help.

just remember...you're not alone

Des/Joyce

[Guest guest]

My son was diagnosed at 12 last year. It has been tough. We have

been slowly weaning him off some of his routines, but he can be

dreadful, particularly now that puberty has set in. we have read

that it gets better after puberty, and all I can say is that I hope

so.

In terms of getting him to break routine, we have found that it works

better if we give him a written agenda on days when the routine might

get broken and then start warning him. HOwever, that said, getting

him to break pattern for something he wants ... well, let's just say

it is tough.

our problem has been getting him out of obsessive behaviors.

CB

> Hi, I am the mother of a 12 year old boy with aspergers. I really

> need support. I asked my daughter to set this up for me. Please

> help. My son is socially challenged and it affects every aspect of

> our lives. He's very short tempered and it can be frustrating. When

> he has a good day, he's VERY sweet, polite and enthusiastic. But

> generally speaking he require extreme structure and routine in his

> schedule. He is unable to handle change easily. Please help.

>

> -c. davis

[Guest guest]

I don't mean to be negative, but those obsessions....I don't know if they can be 'broke.' My daughters were things that she liked....so we basically let her run with them. Let's see, we've gone through whales when she was 4, then Scooby Doo, Titanic, YuGiOh, anything Japanese and animated (cartoons), Harry Potter, Lord of the Rings....and they continue. She just focues on one thing usually to the exclusion of anything else. I must say that they are getting better now that she is 14 though. She seems to be broadening her interests. Yay!!!

I started 'warning' my daughter about school coming on Aug 20 and she now screams at me that I "ruined her summer." I can' win.

Toodles!>^..^<Maralee

Re: Newbie

My son was diagnosed at 12 last year. It has been tough. We have been slowly weaning him off some of his routines, but he can be dreadful, particularly now that puberty has set in. we have read that it gets better after puberty, and all I can say is that I hope so.In terms of getting him to break routine, we have found that it works better if we give him a written agenda on days when the routine might get broken and then start warning him. HOwever, that said, getting him to break pattern for something he wants ... well, let's just say it is tough.our problem has been getting him out of obsessive behaviors.CB> Hi, I am the mother of a 12 year old boy with aspergers. I really > need support. I asked my daughter to set this up for me. Please > help. My son is socially challenged and it affects every aspect of > our lives. He's very short tempered and it can be frustrating. When > he has a good day, he's VERY sweet, polite and enthusiastic. But > generally speaking he require extreme structure and routine in his > schedule. He is unable to handle change easily. Please help. > > -c. davis

[Guest guest]

Hi Barb and welcome back. when did you have your biopsy saying cirrhosis? i

suppose it has it's early stages along with everything else. But once cirrhosis

is detected you are called stage 4 or end stage. Don't let it freak you

out.people live a long time with cirrhosis and some have few symptoms, like you

and me : )))..............ric

she lingers long on love street it's the place where all the hippies meet

__________________________________________________

[Guest guest]

hi Ric.....Thanks for the welcome. I had a biopsy about 5 yrs ago and it said

that I had Fibrosis.....I don't remember what stage......I've had annual CAT

scans though and I think the last time was about a year or so ago....I have

another one next Thurs. They said at that time, if I remember correctly that I

have a little bit of chirrosis. My GP called it " microscopic. " Hmmmm....you DO

have me freaked out a bit but nothing is really surprising me lately. Thanks for

the info......

Barb

[Guest guest]

Ric....Yeah, a lot of things have happened lately....I've been sick with

something else, but that's a long long story. Way too much to write about here.

But that wouldn't have included nausea. My doc (GP) says he doesn't know

why I'm so sick....and with the other sickness I had muscle/joint pain....really

bad...I had to walk with a walker for a month!!! But AGAIN, my doc said that I

shouldn't STILL hurt....he doesn't understand it and has no explaination for it.

.....and I'm taking oxycodone for it still. My LFTs went sky high last July 30 so

I'm wondering if this is related to that. I started the nausea on July 5th and

my

LFTs were " fine " back then.....I've been nauseaus for YEARS on and off, but

not THIS bad...this is just ridiculous! I'm just glad I'm seeing my liver doc

next

week. That's all I know. Thanks again for the info....

Barb

[Guest guest]

make sure you write down all your questions for him Barb. and stay there till he

answers them all. Don't be bullied. let us know how it goes. also check out this

site. www.HCVAdvocate.org and look for peppermint patty's FAQ.....good

info........ric

she lingers long on love street it's the place where all the hippies meet

__________________________________________________

[Guest guest]

Thanks for everything Ric.......you've been great. I will keep you posted.

Sweet dreams.

barb

[Guest guest]

Well, now you got us! Warts and all! Seriously, there is a lot of valuable

information and meaningful support to be had in this group of " misfits "

In a message dated 9/6/2004 3:12:15 PM Eastern Daylight Time,

entertain7777@... writes:

My relationship ended the same way. Now I dont want to meed anyone,

b/c it hurts too much.

I agree with you, there is very little info and very little support

out there.

> > Naomi, I was up front and told all of my friends and family I

had

> > hcv. All of them were very supportive for me. I would think

that

> if

> > any of your friends rejected you because of hcv then they were

not

> > true friends. I think people get scared because they do not have

> any

> > knowelege of this virus. Anyway that was my experience and most

of

> > my friends I have known for 25-30 years.

> >

> > These are just my own thoughts.

> > -----Alan.

>

>

>

> It's a pleasure having you join in our conversations. We hope you

have found the support you need with us.

>

> If you are using email for your posts, for easy access to our

group, just click the link--

http://groups.yahoo.com/group/Hepatitis_C_Central/

>

> Happy Posting

>

>

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>

>

> ---------------------------------

>