Guest guest Posted February 10, 1999 Report Share Posted February 10, 1999 Oluf nson writes: << ... But NO, Michel, if the patient does not respond to sulfonyureas, because the genuine insulin-production is more or less ”cero”. In that situation, though originally and from definition a T2, this patient must be treated as a T1. Accordingly, in that situation, with an extreme rise in BG, ketoacidosis will be a result and the risks are exactly identical, if insulin is not substituted ... >> Oluf, I am relieved that you responded to this. I am concerned about newly-diagnosed diabetics being misled by incorrect info. Type 2's can indeed lose pancreas function - from long-term use of sulfonylureas or from beta cell burnout caused by years on a high-carbohydrate diet. And type 2's with no pancreas function would indeed respond just like type 1's, including experiencing ketoacidosis. Susie Quote Link to comment Share on other sites More sharing options...
Guest guest Posted February 10, 1999 Report Share Posted February 10, 1999 A portion of the exchange between Oluf nson and Rosie: {Rosie} It is also possible that diabetes, in the very early stages, CAUSES weight gain. {Oluf} Rosie, that's new to me. How do you explain that? I would be very interested in knowing how and why? Have you any scientific references on that? Type 2's eat the modern diet, which is relatively low in protein but high in carbohydrates and relatively high in fats, as well as being too high in calories for our sedentary lifestyle. Their bodies churn out insulin to handle the carbohydrate/calorie load, but over time, because they are prone to diabetes and are overeating and " overcarbing " for their energy output, they become resistant to all the insulin being churned out. They eat a lot of carbohydrates, their blood glucose spikes, their insulin level spikes, their b.g. comes crashing down, and their body - quickly realizing that it is carbs that brings glucose levels back up the fastest - begins demanding more carbs. If they give in, their glucose once again soars, the insulin soars, and so on. Because they are eating too many calories for their energy expenditure, and because our bodies cannot store protein, the body burns the fat and uses the protein, and stores the excess carbohydrates as body fat. The fatter and more insulin-resistant we become, the more diabetic we become. It is a vicious cycle that can nevertheless often be broken with caloric restriction, wise food choices, and sufficient energy levele and weight loss. A weight loss of even 10-15 pounds can produce marked improvement. Studies have revealed improvements in glucose uptake from as few as four days of low-cal eating. Twins are no longer separated at birth, but old studies with identical and fraternal twins who were separated at birth did show that nature was more powerful than nurture ... their body shape and diabetes incidence more closely matched their siblings and birth parents than their adoptive families. But that does not mean that we can or should resign ourselves to a lifetime of accumulating diabetes complications, just because we have/had diabetic ancestors. We have the power to make significant improvements in our health. Susie Quote Link to comment Share on other sites More sharing options...
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