Re: MEDICAL, DOCUMENTARY EXPOSING $27 BILLION PSYCHIATRIC...

[Guest guest]

i know, they know, and have for a long time. how they sleep is beyond

me, i've wondered for years how some people can do things and still

sleep at night, now that is something that we need a cure for.

money,greed is one very big reason, a bonus, like not wanting people

to start realizeing the dose factor involved in exposures indoors to

many things besides second hand smoke, like perfumes,pesticides,

chemicals and of corse mold and mold toxins,ect.and that all these

things can come in from your outside environment and make you

ill.just think of what this alone has done for the sale of drugs and

repeated doctor visits not to mention allergy medicines,allergy

shots,asthma inhalents and on and on. but oh, i just mentioned

them.well dang, the cats out of the bag now.

>

>

> In a message dated 12/14/2006 4:12:06 P.M. Pacific Standard Time,

> jeaninem660@... writes:

>

> realizeing that anixity and depression LIKE symptoms can be

> from a physological illness and doing testing first should be a

must.

>

>

>

> They know that. The more and more I get into this matter, the

more and more

> I understand that this technique has been around for a long time.

Deny

> illness to save others money. It's disgusting!

>

> Below is a link from the tobacco docs. It's talks of Pres. Bush's

man on

> Tort " Reform " , Lester Brinkman, speaking out for the Manhattan

Institute, but

> being an " academic " so it's okay. Tort reform basically means, re

shape the

> litigation laws to save industry money while tromping on the

rights of the

> citizens.

>

> _http://legacy.library.ucsf.edu/cgi/getdoc?

tid=mcl46a00 & fmt=pdf & ref=results_

> (http://legacy.library.ucsf.edu/cgi/getdoc?

tid=mcl46a00 & fmt=pdf & ref=results)

>

>

> How do these people sleep at night?

>

> Sharon

>

>

>

>

>

[Guest guest]

Basically, the main premises behind the so-called " Tort Reform " media

campaign are lies..

" tort reform " is anything but. it is a term propagated by companies -

such as those in the tobacco and asbestos industries - vulnerable to

legal actions seeking damages for the impacts of their products.

Proponents use the term to refer to legislative measures designed to

limit the ability and potential damages available to individuals who

take legal actions against companies.

basically, the people who are promoting this so called reform want to

make our already incredibly unjust tort system even more unjust.

The number of people managing to file lawsuits in situations where

they were injured has been falling for years. The awards have also

been falling. The percentage of business profits paid out in lawsuits

has also been falling. Everything that they claim is basically

false.. seriously..

[Guest guest]

There is also an emergent " neurogenic " theory of depression that is backed

by a substantial amount of science that shows that any toxin that blocks the

creation of new neural connections (and some mycotoxins are very much in

that category) will cause a form of depression by preventing the brain from

a necessary process of self-repair and rewiring due to new knowledge

integration that occurs each night when we sleep.

So in that situation, no amount of antidepressants will address the

underlying issue...

See http://molinterv.aspetjournals.org/cgi/content/full/3/8/441

Here is one article about this...

_____cut here_______

A Neurogenic Theory of Depression Gains Momentum * nne M. and

A. *

Department of Neuroscience, The Chicago Medical School North Chicago, IL

USA

*SUMMARY*

The rate of adult neurogenesis fluctuates in response to several environmental

factors. Chronic stress, which can lead to neuronal apoptosis and dendritic

atrophy, certainly affects the overall rate of neurogenesis in the adult

brain. Depression, which arises from several causes, including chronically

stressful situations, is known to correlate with altered hippocampal

morphology. But is the link between depression and neuronal regeneration

merely coincidental? Recent studies indicate that ingestion of

antidepressants leads to increased neurogenesis in the hippocampus.

However, the

hippocampus is generally thought important for learning and memory—not for

" mood " state—thus, there is much more to the story that requires

clarification. Also, caveats abound in the interpretation of neurogenesis in

the amelioration of depression; nonetheless, these results are quite

intriguing and might point to better design and prediction of new-generation

antidepressants.

No neuropathological hallmarks exist for depression, despite decades of

research investigating the cause of this significant public health problem.

The hippocampus increasingly has been implicated as a key brain system

involved in depressive disorders, as it influences many functions impaired

in depressed individuals, including mood and cognitive difficulties, adverse

responses to stress, and altered neuroendocrine functions. An emerging

hypothesis

linking hippocampal neurogenesis and its control to the cause and subsequent

cure of depression is attractive to neuroscientists and clinicians alike.

Alterations in hippocampal morphology––including decreased synaptic

plasticity and decreased neurogenesis––occur both in animal models of

depression and in humans

*(1<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R1>

)*. The hippocampus is one of only two areas in the brain clearly shown to

exhibit neurogenesis throughout the life of mammals, including humans

*(2<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R2>,

3 <http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R3>)*.

Recent evidence

demonstrates that these newly generated neurons display action potentials,

establish synapses, and may function as mature dentate granule cells

*(4<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R4>

)*.

The functional purpose of continued neuronal birth has received much

speculation. As environmental enrichment increases the rate of neurogenesis,

one possibility is that the decreased hippocampal neurogenesis found with

depression may reflect an impaired ability to cope with novelty and

complexity *(5<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R5>

)*. This hypothesis is supported by work correlating neurogenesis with

reactivity

to novelty where hyperactive rats exhibited lower rates of cell

proliferation in response to novelty compared to low-reactive animals

*(6<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R6>

)*. This linkage between neurogenesis and depression was recently advanced

by a report from the laboratory of Rene Hen and colleagues advocating a

causal relationship between increased neurogenesis and amelioration of

depressive behavior in mice

*(7<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R7>

)*.

Neurogenesis is a complex series of events resulting in the generation of

new neurons from a pool of more primitive progenitor or stem cells

(Figure 1[image:

Go] <http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#F1>). This

process occurs during development of the nervous system and, to a more

limited extent, during adulthood

*(8<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R8>

)*. The rate of adult neurogenesis is not static, but changes in response to

environmental factors. Whether such modulation affects the rate of

proliferation or the survival of new neurons will differentially influence

the net outcome on neuronal number

*(9<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R9>

)*. It is the integration of these differentiated, new neurons into the

existing neural circuitry that will likely determine the long-term effect of

neurogenesis. A variety of stimuli may positively or negatively alter

neurogenesis (Figure 2[image:

Go]<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#F2>)

*(10

<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R10>–14<http://moli\

nterv.aspetjournals.org/cgi/content/full/3/8/441#R14>

)*; for example, stress is a potent reducer of adult neurogenesis

*(15<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R15>

–17 <http://molinterv.aspetjournals.org/cgi/content/full/3/8/441#R17>)*.

Given that stress is a factor implicated in psychiatric illness such as

depression, it is plausible that this stress-induced reduction in

neurogenesis may be one aspect of the pathophysiology of depression.

<http://molinterv.aspetjournals.org/cgi/content/full/3/8/441/F1>

http://molinterv.aspetjournals.org/cgi/content/full/3/8/441

(See the URL above for the rest of the paper)

[Guest guest]

A different take on this discussion from the Dec 9 issue of Science

News about an article from the Nov 22 Journal of Neuroscience. I

think most of us on this group will thoroughly understand and

appreciate the concluding statement.

Pain Type Matters to Brain

Chronic back pain affects different parts of the brain than acute

back pain does, magnetic resonance images reveal. Researchers say

that the area of the brain responding to chroinc pain is also

associated with emotional distress.

A. Vania Apkarian and his colleagues at Northwestern University

Medical Center in Chicago asked people with chronic backaches to

undergo magnetic resonance imaging of their brains. While in the

scanner, the patients rated their pain levels, which fluctuated

spontaneously.

During sustained periods of pain, nerve activity increased in the

brain's medial prefrontal cortex, which has been associated with

negative emotions, emotional memories, and self image.

In another experiment, the researchers put a hot probe onto the backs

of chronic-back-pain patients and volunteers with no history of back

pain. The scientists scanned brain activity while escalating the

intenstity of the heat up to painful levels.

When either chronic-back-pain patients or healthy volunteers felt the

painful heat, their medial prefrontal cortices were quiet. However,

the scans showed activation of the insula, a brain area associated

with acute pain.

These results show that chronic pain " impinges on a person's very

sense of being, " Apkarian says.

I interpret this as demonstrating a physical cause for what is

commonly assumed to be imagined or made-up because it is " all in our

head. " For me it also helps explain how chronic pain and illness

triggers the deep psychological and emotional issues we have all

experienced when we can't stop our exposure.

Carl Grimes

Healthy Habitats LLC

[Guest guest]

" How do these people sleep at night?

Sharon "

Forgive me but I couldn't resist--we're the ones who can't sleep at

night, and with a whole host of other symptoms. If " they " were sick the

research would be getting done and treatment improved. Maybe we should

hope that " they " get sick too.

--