Re: Hardin, Kelman, Saxon reply to JACI regarding ACOEM Mold ...

[Guest guest]

Quackadillian,

I thought it was pretty lame, too. If I were a member of AAAAI, even if

knew nothing of mycotoxin illness, I would be asking myself what the heck is

going on. I think the cat is out of the bag. I haven't actually written the

letter to them yet. But will this week.

Sharon

I hope that they print your letter..

All other considerations aside, I think the language used in the

Hardin, Kelman and Saxon

rebuttal letter shows that they know they are on shaky ground,

scientifically.s

At least it raises a lot of red flags for me.. And they seem to be

avoiding any real substantiative debate on the inhalational toxicity

issues, which are many, but discussion is conspicuously absent.

[Guest guest]

Thank you!

All I can say is that if I were a scientist, I would expect them to address

the inhalation toxicity issue much more.

I have a number of

papers on thrichothecene toxicology, as well as other mycotoxins, that show

that in various animals (some more closely related to us than others, like

monkeys, cats, guinea pigs, rats, chickens,) the inhalation toxicity is

always at least 5 times higher and sometimes 20 or more times higher than

intratracheal, dermal, intragastric, ingestion in food, dermal absorbtion,

etc... The only more toxic method is directly injecting into the brain.

(intracerebral)

They have avoided mentioning that and its very important.. Because when you

combine that little inconvenient fact with the other inconvenient fact that

the actual mycotoxin load in sick buildings is largely tiny particles, which

ends up making it much higher than the load measured on microscopically

identifiable spores.. (Straus, , etc. 2 papers on trichothecenes on

'particles smaller than conidia' and 'in the indoor environment', and also

consider the Indoor Air article (FUNGAL SPORE TRANSPORT THROUGH A BUILDING

STRUCTURE, M Airaksinen, J Kurnitski, P Pasanen, O Seppanen) on fungal

transport inside a buildings walls esp. for smaller particles - the most

respirable.. then you have a triple whammy, or trifecta of whatever that

damns this faulty defense argument wholly.)

There are also a whole bunch of papers I have on the effects of CHRONIC

ochratoxin exposure on brain neuroamines that parallel my own experience

with mold - they show why people get so tired and confused..and at best

suffer permanent word finding issues and ADD and can't sleep well and at

worst end up with diseases like parkinsonism and MS or even die.. (usually

of other causes, car accidents, accidental whatever, BUT Kaye Kilburn in his

'Endangered Minds' book REPEATEDLY brings up the IMPORTANT POINT that his

patients - and he has had lots of them, die. That they often don't survive

long.. people with brain injuries he has observed -and he is not a teenager,

he is probably in his 70s or 80s and he has been doing this work for AT

LEAST 40 YEARS - don't survive long..for the reason that they are -

suddenly- NOT able to cope with what they USED to cope with and THAT kills

them in many ways..)

Also, these toxic injuries literally AGE people.. Its probably by

dramatically increasing the reactive oxygen species or whatever they call

it.. cellular stress that the body HAS to repair to prevent cancer and

apoptopsis..The increases in ROS DRAMATICALLY accelerate the aging process

so what we really need is a new way to recognize this kind of injury..

because this kind of poisoning is one of the more common ways toxic

chemicals etc. poison people.. They steal your life away from you..

literally.. the changes in your biological age can be measured

physiologically.

Anyway, thats not rocket science, its just putting some easily available

facts together.. If they can't do that there is something wrong going on.