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Re: SURE SOUNDS LIKE INDOOR MOLD POISONING TO ME

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WHAT I'M SAYING IS THIS IS IRRITANT/TOXIN DAMAGE, DEPENDANT ON DOSE AND

LENGTH OF EXPOSURE.

MULTIPLE ORGAN DAMAGE.

I POPPED INTO THE NEUROSARCOIDOSIS GROUP AND FOUND THEM TALKING

ABOUT MANY OF THE SAME SYMPTOMS AND ALSO MOST NOT GIVEN A KNOWN CAUSE-

IDIOPATHIC. HOWEVER THEIR CNS ENVOLVEMENT IS RECONIZED. THEIR NERVE

DAMAGE IS KNOWN AS SMALL FIBER NEUROPATHY.

MANY TALKED ABOUT DOCTORS HAVEING A HARD TIME DESIDEING IF THEY HAVE MS

OR NEUROSARCOIDOSIS. A LOT OF THEM DEAL WITH EYE PROBLEMS.

>

> NEUROSARCOIDOSIS,SYMPTOMS AND SIGNS

> http://health.allrefer.com/health/neurosarcoidosis-symptoms.html

> DIAGNOSES AND TESTS

> http://health.allrefer.com/health/neurosarcoidosis-diagnosis-

tests.html

>

> SEARCHES

> TDIOPATHIC SYSTEMIC SARCOIDOSIS

> NEUROSARCOIDOSIS,MOLD EXPOSURE

>

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Mold is clearly an etiological factor for sarcoidosis/neurosarcoidosis.

At least they always mention strong statistical links to damp buildings,

mold, bacterial endotoxins.

(I did a search earlier and a LOT of news articles popped up in which health

depts. brought this connection up, something

they seem unwilling to do with other diseases cuased by mold.. BUT.. here's

the weird thing..

Since neurosarcoidosis is known to cause brain damage, and mold causes

neurosarcoidosis

HOW CAN THEY SAY MOLD CAN NOT CAUSE BRAIN DAMAGE???

Seems like any fourth of fifth grader could figure that logical 'proof'

out...

WHY NOT THE ACOEM?

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exactly. here's another piece to the puzzle I was looking into.

and try to make sence of.

chemotaxis.

chemotaxis,neutrophil responce to toxins

http://www.medscape.com/viewarticle/535033_print

some antifungals cause chemorepulsion(see what I'm getting at?)this

is a very telling read.

http://www.nanomedicine.com/NMIIA/15.4.3.6.1.htm

various endotoxins inhibit neutrophol chemotaxis.

chemoattractants are basicly toxins. but chemotaxis doesn't just

happen when self attacks self(autoimmunity) but also when exposued to

toxins. neutrophils are called on to fight toxins as they get inhaled

and start distroying mucus lineings, neutrophils die doing this, at

the site. granuloma is failure of optopsis and necrosis. optopsis-

self toxins, necrosis-non-self toxins as in mycotoxin exposure.

neutrophil chemotaxos, maybe better called(necrotaxis)

necrosis is responce to clean dead neutrophils from our system that

die in the dight of toxic invaders.

necrotaxis-I've been looking for this!!!!

a paticular form of chemotaxis: necrotaxis.

http://www.medscape.com/medline/abstract/8400312

negative necrotaxis-mivement away from dead cells, as in necrosis

does not accur,granuloma?

http://www.ionchannels.org/showabstract.php?pmid=8400315

what happens when neutrophils run low or are depleted? a severe

reaction in the body when exposed to more toxins.

necrosis vs. chemotaxis,3 links come up in this search., dont let the

one on vegitables throw you off ,take a look, this site come up alot

in my searches, alot of diggubf but its not about veggies.

http://olericulture.org/002/899/index.html.

chemotaxis usually refers to self attacking self and

chemotaxis/optopsis rids body of dead cells.

couldn't find any mention of neutrophils called on with self

attacking self.

if I'm remembering right, this is part of the innate immune

responce.neutrophils attacking non-self toxins.

--- In , LiveSimply <quackadillian@...>

wrote:

>

> Mold is clearly an etiological factor for

sarcoidosis/neurosarcoidosis.

> At least they always mention strong statistical links to damp

buildings,

> mold, bacterial endotoxins.

>

> (I did a search earlier and a LOT of news articles popped up in

which health

> depts. brought this connection up, something

> they seem unwilling to do with other diseases cuased by mold..

BUT.. here's

> the weird thing..

>

> Since neurosarcoidosis is known to cause brain damage, and mold

causes

> neurosarcoidosis

>

> HOW CAN THEY SAY MOLD CAN NOT CAUSE BRAIN DAMAGE???

>

> Seems like any fourth of fifth grader could figure that

logical 'proof'

> out...

>

> WHY NOT THE ACOEM?

>

>

>

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remember, aplastic anemia is known to be caused by depletion of

neutraphils from bone marrow. and known that mycotoxins and other

toxin exposure cause this.

from what I've read we dont have a abondant supply of neutrophils,

they might keep up with low exposures but for how long? ad a shorter

term high exposure on top of that, or anything inbetween.

question mught be, how does this effect our outcome and level of

reactions when faced with toxin exposure after we have been injured

this way. how does our body respond to toxins exposures if we are low

on neutrophils or they are tottally depleted?

>

> exactly. here's another piece to the puzzle I was looking into.

> and try to make sence of.

> chemotaxis.

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This is similar to what I have been reading about neural progenitor

cells/neural stem cells and damage by trichothecene mycotoxins and / or

ochratoxin A - the toxins are responded to by the body repairing damage

using what appears to be a finite supply of these repair-capable cells. But

the cells can get used up and when they are used up, things like hippocampal

neurogenesis are reduced.

This is what also happens with many chemotherapy drugs.. thats why they call

the neuro aspects of this condition 'chemo brain'.

In experimental animals they can also permanently end hippocampal

neurogenesis by irradiating them with high amounts of gamma radiation.

Animals so treated lose an important kind of memory.. it apears to be a link

between short term and long term memory..there is a lot of research that

ties this all together - its beginning to be obvious that, for example,

chemo brain and depression are linked, and the theory goes that the

depression comes because the storing of emotional learning is important for

people to be able to integrate the things they learn in life and that

blocking that storing doesn't just threaten their ability to support

themselves, it also threatens their emotional health by blocking healing of

depression..

People with chemo brain also find themself with a kind of ADD/ADHD. Impaired

hippocampal neurogenesis destroys aspects of short and medium term memory

and long term potentiation and it also impairs 'executive function' which is

the process of turning ideas into actions.

....

Perhaps the process you are describing with neutrophils is basically what

happens in the immune system when the immune system''s counterpart of the

neural stem cells is used up?

Don't expect the US medical system to solve this problem. If they were going

to, they already would have.

*If people like us can figure it out, THEY ALREADY KNOW - and they are in

cover it up mode..

Perhaps because this is what would happen all over the world if there was

nuclear war? (the gamma radiation connection)

On 7/8/07, who <jeaninem660@...> wrote:

>

> remember, aplastic anemia is known to be caused by depletion of

> neutraphils from bone marrow. and known that mycotoxins and other

> toxin exposure cause this.

> from what I've read we dont have a abondant supply of neutrophils,

> they might keep up with low exposures but for how long? ad a shorter

> term high exposure on top of that, or anything inbetween.

> question mught be, how does this effect our outcome and level of

> reactions when faced with toxin exposure after we have been injured

> this way. how does our body respond to toxins exposures if we are low

> on neutrophils or they are tottally depleted?

>

>

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Live, you make me laugh.... What idiots we are dealing with... The

hypocrisy of this world; sometimes all you can do is have a good

laugh at it to keep from crying about it.

COMMON SENSE!... I don't think they have any!

Dana

--- In , LiveSimply <quackadillian@...>

wrote:

>

> Mold is clearly an etiological factor for

sarcoidosis/neurosarcoidosis.

> At least they always mention strong statistical links to damp

buildings,

> mold, bacterial endotoxins.

>

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LIVE, I'M PRETTY CONVIENCED THAT NEUROSAECOIDOSIS AND MYCOSIS =

SBS/MYCOTOXICOSIS.

THE DIFFERENCE IN HAVEING SARCOIDOSIS AND NEUROSARCOIDOSIS COULD

EASOLY BE BASED ON LEVEL/AMOUNTS OF TOXIC EXPOSURE.

> >

> > Mold is clearly an etiological factor for

> sarcoidosis/neurosarcoidosis.

> > At least they always mention strong statistical links to damp

> buildings,

> > mold, bacterial endotoxins.

> >

>

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How do you know if what you have is sarcoidosis or if mold or mold and

endotoxins causes sarcoidosis?

The online resources I have seen are like what I have seen on mold, the

professionals OFTEN allude to a connection, but they won't come out and say

that A causes B.

Why wont they say it?

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LIVE,

SARCOIDOSIS OVERVIEW-FOREIGN PARTICLES AS BACTERIAL,VIRUSES,CHEMICAL

TOXINS, FUNGI,TB

SARCOIDOSIS IS NONCASEATING, THIS SUGGESTS AN INFECTION SUCH AS TB OR

FUNGAL INFECTION.

http://www.pulmonologychannel.com/sarcoidosis/

DID YOU TRY A SEARCH " SARCOIDOSIS,ENDOTOXINS "

SEARCHES OF INTEREST

SARCOIDOSIS,PHEURAL EFFUSION

" " " " " " " " " " " " " " " " " " ,FUNGAL LUNG INFECTION

" " " " " " " " " " " " " " " " " " ,ENDOTOXINS

NEUROSAECOIDOSIS,APLASTIC ANEMIA

SARCOIDOSIS,DEPLETION OF NEUTROPHILS

SARCOIDOSIS

NEUROSARCOIDOSIS

MIS THEM AROUND A LITTLE FOR MORE FINDS

--- In , LiveSimply <quackadillian@...>

wrote:

>

> How do you know if what you have is sarcoidosis or if mold or mold and

> endotoxins causes sarcoidosis?

>

> The online resources I have seen are like what I have seen on mold,

the

> professionals OFTEN allude to a connection, but they won't come out

and say

> that A causes B.

>

> Why wont they say it?

>

>

>

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ANOTHER SEARCH TO DO " SARCOIDOSIS,TOXIN EXPOSURE "

> >

> > How do you know if what you have is sarcoidosis or if mold or

mold and

> > endotoxins causes sarcoidosis?

> >

> > The online resources I have seen are like what I have seen on

mold,

> the

> > professionals OFTEN allude to a connection, but they won't come

out

> and say

> > that A causes B.

> >

> > Why wont they say it?

> >

> >

> >

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Jeanine

Do you think that you have sarcoidosis? I have some of the symptoms, but

obviously, they also look like other mold illness symptoms that are not

sarcoidosis, clearly.

Also, not that that makes me skeptical at all anymore, but its not seen as a

mold caused illness by the medical establishment yet, although many health

officials, especially seem to be trying to track incidences of it.

There do seem to be a lot of things suggesting a strong connection. That is

obvious. Thank you for posting these links, I have been looking at them.

It would be useful to hear more from people who had it whether they had been

exposed to toxinogenic molds. But how many would know even if they had been?

Not many. Just a few people like us have done testing. We are a tiny number,

percentage wise.

One thing that can complicate recognizing these things is when a lot of

people have an exposure it becomes VERY hard to discover it using

statistics. A lot of people have had exposure to molds to different degrees.

That is why it took them so long to figure out the connection between

tobacco and cancer. A lot of people then had exposure to tobacco, massive

exposure. Even people who never had themselves smoked.

On 7/9/07, who <jeaninem660@...> wrote:

>

> LIVE,

> SARCOIDOSIS OVERVIEW-FOREIGN PARTICLES AS BACTERIAL,VIRUSES,CHEMICAL

> TOXINS, FUNGI,TB

> SARCOIDOSIS IS NONCASEATING, THIS SUGGESTS AN INFECTION SUCH AS TB OR

> FUNGAL INFECTION.

> http://www.pulmonologychannel.com/sarcoidosis/

>

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