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Re: 2007 DEPARTMENT OF VETERANS AFFAIRS/SARCOIDOSIS

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Hmm... many of us have an exposure to trichothecene mycotoxins

(stachybotrys, fusarium), right? (at least I do)

Trichothecenes can cause primary bilary cirrohsis - so evidently they can

(via that) cause sarcoid-like granulomas that need to be ruled out via

tests. I am pretty sure I have also read that they can directly cause these

weird granulomas..

We have people on this list who have had lots of these granulomas.. and the

pain from them.

IMO, they may be caused by chronic exposure to some mycotoxins, but that's

not something most doctors are going to look for, partly because it is SO

disturbing.

However, with the new exposure data from Straus and others (like the levels

described in the paper Joe mentioned the other day)

they REALLY need to be considering that possibility. If that's the case,

these granulomas ARE, not " may be " - ARE the incontrovertable, unassailable

proof that the exposure to them in the indoor environment people are getting

IS CAUSING SERIOUS SERIOUS SERIOUS DAMAGE TO PEOPLE....

" e. Sarcoidosis can be mistakenly diagnosed for other known causes of

granulomatous

inflammation and other chronic lung diseases. The differential diagnosis

includes

hypersensitivity pneumonitis, mycobacterial infection, fungal infection,

beryllium exposure, and

exposure to metal dusts or organic antigens. Therefore, additional tests

need to be considered

when noncaseating granulomas are found by a biopsy including:

(1) Antineutrophil cytoplasmic antibodies for Wegener's granulomatosis,

(2) Anti-mitochondrial antibodies for primary biliary cirrhosis,

(3) Serologic and skin tests for fungal infections,

(4) Sputum culture for mycobacteria, and

(5) Beryllium lymphocyte proliferation test for beryllium exposure. "

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I dont think sarcoidosis or neurosarcoidosis would be a misdiagnoses

at all. WTC and Katrina may be proving its exposure to high amounts

or long term at lower amounts to irritants and toxins, differentails

may just be part of that exposure. irritants(mold included) and

toxins damage the mucosa and cause oxidative stress which damage the

tissue. asthma is a chronic infection/inflamation in the lungs, after

exposure and damage is done that damaged tissue and granulomas can be

at a certain level of inflamation constantly and wouldn't take much

to bring on infection. maybe with advoidance, some damaged tissue

become fibrotic. now even if someone gets this damage without

exposure to ubfectious agents you can bet those agents will become

problematic in there lives afterwards. example: those diagnosed with

tb, whos to say they didn't aquire after the fact of sarcoidosis? or

maybe it was part of their exposure to many things. nothings going to

be cut and dry here.

theres some cery interesting research thats been many from the navy

on this. think ships,water,mold/mycotoxins.

did you catch the sentence in there stateing that educators(as in

teachers) also get this disease? thinking it's in this one, if not I

have the one that states it.

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