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Fwd: Solutions for Digestive Disorders

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Forwarded from another group due to number of

people here with digestive problem. The Weston

Price Foundation on nutrition has alot of

enthusiastic followers.

In regards to high fiber discussed below, I think

this may be true for people with gi

inflamation/irritable bowel, since fiber could

further irritate gi tract and cause more

inflamation. However if gi tract has healed, my

personal opinion, is that high fiber might be

okay or beneficial then. However I will not be

eating alot of grains anymore due to knowledge of

'storage' conditions of grains causing mold

growth, but fresh sources of fiber such as fresh

veggies and fruits as Dr Marinkovich advises.

Since juicing greens takes the fiber out and

allows you to take in alot of chlorophll and

other important minerals and vitamins from fruits

and vegetables, I think that should be added in

after washing veggies and fruits very well. I

wouldn't buy freshly juiced veggy drinks out

since you don't know if they were washed well.

If you have a leaky gut, you have to be careful

about ingesting too many pathogens, which can get

directly into your blood stream through a damaged

gut lining.

--- wrote:

>

> Here's a great article that list treatments for

> Acid-Reflux & Digestive

> Disorders - " Pain Free After 25 Years " :

>

http://nourishedmagazine.com.au/blog/articles/pain-free-after-25-years

>

>

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Barb,

I have some other ideas that may work as good or maybe better.

Eating fiber through things like brown rice, rye seed or anything with

bran has a form of vitamin E that is " neuroprotective " helps protect

nerve cells. You will not get this from your multivitamins.

Their is also a association between low fiber, high fat diets and colon

cancer. I eat red meat but, I also eat quality grains with bran.

L-carnitine is responsible for bring fat or triglycerides to the

mitochondria (cellular power plant, genetic brain and garbage compacter)

where the glycerol is cleaved for ribose in ATP sythesis. The chemicals

leftover are ketones and alcohols which are taken away by the

L-carnitine. Weight lifters have been using this supplement for years.

It is a branched chain amino acid (BCAA) that the liver can synthesize

if the liver is actually working good. Dr. Shoemaker can give more

information on this area.

If you want to have a calm stomache, eat yogurt everyday without fail.

The yogurt bacteria put your intestinal flora in balance and the

bacteria eats acid as food - Lactobacilli acidophilus (spelling). Look

at Dannon and their research with Activa.

The most important thing is : EXERCISE filters the body due to faster

metabolism than sedentary lifestyles. Start small and keep progessing

without excuses. I have noticed that some of Dr. Shoemaker's patients

who excersize do much better to recover and get back to living life.

This is important for mold survivors and chemical sensitivity sufferors.

Metabolism, metabolism, metabolism!

A doctor can only do so much but you can do more. The smartest health

statement I ever heard was, " You don't have to get sick to get better " .

I can't remember his name.

Regards,

Greg Weatherman

Environmenal Contractor & Consultant

************************************************************

> >

> > Here's a great article that list treatments for

> > Acid-Reflux & Digestive

> > Disorders - " Pain Free After 25 Years " :

> >

> http://nourishedmagazine.com.au/blog/articles/pain-free-after-25-years

> >

> >

>

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Hi Greg, thanks for the suggestions. There are so many schools of

thought on diet. Just passed this along that someone passed on to

me. I agree with you on fiber but think that people with

*inflamation in the gi tract*, may benefit from low fiber until it

is healed over. I know the benefits of raw veggies and fruit but

may cook mine for awhile for same reason. This is what I may try

next. High fiber has caused me some trouble lately but my gi tract

is not healthy even though I have been doing the yogurt, probiotics,

etc and eating nutritious food. I have been eating brown rice and

also oatmeal.

--- In , " Greg Weatherman " <gw@...>

wrote:

>> Barb,

>> I have some other ideas that may work as good or maybe better.

>

> Eating fiber through things like brown rice, rye seed or anything

with

> bran has a form of vitamin E that is " neuroprotective " helps

protect

> nerve cells.

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Barb, you may want to consider trying to eliminate gluten. Dr.

Shoemaker's research shows that some people develop gluten intolerance

following mold exposure.

>

> Hi Greg, thanks for the suggestions. There are so many schools of

> thought on diet. Just passed this along that someone passed on to

> me. I agree with you on fiber but think that people with

> *inflamation in the gi tract*, may benefit from low fiber until it

> is healed over. I know the benefits of raw veggies and fruit but

> may cook mine for awhile for same reason. This is what I may try

> next. High fiber has caused me some trouble lately but my gi tract

> is not healthy even though I have been doing the yogurt, probiotics,

> etc and eating nutritious food. I have been eating brown rice and

> also oatmeal.

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You know, something interesting which I had not thought to post here, but

evidently the 9/11 first responders and survivors have a cluster of GI

issues that are common to them. Along with all of their lung issues.

This just came out.

I wonder if the causes are similar.

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Hi , I have alot of food allergies. I try to avoid gluten as

much as I can. I get hungry and have to eat, sometimes out but when

I have control, I have been trying to avoid. Sugar really bothers

me the most. I feel sure I have gut infection. I think I have been

undertreated, even by mold doctors since I am already 'self

treating' with low carbs and low to no sugar, otc antifungals/anti-

candida stuff, that my symptoms as shown on tests are at low level,

but will keep trying. I asked Dr Rae about taking some antifungals

and he said he would if I wanted, so if I give up on milder

solutions, will get back together with him on this. It's finding

the time. My house really gets more attention than I give myself.

HOWEVER, how clean my environment is I have found to be rewarded

physically, so not a waste of time certainly. Just time consuming.

I could move but I feel I have more control over my own home and may

get something just as bad somewhere else. Plus I have made great

strides at home and learned so much that will help me if I ever do

have to find another place.

--- In , " " <smarshwar@...>

wrote:

>> Barb, you may want to consider trying to eliminate gluten. Dr.

> Shoemaker's research shows that some people develop gluten

intolerance following mold exposure.

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That's interesting. While working down there they probably also

*swallowed asbestos, ***fiberglass, and concrete also, which causes

tiny cuts in tissue. I feel so sorry for them. They should be

getting excellent health care associated with 911 and it should all be

paid for by the government. The people who were done there died

instantly. The people who went down there afterwards to help, are

suffering and dying now. It's a second wave of tragedy following 911.

--- In , LiveSimply <quackadillian@...>

wrote:

>

> You know, something interesting which I had not thought to post

here, but evidently the 9/11 first responders and survivors have a

cluster of GI issues that are common to them. Along with all of their

lung issues. This just came out. I wonder if the causes are similar.

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Most of the worst lung damage came from inhalation of the tiny particles of

toxic dust...

http://www.nytimes.com/2006/10/24/nyregion/24toxic.html

http://video.on.nytimes.com/index.jsp?fr_story=d2c6e51adef45d557627215fd1cca1476\

e86e48f

On 9/1/07, barb1283 <barb1283@...> wrote:

>

> That's interesting. While working down there they probably also

> *swallowed asbestos, ***fiberglass, and concrete also, which causes

> tiny cuts in tissue. I feel so sorry for them. They should be

> getting excellent health care associated with 911 and it should all be

> paid for by the government. The people who were done there died

> instantly. The people who went down there afterwards to help, are

> suffering and dying now. It's a second wave of tragedy following 911.

>

>

> >

> > You know, something interesting which I had not thought to post

> here, but evidently the 9/11 first responders and survivors have a

> cluster of GI issues that are common to them. Along with all of their

> lung issues. This just came out. I wonder if the causes are similar.

>

> _

>

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Barb, it sounds like we have many of the same problems. I am now

working hard at cutting sugar and taking Diflucan & probiotics. I eat

no gluten or dairy, but still have digestive " issues " and other food

allergies. It could be my low MSH which is contributing to the

problem.

Your earlier post which you forwarded about diet and the changes in

our food made lots of sense to me. My husband comments how his

grandmother cooked with animal fats yet they both lived into their

90's. Same with my Mom who will soon turn 90.

I came across a book called " Diet Wise " the theory of which is that

we each have unique dietary needs due to genetics and supposedly

helps you to figure that out.

Good luck with finishing your house. I think that we all have so much

to juggle it's hard to devote time to taking care of ourselves,

especially since our health issues are so complex.

>

> Hi , I have alot of food allergies. I try to avoid gluten

as

> much as I can. I get hungry and have to eat, sometimes out but

when

> I have control, I have been trying to avoid. Sugar really bothers

> me the most. I feel sure I have gut infection. I think I have

been

> undertreated, even by mold doctors since I am already 'self

> treating' with low carbs and low to no sugar, otc antifungals/anti-

> candida stuff, that my symptoms as shown on tests are at low level,

> but will keep trying. . . .

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everything that goes up your nose ends up in your stomach. if your

mucus system is working normaly. and when that becomes disfunctional

your in trouble.

--- In , LiveSimply <quackadillian@...>

wrote:

>

> You know, something interesting which I had not thought to post here,

but

> evidently the 9/11 first responders and survivors have a cluster of GI

> issues that are common to them. Along with all of their lung issues.

>

> This just came out.

>

> I wonder if the causes are similar.

>

>

>

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Share on other sites

You are right. If your mucus is not able to attack and defeat pathogens, it

falls on the rest of your body..

The mucus is the first line of defense, and it needs to be there.

I know for a fact that stachybotrys damages the protective coating of mucus

- the surfactant - in your lungs..

I don't know..

http://toxsci.oxfordjournals.org/cgi/content/full/84/1/186

Toxicol Sci. <javascript:AL_get(this, 'jour', 'Toxicol Sci.');> 2005

Mar;84(1):186-94. Epub 2004 Dec 1.

<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed & DbFrom=PubMed & Cmd=Link & LinkN\

ame=pubmed_pubmed & LinkReadableName=Related%20Articles & IdsFromResult=15574675 & ord\

inalpos=2 & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVAbstra\

ct>

*Stachybotrys chartarum alters surfactant-related phospholipid synthesis and

CTP:cholinephosphate cytidylyltransferase activity in isolated fetal rat

type II cells.*

*Hastings

C*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Hasting\

s%20C%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubm\

ed_RVAbstract>,

*Rand

T*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Rand%20\

T%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_R\

VAbstract>,

*Bergen

HT*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Bergen\

%20HT%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubm\

ed_RVAbstract>,

*Thliveris

JA*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Thlive\

ris%20JA%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.P\

ubmed_RVAbstract>,

*Shaw

AR*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Shaw%2\

0AR%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed\

_RVAbstract>,

*Lombaert

GA*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Lombae\

rt%20GA%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pu\

bmed_RVAbstract>,

*Mantsch

HH*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Mantsc\

h%20HH%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pub\

med_RVAbstract>,

*Giles

BL*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Giles%\

20BL%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubme\

d_RVAbstract>,

*Dakshinamurti

S*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22Dakshin\

amurti%20S%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel\

..Pubmed_RVAbstract>,

*

JE*<http://www.ncbi.nlm.nih.gov/sites/entrez?Db=PubMed & Cmd=Search & Term=%22%\

20JE%22%5BAuthor%5D & itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubme\

d_RVAbstract>

..

Department of Oral Biology, Faculties of Dentistry & Medicine, University of

Manitoba, Winnipeg, Manitoba, Canada R3E0W2.

Stachybotry chartarum, a fungal contaminant of water-damaged buildings

commonly grows on damp cellulose-containing materials. It produces a complex

array of mycotoxins. Their mechanisms of action on the pulmonary system are

not entirely clear. Previous studies suggest spore products may depress

formation of disaturated phosphatidylcholine (DSPC), the major

surface-active component of pulmonary surfactant (PS). If S. chartarum can

indeed affect formation of this phospholipid, then mold exposure may be a

significant issue for pulmonary function in both mature lung and developing

fetal lung. To address this possibility, fetal rat type II cells, the

principal source of DSPC, were used to assess effects of S. chartarum

extract on formation of DSPC. Isolated fetal rat lung type II cells

prelabeled with 3H-choline and incubated with spore extract showed decreased

incorporation of 3H-choline into DSPC. The activity of CTP:cholinephosphate

cytidylyltransferase (CPCT), the rate-limiting enzyme in phosphatidylcholine

synthesis was reduced by approximately 50% by a 1:10 dilution of spore

extract. Two different S. chartarum extracts (isolates from S. chartarum

(Cleveland) and S. chartarum (Hawaiian)) were used to compare activity of

CPCT in the presence of phosphatidylglycerol (PG), a known activator. PG

produced an approximate two-fold increase in CPCT activity. The spore

isolate from Hawaii did not alter enzyme activity. S. chartarum (Cleveland)

eliminated the PG-induced activation of CPCT. These results support previous

observations that mold products alter PS metabolism and may pose a risk in

developing lung, inhibiting surfactant synthesis. Different isolates of the

same species of fungus are not equivalent in terms of potential exposure

risks.

also it damages the cell structures..

Toxicological Sciences *65*, 239-245 (2002)

RESPIRATORY TOXICOLOGY Microanatomical Changes in Alveolar Type II Cells in

Juvenile Mice Intratracheally Exposed to *Stachybotrys chartarum* Spores and

Toxin * T. G.

Rand*,1<http://toxsci.oxfordjournals.org/cgi/content/full/65/2/239#FN1>,

M. Mahoney*, K. White* andM. Oulton

**Stachybotrys chartarum* is an important environmental fungus. We have

shown recently that alveolar type II cells are sensitive to exposure

to *Stachybotrys

chartarum* spores and to the trichothecene, isosatratoxin-F, both *in vitro*and

*in vivo*, in a juvenile mouse model. This sensitivity is manifest as

significant changes in the composition and normal metabolic processing of

pulmonary surfactant. This study evaluated the effects of a single

intratracheal exposure of *S. chartarum* spores and toxin on ultrastructure and

dimensions of alveolar type II cells from juvenile mice. This was to

determine whether there are concurrent morphological and dimensional changes

in the alveolar type II cell that reflect the metabolic alterations in

pulmonary surfactant that we observed in the treated mice. Marked

ultrastructural changes were associated with alveolar type II cells in both

*S. chartarum* and isosatratoxin-F treated animals compared to untreated,

saline, and *Cladosporium cladosporioides* spore treated animals. These

ultrastructural changes included condensed mitochondria with separated

cristae, scattered chromatin and poorly defined nucleolus, cytoplasmic

rarefaction,

and distended lamellar bodies with irregularly arranged lamellae. Point

count stereological analysis revealed a significant increase (*p* < 0.05) in

lamellar body volume density in *S. chartarum* and isosatratoxin-treated

animals after 48 h exposure. Mitochondria volume density was

significantly lower

in the isosatratoxin-F (48 h exposure) and *S. chartarum* treated (24 and 48

h exposure) animals compared to those in the other treatment groups. These

results reveal that exposure to *S. chartarum* spores and toxin elicit

cellular responses *in vivo* differently from those associated with exposure

to spores of a nontoxigenic mold species. They also indicate that

accumulation of newly secreted pulmonary surfactant in the alveolar space of

*S. chartarum* and isosatratoxin-F treated animals might be a consequence of

cellular trauma resulting in lamellar body volume density changes leading to

increased release of pulmonary surfactant into the alveolar space.

*Key Words:* ultrastructure; alveolar type II cells; *Stachybotrys chartarum

*; trichothecenes; intratracheal instillation; fungal conidia;

morphometrics.

These also look like they might be related..

Z. Islam, C. J. Amuzie, J. R. Harkema, and J. J. Pestka

*Neurotoxicity and Inflammation in the Nasal Airways of Mice Exposed to the

Macrocyclic Trichothecene Mycotoxin Roridin A: Kinetics and Potentiation by

Bacterial Lipopolysaccharide Coexposure*

Toxicol. Sci., August 1, 2007; 98(2): 526 - 541.

[Abstract] <http://toxsci.oxfordjournals.org/cgi/content/abstract/98/2/526>

[Full

Text] <http://toxsci.oxfordjournals.org/cgi/content/full/98/2/526>

[PDF]<http://toxsci.oxfordjournals.org/cgi/reprint/98/2/526>

full text PDF @

http://toxsci.oxfordjournals.org/cgi/reprint/98/2/526

M. S. Leino, H. T. Alenius, N. Fyhrquist-Vanni, H. J. Wolff, K. E.

Reijula, E.-L. Hintikka, M. S. Salkinoja-Salonen, T. Haahtela, and M. J.

Makela

*Intranasal Exposure to Stachybotrys chartarum Enhances Airway Inflammation

in Allergic Mice*

Am. J. Respir. Crit. Care Med., March 1, 2006; 173(5): 512 - 518.

[Abstract] <http://www.ajrccm.org/cgi/content/abstract/173/5/512> [Full

Text] <http://www.ajrccm.org/cgi/content/full/173/5/512>

[PDF]<http://www.ajrccm.org/cgi/reprint/173/5/512>

full text PDF @

http://www.ajrccm.org/cgi/reprint/173/5/512

I. Yike, T. G. Rand, and D. G. Dearborn

*Acute Inflammatory Responses to Stachybotrys chartarum in the Lungs of

Infant Rats: Time Course and Possible Mechanisms*

Toxicol. Sci., April 1, 2005; 84(2): 408 - 417.

[Abstract] <http://toxsci.oxfordjournals.org/cgi/content/abstract/84/2/408>

[Full

Text] <http://toxsci.oxfordjournals.org/cgi/content/full/84/2/408>

[PDF]<http://toxsci.oxfordjournals.org/cgi/reprint/84/2/408>

full text PDF @

http://toxsci.oxfordjournals.org/cgi/reprint/84/2/408

J. Flemming, B. Hudson, and T. G. Rand

*Comparison of Inflammatory and Cytotoxic Lung Responses in Mice after

Intratracheal Exposure to Spores of Two Different Stachybotrys chartarum

Strains*

Toxicol. Sci., April 1, 2004; 78(2): 267 - 275.

[Abstract] <http://toxsci.oxfordjournals.org/cgi/content/abstract/78/2/267>

[Full

Text] <http://toxsci.oxfordjournals.org/cgi/content/full/78/2/267>

[PDF<http://toxsci.oxfordjournals.org/cgi/reprint/78/2/267>

full text PDF @

http://toxsci.oxfordjournals.org/cgi/reprint/78/2/267

I'll try to find some links talking about the GI issues..

**On 9/2/07, who <jeaninem660@...> wrote:

>

> everything that goes up your nose ends up in your stomach. if your

> mucus system is working normaly. and when that becomes disfunctional

> your in trouble.

>

>

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do you remember me posting infp. about neutriphils sent to attack

toxins that are attacking the mucus lineing in the stomach?

seems tp me that all myco's would play a role here. theres no dought

that what I passed at my second home was my distroyed mucus lineing

and it left my stomach as raw as my sinuses and later I caughed up

goobs of mucus from my lungs and got some of my breathing compasity

back but it also left my lungs raw and burning and dry rales.

this plays a big part in hypersensative reactions now.

to my sonuses,airways,stomach and bowels. theres nothing there to

protect the tissues now. only me,trying to advoid everything that

aggervates it. when toxins are attacking tissue and bpne it not good.

I think it more easily gets into the blood stream at this point too.

I believe that damage or loss of the protective mucus lineings is

what causes MCS/chemical inyolerence and symptoms of it are dependant

on just how damaged it is. in the sinuses it leads to olfactory

damage and limbic siezures,trigimenal and other nerve damage,leisons

in tissue and bone, damaged mucus glands. and I think damaged tissue

makes for easy paths to the brain.

in the stomach it leaves you very reactive to toxins breathed in or

in foods,exc. and a very reactive bowel and with liver damaged it can

be very painful.

lungs become very reactive to many things in the air.

this is how I look at why I'm chemical/irritsny intolerable.

kind of takes the mistery out of MCS.

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