Guest guest Posted October 6, 2007 Report Share Posted October 6, 2007 Jeanine.. I appreciate your htoughts...but there are major mistakes in your references. Rather than list all of them...one that needs to be addressed: is the president of the CIIN she did not develop TILT, that was , MD, MS out of San , TX On Fri, 5 Oct 2007, who wrote: > Date: Fri, 05 Oct 2007 18:18:44 -0000 > From: who <jeaninem660@...> > Reply- > > Subject: [] my thoughts on MCI > > as my favorite actor " Jsck N. " said in " one flew over the > cpp-coo nest " , " I tried, by golly,at least I tried " . > > WHAT IS MULTIPLE CHEMICAL INTOLERANCE > mci in a hypersensitive airway disease induced by exposure to toxins > where it damages the mucus linings causeing hypersensitive in the > airways. TILT, hepatotixicity(toxic liver) overload is the other > major factor as ir breaks down brain barriers, causes immune and > matabolism disfunction, and in itself adds to toxicity by adding > other effects related to liver malfunction. > damage of the limbic system,nervous system and olfactory system and > blood system are all involved and MCI is a disease involveing all > these factors. > below info. shows some evidence that MCI is not a factor of any one > of these conditions. therefore it has to be a factor of all the > condictions together. > MCI is hypersensitivity reaction of tottal organ involvement. > airways,brain,blood,nerves,immune,mataboism,involves cns/pns and all > organs are involved. a tottal body hypersensitivity to toxins and > effects of re-exposure varies depending on type of toxin and what > organs it affects and severity of responce will depend on > dose/quanity and level of damage of the organs it affects. > --------------------------- > olfactory disfunction-parkinsons,alztimers,schizophrenia > http://chemse.oxfordjournals.org/cgi/content/full/28/6/537 > -------------------------- > one type of schizophrenia that looks to be induced. > cellular evidence suggests BBB impairment in a % of schizophrenics > short-term and spatial mrmoty deficit > time estimation changes > deficits in sequencing > coordination deficit > numerous electrophysiologic changes > startle decrease > neurotransmitter changes > hormone alterations > immune alterations > mitochondria deficits > lipid phosphorylation decrease > lipid peroxidation > deleterious histologic changes in disease reduced brain areas > activation of hallucination involved brain areas > and ocular disease > . (and others) > ---------------------------------- > schizophrenia,hyperbilirubinemia(liver) > frontal temp cortex,limbic system,basil ganglia > http://www.medscape.com/medline/abstract/16018925?src=emed_ckb_ref_0 > bilirubin-jaundice related to inadequate liver function. > (?, would someine like this have a type of MCI because I know someone > with schizophrenia and often think it is a toxin exposure that brings > on their arracks. but its not quite this same so I'm wondering, if > they dont have sensatized olfactory system from a exposure situation. > maybe theirs is mainly from liver toxicity effects to the brain. > airisol exposure is not the only way ro have exposure to toxic > substances). > ------------------------------------------ > multiple chemical intolerance > immune and matabolism disorders result from toxic overload in the > liver. liver malfunctions cause higher blood ammonia,bilirubin, BBB > breakdown causeing damage to linbic system and with oxidative stress > in vessels,blood leaks in the brain causes leisons where ever they > happen. > and increases intercranial pressure, C. is correct in that > TILT, is induced by toxic liver overload and is the cause of MCI > hepatotoxicity damages blood brain barrier resulting in easy acsess > to the limbic system. intervranial pressure and breakdown of arcgnoid > villa allows csf in brain.(alsomdamage to myelin sheath,derma layer > allows csf leaks in brain) > MCI is than regulated based on total amout if organ damage as the > more organs are damaged the more hypersensative they are. re-exposure > to a toxin and depending on the type of toxin and what organs it > affects. > intake of a toxin through airways,olfactory responce,nerve > responce,blood responce. olfactory goes directly to limbic system and > frontal cortex(immediate brain effects,, lung responce,stomach, > liver,kidney responce,GI,ect. > -------------------------- > toxic liver overload, 2nd. home exposure,high amounts of several > toxic molds,high hunidity in home,very possible bacteria involved. > liver pain,brain swelling,csf leaks from ears and nose with burning > in these areas. cause distorted hearing and no dought more olfactory > and trigeminal damage > phychologicol exam while in first home exposure(anixity and > depression). after 2nd. home exposure(post tramatic stress disorder) > first home luver still detoxing lower amounts of mycotoxin exposure. > no indications of MCI > in first home.. > after 2nd. home exposure remember perfumes causeing burning up in the > nose.and caused increased brain responce on top of severe brain > malfunctions I was still suffering at the time. thought I had lost my > mind. > with MCI you have a reaction to toxin exposure weather you smell it > or not. normal people report smells that their sence of smell takes > affence to,everyone does.this is not MCI. > with a toxic subdtance envolved and longer exposure to that substance > they may get a headacke > but it doesn't cause brain fog in the frontal cortex or a seizure in > the limbic system. > http://en.wikipedia.org/wiki/Olfactory_system > ----------------------------------------------- > olfactory,limbic and liver connections > hepatotoxicity overload, cns effects > olfactory system,liver and limbic system,hypothalamus > hypothalamus modulates physilogic functions through enzymes/metabolic > function in the liver,GI system,lungs and adrenal medulla. > alcoholics have olfactory malfunction from brain injury. .benge > drinking causes brain damage in the limbic system,blood > poisoning,.BBB breakdown. > ------------------------- > BLOOD AMMONIA AND ACUTE LIVER FAILURE > metabolic encephalopathy: mechsnisms affecting limbic function > http://www.level1diet.com/research/id/51498 > the pathogenesis of intercranial hypertension in acute liver > failure:inflammation,ammonia(blood ammonia) and cerebral blood flow > http://www.level1diet.com/research/id/98112 > ----------------------------- > liver and cns . > intercranial pressure worsens the outcome of acute liver failure. > -liver malfunction increases interceanial pressure ---------------- > so in second home exposure it was either my brain or my liver. I > guess its good that it was my brain. this article also goes with what > I talked about before about drinking a cold milkshake tp bring down > the temp. in the head with reaccurance of cns/brain infection and > swelling. rapping a cold towel around the head also helps. getting > the temp. down is very important. > moderate hypothernia for uncontrolled intracranial hypertension in > acure liver failure > http://www.level1diet.com/research/id/221275 > ------------ > olfactory tract demyelination > olfaction and vision > http://www.dartmouth.edu/~dons/part_1/chapter_3.html > --------------- > nerve damage > trigeminal neuralgia results from nerve damage in MS. > fibromyalgia is a diagnoses of pns damage. > fibromyalgia involves the amygdala in the limbic system > the amygdala recieves and sends sensory messages to the trigeminal > nerve and the nerves in the pns. > demyelination of myelin sheaths inhibit sensory message pathway. > toxins make it to the limbic system through the olfactory system and > BBB from blood poisoning. > damage to the amygdala in the linbic system, unregulated nerve > responces in the cns and pns with re-exposure to toxic substances > damage to the myelin sheaths, nerve palsy symptoms . total > demyelination results in permenant palsy to the nerve system > involved. > ------------------------- > > . > > > > > FAIR USE NOTICE: > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 7, 2007 Report Share Posted October 7, 2007 heres some info. on olfactation I syarted by got sidetract on mucoceles. olfaction http://en.wikipedia.org/wiki/Olfaction the mucus overlying the epithelium contains mucopolysaccharodes,salts,enzymes and antibodies. these are lighly inportant as the olfactory neurons provide a direct passage for infection to pass to the brain. mucus http://en.wikipedia.org/wiki/Mucus olfactory epithelium http://en.wikipedia.org/wiki/Olfactory_epithelium the tissue is made of three types of cells. the olfactory receptor neurons which transduce the oder to wlectrical signals, the supporting cells which protect the neurons and secrete mucus, and the basal cells which are a type of stem cell that differentiate into olfactory receptor neurons to replace dead receptor cells. the olfactory epithelium can be damage by inhalarion of toxic fumes because of its refenerative capacity, damage to the olfactory epithelium can be temporary, but ib extreme cases, injury can be permanent. transduction http://en.wikipedia.org/wiki/Transduction_%28physiology%29 olfactory receptor neuron http://en.wikipedia.org/wiki/Olfactory_receptor_neuron http://en.wikipedia.org/wiki/Olfactory_system -------------------------------------- mucoceles of the paranasal sinuses http://www.jalanetipot.com/sinus-articles/paranasal-sinuses.html ouch frontal sinus obliteration http://www.doctorhoffman.com/wwoblit.htm > WHAT IS MULTIPLE CHEMICAL INTOLERANCE > mci is a hypersensitive airway disease induced by exposure to toxins > where it damages the mucus linings causeing hypersensitive in the > airways. TILT, hepatotixicity(toxic liver) overload is the other > major factor as ir breaks down brain barriers, causes immune and > matabolism disfunction, and in itself adds to toxicity by adding > other effects related to liver malfunction. > damage of the limbic system,nervous system and olfactory system and > blood system are all involved and MCI is a disease involveing all > these factors. during and after exposure. > below info. shows some evidence that MCI is not a factor of any one > of these conditions. therefore it has to be a factor of all the > condictions together. > MCI is hypersensitivity reaction of tottal organ involvement. > airways,brain,blood,nerves,immune,mataboism,involves cns/pns and all > organs are involved. a tottal body hypersensitivity to toxins and > effects of re-exposure varies depending on type of toxin and what > organs it affects and severity of responce will depend on > dose/quanity and level of damage of the organs it affects. > --------------------------- > olfactory disfunction-parkinsons,alztimers,schizophrenia > http://chemse.oxfordjournals.org/cgi/content/full/28/6/537 > > -------------------------- > one type of schizophrenia that looks to be induced. > cellular evidence suggests BBB impairment in a % of schizophrenics > short-term and spatial memory deficit > time estimation changes > deficits in sequencing > coordination deficit > numerous electrophysiologic changes > startle decrease > neurotransmitter changes > hormone alterations > immune alterations > mitochondria deficits > lipid phosphorylation decrease > lipid peroxidation > deleterious histologic changes in disease reduced brain areas > activation of hallucination involved brain areas > and ocular disease > . (and others) > ---------------------------------- > schizophrenia,hyperbilirubinemia(liver) > frontal temp cortex,limbic system,basil ganglia > http://www.medscape.com/medline/abstract/16018925? src=emed_ckb_ref_0 > > bilirubin-jaundice related to inadequate liver function. > (?, would someine like this have a type of MCI because I know someone > with schizophrenia and often think it is a toxin exposure that brings > on their arracks. but its not quite this same so I'm wondering, if > they dont have sensatized olfactory system from a exposure situation > where their olfactory system is not as envolved as ours. maybe theirs > is mainly from liver toxicity effects to the brain. airisol exposure > can cause effects by other routes besides olfactory route as in lung > to cns/kidney,stomach,ect. to liver. > ------------------------ > immune and matabolism disorders also result from toxic overload in > the liver. liver malfunctions cause higher blood ammonia,bilirubin, > BBB breakdown causeing damage to linbic system and with oxidative > stress in vessels,blood leaks in the brain causes leisons where ever > they happen. > and increases intercranial pressure, 's term TILT, is a > good term to explain what basicly happens to us. toxic liver overload > does play a part in the cause of MCI, but damage to the limbic system > from toxin exposure through the nose to the limbic system adds to > liver malfunction. these are two way circuts. olfactory to limbic and > liver to limbic. limbic to olfactory and limbic to luver. > I also agree with Moller that allergies have nothing to do > with MCI. > it is a result of irritants and toxins. > hepatotoxicity damages blood brain barrier resulting in easy acsess > to the limbic system. intercranial pressure and breakdown of archnoid > villa allows csf in brain.(also damage to myelin sheath,derma layer > allows csf leaks in brain) > MCI is than regulated based on total amount of organ damage as the > more organs are damaged the more hypersensative they are. re- exposure > to a toxin and depending on the type of toxin and what organs it > affects. > intake of a toxin through airways,olfactory responce,nerve > responce,blood responce. olfactory goes directly to limbic system and > frontal cortex(immediate brain effects, and lung responce,stomach. > liver,kidney responce,GI,ect. > there can also be some delayed respomces > -------------------------- > toxic liver overload, 2nd. home exposure,high amounts of several > toxic molds,high humidity in home,very possible bacteria involved. > liver pain,brain swelling,csf leaks from ears and nose with burning > in these areas. cause distorted hearing and no dought more olfactory > and trigeminal damage > phychologicol exam while in first home exposure(anixity and > depression). after 2nd. home exposure(post tramatic stress disorder) > first home liver still detoxing lower amounts of mycotoxin exposure. > no indications of MCI > in first home.. > after 2nd. home exposure remember perfumes causeing burning up in the > nose.and caused increased brain responce on top of severe brain > malfunctions I was still suffering at the time. thought I had lost my > mind. > ------------------- > with MCI you have a reaction to toxin exposure weather you smell it > or not. normal people report smells that their sence of smell takes > affence to,everyone does.this is not MCI. > with a toxic substance involved and longer exposure to that substance > they may get a headacke > but it doesn't cause brain fog in the frontal cortex or a seizure in > the limbic system. > http://en.wikipedia.org/wiki/Olfactory_system > not everything is known about the olfactory system but trying to > judge anything by smell seams silly to me because my sence of smell > can be acute one day and not the next. it's better after I wash my > sinuses and worse if my nose stuffy. > but I have noticed differences in toxin exposures and that different > parts of the olfactory system can be affected by different toxins. > ----------------------------------------------- > olfactory,limbic and liver connections > hepatotoxicity overload, cns effects > olfactory system,liver and limbic system,hypothalamus > hypothalamus modulates physilogic functions through enzymes/metabolic > function in the liver,GI system,lungs and adrenal medulla. > alcoholics have olfactory malfunction from brain injury. .benge > drinking causes brain damage in the limbic system,blood > poisoning,.BBB breakdown. > ------------------------- > BLOOD AMMONIA AND ACUTE LIVER FAILURE > metabolic encephalopathy: mechsnisms affecting limbic function > http://www.level1diet.com/research/id/51498 > > the pathogenesis of intercranial hypertension in acute liver > failure:inflammation,ammonia(blood ammonia) and cerebral blood flow > http://www.level1diet.com/research/id/98112 > > ----------------------------- > liver and cns . > intercranial pressure worsens the outcome of acute liver failure. > -liver malfunction increases interceanial pressure ---------------- > so in second home exposure it was either my brain or my > liver.actually it was both but I fell my brain suffered worse. I > guess its good that it was my brain instead of liver failure. this > article also goes with what I talked about before about drinking a > cold milkshake tp bring down the temp. in the head with reaccurance > of cns/brain infection and swelling. rapping a cold towel around the > head also helps. getting the temp. down is very important. > moderate hypothermia for uncontrolled intracranial hypertension in > acure liver failure > http://www.level1diet.com/research/id/221275 > > ------------ > olfactory tract demyelination > olfaction and vision > http://www.dartmouth.edu/~dons/part_1/chapter_3.html > > --------------- > nerve damage > trigeminal neuralgia results from nerve damage in MS. > fibromyalgia is a diagnoses of pns damage. > fibromyalgia involves the amygdala in the limbic system > the amygdala recieves and sends sensory messages to the trigeminal > nerve and the nerves in the pns. > demyelination of myelin sheaths inhibit sensory message pathway. > toxins make it to the limbic system through the olfactory system and > BBB from blood poisoning. > damage to the amygdala in the linbic system, unregulated nerve > responces in the cns and pns with re-exposure to toxic substances > damage to the myelin sheaths, nerve palsy symptoms . total > demyelination results in permenant palsy to the nerve system > involved. > ------------------------- > Quote Link to comment Share on other sites More sharing options...
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