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Re: Cellular Hypoxia

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Hey I just googled him, there is an interview with him posted about this book

Its really good

salzberglver3 <salzberglver3@...> wrote: For those of you who

have MCS from your exposures, I just finished a

very short, well written book by Dr. S. Bell, called Cellular

Hypoxia and Neuro-Immune Fatigue. Many of the concepts rang true for

me with my chemical intolerance. The main focus is on vasculopathy,the

abnormal dilation/contraction of blood vessels which shows itself as

exhaustion, cognitive issues, pallor, and headache. Next, central

sensitization of the central nervous system with light, noise,

chemical sensitivity, and last, cellular hypoxia which is a disruption

of energy production in mitochondria. My library purchased it for me

in case you can't afford new books.(NFI)

---------------------------------

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immunesupport.com

Q & A with Dr. Bell. I think he is a little behind on his research for

patients who are pre-dispostioned to get these illnesses thou. Still worth the

read.

a Townsend <kmtown2003@...> wrote:

Hey I just googled him, there is an interview with him posted about

this book Its really good

salzberglver3 <salzberglver3@...> wrote: For those of you who have MCS

from your exposures, I just finished a

very short, well written book by Dr. S. Bell, called Cellular

Hypoxia and Neuro-Immune Fatigue. Many of the concepts rang true for

me with my chemical intolerance. The main focus is on vasculopathy,the

abnormal dilation/contraction of blood vessels which shows itself as

exhaustion, cognitive issues, pallor, and headache. Next, central

sensitization of the central nervous system with light, noise,

chemical sensitivity, and last, cellular hypoxia which is a disruption

of energy production in mitochondria. My library purchased it for me

in case you can't afford new books.(NFI)

---------------------------------

Never miss a thing. Make your homepage.

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I wouldn't be too concerned about issues of 'pre-disposition' when

discussing disorders resulting from poisoning. There are no genes

which prevent the toxic effects of chemicals from damaging the

system. Also, basic genetic diversity rather than defects, may

dictate the timing of apparent injuries (e.g. how much of an

organophosphate may be needed to inhibit paroxonase enzymes to a

dangerous level). Causality ought not to be confused with co-

occurrence of various factors that have some protective effect on an

organism. Poisoning still sets off a chain of events that causes

physiological stress and inflammation.

Its about the chemicals since human evolution isn't going to alter

such effects.

The chemicals which damage are pretty recent (say 60 years) and are

now ubiquitious with exposures actually unknown to most individuals

(see statistics on pesticide metabolites in urine/residues in

dwellings). The harmful effects may be displayed in varying

forms/degrees from person to person but no one is immune to them.

Also, the mutagenic properties of chemicals, in which they disrupt

the expression of genes (inhibition or activation at the wrong

times) is still induced and therefore can be considered poisoning.

Barb Rubin

=======================================

>

> immunesupport.com

> Q & A with Dr. Bell. I think he is a little behind on his

research for patients who are pre-dispostioned to get these

illnesses thou. Still worth the read.

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the pre-dospodiyion would only be to the mold itself not it's

mycotoxins as I understand it.

> >

> > immunesupport.com

> > Q & A with Dr. Bell. I think he is a little behind on his

> research for patients who are pre-dispostioned to get these

> illnesses thou. Still worth the read.

>

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Barb is right. That is basically what many epidemiologists have been

saying for 30 or 40 years now.

If we focus on diverting blame from the toxic poisons onto the

victims, we are signing our own death warrants.

The toxic chemicals both as a whole, and individually, have toxicity

to animals in ALL different genotypes.

As I understand it, with some mycotoxins (Dr. Shoemaker's work) its

only the *rate at which they are removed* which is different which

doesn't come into play until the toxins have ALREADY gone through the

bloodstream doing a HUGE amount of damage to everything they touch, in

EVERYBODY. And making the body go into 'we need to mobilize our

defenses, NOW!' mode)

I read recently that all toxic chemicals have some variability in how

they effect different animals and that variability is not consistent,

it cuts every which way, so if you tried to legalize all toxic

chemicals based on one or two or even three aspects of some peoples

individual genetics (which is what some people, for example, companies

that pre-screen workers for toxic jobs based on their genetics are

trying to do!) it would fall every which way and the net result would

be a public health DISASTER.

On Nov 25, 2007 8:23 AM, agasaya <agasaya@...> wrote:

>

> I wouldn't be too concerned about issues of 'pre-disposition' when

> discussing disorders resulting from poisoning. There are no genes

> which prevent the toxic effects of chemicals from damaging the

> system. Also, basic genetic diversity rather than defects, may

> dictate the timing of apparent injuries (e.g. how much of an

> organophosphate may be needed to inhibit paroxonase enzymes to a

> dangerous level). Causality ought not to be confused with co-

> occurrence of various factors that have some protective effect on an

> organism. Poisoning still sets off a chain of events that causes

> physiological stress and inflammation.

>

> Its about the chemicals since human evolution isn't going to alter

> such effects.

>

> The chemicals which damage are pretty recent (say 60 years) and are

> now ubiquitious with exposures actually unknown to most individuals

> (see statistics on pesticide metabolites in urine/residues in

> dwellings). The harmful effects may be displayed in varying

> forms/degrees from person to person but no one is immune to them.

>

> Also, the mutagenic properties of chemicals, in which they disrupt

> the expression of genes (inhibition or activation at the wrong

> times) is still induced and therefore can be considered poisoning.

>

> Barb Rubin

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Did you know that some colonies of stachybotrys in water damaged

buildings contain a version of cyclosporin?

Cyclosporin is well documented as producing a very long lived kind of

cerebral hypoperfusion.

For an example, see

http://www.journalarchive.jst.go.jp/english/jnlabstract_en.php?cdjournal=interna\

lmedicine1992 & cdvol=42 & noissue=8 & startpage=750

On Nov 24, 2007 8:34 PM, salzberglver3 <salzberglver3@...> wrote:

>

>

>

> For those of you who have MCS from your exposures, I just finished a

> very short, well written book by Dr. S. Bell, called Cellular

> Hypoxia and Neuro-Immune Fatigue. Many of the concepts rang true for

> me with my chemical intolerance. The main focus is on vasculopathy,the

> abnormal dilation/contraction of blood vessels which shows itself as

> exhaustion, cognitive issues, pallor, and headache. Next, central

> sensitization of the central nervous system with light, noise,

> chemical sensitivity, and last, cellular hypoxia which is a disruption

> of energy production in mitochondria. My library purchased it for me

> in case you can't afford new books.(NFI)

>

>

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