Re: Docs against Miralax

[Guest guest]

Put in the Facebook page, too.

Fabulous! Thanks for finding and posting this! To: "miralax " <miralax > Sent: Tuesday, February 28, 2012 8:35 PM Subject: Docs against Miralax [1 Attachment]

Quote from the article.................The saving grace for PEG – at least in theory – is that it has a molecular weight of 3350, too large to be absorbed by a normal digestive tract. Studies performed in healthy, non-constipated adults show that pretty much all the Miralax taken by mouth comes out at the opposite end, meaning

that none of the PEG is absorbed.However, in constipated adults (those who actually take the Miralax) some of the PEG is always absorbed, and studies have not been able to match the amount that is excreted to what is taken in. This, incidentally, confirms that individuals with constipation have an unhealthy and excessively porous intestinal tract. If some of the PEG is retained in the body the unanswered questions are where does it go, how long does it stay there and what does it do? You might also be wondering what happens to PEG in children, but the reality is that Miralax was not studied in children.Perhaps the most disconcerting aspect of PEG is its well-known use in research laboratories. Here PEG is used to cause different cell

lines to merge, forming new ìmutantî cells, or to facilitate viral infection of resistant cells. Under normal circumstances, a given virus can only infect certain types of cells because it needs a specific attachment site that is found only on some cells and not others. Add a little PEG to the mix and the virus can infect any cell type and no longer needs an attachment site to get in. To be fair, this characteristic of PEG has not been demonstrated in the human body, although that doesnít reassure me.So if PEG and Miralax are out, what options do we have? The first and most obvious one is to find what is causing the constipation and correct it. By far the most common cause is food allergy or sensitivity and, among these, milk wins the contest by a long shot.

[Guest guest]

We should be writing Ayus who wrote the fatal disnatremia caused by elective colonoscopy article, his e is on the article, I think I've sent it to you before, I have his phone number too, I've spoke with him myself. Let me know........DiPalma is another one we should contact, he's the one who submitted all the info that our medical professionals are going by, he's the medical director of braintree labsjeanie From: "CAChitter@..."

To: miralax Sent: Wed, February 29, 2012 7:52:20 AMSubject: Re: Docs against Miralax

It is a great article and probably all true, but just be aware that he's not an md if you take to your dr, so they don't pick it apart on that basis. He doesn't cite the articles he refers to - I think I'll write to him to ask for it.

Carol

Quote from the article.................

The saving grace for PEG – at least in theory – is that it has a molecular weight of 3350, too large to be absorbed by a normal digestive tract. Studies performed in healthy, non-constipated adults show that pretty much all the Miralax taken by mouth comes out at the opposite end, meaning that none of the PEG is absorbed.

However, in constipated adults (those who actually take the Miralax) some of the PEG is always absorbed, and studies have not been able to match the amount that is excreted to what is taken in. This, incidentally, confirms that individuals with constipation have an unhealthy and excessively porous intestinal tract. If some of the PEG is retained in the body the unanswered questions are where does it go, how long does it stay there and what does it do? You might also be wondering what happens to PEG in children, but the reality is that Miralax was not studied in children.

Perhaps the most disconcerting aspect of PEG is its well-known use in research laboratories. Here PEG is used to cause different cell lines to merge, forming new ìmutantî cells, or to facilitate viral infection of resistant cells. Under normal circumstances, a given virus can only infect certain types of cells because it needs a specific attachment site that is found only on some cells and not others. Add a little PEG to the mix and the virus can infect any cell type and no longer needs an attachment site to get in. To be fair, this characteristic of PEG has not been demonstrated in the human body, although that doesnít reassure me.

So if PEG and Miralax are out, what options do we have? The first and most obvious one is to find what is causing the constipation and correct it. By far the most common cause is food allergy or sensitivity and, among these, milk wins the contest by a long shot.

[Guest guest]

I don't think we should write to anyone with ties to industry, you know?

We should be writing Ayus who wrote the fatal disnatremia caused by elective colonoscopy article, his e is on the article, I think I've sent it to you before, I have his phone number too, I've spoke with him myself. Let me know........ DiPalma is another one we should contact, he's the one who submitted all the info that our medical professionals are going by, he's the medical director of braintree labs

jeanie

To: miralax Sent: Wed, February 29, 2012 7:52:20 AMSubject: Re: Docs against Miralax

It is a great article and probably all true, but just be aware that he's not an md if you take to your dr, so they don't pick it apart on that basis. He doesn't cite the articles he refers to - I think I'll write to him to ask for it.

Carol

Quote from the article.................

The saving grace for PEG – at least in theory – is that it has a molecular weight of 3350, too large to be absorbed by a normal digestive tract. Studies performed in healthy, non-constipated adults show that pretty much all the Miralax taken by mouth comes out at the opposite end, meaning that none of the PEG is absorbed.

However, in constipated adults (those who actually take the Miralax) some of the PEG is always absorbed, and studies have not been able to match the amount that is excreted to what is taken in. This, incidentally, confirms that individuals with constipation have an unhealthy and excessively porous intestinal tract. If some of the PEG is retained in the body the unanswered questions are where does it go, how long does it stay there and what does it do? You might also be wondering what happens to PEG in children, but the reality is that Miralax was not studied in children.

Perhaps the most disconcerting aspect of PEG is its well-known use in research laboratories. Here PEG is used to cause different cell lines to merge, forming new ìmutantî cells, or to facilitate viral infection of resistant cells. Under normal circumstances, a given virus can only infect certain types of cells because it needs a specific attachment site that is found only on some cells and not others. Add a little PEG to the mix and the virus can infect any cell type and no longer needs an attachment site to get in. To be fair, this characteristic of PEG has not been demonstrated in the human body, although that doesnít reassure me.

So if PEG and Miralax are out, what options do we have? The first and most obvious one is to find what is causing the constipation and correct it. By far the most common cause is food allergy or sensitivity and, among these, milk wins the contest by a long shot.