Re: Mold study finds insufficient evidence

[Guest guest]

Joe,

The wording, " These findings extend the conclusions of

insufficient evidence, " which appears at the conclusion of the

study, are clumsy and ambiguous.

When I first read it I interpreted it the same as you. But in the

context of the full paragraph it seems to mean something

different, that the conclusions go beyond (extend) the insufficient

evidence and justifies more research. If the evidence supported

the denial of asthma from mold exposure I doubt they would

suggest further study. So which is true?

The conclusion statement in the Abstact has different wording

and is quite clear (thanks for including http://tinyurl.com/4fro2x ):

Conclusions. Our findings extend the 2004 conclusions of

the Institute of Medicine ... by showing that mold levels in

dust were associated with new-onset asthma in this damp

indoor environment. Hydrophilic fungi and ergosterol as

measures of fungal biomass may have promise as

markers of risk of building-related respiratory diseases in

damp indoor environments.

Interpreting studies can be as difficult as dissecting marketing

claims.

Carl Grimes

Healthy Habitats LLC

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> National Institute for Occupational Safety and Health (NIOSH) continues to

find " insufficient evidence " to link indoor mold in " water-damaged [office]

building " to asthma. [Toxic Mold? What's toxic mold?] These tax-dollar-financed

bozos are definitely on the fast-track (sarcasm), to " discover " what we ALL know

to be true about the cause of our suffering. They confirmed their own 2004

study, but discovered a really " neat " new measuring tool, for mold, to allow

them to come to the same stupid conclusion they reached in their 2004 study. Way

to go guys! (sarcasm)

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> Joe

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> (This is the last paragraph of their study)

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> In conclusion, we showed that among employees in a building with a long

history of water damage, respiratory symptoms and post-occupancy asthma were

strongly associated with fungi in a linear exposure-response manner, especially

the levels of hydrophilic fungi (including yeasts) in dust. These findings

extend the conclusions of insufficient evidence for the development of asthma in

relation to the presence of mold or other agents in damp indoor environments

reported by the Institute of Medicine (2004). Because the markers (totl

culturable fungi, hydrophilic fungi, and ergosterol) of potential mold exposure

were associated with health outcomes, we suggest that further research to

understand respiratory health effects in water-damaged indoor environments

include measurements of both ergosterol and speciated culturable fungi in dust.

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> For Complete Study, Click this link:

> http://tinyurl.com/4fro2x

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