Guest guest Posted June 20, 2008 Report Share Posted June 20, 2008 I also worked in a moldy hospital. It flooded twice during tropical storms. I believe that Administration knows there is a problem but is keeping it a secret. I think it is very common in hospitals, at least that is what my Toxicologist says, as he has treated many MD's & hospital personnel. I recommend you find another job before it kills you. I quit and found another job as soon as I figured out that was why me & so many of my coworkers were ill and some have died. I have tried to get the hospital to let me see copies of air quality testing, but they refused. It seems like that should be available upon request since our health is in their hands. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 20, 2008 Report Share Posted June 20, 2008 I also worked in a Hospital I had a double dose! of " Sick Building " and then some! Many of my Co-Workers who worked in and around an area of a large amount of MOULD died. After the MOULD was discovered and the part of the infected wall was replaced, even more people died! After I became sick and disabled and went in search of a cause, I discovered MOULD, as something that could cause illness. Well no wonder I kept having continuous joint pain and body aches....severe flu-like symptoms and my lungs felt as if they were on fire! I suffered along whith everyone else and most likely nobody knew or thought of " sickbuilding " When I inquired about any testing on the MOULD and was told, " What MOULD? and if there was MOULD, it was caused by a plant, or some other cause, but not by water damage! I had a Co-Worker do a Tape Lift of the floor and the part of the wall that had not been replaced, about an inch away from the part that had been cut out and replaced. An Environmental Lab did the analysis and called me at home immediately with a result. The gentlemans voice was much with concern as he told me of this MOULD. He said it was a very dangerous type of MOULD. He told me of friends in the Constuction Field had died as a result of exposure. The Mold he reported was Chaetomium...a Trichothecene/ T-2 producing type. My Dept. had monthly birthday celebrations in this room...Pot-Lucks. Birthday Cakes, Punch, Soda, and WOW! I bet quite a few spores went flying as the free standing cabinet was leaned up against. Spores inhaled as we spoke! Spores swallowed as we talked! Spores swallowed as we ate! Spores being taken home and shared possibly by consuming the left-overs...as I certainly know the Ladies in the Dept. would " divy " up what was left. One of the Ladies sons mysteriously came down with a case of Guillome-Barre'Syndrome. I have read where it is thought that diabetes may even be linked to the Toxins. Hmmm our Assist. Admin. passed out in her bathroom and was diagnosed with Diabetes.... Another Lady Thyroid Cancer. Another Lady Liver and Pancreatic Cancer. I recall one afternoon as I came from the restroom (adjacent to our Work-room/Patient examroom/ Pot-luck room), my Co-Worker said I was white as a ghost! No doubt...I told him why... It seems I was bleeding internally and had just passed a Large amount of blood he said he had been doing the same thing. Not one of us ever knew the term " Sick Building " My Dept. was relocated to a " New Building " . A 4 floor Medical Office Building that was the first built, and is now part of the New Regional Medical Center. I didn't become disabled until spending 17 mos. in our new building. Not only did I learn Mould can make a person sick....I also learned my Employer had built their New Medical Center on a highly contaminated " SUPERFUND " Site! Well no wonder the plants wouldn't grow at the entrance to our New Building! Anyone interested, can view on google map ariel view at 12214 Lakewood Blvd. Downey, CA 90242.... thats just a general address of the 160 acre contaminated area.... there is a 3000 lb herbicide underground tank that they didn't locate...I guess they said, " Oh Well " and they went ahead and built over it. The News about this Site is now VERY PUBLIC ....at (downey.kaiserpapers.info) I just thought I'd share ... Oh and I found some interesting FACTS from the WHO dated 2001 220 * Public health response to biological and chemical weapons—WHO guidance 2.3 Aflatoxins and other fungal toxins Before the 1960s, there was little systematic attention to fungal toxins as important causes of illness; the literature has developed mostly since the conclusion of the Biological and Toxin Weapons Convention. It is now well known that some fungi produce a single toxin, others may produce many, and different fungal genera may produce the same mycotoxin. Many genera, including Acremonium, Alternaria, Aspergillus, Claviceps, Fusarium and Penicillium produce mycotoxins. While the evidence indicates that ingestion of mouldy fodder is the primary route to animal mycotoxicoses, airborne fungal spores and infested/infected plant particulates may also induce disease leading to death in both animals and humans (18). The weapons potential of such airborne toxins has not been disregarded, although, in the 1992 returns of information under the BWC confidence-building measures in which the Russian Federation declared offensive biological research and development programmes during the period since 1946, it is stated that " in the opinion of the experts, mycotoxins have no military significance " (19 Nevertheless, two categories of mycotoxin have been considered as warfare agents, namely the aflatoxins and the trichothecenes, and will be considered briefly here. The United Nations Special Commission (UNSCOM) on Iraq mentioned weaponization of an aflatoxin in its synoptic report of January 1999, stating that the " question remains open regarding the aims and reasons of the choice of aflatoxin as an agent " . However, it went on to report that one Iraqi document " refers to military requirements to produce liver cancer using aflatoxin and the efficacy against military and civilian targets " (20). The trichothecenes were the subject of allegations of weapons use ( " yellow rain " ) in Cambodia and the Lao People's Democratic Republic during 1975–1984 that have since been discredited (21). Aflatoxicosis in humans is associated with the consumption of aflatoxin from food contaminated with the mould Aspergillus flavus. A number of aflatoxins with a range of potency (B1 > G1 > B2 > G2) are produced by Aspergillus, the relative proportions depending on the species of mould. Jaundice, fever, ascites, oedema of the feet, and vomiting are the symptoms associated with aflatoxicosis. In 397 patients estimated to have consumed 2–6 mg aflatoxin daily for a month, 106 fatalities occurred. Fatalities also followed estimated intakes of 12 mg/kg of aflatoxin B1. Five-year follow-up of survivors of acute poisoning (including liver biopsies) showed almost complete recovery. The principal concern with aflatoxin (particularly B1) is the possibility of liver cancer associated with the chronic consumption of mouldy food. Aflatoxin chemistry and metabolism are well described. Aflatoxin B1 is metabolized to a range of metabolites by microsomal systems. The active metabolite is presumed to be aflatoxin B1 8-9 epoxide. Inactivation is dependent on glutathione conjugation, with susceptibility to acute intoxication dependent on the activity of the enzyme glutathione-Stransferase. The B1 epoxide binds covalently to a range of proteins that have both structural and enzymic functions. Protein phosphorylation isalso altered by aflatoxin B1. All aflatoxins are genotoxic (22, 23). Trichothecene mycotoxins are a group of structurally related toxins produced by the Fusarium fungi found on many crops, and also by other mould genera such as Stachybotrys. They are sesquiterpinoids of low molecular weight, in the range 250–550 Da. Two of the better known toxins are T-2 and deoxynivalenol (or vomitoxin). Symptoms caused by the toxins are wide-ranging and include vomiting, diarrhoea, ataxia and haemorrhaging. The toxins are immunosuppressants and inhibit protein synthesis at the ribosomal level. They bind to the 60S subunit of eukaryotic ribosomes, altering peptidyl transferase activity. Inhibition of enzyme activity depends on toxin structure, and results in the failure either of polypeptide chain initiation or elongation. Toxicity of the toxins in in vitro test systems varies by as much as four orders of magnitude (24). In animals, the toxicity of T-2 is markedly species-dependent. Vomiting is induced in cats at 0.1–0.2 mg/kg after oral dosing. Guinea-pigs are unaffected at 0.75 mg/kg per day in the diet, but develop irritation and ulceration of the gut at 2.5 mg/kg per day. Immunosuppression is observed in rhesus monkeys at 0.5 mg/kgand in mice at 20 mg/kg. The LD50 in mice following intraperitoneal administration is reported to be 5.2 mg/kg. The toxicity of the trichothecenes in comparison with other toxins is therefore relatively low. They are, however, unusual among toxins in their ability to damage the skin, causing skin pain, pruritis, vesicles, necrosis and sloughing of epidermis. The Joint FAO/WHO Expert Committee on Food Additives assessed the safety of aflatoxins and trichothecenes in food at its 56th meeting in February 2001 > > I also worked in a moldy hospital. It flooded twice during tropical storms. > I believe that Administration knows there is a problem but is keeping it a secret. I think it is very common in hospitals, at least that is what my Toxicologist says, as he has treated many MD's & hospital personnel. I recommend you find another job before it kills you. I quit and found another job as soon as I figured out that was why me & so many of my coworkers were ill and some have died. > > I have tried to get the hospital to let me see copies of air quality testing, but they refused. It seems like that should be available upon request since our health is in their hands. > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 20, 2008 Report Share Posted June 20, 2008 Did you read this? http://www.reuters.com/article/healthNews/idUSL0383329520080603 llaci Quote Link to comment Share on other sites More sharing options...
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