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axonal demyelination,repair

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these downloads are definity worth reading

GENERAL MECHANISMS /AXONAL DAMAGE

together, these data present evidence that mechanisms of injury to

white matter in anoxia/ischemia and neuronalinflammatory diseases may

be surprisingly similar.

CELL DEATH BY NECROSIS

AH, FINALLY A EXPLANATION?

opoptosis vs. necrosis

sounds to me that necrosis may play a role in inflamation and cancer

but I have read all this yet.

MITCHONDRAIL DYSFUNCTION AS A CAISE OF AXONAL DEGENERATION IN MS

our data supports dysfunction of mitchondrail genes in the motor

cortex with MS, not definiencysGENERAL MECHANISMS /AXONAL DAMAGE

together, these data present evidence that mechanisms of injury to

white matter in anoxia/ischemia and neuronalinflammatory diseases may

be surprisingly similar.

CELL DEATH BY NECROSIS

AH, FINALLY A EXPLANATION?

opoptosis vs. necrosis

sounds to me that necrosis may play a role in inflamation and cancer

but I have read all this yet.

MITCHONDRAIL DYSFUNCTION AS A CAISE OF AXONAL DEGENERATION IN MS

our data supports dysfunction of mitchondrail genes in the motor

cortex with MS, not definiencys

NEUROBIOLOGY OF DISEASE/AXONAL INJURY

http://www.ohsu.edu/nod/2007-05-29.shtml

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necrosis,inflammation,no gene involvement known yet, mycotoxins

attaching and getting into cells, so this article leaves alot of

questions still but geem I spent alot on time trying to research the

difference in apoptosis and necrosis and necrosis sure fit in with

everything else and this just convences me more that we suffer more

from necrosis than apoptosis.

> CELL DEATH BY NECROSIS

> AH, FINALLY A EXPLANATION?

> opoptosis vs. necrosis

> sounds to me that necrosis may play a role in inflamation and

cancer

> but I have read all this yet.

>

>

> MITCHONDRAIL DYSFUNCTION AS A CAISE OF AXONAL DEGENERATION IN MS

> our data supports dysfunction of mitchondrail genes in the motor

> cortex with MS, not definiencysGENERAL MECHANISMS /AXONAL DAMAGE

> together, these data present evidence that mechanisms of injury to

> white matter in anoxia/ischemia and neuronalinflammatory diseases

may

> be surprisingly similar.

>

> CELL DEATH BY NECROSIS

> AH, FINALLY A EXPLANATION?

> opoptosis vs. necrosis

> sounds to me that necrosis may play a role in inflamation and

cancer

> but I have read all this yet.

>

>

> MITCHONDRAIL DYSFUNCTION AS A CAISE OF AXONAL DEGENERATION IN MS

> our data supports dysfunction of mitchondrail genes in the motor

> cortex with MS, not definiencys

>

> NEUROBIOLOGY OF DISEASE/AXONAL INJURY

> http://www.ohsu.edu/nod/2007-05-29.shtml

>

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