Guest guest Posted September 2, 2008 Report Share Posted September 2, 2008 DECTIN-1 RECEPTOR IN CONTROL OF FUNGAL INFECTION http://www.jleukbio.org/cgi/content/abstract/82/2/253 DECTIN-2 COUPLES WITH Fc RECEPTOR TO INDUCE INNATE IMMUNE RESPONCE http://www.jbc.org/cgi/content/full/281/50/38854 Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 3, 2008 Report Share Posted September 3, 2008 humm, well I was looking for the answer to wiether oxidative stress inhibited histamine release which took me into the world of IgE and IgG,infection,inflammation, and receptors, what a confuseing mess. either science cant sort it out or my head just gets lost in it. IgE>inflammation, IgG>infection, ? seems IgG may influence IgE or inhobit it. if I understand the with HLA FACTORS molds/myco's might not even get presented to the receptors but does that include all chemicals. I cant help but think the HLA factors play more of a role with low dose /beginning or after exposure.? and may actually protect the receptors in that way.? maybe I'm not understanding that right. ofcorse that means other routes would suffer more at even low doses. the omentum being connected to the liver functions and being known as our second brain plays a big role. thinking about the sinuses which aren't seperate from our bodies but largely has it's own immune system in a way,the mucus system but it's not full proff,good with low dose but not high doses, the myco's and other chemicals get threw to the brain not the mold spores, they are to big. so it seems the myco's and chemicals are the ones that mess up electrofields/transporting and receiveing across the receptors with that route but in my case where obviously some very toxic csf swelled my brain and toxic blood leaked out vessels in my head I'd say that did more damage to matter and nerve/myelination which also hinders transport of neurons. I've read that the shocking/touching thing is do to the receptor malfunctions so they've diffenitly been involved. but to what extent is damage to them or just failure of transmitting sugnals reaching them may very. seems this is a big factor in healing or not healing. many receptors under classes of receptors. yikes, to much there to try to sort out and to many bad articles floating around. why aren't the gate keepers who should be the " world experts " keeping the trash articles out of circulation? once they've been proved to be trash they should be thrown out. how confuseing to read something than turn around and read the exact opposite. seems theres more investagaters than there are activaters, it's really crazy when you read articles one pit's out but than talk to them and they act like they dont have a clue, like they dont practice what they preach, makes you wonder who's pulling the strings on the puppet. > > DECTIN-1 RECEPTOR IN CONTROL OF FUNGAL INFECTION > http://www.jleukbio.org/cgi/content/abstract/82/2/253 > DECTIN-2 COUPLES WITH Fc RECEPTOR TO INDUCE INNATE IMMUNE RESPONCE > http://www.jbc.org/cgi/content/full/281/50/38854 > Quote Link to comment Share on other sites More sharing options...
Recommended Posts
Join the conversation
You are posting as a guest. If you have an account, sign in now to post with your account.
Note: Your post will require moderator approval before it will be visible.