Re: anyone else out there?

[Guest guest]

Hi ,

Your throat can enlarge in both hypothyroidism and in hyperthyroidism. In

hypo the cells enlarge as they try to trap more iodine to make more hormone.

In hyper the cells enlarge in their efforts to keep churning out thyroid

hormone.

So either of your imbalances can cause this. Also, some of us have thyroid

glands that are situated further back in our throats. When we develop

goiters, they're called retrosternal and can interfere with breathing,

causing the symptoms you describe. Your swelling should subside as your

thyroid hormone levels return to the normal range unless you have a

co-existing conditon of viral or bacterial thyroiditis. The treatment for

this disorder is usually antibiotics and anti-inflammatory meds. Be sure to

let your doctor know if this swelling is interfering with your ability to

breathe. Take care,Elaine

[Guest guest]

>>> Re: anyone else out there?

>>>

>>>

>>>Hi ,

>>>Your throat can enlarge in both hypothyroidism and in

>>>hyperthyroidism. In

>>>hypo the cells enlarge as they try to trap more iodine to

>>>make more hormone.

>>>In hyper the cells enlarge in their efforts to keep

>>>churning out thyroid

>>>hormone.

>>>

Thanks for that answer. I've always wondered why as a HYPER I have a very

large

sub-sternal thyroid. I never have understood that part of the equation.

Sue

[Guest guest]

I've noticed that coffee makes me sleepy many times and I test allergic to it,

but I still drink a small cup in the am, then sometimes I lay back down until

time to go to work. Sometimes it is stimulating and sometimes makes me sleepy.

If my grandmother woke up in the middle of the night, she had a cup of coffee

and went back to bed, so maybe it's genetic or something.

>

> sence my exposure, there has been something involved in some foods that cause

me to have to go to sleep. it's not related to eating to much or based on how

tired I am. I'm wondering if anyone else has experienced this ?

>

[Guest guest]

yes, it's probably a allergy or intolerance to something that I just haven't

quite figured out yet. I need more testing done in that area to help me out.

allergic to coffee, you poor girl, I know some brands are worse about being

moldy, some I just cant tolerate but coffee is the only way I can start

functioning in the mornings.

> >

> > sence my exposure, there has been something involved in some foods that

cause me to have to go to sleep. it's not related to eating to much or based on

how tired I am. I'm wondering if anyone else has experienced this ?

> >

>

[Guest guest]

In the morning I take my medication for a neurological problem with coffee

because it helps me swallow and water kind of gives me a belly ache. And soon I

am back to bed.

[Guest guest]

Carl, haven't read that for awhile, is the masking effect based on brain

responces?

>

> What you may be experiencing with coffee is it's ability to " mask "

> other symptoms and complaints. It essentially " replaces "

> environmental reactions with its own. Nicotine is another mask,

> especially for those with chemical sensititivies.

>

> I know it sounds counter-intuitive because both can be severe

> reactors for us. But caffeine (this includes chocolate) and nicotine

> can " overwhelm " the body of some people and essentially " cover

> up " the environmental reactions. I've had a number of clients who

> feel better while smoking because when they quit their reactivity

> to their environment is overwhelming. Nicotine and caffeine block

> much of it and they report " feeling better. "

>

> Check out 's testimony on the role of masking with

> Gulf War Veterns at:

> http://veterans.house.gov/hearings/schedule106/oct99/10-26-

> 99/miller.htm

>

[Guest guest]

frutose vs. sugar

http://www.thegoodhuman.com/2007/09/10/high-fructose-corn-syrup-vs-pure-sugar-is\

-one-worse-than-the-other/

well I may have partly figured out what it is that causes me to fall asleep like

that. my daughter brought me a few granola oat bars with honey,raisins and a few

nuts. not thinking the other morning I ate one and shorty after I couldn't stay

awake, I pretty much spent the day in and out of bed. so anyway, while my

intolerance to corn may have mellowed out some as far as my stomach, sometimes

what hurts most is what you notice the most and other symptoms get ignored.

but maybe the process of makeing corn surup or frutose has something to do with

it and might also explain some stronger reactions depending on this process.

anyway, even though the granola bars had honey ( which also has a natural

frutose) there was also corn surup in them. I also had lose stools but it wasn't

the vilant,hurt the stomach kind like I would have reconized before.

the other day my grandson handed me a drink we got at a drive through, mine was

supposed to be tea, his was Dr. pepper, I took a sip of Dr. pepper thinking it

was my tea. this used to be all it would take to set my stomach rolling and

running to the bathroom about 10 minutes later. I quickly watered it down with

tea and although I felt it in my tummy and quickly got home just in case, I

didn't have that horrable stomach reaction.

my dad started back with his bee hives, so I got a big chunk of honey and cone,

yum. wasn't thinking about it so I cant say if it made me sleepy or not , only

that if it did it wasn't the same very noticeable effect that I had with this

granola bar.

than theres the time I took the sorbitol and CSM, not being able to see well at

the time I didn't realize frutose was in both of them,

however I got constipated and kept getting sicker and sicker and do remember

that I was very tired too. but something else happened that was pretty weird, I

loss complete balance. but my bowels were very inflamed and that may have been

why. if toxins can unbind in the bowel with constipation, I'd say thats what

happened.

still have discounted that there could have been fillers or something else there

that caused the loss of balance, but sence that is a brain reaction, I fell like

the inflamatory bowels could have been the cause.

so anyway, maybe the way corn surups are processed has something to do with some

effects showing theirself more than others.

I usally try to eat dark chocalate if I get that craving but of corse sometime I

cheat. I ate some milk chocalate yesterday. I could fell the blood rushing in my

viens but no other obvious effects.

later on I was tired but it wasn't the same as just not being able to stay awake

at all.

>

> Thanks very much for this information Carl. It is eye-opening. I've noticed

that a small amount of coffee would give me more energy for a while, but also

sometimes I would feel emotionally like my old self. It might not last for more

than an hour or so. And then if I started to drink coffee every day after a

while that would not happen as much.

> Sam

[Guest guest]

Carl, dont know if you saw these, pretty interesting. also THC theropy is being

reconized for endocannibinoid treatment.

I wonder if caffine might be a theropy for dopamine loss. ?

makes me wonder if the masking effect may be a good thing if ours bodys are

craveing something we need. a theropy,and if this could be showing that we have

some of these brain effects and thats why some things maybe not so good for you

might actually have some theropy values. ?

NICOTINE TREATMENT/PARKINSON'S AND HUNTINGSTON'S DISEASE

2005 Sep 30;67(1-2):161-8.

Neuroprotective effect of nicotine against 3-nitropropionic acid (3-NP)-induced

experimental Huntington's disease in rats.

Tariq M, Khan HA, Elfaki I, Al Deeb S, Al Moutaery K.

Neuroscience Research Group, Armed Forces Hospital, P.O. Box 7897 (W-912),

Riyadh 11159, Saudi Arabia. rkh_research.

Nicotinic acetylcholine receptors (nAChRs) are regarded as potential therapeutic

targets to control various neurodegenerative diseases. Owing to the relevance of

cholinergic neurotransmission in the pathogenesis of Huntington's disease (HD)

this investigation was aimed to study the effect of nicotine, a nAChR agonist,

on 3-nitropropionic acid (3-NP)-induced neurodegeneration in female Wistar rats.

Systemic administration of 3-NP in rats serves as an important model of HD. The

animals received subcutaneous injections of nicotine (0, 0.25, 0.50 and 1.00

mg/kg) daily for 7 days. 3-NP (25 mg/kg, i.p.) was administered daily 30 min

after nicotine for the same duration. One additional group of rats served as

control (vehicle only). On day 8, the animals were observed for neurobehavioral

performance (motor activity, inclined plane test, grip strength test, paw test

and beam balance). Immediately after behavioral studies, the animals were

transcardially perfused with neutral buffered formalin (10%) and brains were

fixed for histological studies. Lesions in the striatal dopaminergic neurons

were assessed by immunohistochemical method using tyrosine hydroxylase (TH)

immunostaining. Treatment of rats with nicotine significantly and

dose-dependently attenuated 3-NP-induced behavioral deficits. Administration of

3-NP alone caused significant depletion of striatal dopamine (DA) and

glutathione (GSH), which was significantly and dose-dependently attenuated by

nicotine. Preservation of striatal dopaminergic neurons by nicotine was also

confirmed by immunohistochemical studies. These results clearly showed

neuroprotective effect of nicotine in experimental model of HD. The clinical

relevance of these findings in HD patients remains unclear and warrants further

studies.

PMID: 16140176 [PubMed - indexed for MEDLINE]

2009 Oct 1;78(7):677-85. Epub 2009 May 9.

Multiple roles for nicotine in Parkinson's disease.

Quik M, Huang LZ, Parameswaran N, Bordia T, Campos C, XA.

The Parkinson's Institute, Sunnyvale, CA 94085, United States.

mquik@...

There exists a remarkable diversity of neurotransmitter compounds in the

striatum, a pivotal brain region in the pathology of Parkinson's disease, a

movement disorder characterized by rigidity, tremor and bradykinesia. The

striatal dopaminergic system, which is particularly vulnerable to

neurodegeneration in this disorder, appears to be the major contributor to these

motor problems. However, numerous other neurotransmitter systems in the striatum

most likely also play a significant role, including the nicotinic cholinergic

system. Indeed, there is an extensive anatomical overlap between dopaminergic

and cholinergic neurons, and acetylcholine is well known to modulate striatal

dopamine release both in vitro and in vivo. Nicotine, a drug that stimulates

nicotinic acetylcholine receptors (nAChRs), influences several functions

relevant to Parkinson's disease. Extensive studies in parkinsonian animals show

that nicotine protects against nigrostriatal damage, findings that may explain

the well-established decline in Parkinson's disease incidence with tobacco use.

In addition, recent work shows that nicotine reduces l-dopa-induced abnormal

involuntary movements, a debilitating complication of l-dopa therapy for

Parkinson's disease. These combined observations suggest that nAChR stimulation

may represent a useful treatment strategy for Parkinson's disease for

neuroprotection and symptomatic treatment. Importantly, only selective nAChR

subtypes are present in the striatum including the alpha4beta2*, alpha6beta2*

and alpha7 nAChR populations. Treatment with nAChR ligands directed to these

subtypes may thus yield optimal therapeutic benefit for Parkinson's disease,

with a minimum of adverse side effects.

PMID: 19433069 [PubMed - indexed for MEDLINE]

http://www.ncbi.nlm.nih.gov/pubmed/19433069?ordinalpos=1 & itool=EntrezSystem2.PEn\

trez.Pubmed.Pubmed_ResultsPanel.Pubmed_SingleItemSupl.Pubmed_Discovery_RA & linkpo\

s=5 & log$=relatedreviews & logdbfrom=pubmed

[Guest guest]

humm, had to take a look and grabbed this link.

General Hospital, revealed that caffeine could protect against dopamine loss in

a mouse model of Parkinson's

http://focus.hms.harvard.edu/2001/May18_2001/neurology.html

>

> She doesn't say which part of the body is the mechanism, as best

> I remember, but I'll ask her.

>

> Carl Grimes

> Healthy Habitats LLC

>

> -----

[Guest guest]

addiction is a brain disease.

>

> Sam,

>

> Good observation. Not only is that part of " masking " but you also

> described what Dr says is a cornerstone to addiction of

> coffee, cigarettes, chocolate and even street drugs. It is also

> relevant to several discussions here recently about how stopping

> mold exposure makes some of us feel even worse, at least for

> awhile.

>

> Using your example,

[Guest guest]

heres a example of the point I'm trying to make.

if toxic mold exposure causes dopamine loss than maybe we crave caffine to make

up for that.

if a person takes a drug that causes dopamine overproduction, maybe that fells

good or gives them more energy but maybe some chemicals ot toxins in that drug

can ot just the over expression of dopamine might than cause a lack of dopamine

when they come down off that drug ,and no understanding how they may be hurting

there brain and just wanting that felling again leads them to doing it again and

eventually the toxins may cause permentant damage we we may have from

mycotoxin/other toxins exposures getting to the brain.

both might be considered a addiction but we might just be striveing to get a

normal dopamine responce while addict's our striveing to get that over

expression of dopamine cause it felt good, but later on in their addiction if

they end up with dopamine loss than their addiction would be fueled even more so

to make up for that.

does that make sence?

> >

> > Sam,

> >

> > Good observation. Not only is that part of " masking " but you also

> > described what Dr says is a cornerstone to addiction of

> > coffee, cigarettes, chocolate and even street drugs. It is also

> > relevant to several discussions here recently about how stopping

> > mold exposure makes some of us feel even worse, at least for

> > awhile.

> >

> > Using your example,

>

[Guest guest]

Carl, my interest in this paticular topic has been long time.

I fell that putting names on somethings which could lead to different

inturpatations can be harmful to all as well.

my interest is not a addictive process, I have always been this way, I like

anwers, understandable answers, if I have to dig for the truth myself.

sometimes I have to deside if I need to put something in a more matter of fact

view or a ? view depending on how I'm addressing or what I'm trying to achive.

theres been something very curious on my mind for quite awhile conserning the

masking effect. it's ok, subject dropped.

>

> Jeanine,

>

> It makes logical sense but I have no idea if that is actually what

> happens in our brain.

>

>