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FYI,

It was proposed in 1997 that neuroprotein manufacturing

processes and, the amygdale are involved in identifying the etiology of Autism.

11 years later, it is more than just a hypothesis.

See below,

From:

sarnets-bounces@... On Behalf Of schafer

Sent: Wednesday, June 25, 2008

1:47 PM

To: Schafer Autism Report

Subject: Postech May Unlock A

Secret to Autism

Wednesday, June 25, 2008

Reader Supported

Vol. 12 No. 93p

In This Issue:

RESEARCH

Postech May Unlock A Secret to Autism

PUBLIC

HEALTH

NPR: Raw Milk: Panacea or Poison?

PEOPLE

Autism Boy Jumps To Death Over Cancelled School Trip

Computer Program Helped Find Autistic Man

Missing Autistic Wisconsin Man

Man, 21, Accused of Molesting Autistic Boys

Mother of Autistic Child Puts Priest On Stand

FINANCE

Autistic Australian Children To See $190m Support

MEDIA

The Autism File Magazine Debuts In The Us & Canada

Kirby on WOR Radio in New

York

EVENTS

National Autism Association' s National Autism Conference

COMMENTARY

The “Dark Age Of Autism” Is Dead

LETTERS

Vaccine Action

DEADLINE TODAY!

WEDS. June 25

For July

Autism Events Calendar

Submit listing here free!

the Autism Calendartm here

Hundreds of Local Autism Events

RESEARCH

Postech May Unlock A Secret to Autism

By Ha. tinyurl.com/3nk4rf

A team of researchers at Korea's Pohang University

of Science and Technology (Postech) has found new clues in understanding

the development process for autism, a brain disorder that impairs social

interaction and communication skills.

Kim Joung-Hun, a life sciences professor at

Postech, and his research team have found a brain mechanism through which

the disorder is created. The new finding was published this week by

Proceedings of the National

Academy of Sciences.

Professor Kim and his team have found that a

shortage of protein called " neuroligin " secreted by amygdala may

lead to the brain disorder. Amygdala is an almond-shaped structure in the

brain, involved in the generation of emotions and memories.

The Postech research team said the shortage

of neuroligin can interfere with normal activities between neurons and

their synapses, leading to the onset of autism.

" Neuroligin-1 is a potent trigger for

the de novo formation of synaptic connections, and it has recently been

suggested that it is required for the maturation of functionally competent

excitatory synapses, " according to the findings.

The research team manipulated brain

chemicals of laboratory mice to mimic this condition. When laboratory mice

no longer produced neuroligin protein in their brains, their neuron

synapses' functional speed and adaptability dropped markedly. Such

deterioration in brain synapses leads to autistic symptoms. This is

reportedly the first time that a link between neuroligin protein and autism

has been demonstrated.

The research team says the finding will help

further the understanding of autistic development in human brains and in

developing possible treatments for the disorder.

For rest of today's SAR click here:

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personal education or research purposes only and provided at their request.

Articles may not be further reprinted or used commercially without consent

from the copyright holders. To find the copyright holders, follow the

referenced website link provided at the beginning of each item.

Lenny Schafer editor@...

The Schafer Autism Report is a non-profit corporation

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